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Genetically Modifying Humans Via Antibiotics? Something You Need To Know

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lisaA new kind of antibiotic has been developed by researchers at Oregon State University.  The new antibiotics are called PPMOs, which stand for peptide-conjugated phosphorodiamidate morpholino oligomers.  They are “a synthetic analog of DNA or RNA that has the ability to silence the expression of specific genes.” (1) The way that PPMO antibiotics will work is to, “specifically target the underlying genes of a bacterium.”  In plain English, PPMOs will genetically modify bacteria.

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This may not sound like a horrible thing on initial glance.  Bacteria are generally thought of as evil (soap commercials have conditioned us all), something to fight because some bacteria can make people sick and even kill them if their body is overwhelmed by “bad” bacteria.  However, bacteria and the other single-celled organisms that make up the human microbiome are intimate parts of each human being.  Per the Human Microbiome Project:

The healthy adult body hosts ten times as many microbial cells as human cells, including bacteria, archaea, viruses, and eukaryotic microbes resident on nearly every body surface. The metagenome carried collectively by these microbial communities dwarfs the human genome in size, and their influences on normal development, diet and obesity, immunity, and disease are under active research. (2)

The average 200 pound human body contains 6 pounds of microbiome organisms, including several billion bacteria (3).  These bacteria act symbiotically with us, helping to digest food, extract vitamins and other nutrients from food, regulate the immune system and even  contribute to each individual’s personality.  Per an article published in Molecular Psychology, “CNS neurotransmission can be profoundly disturbed by the absence of a normal gut microbiota.” (4)  Multiple neurochemicals are produced by gut bacteria, including 95% of the serotonin in each human body (5).  Studies of mice have shown that behavioral changes can be triggered by changes in the gut bacteria and it has been observed that people with Crohn’s Disease and other GI disorders often suffer from anxiety and depression.  The health of each person’s microbiome is intimately connected to both their physical and the mental health.

The bacteria that compose our microbiome work so synergistically with our human cells that the difference between “us” and “the bacteria” is difficult to decipher.  Where do “we” begin and “they” end?  If all of the bacteria in a person’s microbiome were killed off, that person would die.  Bacteria are an intimate and important part of “us.”  In genetically modifying “them,” are we genetically modifying “us?”  How could genetically modified bacteria affect the balance of the human microbiome?  How could they affect the bodily systems that the microbiome controls?  How could a GM bacteria adversely affect human health including personality and behavior?

One of many other things to consider is that mitochondria, the energy centers of our cells, are very similar in structure and design to bacteria. (6)  Mitochondrial DNA is also much more vulnerable to environmental toxins than the rest of the human DNA. (7)

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Could PPMOs (or other drugs that genetically modify bacteria) modify human mitochondria?  If so, what are the consequences of having genetically modified mitochondria?  One consequence is that humans truly would be genetically modified.  Perhaps that should be taken into consideration before developing drugs that genetically modify bacteria.

There are thousands of medical and ethical questions that should be asked about the development of drugs that genetically modify bacteria.  Sadly, I suspect that many people will look the other way, assuming that PPMOs are just another antibiotic that are as innocuous as penicillin, rather than asking the really difficult questions that should be asked before our mitochondrial DNA is permanently and irreversibly altered.  I suspect that the questions about whether or not antibiotics that alter the human microbiome should be created or not will not be asked though, because human mitochondrial DNA has been being altered and damaged by a certain class of antibiotics, fluoroquinolones, for years without anyone saying a peep.

Genetic Modification via Antibiotics is Already Occurring

Fluoroquinolone antibiotics, more popularly known as Cipro (Ciprofloxacin), Levaquin (Levofloxacin), Avelox (Moxifloxacin), Floxin (Ofloxacin) and a few other less commonly used ones, are topoisomerase interrupters.  They unravel bacterial DNA and lead to apoptosis, programmed cell death.  This video explains how they work:

The chemical backbone of fluroquinolone antibiotics, nalidixic acid, was developed in 1962 by George Lesher. (8)  They became popular starting in the 1980s when pharmaceutical companies pressured the FDA to accept them as a “first line of defense” antibiotic despite the fact that they had  shown to be toxic to mammalian cells.  They increased in popularity after the 2001 anthrax scare.  They are used to treat urinary tract infections, sinus infections, bronchial infections, strep throat, etc. despite the fact that the side effects include psychosis (9) and destruction of every tendon in the body. A side-effect that is lightly referred to as “tendinitis” on the warning label.  (A more complete list of effects of fluoroquinolones can be found on www.ciproispoison.com.  The person who wrote that list of things that happened to him as a result of taking Cipro was a happy, healthy, employed 31 year old when he took Cipro.  He is now disabled.)

