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Studies Link DNA Damage from Synthetic Antibiotics To Autism

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antibioticsBayer, the maker of the world’s most unsafe brands of birth control – the Yasmin line and the Mirena/Skyla IUDs, as well as a maker of neocontinid pesticides killing the bees – can now be held responsible for the rise in Autism rates over the last 30 years, as they are the makers and manufacturers of Cipro and Avelox, two of the world’s most popular fluoroquinolone antibiotics, which have recently been shown to alter DNA in ways that encourage expression of Autism related genes. Johnson & Johnson, the maker of Levaquin, also a popular fluoroquinolone antibiotic, can also be held responsible for the atrocity of one in eighty-eight children being autistic, as Levaquin is also a fluoroquinolone antibiotic that does the same thing.

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Cipro (ciprofloxacin) is a second generation fluoroquinolone patented in 1983 by Bayer, Levaquin (levofloxacin) is a third generation fluroquinolone patented in 1987 by Ortho-McNeil-Janssen (a division of Johnson & Johnson), and Avelox (moxifloxacin) is a fourth generation fluoroquinolone patented in 1991 by Bayer. In the 1980s the incidence of Autism was 1 per 1,000 children, today it is 1 per 88 children. The incidence of Autism has gone up hand in hand with the use of fluoroquinolone antibiotics.

Of course, this correlation between the introduction of fluoroquinolone antibiotics to the market and increasing rates of Autism, proves nothing. For proof, studies, experimentation and scientific exploration are needed. In September, 2013 the studies of topoisomerase inhibitors like fluoroquinolones as they relate to Autism commenced with an article in Nature entitled “Topoisomerases facilitate transcription of long genes linked to autism.”

Fluoroquinolone Antibiotics Lead to Autism Gene Expression

Fluoroquinolone antibiotics are eukaryotic DNA gyrase (also known as topoisomerase II) and topoisomerase IV inhibitors. Topoisomerases “are integral to gene expression, as they resolve DNA supercoiling that is generated during transcription.” Here is a video describing what that means and how fluoroquinolones work –


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A 1999 study in Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis showed that fluoroquinolone antibiotics damage and destroy mitochondrial DNA. It makes sense that they would do so because mitochondria are the parts of our cells that are most closely related to bacteria.  Bacteria are destroyed by fluoroquinolone antibiotics through the unraveling of bacterial DNA and resulting apoptosis (programmed cell death). Sadly, mitochondria also suffer the same fate.

An article published in September, 2013 in Nature entitled “Topoisomerases facilitate transcription of long genes linked to autism” noted that topoisomerase inhibitors can adversely affect gene expression and, “topoisomerases facilitate the expression of a large number of ASD (Autism Spectrum Disorder) candidate genes, including many that are long and that are thought to have large effects on ASD pathology.” Basically, drugs that effect topoisomerases, chemotherapy drugs and fluoroquinolone antibiotics (which are chemotherapy drugs) can cause Autism genes to be expressed.

The article concludes by stating that:

“Our data suggest that chemicals or genetic mutations that impair topoisomerases, and possibly other components of the transcription elongation machinery that interface with topoisomerases, have the potential to profoundly affect the expression of long ASD candidate genes. Length-dependent impairment of gene transcription, particularly in neurons and during critical periods of brain development, may thus represent a unifying cause of pathology in in many individuals with ASD and other neurodevelopmental disorders.”

THIS IS HUGE. It is an environmental factor, topoisomerase inhibiting chemotherapy drugs including fluoroquinolone antibiotics, that cause the expression of Autism genes.

It should be noted that the specific drug studied in “Topoisomerases facilitate transcription of long genes linked to autism” is Topotecan, not fluoroquinolones. Topotecan is a topoisomerase I inhibitor whereas fluoroquinolone atibiotics are topoisomerase II (also known as DNA gyrase) and topoisomerase IV inhibitors. Further studies need to be done to definitively show whether or not fluoroquinolones have the same adverse effects as Topotecan.

