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Studies Link DNA Damage from Synthetic Antibiotics To Autism

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antibioticsBayer, the maker of the world’s most unsafe brands of birth control – the Yasmin line and the Mirena/Skyla IUDs, as well as a maker of neocontinid pesticides killing the bees – can now be held responsible for the rise in Autism rates over the last 30 years, as they are the makers and manufacturers of Cipro and Avelox, two of the world’s most popular fluoroquinolone antibiotics, which have recently been shown to alter DNA in ways that encourage expression of Autism related genes. Johnson & Johnson, the maker of Levaquin, also a popular fluoroquinolone antibiotic, can also be held responsible for the atrocity of one in eighty-eight children being autistic, as Levaquin is also a fluoroquinolone antibiotic that does the same thing.

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Cipro (ciprofloxacin) is a second generation fluoroquinolone patented in 1983 by Bayer, Levaquin (levofloxacin) is a third generation fluroquinolone patented in 1987 by Ortho-McNeil-Janssen (a division of Johnson & Johnson), and Avelox (moxifloxacin) is a fourth generation fluoroquinolone patented in 1991 by Bayer. In the 1980s the incidence of Autism was 1 per 1,000 children, today it is 1 per 88 children. The incidence of Autism has gone up hand in hand with the use of fluoroquinolone antibiotics.

Of course, this correlation between the introduction of fluoroquinolone antibiotics to the market and increasing rates of Autism, proves nothing. For proof, studies, experimentation and scientific exploration are needed. In September, 2013 the studies of topoisomerase inhibitors like fluoroquinolones as they relate to Autism commenced with an article in Nature entitled “Topoisomerases facilitate transcription of long genes linked to autism.”

Fluoroquinolone Antibiotics Lead to Autism Gene Expression

Fluoroquinolone antibiotics are eukaryotic DNA gyrase (also known as topoisomerase II) and topoisomerase IV inhibitors. Topoisomerases “are integral to gene expression, as they resolve DNA supercoiling that is generated during transcription.” Here is a video describing what that means and how fluoroquinolones work –


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A 1999 study in Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis showed that fluoroquinolone antibiotics damage and destroy mitochondrial DNA. It makes sense that they would do so because mitochondria are the parts of our cells that are most closely related to bacteria.  Bacteria are destroyed by fluoroquinolone antibiotics through the unraveling of bacterial DNA and resulting apoptosis (programmed cell death). Sadly, mitochondria also suffer the same fate.

An article published in September, 2013 in Nature entitled “Topoisomerases facilitate transcription of long genes linked to autism” noted that topoisomerase inhibitors can adversely affect gene expression and, “topoisomerases facilitate the expression of a large number of ASD (Autism Spectrum Disorder) candidate genes, including many that are long and that are thought to have large effects on ASD pathology.” Basically, drugs that effect topoisomerases, chemotherapy drugs and fluoroquinolone antibiotics (which are chemotherapy drugs) can cause Autism genes to be expressed.

The article concludes by stating that:

“Our data suggest that chemicals or genetic mutations that impair topoisomerases, and possibly other components of the transcription elongation machinery that interface with topoisomerases, have the potential to profoundly affect the expression of long ASD candidate genes. Length-dependent impairment of gene transcription, particularly in neurons and during critical periods of brain development, may thus represent a unifying cause of pathology in in many individuals with ASD and other neurodevelopmental disorders.”

THIS IS HUGE. It is an environmental factor, topoisomerase inhibiting chemotherapy drugs including fluoroquinolone antibiotics, that cause the expression of Autism genes.

It should be noted that the specific drug studied in “Topoisomerases facilitate transcription of long genes linked to autism” is Topotecan, not fluoroquinolones. Topotecan is a topoisomerase I inhibitor whereas fluoroquinolone atibiotics are topoisomerase II (also known as DNA gyrase) and topoisomerase IV inhibitors. Further studies need to be done to definitively show whether or not fluoroquinolones have the same adverse effects as Topotecan.

HOWEVER, the Researchers note that:

TOP2 (topoisomerase II) enzymes, (particularly TOP2B) also participate in gene transcription. We next tested whether genetic or pharmacological inhibition of TOP2 enzymes could reduce the expression of long genes. Indeed, with new experiments and by re-analyzing data from others, we found that the TOP2A/TOP2B inhibitor ICRF-193 reduced gene expression in a length-dependent manner in cultured mouse cortical neurons, embryonic stem (ES) cells and ES-cell-derived neurons. There was extensive overlap between genes affected by ICRF-193 and topotecan in cortical neurons, particularly for long genes, and the magnitudes of these effects were highly correlated. Thus, TOP1 (topoisomerase I) and TOP2 (topoisomerase II) enzymes regulate the expression of many of the same genes.