Multiple studies have shown that quinolones/fluoroquinolones adduct to bacterial DNA. (10)(11) This means that they attach to and change DNA, that the DNA has altered molecules hooked onto it and that all duplicate versions of the cells have been altered.  An example of another chemical that adducts to DNA is Agent Orange.

Some DNA tests performed on people who have experienced severe adverse reactions to fluoroquinolone antibiotics have shown that the quinolone/fluoroquinolone molecules have adducted to their human DNA, attaching to and changing their DNA into perpetuity.  (As cells replicate, the altered DNA replicates too.)  A DNA Adduct Mass Spectrogram Analysis showed that the quinolone/fluoroquinolone molecules had attached to every cell in the subjects’ bodies, not just the bacteria that make up their microbiome; the drug adducted to their DNA, to THEM.

They, along with thousands of other people who have had an adverse reaction to a fluoroquinolone, have been genetically modified by an antibiotic.

A large portion of those who have been genetically modified by a fluoroquinolone antibiotic have been subjected to irreversible damage to their DNA for no sensible reason at all.  Fluoroquinolone antibiotics are given out to treat benign infections like sinus and urinary tract infections, that can be treated with other, safer antibiotics.  A 2011 study (12) found that 39% of patients given fluoroquinolone antibiotics were given them unnecessarily (and the necessity of them was determined without it being taken into consideration that DNA damage can be done by these drugs as this fact is not acknowledged, despite the peer reviewed studies noted above.)

26.9 million prescriptions for fluoroquinolone antibiotics were dispensed in America in 2011 alone (13).  Similarly massive numbers of prescriptions of these drugs have been dispensed each year since Bayer patented Cipro in 1983.  Humanity has not stopped existing since these DNA modifying drugs were introduced to the market, but before you find that to be reassuring, the following should be noted.

  1. An article in the September, 2013 issue of Nature entitled “Topoisomerases facilitate transcription of long genes linked to autism” (14) noted that, “Our data suggest that chemicals or genetic mutations that impair topoisomerases, and possibly other components of the transcription elongation machinery that interface with topoisomerases, have the potential to profoundly affect the expression of long ASD candidate genes.”  Fluoroquinolone antibiotics impair topoisomerases.  A post about this is on Collective Evolution – http://www.collective-evolution.com/2013/09/18/a-horrifying-cause-of-autism-dna-damage-from-synthetic-antibiotics
  2. Anthraquinone was found in the subject who underwent The DNA testing.  Anthraquinone causes an inflammatory process within the body and causes pain, burning, and hurting sensations, a condition that is often confused with fibromyalgia. (15)
  3. Fluoroquinolone antibiotics have been shown to damage mitochondria (16)(17)(18) and “Damage to mitochondria is now understood to play a role in the pathogenesis of a wide range of seemingly unrelated disorders such as schizophrenia, bipolar disease, dementia, Alzheimer’s disease, epilepsy, migraine headaches, strokes, neuropathic pain, Parkinson’s disease, ataxia, transient ischemic attack, cardiomyopathy, coronary artery disease, chronic fatigue syndrome, fibromyalgia, retinitis pigmentosa, diabetes, hepatitis C, and primary biliary cirrhosis.” (19)

So, if you’re wondering what happens when humans are genetically modified, the experiment is being conducted as you read this post.  Since fluoroquinolone antibiotics have been popularized, rates of autism, schizophrenia, bipolar disease, dementia, Alzheimer’s disease, epilepsy, migraine headaches, strokes, neuropathic pain, Parkinson’s disease, ataxia, transient ischemic attack, cardiomyopathy, coronary artery disease, chronic fatigue syndrome, fibromyalgia, retinitis pigmentosa, diabetes, hepatitis C, and primary biliary cirrhosis have risen substantially.

Perhaps the question of the intelligence of altering human DNA with antibiotics can be questioned before PPMOs are introduced to the market, as opposed to 30+ years afterward, as is the case with fluoroquinolone antibiotics.  It would show wisdom and desire for sustainability as a species.  Unfortunately, neither wisdom nor sustainability are valued at the moment and I suspect that the travesty of people being genetically altered by fluoroquinolones will continue and that the travesty of people being altered by PPMOs will begin.