HOWEVER, the Researchers note that:

TOP2 (topoisomerase II) enzymes, (particularly TOP2B) also participate in gene transcription. We next tested whether genetic or pharmacological inhibition of TOP2 enzymes could reduce the expression of long genes. Indeed, with new experiments and by re-analyzing data from others, we found that the TOP2A/TOP2B inhibitor ICRF-193 reduced gene expression in a length-dependent manner in cultured mouse cortical neurons, embryonic stem (ES) cells and ES-cell-derived neurons. There was extensive overlap between genes affected by ICRF-193 and topotecan in cortical neurons, particularly for long genes, and the magnitudes of these effects were highly correlated. Thus, TOP1 (topoisomerase I) and TOP2 (topoisomerase II) enzymes regulate the expression of many of the same genes.

This means that topoisomerase II inhibitors, also known as DNA gyrase inhibitors, such as fluoroquinolones do the SAME THING as the topoisomerase I inhibitors studied. Fluoroquinolone antibiotics – Cipro, Levaquin, Avelox and a few more – “profoundly affect the expression of long ASD candidate genes.”

26.9 MILLION prescriptions for fluoroquinolone antibiotics were given to people in 2011 alone. Quantities of prescriptions of these DNA altering drugs have been in the millions since their rise in popularity in the 1980s.

These popular antibiotics, prescribed for sinus infections, urinary tract infections, strep throat, traveler’s diarrhea, prostate infections, etc. ALTER DNA AND NEGATIVELY INFLUENCE GENE EXPRESSION (EPI-GENETICS) AND LEAD TO THE EXPRESSION OF AUTISM GENES. Genes. DNA. Epi-genetics. The bits of information that our cells pass from one generation to the next. The damaged DNA is passed from parent to child and from that child down to their children, and so on and so on.

Gene expression, epi-genetics, is tricky because so many factors influence how genes are expressed, and it is only recently that ways of measuring and describing epi-genetics have been discovered. Neil deGrasse Tyson is probably better at explaining epi-genetics than I am, so please check out this video –

One of the research scientists who Dr. deGrasse Tyson interviews notes that, “you’re not only what you eat, (you are) potentially what your mother ate and even what your grandparents ate” to show the power of the heritability of epi-genetic markers. Dr. deGrasse Tyson and the research scientist are discussing how food can effect epi-genetic markers and influence obesity throughout generations of mice. If food can effect epi-genetics that dramatically, just imagine how dramatically a drug that is intentionally designed to interfere with and unravel mitochondrial DNA can effect human heredity. Not only can a drug that a parent (not just mothers, the DNA of fathers contributes to 50% of everyone’s genes) took influence the epi-genetics of their child, but the epi-genetics of their grandchildren and great-grandchildren can be adversely effected as well.

So, children are inheriting damaged genes, the genes that control whether or not a person is Autistic, because their parent (or even grandparent – for future generations – fluoroquinolones haven’t been around long enough to mess up multiple generations of people – yet) took an ANTIBIOTIC that messed up their mitochondrial DNA. This is absurd. Generations of humans will continue to be plagued by high autism rates, possibly indefinitely, because a certain class of antibiotics that has dangerous and severe side-effects even to those who take them directly, has damaged their mitochondrial DNA. This entire horrifying situation is Bayer’s and Johnson & Johnson’s fault and they should be held accountable to humanity in every way possible.

Let me just back-pedal a little bit and say that many women who have been severely adversely effected by fluoroquinolone antibiotics have had healthy, happy, beautiful, smart babies. Many factors go into how genes are expressed. What you eat, music, positive thoughts and words, etc. can influence how your genes are expressed. If you have taken a fluoroquinolone antibiotic, you are not doomed to have an autistic child. But it really seems shameful, horrifying actually, to increase someone’s odds of having an autistic child by encouraging the expression of autism related genes synthetically, through an unneeded and destructive antibiotic, when other, safer, non-DNA-damaging antibiotics are available.

If I can figure out that fluoroquinolones have the same effects as Topotecan, and that damaged DNA and gene expression / epi-genetics markers can be passed on from generation to generation, with my Cipro frazzled brain, you can bet that Dr. Zylka and his team of undoubtedly brilliant Scientists also realize that topoisomerase II / DNA gyrase (and topoisomerase IV) inhibitors like fluoroquinolones are leading to the expression of autism genes. I thank these Scientists for what they have uncovered and published from the bottom of my heart, and I beg of them, please have enough courage and moral fortitude to stand up for what you know to be true – that fluoroquinolone antibiotics made by Bayer and Johnson & Johnson have severely, and possibly irreversibly, damaged the human gene pool in a way that is causing children, innocent children, to be hurt. Though there is little that can be done to stop the damage from being passed down through the generations, the people who have already been hurt by Bayer and J&J deserve compensation. The children who are living with Autism deserve compensation. Bayer and J&J hurt them, they hurt humanity, and they should pay for their sins. Without the word of Scientists to back up these assertions, no justice will ever come to the families. Dr. Zylka and others… please, do what’s right.