This means that topoisomerase II inhibitors, also known as DNA gyrase inhibitors, such as fluoroquinolones do the SAME THING as the topoisomerase I inhibitors studied. Fluoroquinolone antibiotics – Cipro, Levaquin, Avelox and a few more – “profoundly affect the expression of long ASD candidate genes.”

26.9 MILLION prescriptions for fluoroquinolone antibiotics were given to people in 2011 alone. Quantities of prescriptions of these DNA altering drugs have been in the millions since their rise in popularity in the 1980s.

These popular antibiotics, prescribed for sinus infections, urinary tract infections, strep throat, traveler’s diarrhea, prostate infections, etc. ALTER DNA AND NEGATIVELY INFLUENCE GENE EXPRESSION (EPI-GENETICS) AND LEAD TO THE EXPRESSION OF AUTISM GENES. Genes. DNA. Epi-genetics. The bits of information that our cells pass from one generation to the next. The damaged DNA is passed from parent to child and from that child down to their children, and so on and so on.

Gene expression, epi-genetics, is tricky because so many factors influence how genes are expressed, and it is only recently that ways of measuring and describing epi-genetics have been discovered. Neil deGrasse Tyson is probably better at explaining epi-genetics than I am, so please check out this video –

One of the research scientists who Dr. deGrasse Tyson interviews notes that, “you’re not only what you eat, (you are) potentially what your mother ate and even what your grandparents ate” to show the power of the heritability of epi-genetic markers. Dr. deGrasse Tyson and the research scientist are discussing how food can effect epi-genetic markers and influence obesity throughout generations of mice. If food can effect epi-genetics that dramatically, just imagine how dramatically a drug that is intentionally designed to interfere with and unravel mitochondrial DNA can effect human heredity. Not only can a drug that a parent (not just mothers, the DNA of fathers contributes to 50% of everyone’s genes) took influence the epi-genetics of their child, but the epi-genetics of their grandchildren and great-grandchildren can be adversely effected as well.

So, children are inheriting damaged genes, the genes that control whether or not a person is Autistic, because their parent (or even grandparent – for future generations – fluoroquinolones haven’t been around long enough to mess up multiple generations of people – yet) took an ANTIBIOTIC that messed up their mitochondrial DNA. This is absurd. Generations of humans will continue to be plagued by high autism rates, possibly indefinitely, because a certain class of antibiotics that has dangerous and severe side-effects even to those who take them directly, has damaged their mitochondrial DNA. This entire horrifying situation is Bayer’s and Johnson & Johnson’s fault and they should be held accountable to humanity in every way possible.

Let me just back-pedal a little bit and say that many women who have been severely adversely effected by fluoroquinolone antibiotics have had healthy, happy, beautiful, smart babies. Many factors go into how genes are expressed. What you eat, music, positive thoughts and words, etc. can influence how your genes are expressed. If you have taken a fluoroquinolone antibiotic, you are not doomed to have an autistic child. But it really seems shameful, horrifying actually, to increase someone’s odds of having an autistic child by encouraging the expression of autism related genes synthetically, through an unneeded and destructive antibiotic, when other, safer, non-DNA-damaging antibiotics are available.

If I can figure out that fluoroquinolones have the same effects as Topotecan, and that damaged DNA and gene expression / epi-genetics markers can be passed on from generation to generation, with my Cipro frazzled brain, you can bet that Dr. Zylka and his team of undoubtedly brilliant Scientists also realize that topoisomerase II / DNA gyrase (and topoisomerase IV) inhibitors like fluoroquinolones are leading to the expression of autism genes. I thank these Scientists for what they have uncovered and published from the bottom of my heart, and I beg of them, please have enough courage and moral fortitude to stand up for what you know to be true – that fluoroquinolone antibiotics made by Bayer and Johnson & Johnson have severely, and possibly irreversibly, damaged the human gene pool in a way that is causing children, innocent children, to be hurt. Though there is little that can be done to stop the damage from being passed down through the generations, the people who have already been hurt by Bayer and J&J deserve compensation. The children who are living with Autism deserve compensation. Bayer and J&J hurt them, they hurt humanity, and they should pay for their sins. Without the word of Scientists to back up these assertions, no justice will ever come to the families. Dr. Zylka and others… please, do what’s right.