Post Script:

  1. If enough people gathered together, got their DNA tested, got those test results interpreted by a Toxicologist, and appropriate research was published on the results, this atrocity could stop.  Please note that both Bayer (producer of Cipro and Avelox) and Johnson and Johnson (producer of Levaquin), and even the generic producers of these drugs, have very deep pockets.
  2. The author’s blog is www.floxiehope.com.

Sources:

  1. Drug Discovery and Development, “Beyond Antibiotics: New Approach to Bacterial Infections” published online on 10/16/13 – http://www.dddmag.com/news/2013/10/beyond-antibiotics-new-approach-bacterial-infections?et_cid=3541647&et_rid=45519727&location=top
  2. PLOS Collections, “Table of Contents: The Human Microbiome Project Collection”  http://www.ploscollections.org/article/browseIssue.action?issue=info:doi/10.1371/issue.pcol.v01.i13
  3. Neergaard, Lauran, “Human Microbiome Project: 10,000 Species Of Microbes In And On Our Bodies,” Huffpost Healthy Living, 06/13/2012  http://www.huffingtonpost.com/2012/06/13/human-microbiome-project-100-trillion-bacteria_n_1594430.html
  4. Mol Psychiatry. 2013 Jun;18(6):666-73. doi: 10.1038/mp.2012.77. Epub 2012 Jun 12. The microbiome-gut-brain axis during early life regulates the hippocampal serotonergic system in a sex-dependent manner. Clarke G, Grenham S, Scully P, Fitzgerald P, Moloney RD, Shanahan F, Dinan TG, Cryan JF. http://www.ncbi.nlm.nih.gov/pubmed/22688187
  5. Carpenter, Siri.  “That Gut Feeling: With a sophisticated neural network transmitting messages from trillions of bacteria, the brain in your gut exerts a powerful influence over the one in your head, new research suggests.”  Monitor on Psychology.  American Psychological Association.  September 2012, Vol 43, No. 8 Print version: page 50   http://www.apa.org/monitor/2012/09/gut-feeling.aspx
  6. http://en.wikipedia.org/wiki/Mitochondria
  7. John Neustadt and Steve R. Pieczenik.  “Medication-induced mitochondrial damage and disease.”  Mol. Nutr. Food Res. 2008,52, 780 – 788 http://psychrights.org/Research/Digest/NLPs/DrugsCauseMitochondrialDamage.pdf
  8. http://en.wikipedia.org/wiki/Fluoroquinolone_antibiotic
  9. Nagaraja Moorthy, N. Raghavendra, and P. N. Venkatarathnamma.  “Levofloxacin-induced acute psychosis.”  Indian J Psychiatry. 2008 Jan-Mar; 50(1): 57–58. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2745871/
  10.  Arkady B. Khodursky and Nicholas R. Cozzarelli.  “The Mechanism of Inhibition of Topoisomerase IV by Quinolone Antibacterials”  The Journal of Biological Chemistry.  August 5, 1998.   http://www.jbc.org/content/273/42/27668.full
  11. G. PALLJ*, S. VALISENA*, G. CIARROCCHI, B. GATTO, AND M. PALUMBO.  “Quinolone binding to DNA is mediated by magnesium ions.”  Proc. Natl. Acad. Sci. USA Vol. 89, pp. 9671-9675, October 1992 Biochemistry.  http://www.ncbi.nlm.nih.gov/pmc/articles/PMC50194/pdf/pnas01094-0315.pdf
  12. Nicole L Werner, Michelle T Hecker, Ajay K Sethi and Curtis J Donskey.  “Unnecessary use of fluoroquinolone antibiotics in hospitalized patients.” BMC Infectious Diseases.  Volume 11.  http://www.biomedcentral.com/1471-2334/11/187
  13.  “FDA Drug Safety Communication: FDA requires label changes to warn of risk for possibly permanent nerve damage from antibacterial fluoroquinolone drugs taken by mouth or by injection” 08/15/2013 http://www.fda.gov/downloads/Drugs/DrugSafety/UCM365078.pdf
  14. Ian F. King, Chandri N. Yandava, Angela M. Mabb,  Jack S. Hsiao, Hsien-Sung Huang, Brandon L. Pearson, J. Mauro Calabrese, Joshua Starmer, Joel S. Parker, Terry Magnuson,           Stormy J. Chamberlain, Benjamin D. Philpot & Mark J. Zylka.  “Topoisomerases facilitate transcription of long genes linked to autism.”  Nature 501, 58–62 (05 September 2013) doi:10.1038/nature12504 Received 17 January 2013 Accepted 24 July 2013 Published online 28 August 2013 http://www.nature.com/nature/journal/v501/n7465/full/nature12504.html
  15. http://en.wikipedia.org/wiki/Anthraquinone
  16. “Dodging Antibiotic Side Effects.”  July 3, 2013. http://wyss.harvard.edu/viewpressrelease/117/
  17. “Pinpointing How Antibiotics Work” April 19, 2012.  MIT Media Relations.  http://web.mit.edu/press/2012/pinpointing-how-antibiotics-work.html
  18. J W Lawrence, D C Claire, V Weissig and T C Rowe.  “Delayed cytotoxicity and cleavage of mitochondrial DNA in ciprofloxacin-treated mammalian cells.”  Molecular Pharmacology November 1996 vol. 50 no. 5 1178-1188 http://m.molpharm.aspetjournals.org/content/50/5/1178.abstract
  19. John Neustadt and Steve R. Pieczenik.  “Medication-induced mitochondrial damage and disease.”  Mol. Nutr. Food Res. 2008,52, 780 – 788 http://psychrights.org/Research/Digest/NLPs/DrugsCauseMitochondrialDamage.pdf