The Dangers of Fluoroquinolones Shouldn’t be a Surprise

This coalmine is littered with dead canaries. The Scientists who designed fluoroquinolones always knew that they were topoisomerase inhibitors that unraveled bacterial DNA. They may not have known that fluoroquinolones caused the expression of Autism related genes, that’s a recent discovery, but any claims not to know that these drugs are dangerous involves a huge amount of willful ignorance. In 1998 Stephen Fried published Bitter Pills: Inside the Hazardous World of Legal Drugs, describing his wife Diane’s severe adverse reaction to Floxin (a fluoroquinolone that is no longer popular, but is still available) that included severe CNS issues. Before and since publication of Bitter Pills, article after article, research paper after research paper has been published noting one danger of fluoroquinolones after another. Here is just a small sample of the information about the immediate dangers of fluoroquinolone antibiotics – http://floxiehope.com/links-resources/. So many people have been needlessly hurt by these drugs. Many of those who have been hurt have been screaming about their pain, trying to get people to listen, trying to save others from their sad fate – and their warnings have been unheeded. It is to be determined whether or not this article in Nature will change anything, whether or not people will pay attention to the canaries in the coalmine. I hope so. Regardless of whether or not the harm that has been done to human DNA is reversible, people deserve to know the truth. They deserve to know why autism rates have gone from 1 in 1,000 children in 1980 to 1 in 88 children in 2013. They deserve to be compensated for their losses. They deserve to be able to make appropriate and informed decisions regarding the drugs they take and, sadly, the reproduction choices they make.

Nalidixic Acid, the foundation of all quinolone and fluoroquinolone antibiotics was discovered in 1962 by George Lesher.  It took 51 years and indescribable damage to the human gene pool for the dangers of this DNA altering substance to be revealed.  May this be a lesson for all people intentionally altering the genes of humans, animals or plants.

Other Factors

Of course, there are some factors other than gene expression that contribute to Autism Spectrum Disorders. Sadly, many of those can also be explained by fluoroquinolone use. Direct application of fluoroquinolones (ear and eye drops that are fluoroquinolone based are commonly prescribed to children as young as 1 to treat their ear and eye infections, as opposed to the inherited exposure described above, has been shown to cause disruption of tubulin assembly, mitochondrial damage, and a cascade of interrelated brain and nervous system damage stemming from the ability of certain drugs and substances to inhibit deacetylation of histone.  All of these things have also been linked to Autism.

A Note About Vaccines

Studies have shown that, “certain individuals with a mild mitochondrial defect may be highly susceptible to mitochondrial specific toxins like the vaccine preservative thimerosal.”  Drugs that damage mitochondria, fluoroquinolones are not the only drugs that do so, combined with vaccines, can be toxic and can lead to a marked increase in oxidative stress, production of reactive oxygen species, cell death and possibly Autism Spectrum Disorders.