The Dangers of Fluoroquinolones Shouldn’t be a Surprise

This coalmine is littered with dead canaries. The Scientists who designed fluoroquinolones always knew that they were topoisomerase inhibitors that unraveled bacterial DNA. They may not have known that fluoroquinolones caused the expression of Autism related genes, that’s a recent discovery, but any claims not to know that these drugs are dangerous involves a huge amount of willful ignorance. In 1998 Stephen Fried published Bitter Pills: Inside the Hazardous World of Legal Drugs, describing his wife Diane’s severe adverse reaction to Floxin (a fluoroquinolone that is no longer popular, but is still available) that included severe CNS issues. Before and since publication of Bitter Pills, article after article, research paper after research paper has been published noting one danger of fluoroquinolones after another. Here is just a small sample of the information about the immediate dangers of fluoroquinolone antibiotics – http://floxiehope.com/links-resources/. So many people have been needlessly hurt by these drugs. Many of those who have been hurt have been screaming about their pain, trying to get people to listen, trying to save others from their sad fate – and their warnings have been unheeded. It is to be determined whether or not this article in Nature will change anything, whether or not people will pay attention to the canaries in the coalmine. I hope so. Regardless of whether or not the harm that has been done to human DNA is reversible, people deserve to know the truth. They deserve to know why autism rates have gone from 1 in 1,000 children in 1980 to 1 in 88 children in 2013. They deserve to be compensated for their losses. They deserve to be able to make appropriate and informed decisions regarding the drugs they take and, sadly, the reproduction choices they make.

Nalidixic Acid, the foundation of all quinolone and fluoroquinolone antibiotics was discovered in 1962 by George Lesher.  It took 51 years and indescribable damage to the human gene pool for the dangers of this DNA altering substance to be revealed.  May this be a lesson for all people intentionally altering the genes of humans, animals or plants.

Other Factors

Of course, there are some factors other than gene expression that contribute to Autism Spectrum Disorders. Sadly, many of those can also be explained by fluoroquinolone use. Direct application of fluoroquinolones (ear and eye drops that are fluoroquinolone based are commonly prescribed to children as young as 1 to treat their ear and eye infections, as opposed to the inherited exposure described above, has been shown to cause disruption of tubulin assembly, mitochondrial damage, and a cascade of interrelated brain and nervous system damage stemming from the ability of certain drugs and substances to inhibit deacetylation of histone.  All of these things have also been linked to Autism.

A Note About Vaccines

Studies have shown that, “certain individuals with a mild mitochondrial defect may be highly susceptible to mitochondrial specific toxins like the vaccine preservative thimerosal.”  Drugs that damage mitochondria, fluoroquinolones are not the only drugs that do so, combined with vaccines, can be toxic and can lead to a marked increase in oxidative stress, production of reactive oxygen species, cell death and possibly Autism Spectrum Disorders.

Adverse reactions to direct application of fluoroquinolones (again, as opposed to inherited exposure and direct application can come in the form of orally administered fluoroquinolones, ie pills, intravenously administered fluoroquinolones or topically administered fluoroquinolones, ie ear and eye drops) are often both delayed and triggered by exposure to another toxin. The 2008 lawsuit that got the Black Box Warning of tendon ruptures added to the warning label of orally and intravenously administered fluoroquinolones accepted drug reaction times of up to four months after the fluoroquinolone was taken as a reasonable time frame. Also, the warning labels for fluoroquinolones note that adverse reactions can occur several months after administration of the drug has stopped. Unfortunately, the evidence that I have for adverse reactions to fluoroquinolones being triggered by another toxin are anecdotal. However, I think that the anecdotes are illustrative. In my personal case, my adverse reaction to Cipro started a full two weeks after I had FINISHED taking the Cipro, when I started taking ibuprofen, a NSAID. Enough other people who are also suffering from fluoroquinolone toxicity also have reported adverse reactions to NSAIDs (and the warning label says, “Non-steroidal anti-inflammatory drugs (but not acetyl salicylic acid) in combination of very high doses of quinolones have been shown to provoke convulsions in pre-clinical studies.” http://www.accessdata.fda.gov/drugsatfda_docs/label/2013/019537s082,020780s040lbl.pdf though I did not take NSAIDs in combination with the quinolone/fluoroquinolone nor have I suffered through convulsions – yet) for me to think that the ibuprofen triggered my severe adverse reaction. The CAUSE of my adverse reaction was the Cipro that I had taken two weeks earlier. The ibuprofen was simply the trigger. It would not have been dangerous on its own. However, since Cipro had started a cellular adverse reaction in my body, the ibuprofen became dangerous to me. (It should also be noted that my adverse reaction began when I started my period and that hormones may be related to fluoroquinolone toxicity as well – again, sorry for the anecdotally based assertion.) It should be explored whether or not vaccines can TRIGGER adverse reactions to other drugs – especially drugs that damage mitochondria. If this is the case, the earlier administered drugs are the cause of the problem and the vaccines are simply the trigger – but they’re not exactly innocent either. All of these assertions should be explored further than I have the resources or expertise to do.