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How X-Ray Mammography Is Accelerating The Epidemic of Cancer

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Article written by Sayer Ji, Founder of Greenmedinfo LLC, posted here with permission.

While a growing body of research now suggests that x-ray mammography is causing more harm than good in the millions of women who subject themselves to breast screenings, annually, without knowledge of their true health risks, the primary focus has been on the harms associated with over-diagnosis and over-treatment, and not the radiobiological dangers of the procedure itself.

In 2006, a paper published in the British Journal of Radiobiology, titled “Enhanced biological effectiveness of low energy X-rays and implications for the UK breast screening programme,” revealed the type of radiation used in x-ray-based breast screenings is much more carcinogenic than previously believed:

Recent radiobiological studies have provided compelling evidence that the low energy X-rays as used in mammography are approximately four times – butpossibly as much as six times – more effective in causing mutational damage than higher energy X-rays. Since current radiation risk estimates are based on the effects of high energy gamma radiation, this implies that the risks of radiation-induced breast cancers for mammography X-rays are underestimated by the same factor.[1]

In other words, the radiation risk model used to determine whether the benefit of breast screenings in asymptomatic women outweighs their harm, underestimates the risk of mammography-induced breast and related cancers by between 4-600%.

The authors continued

Risk estimates for radiation-induced cancer – principally derived from the atomic bomb survivor study (ABSS) – are based on the effects of high energy gamma-rays and thus the implication is that the risks of radiation-induced breast cancer arising from mammography may be higher than that assumed based on standard risks estimates.

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This is not the only study to demonstrate mammography X-rays are more carcinogenic than atomic bomb spectrum radiation. There is also an extensive amount of data on the downside of x-ray mammography.

Sadly, even if one uses the outdated radiation risk model (which underestimates the harm done),* the weight of the scientific evidence (as determined by the work of The Cochrane Collaboration) actually shows that breast screenings are in all likelihood not doing any net good in those who undergo them.

In a 2009 Cochrane Database Systematic Review,** also known as the Gøtzsche and Nielsen’s Cochrane Review, titled “Screening for breast cancer with mammography,” the authors revealed the tenuous statistical justifications for mass breast screenings:

Screening led to 30% overdiagnosis and overtreatment, or an absolute risk increase of 0.5%. This means that for every 2000 women invited for screening throughout 10 years, one will have her life prolonged and 10 healthy women, who would not have been diagnosed if there had not been screening, will be treated unnecessarily. Furthermore, more than 200 women will experience important psychological distress for many months because of false positive findings. It is thus not clear whether screening does more good than harm.[2]

In this review, the basis for estimating unnecessary treatment was the 35% increased risk of surgery among women who underwent screenings. Many of the surgeries, in fact, were the result of women being diagnosed with ductal carcinoma in situ (DCIS), a “cancer” that would not exists as a clinically relevant entity were it not for the fact that it is detectable through x-ray mammography. DCIS, in the vast majority of cases, has no palpable lesion or symptoms, and some experts believe it should be completely reclassified as a non-cancerous condition.