Adverse reactions to direct application of fluoroquinolones (again, as opposed to inherited exposure and direct application can come in the form of orally administered fluoroquinolones, ie pills, intravenously administered fluoroquinolones or topically administered fluoroquinolones, ie ear and eye drops) are often both delayed and triggered by exposure to another toxin. The 2008 lawsuit that got the Black Box Warning of tendon ruptures added to the warning label of orally and intravenously administered fluoroquinolones accepted drug reaction times of up to four months after the fluoroquinolone was taken as a reasonable time frame. Also, the warning labels for fluoroquinolones note that adverse reactions can occur several months after administration of the drug has stopped. Unfortunately, the evidence that I have for adverse reactions to fluoroquinolones being triggered by another toxin are anecdotal. However, I think that the anecdotes are illustrative. In my personal case, my adverse reaction to Cipro started a full two weeks after I had FINISHED taking the Cipro, when I started taking ibuprofen, a NSAID. Enough other people who are also suffering from fluoroquinolone toxicity also have reported adverse reactions to NSAIDs (and the warning label says, “Non-steroidal anti-inflammatory drugs (but not acetyl salicylic acid) in combination of very high doses of quinolones have been shown to provoke convulsions in pre-clinical studies.” http://www.accessdata.fda.gov/drugsatfda_docs/label/2013/019537s082,020780s040lbl.pdf though I did not take NSAIDs in combination with the quinolone/fluoroquinolone nor have I suffered through convulsions – yet) for me to think that the ibuprofen triggered my severe adverse reaction. The CAUSE of my adverse reaction was the Cipro that I had taken two weeks earlier. The ibuprofen was simply the trigger. It would not have been dangerous on its own. However, since Cipro had started a cellular adverse reaction in my body, the ibuprofen became dangerous to me. (It should also be noted that my adverse reaction began when I started my period and that hormones may be related to fluoroquinolone toxicity as well – again, sorry for the anecdotally based assertion.) It should be explored whether or not vaccines can TRIGGER adverse reactions to other drugs – especially drugs that damage mitochondria. If this is the case, the earlier administered drugs are the cause of the problem and the vaccines are simply the trigger – but they’re not exactly innocent either. All of these assertions should be explored further than I have the resources or expertise to do.

Tragedy

As someone who has personally been severely adversely effected by a fluoroquinolone antibiotic, Cipro, which caused central, peripheral and autonomic nervous system problems as well as damage to the connective tissue throughout my body, I always suspected that the dangerous and tragic effects of fluoroquinolones were going to explode into common consciousness at some point. I hoped that it would blow up because of advocacy efforts and that it wouldn’t require a tragedy for the screams of the victims of fluoroquinolones to be noticed. I suspected that my hope was ill-founded though. I thought that a difficult to treat major infection would make its rounds and that everyone would take a fluoroquinolone – and that a large number of people would get sick so that it became undeniable what these drugs did. I imagined a scenario where a foreign leader got sick from a fluoroquinolone and had a fit over it – causing an international relations snafu. Though either of these scenarios would have involved a huge amount of sadness for the victims, they also would have involved some vindication and righteousness on my part over the fact that I KNEW and I tried to warn everyone. Not in my wildest dreams did I imagine that the tragedy caused by fluoroquinolones would be a slow-moving one and that it had been happening over my lifetime. Never would I have imagined that something as insidious and tragic as Autism was the tragedy caused by fluoroquinolones and that one in eighty-eight children would be effected. Never would I have imagined that our DNA would be so profoundly affected by these dangerous drugs that even if everything that I wished for – that their use be severely restricted and that victims of these drugs be compensated – came true, that the havoc that these drugs caused would not be stopped. I am profoundly and deeply saddened by this situation. I cannot express how much my soul aches over the fact that the victims of these drugs are children, the innocent among us, those who need our protection, the babies. They have been let down. Humanity has been let down and I cannot quit sobbing for all of our souls.

Read more from the author at www.floxiehope.com

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8 Natural Alternatives For Commonly Used Antibiotics

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In Brief

  • The Facts:

    While antibiotics are prescribed to be helpful, their bug-killing ability unfortunately is unable to distinguish between good and bad microbiomes. Thankfully, all medications are derived from plants, therefore there are many natural alternatives.

  • Reflect On:

    How many antibiotics do you regularly rely upon? Have you tried any of the outlined alternatives to see if they can provide you the same relief without the long-term side effects?

Antibiotics are medicines used to prevent and treat bacterial infections and can be life-saving. However, most people have no idea how much we are consuming and how they can negatively affect our health.

So many of us are prescribed antibiotics at the first sign of an illness, and we are eating pesticides, and antibiotics in our conventionally raised food. Also, according to many sources including Scientific American, pharmaceuticals including antibiotics are often found in our water supply.

So, What’s The Harm?

Antibiotics are designed to kill “bad bugs” however cannot distinguish between good and bad microbiomes in the body and seeks to destroy all. Leaving the immune system compromised. We see an enormous upswing in autoimmune diseases, and it seems to coincide with the massive overuse of antibiotics. If we wipe out the good gut bacteria that is involved with the strengthening the immune system, there is nothing to stop the autoimmune response which leads to inflammation and disease.