Tragedy

As someone who has personally been severely adversely effected by a fluoroquinolone antibiotic, Cipro, which caused central, peripheral and autonomic nervous system problems as well as damage to the connective tissue throughout my body, I always suspected that the dangerous and tragic effects of fluoroquinolones were going to explode into common consciousness at some point. I hoped that it would blow up because of advocacy efforts and that it wouldn’t require a tragedy for the screams of the victims of fluoroquinolones to be noticed. I suspected that my hope was ill-founded though. I thought that a difficult to treat major infection would make its rounds and that everyone would take a fluoroquinolone – and that a large number of people would get sick so that it became undeniable what these drugs did. I imagined a scenario where a foreign leader got sick from a fluoroquinolone and had a fit over it – causing an international relations snafu. Though either of these scenarios would have involved a huge amount of sadness for the victims, they also would have involved some vindication and righteousness on my part over the fact that I KNEW and I tried to warn everyone. Not in my wildest dreams did I imagine that the tragedy caused by fluoroquinolones would be a slow-moving one and that it had been happening over my lifetime. Never would I have imagined that something as insidious and tragic as Autism was the tragedy caused by fluoroquinolones and that one in eighty-eight children would be effected. Never would I have imagined that our DNA would be so profoundly affected by these dangerous drugs that even if everything that I wished for – that their use be severely restricted and that victims of these drugs be compensated – came true, that the havoc that these drugs caused would not be stopped. I am profoundly and deeply saddened by this situation. I cannot express how much my soul aches over the fact that the victims of these drugs are children, the innocent among us, those who need our protection, the babies. They have been let down. Humanity has been let down and I cannot quit sobbing for all of our souls.

Read more from the author at www.floxiehope.com

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Awareness

CDC’s Recommendation for Hepatitis B Vaccination in Infants. Are There More Risks Than Benefits?

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In Brief

  • The Facts:

    The CDC’s recommendation for universal hepatitis B vaccination of infants puts most children at unnecessary risk of harm from the vaccine. By Jeremy R. Hammond, Contributing Writer, Children’s Health Defense

  • Reflect On:

    How much do physicians really know about vaccines?

Parents are told by public health officials and the media that they should vaccinate their children strictly according to the schedule recommended by the US Centers for Disease Control and Prevention (CDC). The CDC’s routine childhood vaccine schedule is based on solid science, we are told, and it is imperative that all parents comply to reduce the societal disease burden. Anyone who dares to criticize or dissent from public vaccine policy is characterized as dangerously ignorant and irrational. A recent New York Times editorial, for example, characterized anyone who does so as “the enemy” and described all vaccines on the CDC’s schedule as “crucial shots”.

So is the HepB vaccine really necessary for all infants? Why does the CDC treat this vaccine as a one-size-fits-all solution when the vast majority of infants are not at significant risk of infection?

But is it really “crucial” for all children to be so vaccinated? To highlight the rationality and importance of this question, consider the example of the CDC’s recommendation that all newborn babies receive a hepatitis B (HepB) vaccine, typically on their very first day of life. Many parents naturally wonder why it is considered so necessary to vaccinate their baby against a virus that is primarily transmitted sexually or through sharing of needles among injection drug users. The hepatitis B virus (HBV) can also be transmitted to infants at birth if the mother is a carrier, but screening to identify infected pregnant women is done routinely, and an alternative effective treatment has long been available for infants born to carriers. So is the HepB vaccine really necessary for all infants? Why does the CDC treat this vaccine as a one-size-fits-all solution when the vast majority of infants are not at significant risk of infection?

To answer this question, we need look no further than the CDC’s own stated rationale for this policy, which was adopted in 1991. Close examination of the CDC’s reasoning and the evolution of this policy illustrates that, far from being based on science, the decision by the CDC’s vaccine advisory committee to adopt this policy was faith-based and concerned primarily not with the health of infants, but with the agency’s overriding goal of achieving high vaccination rates.Comparing the policy with the science reveals that parents are right to be concerned because the policy unnecessarily puts children who are not at risk of infection at risk of harm from the vaccine.

The Risk to Infants of Hepatitis B Infection

To place the CDC’s stated rationale for this policy into proper context, it’s important to understand a little bit about the nature of the virus and the risk it poses generally to the population and particularly to infants.

According to the CDC’s “Pink Book”, while most acute hepatitis B infections among adults are effectively dealt with by the host’s immune system, chronic infection is a known cause of liver disease, contributing significantly to the disease burden of cirrhosis and hepatocellular carcinomas. Most children and about half of adults with acute infection do not show any symptoms. Those with chronic infection may also be asymptomatic but are known as “carriers” since they still carry and can spread the virus.

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Subpopulations at highest risk therefore include sexually active individuals, injection drug users, health care workers, and children who are born to infected mothers…

Transmission of the virus occurs through infected blood or other bodily fluids. Subpopulations at highest risk therefore include sexually active individuals, injection drug users, health care workers, and children who are born to infected mothers or otherwise come into prolonged close contact with infected household members. Mother-to-infant transmission usually occurs during birth. If an environmental surface is contaminated, the virus can remain stable and infectious for seven or more days, so indirect transmission, while unlikely, is also possible. Replication of the virus occurs only in liver tissue.

Most adults completely recover from acute infection and come away with lasting immunity. However, 1 percent to 2 percent of acute cases result in fulminant disease. Among these cases, 63 percent to 93 percent will result in death. About 200 to 300 deaths occur each year in the US due to severe HBV disease.

Before routine childhood vaccination, more than 80 percent of acute infections occurred in adults, about 8 percent in adolescents, and about 4 percent in children infected through perinatal transmission. Although at lower risk of becoming infected, such children are at higherrisk of their infection becoming chronic, disproportionately accounting for about 24 percent of chronic infections. While chronic infection occurs in only about 5 percent of adult cases, the risk of an acute infection becoming chronic increases as the age of the host decreases. An estimated 30 percent to 50 percent of infections occurring in children aged one to five years become chronic, and for infants infected from their mothers, the rate is as high as 90 percent.

An estimated 25 percent of individuals with chronic infection will die prematurely from liver disease. About 3,000 to 4,000 people die from HBV-related cirrhosis each year, and another 1,000 to 1,500 die from HBV-related liver cancer.

It is primarily these fatal outcomes in adults—the few hundred deaths from fulminant disease and the few thousand deaths from liver disease—that public health officials have aimed to prevent through mass vaccination.

The hepatitis B virus has a number of different antigen components. (This gets a bit technical, but it’s important context, so bear with me.) The CDC defines an “antigen” as any foreign substance in the body, including but not limited to viruses or bacteria, which is capable of causing disease, and the presence of which triggers an immune response, including but not limited to the production of antibodies. As the CDC’s Pink Book explains, “Several well-defined antigen-antibody systems are associated with HBV infection.” These are the HBV core antigen (HBcAg), another protein contained in the viral core called the HBV e antigen (HBeAg), and a surface antigen (HBsAg).

The presence of HBsAg in the blood indicates infection, but only the complete virus is infectious, not individual antigen components. The presence in the blood of antibodies to this antigen, called “anti-HBs”, is considered indicative of immunity. Infection may also stimulate production of antibodies to HBcAg, or “anti-HBc”, the presence of which indicates past infection. The presence of anti-HBc of the immunoglobulin M class (IgM-anti-HBc) indicates recent infection. Chronic infection is determined by a positive result for HBsAg along with a negative result for IgM-anti-HBc.

The HepB vaccine contains just one viral antigen, HBsAg. Unlike natural infection, the vaccine does not stimulate production of anti-HBc.

For nearly three decades now, the CDC has treated vaccination during early childhood as a one-size-fits-all solution despite the variability in individual immune responses, individual risk from the virus, and individual risk from the vaccine.

Despite the advancements of modern science, much remains unknown about the human immune system and the full impact of viral infection or vaccination. And reading through the CDC’s Pink Book chapter on hepatitis B raises as many questions as it answers. Why do some individuals develop protective anti-HBs to fight off infection while others don’t and hence become carriers? What is the clinical significance of the development of anti-HBc in addition to anti-HBs versus the development only of the latter? In what other ways does natural immunity differ from vaccine-conferred immunity? Why would an individual’s immune system—and particularly children’s immune systems—fail to generate protective antibodies in response to the live virus, yet still be capable of doing so in response to the vaccine? Why do some individuals also fail to develop protective antibodies in response to the vaccine?

One would think that such questions would be relevant for understanding how to develop more effective methods of disease prevention, but answers to them cannot be found in the Pink Book. Indeed, answers to them are not readily found by perusing the broader scientific literature. The most obvious reason for this curiosity is the influence of the pharmaceutical industry and government policies on the direction of scientific research.

For nearly three decades now, the CDC has treated vaccination during early childhood as a one-size-fits-all solution despite the variability in individual immune responses, individual risk from the virus, and individual risk from the vaccine.

Summary

The vast majority of children in the US today are not at significant risk of hepatitis B infection, and yet the CDC nevertheless recommends universal infant vaccination.

Why?

To answer that question, in part two of this series, we will examine the evolution of the CDC’s HepB vaccine recommendations, revealing how the agency began recommending vaccination for pregnant women and infants at high risk of infection despite a complete lack of randomized, placebo-controlled trials demonstrating that these practices are safe.