Also, according to the World Health Organization, the overuse of antibiotics is causing antibiotic resistance which is an increasingly severe threat because bacterial infections such as MRSA are becoming harder and harder to treat, which could lead to death. In 2015, antibiotic-resistant pathogens were estimated to cause over 50,000 deaths a year in Europe and the USA. The same medical journal concluded that the frequent use of antibiotics is disrupting and harming our bodies ability to distinguish pathogens from normal cells and bacteria.

Use Natural/ Plant-Based Antibiotics Instead

Most people have no idea that all medications are derived from plants and brought to a factory and created with chemicals, hence side effects. Many plants have antimicrobial, antibacterial and antifungal properties. It’s time to return to tradition.

1. Honey

Honey has been used for centuries and possesses antimicrobial properties. It has been known to heal burns, and wounds and it also provides a protective shield to prevent infection. Manuka honey has a significant antibacterial effect.

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2. Oregano

Oregano fights many strains of bacterial, viral and fungal infections, sometimes better than its prescription counterparts. Oregano is commonly used to fight Streptococcus when combined with olive leaf extract. According to the Microbiology Journal, Oregano even fights intestinal bacterial infections including E.Coli and is an effective treatment for candida overgrowth. It is believed to destroy the cell membrane of dangerous pathogens to stop them from replicating.

3. Shiitake Mushrooms

Shiitake Mushrooms naturally boost the immune system and contain antimicrobial properties. These mushrooms packed with many essential amino acids are a nutrient powerhouse which also improves brain function.

4. Thyme

Thyme heals most wounds, especially in the gut. This fantastic herb treats dysbiosis and helps the body rebalance bacteria in your body. The compound found in thyme oil is called thymol which is an antimicrobial, antifungal and antiparasitic remedy.

5. Garlic

Garlic is one of the best natural antibiotics. The unique Sulphur compound in garlic helps resolve illnesses of all kinds including pneumonia, prevents food poisoning, and it improves intestinal health. Unlike traditional medications, garlic like many herbs is intelligent. Garlic is highly effective in killing off a host of gut pathogens including candida, while at the same time feeding and encouraging the growth of the beneficial microbes.

6. Goldenseal

Goldenseal has a long history of use by Native American Indians to treat a variety of infections. It is commonly used for sore throats, colds, flu, bronchitis, and streptococcus. Containing a compound called Berberine, Goldenseal is greatly useful.

7. Clove

Clove has an antioxidant and antimicrobial activity which is higher than many other fruits, vegetables, and other herbs. Clove is such a powerful natural medicine, it is believed to treat even the most stubborn staph infection.

8. Colloidal Silver

Colloidal Silver is an old-time remedy that destroys disease-causing pathogens in minutes when exposed to small amounts, according to over 650 documents released by Larry C. Ford, MD.

FLU REMEDY – Wipe it out Quickly: Sovereign Silver, organic elderberry syrup, vitamin C, vitamin D, and l-lysine.

Please note: when fighting a more severe infection, a longer course of treatment may be required.

Bonus Natural Alternative: Echinacea

Echinacea has been used for centuries and is making a comeback in recent years. This North American flower It is highly effective in fighting infections, especially the common cold, and the flu. When coupled with Goldenseal, it’s a dynamic duo.

The Takeaway

Avoid eating non-organic, and grain fed meats and find a local organic farmer to help ensure the integrity of the food you are eating. Filter your water with a high-quality filtration system, such as Berkey. Explore natural alternatives to antibiotics with your Functional Medicine M.D. or Naturopathic Physician. They can help you explore your options and discuss the possible risks and benefits of each regimen.

Don’t take antibiotics unless absolutely necessary. And, if you do take antibiotic medication, make sure to take probiotics such as ABX Support by Klaire Labs (four hours away from antibiotics). And, make sure to finish the prescription to help avoid antibiotic resistance.


Learn more about my family’s healing journey (including everything that has worked for me and many of my clients) in my book Healing Without Hurting. And if you’re looking for more personalized solutions and support Dr. Brad Ellisor and I have launched a new Private Group geared to give you the personal attention and answers you need. LEARN MORE

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Antidepressants Cause Severe Withdrawal Symptoms Like Hallucination, Mania, & Anxiety, Study Reveals

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In Brief

  • The Facts:

    Another study has emerged outlining the harmful health consequences of taking antidepressant drugs. Not only do pharmaceutical companies lie about their benefits, but they also conceal their harm.