Then in part three, we’ll examine the CDC’s stated rationale for its 1991 policy shift to recommending that infants be universally vaccinated, typically on the first day of their lives. Part three will show how the CDC itself concluded that its policy was a failure because of low vaccination rates among high-risk groups, as well as illuminate how the agency’s goal of achieving high vaccination rates overrode any considerations of individual risk-benefit analysis, thus placing millions of children at unnecessary risk of neurodevelopmental harm from the vaccine.

Sign up for free news and updates from Robert F. Kennedy, Jr. and the Children’s Health Defense. CHD is planning many strategies, including legal, in an effort to defend the health of our children and obtain justice for those already injured. Your support is essential to CHD’s successful mission.

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Awareness

“I Tried Every Diet & Nothing Worked” How Mucus Free Living Saved This Woman’s Life

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In Brief

  • The Facts:

    After a year on a high-fat/high-protein lifestyle, Livia Macdonald nearly died. After adopting a 'mucus-free' lifestyle, a diet rich in fresh fruit and vegetables, she cured her depression, anxiety, and health issues.

  • Reflect On:

    True healing takes time and commitment, and a willingness to face the emotions and trauma buried beneath our eating habits.

In 2011, Livia Macdonald was looking for answers to her health. At nearly 300 lbs and stuck in the despairs of chronic illness, she was ready to make a big change. The first step—divorcing allopathic medicine all together. Like many others stepping away from conventional medicine, Livia found herself enveloped by the siren of holistic healthcare, adopting the protocols laid out by natural-health celebrity and functional medicine doctor, Mark Hyman.

Following Hyman’s vitality guidelines, Livia cut out grains, starches, and processed sugars, while incorporating more vegetables, ‘healthy’ fats and animal products into her diet.

I was told that high protein and high fats is the way to go because our brain needs fat. I even made my own ghee and ate loads of coconut oil and eggs every day,” she told Collective Evolution.

At first the high-fat diet did wonders for Livia’s health. She felt more energized, had more mental clarity, and even began to drop weight. “I lost almost 80 lbs the first year on the [high-fat] diet,” she said.

But after twelve months of a high-fat lifestyle, Livia said her body began to shut down.

“I started to feel awful. Like everything turned on me. I got severe depression, anxiety, shaking, internal tremors, my organs started to really hurt, I had them checked and my pancreas had so many fat deposits all over it and my cholesterol was through the roof after being optimal. My entire body started to shut down and I became bed ridden for an entire year.”

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During this difficult time Livia came across the work of Dr. Robert Morse, a regenerative detoxification specialist well known in the natural health world. One of the foundations of Dr. Morse’s teachings is that man is a part of the primate family, and therefore we are primarily a frugivore species whose bodies thrive off of fruit, some vegetables and herbs. Livia says that a lightbulb went off in her head immediately upon reading Dr. Morse’s work.

My intuition was screaming that this was the missing piece of my puzzle, and that he speaks the absolute truth.”

Arnold Ehret wrote “The Mucusless Diet Healing System,” a resource for the chronically ill. Ehret’s protocols implement systematic fasting, as well as a diet of raw fruit and vegetables.

Next, Livia discovered the work of a 19th century natural health educator named Arnold Ehret. Ehret’s rise to fame came through his in depth knowledge about the body, specifically in healing chronic disease through systematic fasting and a diet similar to what Morse prescribes—raw fruit and vegetables.

His magnum opus, The Mucusless Diet Healing System, detailed his many years working in a clinic for the chronically ill while implementing his detox protocols to cure their diseases. Ehret’s work garnered a cult-following throughout the early 20th century and inspired the works of well-known detox specialists like Robert Morse himself, Paul Braggs, and Alfredo Bowman.

Adopting A Mucus-Free Lifestyle

But Livia said her biggest aha moment did not come until she discovered the work of South-African detox specialist  Alexandra Cousins. Inspired by the teachings of Robert Morse and Arnold Ehret, Cousins takes their healing principles and merges them with the shamanic and emotional work which she feels is the missing piece for those seeking full-bodied healing.

What I am witnessing is that trauma, PTSD, OCD, addictions are running everyone’s lives,” she writes in her Facebook group, Living Mucus Free. “The degree will vary but we all have it unless we have specifically addressed it. It is safe to say that all my clients, especially the chronically ill suffer from some form of unresolved trauma. If you have adrenal, hormonal, thyroid, or CFS issues, you are dealing with trauma residue. Living mucus free tends to bring up all our unresolved trauma. As we no longer consume foods that numb us or stimulate us, trauma rises to the surface so that it can be felt and dealt with.”

Having endured years of ill-health herself and having tried almost every diet trend out there, Cousins eventually found solace through a lifestyle termed Living Mucus Free (LMF). Mucus, for those wondering, is the residue which builds in the body from eating non-species-specific food, i.e., animal products, grains, or most cooked food. This mucus putrefies and plaques to the intestinal walls, eventually causing acids to build up in the body and damage our organs and glands.

LMF does away with mucus-causing foods while utilizing fruit, vegetables, herbs, systematic fasting, lymphatic movement, and various trauma-release therapies. Today, Cousins teaches what she’s learned at detox retreats around the globe and inspires thousands through her fierce social media presence.

Alexandra Cousins; founder ‘Living Mucus Free’. Cousins teaches people how to heal their chronic illness through the principles of cellular detoxification.

Sweet potato pizza via Living Mucus Free.

Photo by Livia Macdonald.

Livia says she has dedicated herself to the Living Mucus Free principles with great results, incorporating daily intermittent fasting, herbal tinctures, movement and breathing practices targeted at draining the lymphatic system, as well as raw food diet.

“I have been vegan one year and living mucus free for 10 months now. My anxiety and depression cleared up within two months, never to return. I have so much more clarity and mental focus now and that is getting better with time, not worse. I am slowly healing my endocrine system and gaining more energy back, I am no longer bed ridden since the first couple of months on this lifestyle.. all my spiritual and emotional stuff has surfaced to be healed and it’s truly a fascinating and incredible journey to learn the truth and realize just how wrongly we have been conditioned in such a deep way.”

The emphasis in Living Mucus Free is elimination—getting out of the body’s way and allowing it to do its job of eliminating acids, toxins, undigested food material and mucoid plaque. This is primarily achieved through daily dry fasting and eating watery, astringent fruit, which pulls out toxins as it transits the digestive tract.

Another principle to the Living Mucus Free lifestyle is eating little to no fat while detoxing, a principle that goes against many of the high-fat diet trends of today. But as Alexandra Cousins explains, in the case of those who are cellularly degenerate, fats only serve to cover up their issues. Fats are anti-inflammatory, buffering the acidity in the body but never pulling the acids out. A temporary bandaid for true healing.

Livia feels this is what happened in her case, and it is why she thinks so many initially feel great adopting a high-fat diet.

“I feel the high fat diet works for some because it suppresses and clogs their lymphatic system so naturally they will feel instant relief. But now that I understand how the body actually works, of course you are going to show improvement at the beginning if you remove junk food, sugars/grains, dairy etc.”

Cousins also speaks much to the notion that fats, salts, animal products, and processed foods are stimulating to our nervous system which cover up our emotional wounds, so when we begin to remove these foods and focus on detoxifying the body, we are suddenly faced with old emotions or traumatic memories, and this, Alex says, is mostly what Living Mucus Free is about.

“When we detox on a cellular level, we are consistently clearing old information, old cellular memory in the form of emotion which is held in physical waste stored in the body, replacing it with new cellular information,” Alex Cousins, Living Mucus Free.

For those looking for a quick fix, Living Mucus Free probably isn’t the right fit. Those living the Mucus Free lifestyle don’t make false promises that you will be healed after a 30 day detox. The journey is slow and steady, one with bumps along the way known as healing crises. During a healing crisis any number of uncomfortable symptoms can arise as the body expels old debris and toxins. But as Livia says, walking through the discomfort is the only way towards true healing.

I believe that our society has everything so backwards,” says Livia. “We are taught to chase feeling good, and run away from feeling bad, and Living Mucus Free isn’t going to feel good in the beginning as it brings up our weaknesses for healing.”

The reward, as promised by Cousins, Morse, Ehret, and thousands of others who have healed through regenerative detox principles, is beyond anything we can imagine:

Unimaginable health and vitality, weight loss and reversed ageing, improved energy levels, mental clarity and confidence, liberation from anxiety, mood swings and self-doubt, resolution of stored trauma and a deeper connection to source, vastly improved sex life and orgasms.”

Is Living Mucus Free really the key to such incredible feats? The answer, it seems, is to be discovered only by those willing to walk through the fire to find out.

For more information about Living Mucus Free, visit Alexandra Cousins’ website, Living Mucus Free.

For amazing mucus free recipes and to continue following Livia’s journey, check her out Instagram or Facebook, or her website, LiveAlittleRaw.

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Two Doctors Explain Autophagy, How To Induce It (Fasting) & What It Does To The Human Body (Video)

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In Brief

  • The Facts:

    Dr. Guido Kroemer and Rhonda Patrick sit down and discuss autophagy, how to induce it and it's health benefits.

  • Reflect On:

    Why do we never hear about fasting interventions as an 'official' treatment for certain from our federal health regulatory agencies when there is so much scientific proof?