  • Reflect On:

    There are other ways to deal with depression that are more effective than medication. Placebo, for example, exercise, a plant-based diet, meditation etc.

This article was written by Sayer Ji, founder of Greenmedinfo.com. Posted here with permission. You can sign up for their newsletter here.

A concerning new study published in the journal Addictive Behavior and titled, “A systematic review into the incidence, severity and duration of antidepressant withdrawal effects: Are guidelines evidence-based?,” reveals that antidepressants are far more addictive and harmful than previously assumed, and vindicates the long time activism on this issue spearheaded by American psychiatrists like Kelly Brogan, MD and Peter Breggin, MD.

Highlights from the paper are as follows:

  • More than half (56%) of people who attempt to come off antidepressants experience withdrawal effects.
  • Nearly half (46%) of people experiencing withdrawal effects describe them as severe.
  • It is not uncommon for the withdrawal effects to last for several weeks or months.
  • Current UK and USA Guidelines underestimate the severity and duration of antidepressant withdrawal, with significant clinical implications.

This study aimed to assess the veracity of the the U.K.’s current National Institute for Health and Care Excellence and the American Psychiatric Association’s depression guidelines which state that withdrawal reactions from antidepressants are ‘self-limiting’ (i.e. typically resolving between 1 and 2 weeks).

In order to accomplish this goal the systematic review used the following methods:

“A systematic literature review was undertaken to ascertain the incidence, severity and duration of antidepressant withdrawal reactions. We identified 23 relevant studies, with diverse methodologies and sample sizes.”

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The results were reported as follows:

“Withdrawal incidence rates from 14 studies ranged from 27% to 86% with a weighted average of 56%. Four large studies of severity produced a weighted average of 46% of those experiencing antidepressant withdrawal effects endorsing the most extreme severity rating on offer. Seven of the ten very diverse studies providing data on duration contradict the UK and USA withdrawal Guidelines in that they found that a significant proportion of people who experience withdrawal do so for more than two weeks, and that it is not uncommon for people to experience withdrawal for several months.”

Side effects were wide-ranging, lasting several months or longer (including permanent dysfunction), such as: 

“Typical AD withdrawal reactions include increased anxiety, flu-like symptoms, insomnia, nausea, imbalance, sensory disturbances, and hyperarousal. Dizziness, electric shock-like sensations, brain zaps, diarrhoea, headaches, muscle spasms and tremors, agitation, hallucinations, confusion, malaise, sweating and irritability are also reported (Warner, Bobo, Warner, Reid, & Rachal, 2006, Healy, 2012). Although the aforementioned symptoms are the most common physical symptoms, there is also evidence that AD withdrawal can induce mania and hypomania, (Goldstein et al., 1999; Naryan & Haddad, 2011) emotional blunting and an inability to cry, (HolguinLew & Bell, 2013) long-term or even permanent sexual dysfunction (Csoka & Shipko, 2006).”

The study concluded:

“We recommend that U.K. and U.S.A. guidelines on antidepressant withdrawal be urgently updated as they are clearly at variance with the evidence on the incidence, severity and duration of antidepressant withdrawal, and are probably leading to the widespread misdiagnosing of withdrawal, the consequent lengthening of antidepressant use, much unnecessary antidepressant prescribing and higher rates of antidepressant prescriptions overall. We also recommend that prescribers fully inform patients about the possibility of withdrawal effects.”

The researchers also noted that the rising numbers of antidepressant prescriptions used throughout the world may be fueled by the antidepressant drug withdrawal side effects themselves:

“As the lengthening duration of AD use has fuelled rising AD prescriptions over the same time period, we must understand the drivers of such lengthening use. The evidence set out suggests that lengthening use may be partly rooted in the underestimation of the incidence, severity and duration of AD withdrawal reactions, leading to many withdrawal reactions being misdiagnosed, for example, as relapse (with drugs being reinstated as a consequence) or as failure to respond to treatment (with either new drugs being tried and/or dosages increased). This issue is pressing as long-term AD use is associated with increased severe side-effects, increased risk of weight gain, the impairment of patients’ autonomy and resilience (increasing their dependence on medical help), worsening outcomes for some patients, greater relapse rates, increased mortality and the development of neurodegenerative diseases, such as dementia.”

The concerning implications of this study to millions around the world who are on antidepressants were immediately recognized by the media, as evidenced by mainstream reporting on the topic with the following headlines:

Thanks to a small but courageous group of professionals who have been raising awareness of the profound, unintended adverse effects of psychiatric drugs and the abject absence of objective criteria for determining “mental disease,” not only are there already resources available to the public today to better understand the dangers of psychiatric drugs, but there are also programs and protocols in place to help those who are on them to come off of them safely and with the support of others who have done the same already. For instance, the program put together by Dr. Kelly Brogan — Vital Mind Reset — has produced powerful outcomes. Take a look at the testimony wall here to learn from the first hand experiences of those who underwent the program and came out drug-free, often with their psychiatric symptoms and comorbid conditions reduced or completely put into remission.

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In order to stay truly independent, we need your help. We are not going to put up paywalls on this website, as we want to get our info out far and wide. For as little as $3 a month, you can help keep CE alive!

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Scientists Break Down How Aging Is “Plastic” & We Can Manipulate It To Slow Down Aging

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In Brief

  • The Facts:

    Slowing down the ageing process is not about looks, it's about health, and feeling good. Scientists have discovered multiple healthy ways to regenerate our immune systems and repair our DNA, and caloric restriction/fasting is one of them.

  • Reflect On:

    Why are we told to eat three meals a day? Why are our national food guides more of a guide towards bad health rather than health? Why have many people stopped caring about health? To change the world, we have to change ourselves in multiple ways.

Can we reverse age regression, or slow it down? Given our research into Black Budget programs, it’s clear what we know in the mainstream scientific world differs greatly from the world of secrecy. We recently conducted an interview with a neuroscientist from the University of Arizona who also makes a clear distinction between mainstream science and Black Budget science.

From a mainstream scientific standpoint, it is reversible. At least in human cells and in mice. 

This is why it’s always interesting to ponder just how advanced the world might be. The U.S. air strike against Libya in 1986 used the F-111 fighter aircraft, for instance, but not the F-117A Nighthawk. The latter was still classified at the time, and keeping it secret was more important than using it for this mission. Then there’s the National Reconnaissance Office, which was founded in 1960 but remained completely secret for 30 years. What type of technology were/are they using? Does the NSA have computers that are far more advanced than ours? Can we teleport? Can we travel faster than the speed of light? Is there a secret space program? Can humans be cloned?

While these questions might conflict with many people’s belief systems, they represent valid concerns. Another question worth asking is, can we reverse age regression? We have no idea what military technology is capable of, or how far beyond us it has progressed. Considering the advancements in technology in the past century alone within the mainstream scientific/technical world, these things are hardly beyond our grasp.

But let’s take a look at what we do know. We are, after all, living in a world where science fiction is becoming a reality.

Aging Is Reversible

Today, scientists are actually able to tweak genes that turn adult cells back into embryonic-like ones. For example, it wasn’t long ago that researchers at the Salk Institute for Biological Studies reversed the aging of human and mouse cells, in vitro. The study was published in the journal Cell

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According to Juan Carlos Izpisua Belmonte, the study’s senior author and an expert in gene expression at Salk, “aging is something plastic that we can manipulate.” In living mice, they activated what are known as “Yamanaka factors,” which rejuvenated muscles that were damaged, as well as the pancreas in a middle-aged mouse. This extended the lifespan of the mouse, who also had a genetic mutation for Hutchinson-Gilford progeria syndrome, which causes rapid aging in children.

The researchers believe that this study suggests it’s not just possible to slow the aging process, but actually reverse it.

“I fully agree with the conclusions. This work indicated that epigenetic shift is parr responsible for aging, and reprogramming can correct these epigenetic errors. This will be the basis for future exciting developments.”

– Manuel Serrano from the Spanish National Cancer Research Center In Madrid

Epigenetics is the study of changes in organisms caused by gene expression, and gene expression can change due to a myriad of factors.

But, as Scientific American points out“The study also showed how fine the line can be between benefit and harm. When the researchers treated mice continually, some developed tumors and died within a week. When the scientists cut the treatment to two days out of seven, however, the mice benefited significantly.” 