Fasting and caloric restriction, if done correctly in a healthy and appropriate manner, combined with a healthy diet can have tremendous benefits for the human body. Interventions like fasting are gaining tremendous amounts of popularity, and that is in large part due to the fact that this information is being spread across the world via alternative media outlets and independent websites, youtube channels, etc. It’s not really a health topic that we’re hearing from mainstream media sources or our federal health regulatory agencies. Why? Because you can’t make money off of fasting. Perhaps when drugs are developed that mimic the effects of fasting, that’s when its popularity will skyrocket; but unfortunately, modern day health authorities don’t really seem to be as concerned with our health and wellbeing as they are about profiting and making money, and nobody is going to make any money if people starting eating less. That being said, the information revolution cannot be stopped, and fasting is now on the minds of many, and for good reason.

On October 3rd, 2016, the Nobel Assembly at Karolinska Institutet awarded the Nobel Prize in Physiology or Medicine to Yoshinori Ohsumi for his discoveries of mechanisms for autophagy, a term that translates to “self-eat.” In short, autophagy is the body’s self-cleaning system, a mechanism in which cells get rid of all the broken down, old cell machinery (organelles, proteins and cell membranes). It is a regulated, orderly process to degrade and recycle cellular components.

The process of autophagy is like replacing parts in a car—sometimes we need a new engine or battery for the car to function better. The same thing happens within each of our cells. During autophagy, old cellular debris is sent to specialized compartments within the cell called “lysosomes.” Lysosomes contain enzymes that degrade the old debris, breaking it down into smaller components to be reused again by the cell.

Scientists have found that fasting for 12 to 24+ hours triggers autophagy, which is thought to be one of the reasons that fasting is associated with longevity. There is a large body of research that connects fasting to improved blood sugar control, reduced inflammationweight loss, and improved brain function, and Oshumi’s findings provide greater insight into this research.

“Sporadic short-term fasting, driven by religious and spiritual beliefs, is common to many cultures and has been practiced for millennia, but scientific analyses of the consequences of caloric restriction are more recent… short-term food restriction induces a dramatic upregulation of autophagy in cortical and Purkinje neurons. As noted above, disruption of autophagy can cause neurodegenerative disease, and the converse also may hold true: upregulation of autophagy may have a neuroprotective effect.

Food restriction is a simple, reliable, inexpensive and harmless alternative to drug ingestion and, therefore, we propose that short-term food restriction may represent an attractive alternative to the prophylaxis and treatment of diseases in which candidate drugs are currently being sought.”

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If you look at the plethora of studies that’ve been published regarding caloric restriction and fasting, the benefits are overwhelming. These benefits are seen across the board, not just in humans, but in animals as well. Some of these benefits are talked about below in a fascinating interview and discussion between Dr. Rhonda Patrick  and Dr. Guido Kroemer. Dr. Patrick, as her website states, “is dedicated to the pursuit of longevity and optimal health and shares the latest research on nutrition, aging, and disease prevention with her audience. She has a gift for translating scientific topics into understandable takeaways for all levels of education and interest.” She has a lot of great content on her Youtube channel with some very interesting people who are leaders in their respective field.

Dr. Guido Kroemer is currently a Professor at the Faculty of Medicine of the University of Paris Descartes, Director of the research team “Apoptosis, Cancer and Immunity” of the French Medical Research Council (INSERM), Director of the Metabolomics and Cell Biology platforms of the Gustave Roussy Comprehensive Cancer Center, Deputy Director of the Cordeliers Research Center, and Hospital Practitioner at the Hôpital Européen George Pompidou, Paris, France. He is also a Foreign Adjunct Professor at the Karolinska Institutet, Stockholm, Sweden.

The Takeaway

The takeaway here is to recognize the potential of dietary interventions for certain ailments. It’s also to recognize the importance of seeking out knowledge and wisdom, and not just relying on your doctor for advice or prescription medications.

Related CE Articles on Fasting

How To Activate Autophagy: Your Body’s Self-Cleansing System

Autophagy, Fasting & Exercise: Scientist Reveal Multiple Ways You Can Slow Down The Process of Aging

The Complete Guide To Fasting & Reversing Type 2 Diabetes: A Special Interview With Dr. Jason Fung

Neuroscientist Shows What Fasting Does To Your Brain & Why Big Pharma Won’t Study It

Scientists Explain How Fasting Fights Cancer, Triggers Stem Cell Regeneration & Changes Your Brain (In A Good Way)

Help Support Collective Evolution

The demand for Collective Evolution's content is bigger than ever, except ad agencies and social media keep cutting our revenues. This is making it hard for us to continue.

In order to stay truly independent, we need your help. We are not going to put up paywalls on this website, as we want to get our info out far and wide. For as little as $3 a month, you can help keep CE alive!

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