The lead author also told Scientific American that they “currently think the brain’s hypothalamus—known as the seat of control for hormones, body temperature, mood, hunger and circadian rhythms—may also act as a regulator of aging.”

According to the Telegraph, with the success of these animals studies, scientists predict human trials to commence within 10 years.

Caloric Restriction and Fasting 

Did you know that, in all animal model studies, caloric restriction reverses signs of aging, slowing it down, and reverses age-related diseases? Research has shown that it reduces what’s called the PKA enzyme, which has been linked to aging, tumour progression, and cancer.

According to a review of fasting literature conducted in 2003“Calorie restriction (CR) extends lifespan and retards age-related chronic diseases in a variety of species, including rats, mice, fish, flies, worms, and yeast. The mechanism or mechanisms through which this occurs are unclear.”

Fasting and caloric restriction have also shown to have a tremendous effect on the brain. As an article from John Hopkins Magazine reveals:

Dietary changes have long been known to have an effect on the brain. Children who suffer from epileptic seizures have fewer of them when placed on caloric restriction or fasts. It is believed that fasting helps kick-start protective measures that help counteract the overexcited signals that epileptic brains often exhibit. (Some children with epilepsy have also benefited from a specific high-fat, low-carbohydrate diet.) Normal brains, when overfed, can experience another kind of uncontrolled excitation, impairing the brain’s function.

A plate, fork and knife

Fasting has also been shown to regenerate the immune system and our organs. With regards to the brain, fasting challenges it, and your brain responds to that challenge by adapting stress response pathways that help your brain cope with stress and disease risk. The same changes that occur in the brain during fasting mimic the changes that occur with regular exercise — both increase the production of protein in the brain (neurotrophic factors), which in turn promotes the growth of neurons, the connection between neurons, and the strength of synapses. This is why it’s been found to completely reverse age-related neurodegenerative diseases.

Here is an excellent  TEDx talk given by Mark Mattson, the current Chief of the Laboratory of Neuroscience at the National Institute on Aging. He is also a professor of Neuroscience at Johns Hopkins University, and one of the foremost researchers of the cellular and molecular mechanisms underlying multiple neurodegenerative disorders, like Parkinson’s and Alzheimer’s disease.

We’ve published many articles on fasting, and to find out more information on how to do it, different strategies, and more science, you can start here. Below are a select few related articles:

Neuroscientist Shows What Fasting Does To Your Brain & Why Big Pharma Won’t Study It 

The Complete Guide To Fasting & Reversing Type 2 Diabetes: A Special Inter Interview With Dr. Jason Fung

Why Researchers Are Seeking FDA Approval For Fasting & Caloric Restriction For Cancer Treatment 

Scientists Discover That Fasting Triggers Stem Cell Regeneration & Fights Cancer

Reversing the Age of White Blood Cells

Elizabeth Parris, the CEO of Bioviva USA Inc, has become the very first human being to successfully, from a biological standpoint, reverse the age of her white blood cells, thanks to her own company’s experimental therapies. Bioviva utilizes intramural and extramural peer-reviewed research to create therapies for age-related diseases (Parkinson’s, Alzheimer’s, cancer, heart-disease), and now, they have reversed 20 years of ‘telomere shortening’ in a human for the first time.

Telomeres are short segments of DNA that cap the ends of every chromosome and act as a protective feature against wear and tear, which occurs naturally as the human body ages. As we age, these telomeres become shorter and shorter as our cells continue to divide more and more. Eventually, they become too short to protect the chromosome, which is what causes our cells to malfunction and age-related diseases to start setting in.

We published a story about this early last year, and you can read more about it here:

First Human Being Has Their DNA Manipulated To Make White Blood Cells 20 Years Younger

So, as you can see, even within the mainstream scientific world, we’re not too far off from reversing aging, or slowing it down to prevent age-related diseases. This research represents just the tip of the iceberg, and at our current rate of acceleration with regards to scientific and technological advancement, who knows where we will be in 20 years?

Would age reversal be “playing God?” It’s impossible to say. Perhaps “God” meant us to discover our own intelligence and ability and use these findings for good. Perhaps manipulating our own genes is part of our natural process of human evolution and development. This, however, is a completely separate topic, worthy of another article.

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