A more recent study published in the British Medical Journal in 2011 titled, “Possible net harms of breast cancer screening: updated modeling of Forrest report,” not only confirmed the Gøtzsche and Nielsen’s Cochrane Review findings, but found the situation likely worse:

This analysis supports the claim that the introduction of breast cancer screening might have caused net harm for up to 10 years after the start of screening.[3]

So, let’s assume that these reviews are correct, and at the very least, the screenings are not doing any good, and at worst, causing more harm than good. The salient question, however, is how much more harm than good? If we consider that, according to data from Journal of the National Cancer Institute (2011), a mammogram uses 4 mSv of radiation vs. the .02 mSv of your average chest x-ray (which is 200 times more radiation), and then, we factor in the 4-600% higher genotoxicity/carcinogenicity associated with the specific “low-energy” wavelengths used in mammography, it is highly possible that beyond the epidemic of over-diagnosis and over-treatment, mammograms are planting seeds of radiation-induced cancer within the breasts of millions of women.***

With the advent of non-ionizing radiation based diagnostic technologies, such as thermography, it has become vitally important that patients educate themselves about the alternatives to x-ray mammography that already exist.  Until then, we must use our good sense – and research like this – to inform our decisions, and as far as the unintended adverse effects of radiation go, erring on the side of caution whenever possible.

Additional Reading

Is X-ray Mammography Findings Cancer or Benign Lesions?

The Dark Side of Breast Cancer Awareness Month

Does Chemo & Radiation Actually Make Cancer More Malignant?


*This discrepancy in radiation risk models/estimates follows from two fundamental problems: 1) the older risk model was based on higher-energy radiation emissions, such as are given off from atomic bomb blasts 2) it was a crude model, developed before the discovery of DNA and a full understanding of radiotoxicity/genotoxicity.

** Keep in mind that the Cochrane Database Review is at the top of the “food chain” of truth, in the highly touted “evidence-based model” of conventional medicine. Cochrane Database Reviews are produced by The Cochrane Collaboration, which is internationally recognized as the benchmark for high quality, evidence-based information concerning the effectiveness (or lack thereof) of common health care interventions. The organization, comprised of over 28,000 dedicated people from over 100 countries, prides itself on being an “independent” source of information, and historically has not been afraid to point out the corrupting influence of industry, which increasingly co-opts  the biomedical research and publishing fields.

***The low-energy wavelengths cause double strand breaks within the DNA of susceptible cells, which the cell can not repair. Through time these mutations result in “neoplastic transformation”; radiation has the ability to induce a cancerous phenotype within formerly healthy cells that has cancer stem cell-like (CSC) properties.


[1] Enhanced biological effectiveness of low energy X-rays and implications for the UK breast screening programme. Br J Radiol. 2006 Mar ;79(939):195-200. PMID: 16498030

[2] Screening for breast cancer with mammography. Cochrane Database Syst Rev. 2009(4):CD001877. Epub 2009 Oct 7. PMID: 19821284

[3] Possible net harms of breast cancer screening: updated modelling of Forrest report. BMJ. 2011 ;343:d7627. Epub 2011 Dec 8. PMID: 22155336


Sayer Ji is founder of Greenmedinfo.com, a reviewer at the International Journal of Human Nutrition and Functional Medicine, Co-founder and CEO of Systome Biomed, Vice Chairman of the Board of the National Health Federation, Steering Committee Member of the Global Non-GMO Foundation.

If you want to learn more from Greenmedinfo, sign up for their newsletter here

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Why Water Fluoridation Is A Forced Experiment That Needs To Stop

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The United States stands almost entirely alone among developed nations in adding industrial silicofluorides to its drinking water—imposing the community-wide measure without informed consent. Globally, roughly 5% of the population consumes chemically fluoridated water, but more people in the U.S. drink fluoride-adulterated water than in all other countries combined. Within the U.S., just under a third (30%) of local water supplies are not fluoridated; these municipalities have either held the practice at bay since fluoridation’s inception or have won hard-fought battles to halt water fluoridation.

Dozens of studies and reviews—including in top-tier journals such as The Lancet—have shown that fluoride is neurotoxic and lowers children’s IQ.

The fluoride chemicals added to drinking water are unprocessed toxic wasteproducts—captured pollutants from Florida’s phosphate fertilizer industry or unregulated chemical imports from China. The chemicals undergo no purification before being dumped into drinking water and often harbor significant levels of arsenic and other heavy metal contamination; one researcher describes this unavoidable contamination as a “regulatory blind spotthat jeopardizes any safe use of fluoride additives.”

Dozens of studies and reviews—including in top-tier journals such as The Lancet—have shown that fluoride is neurotoxic and lowers children’s IQ. Fluoride is also associated with a variety of other health risks in both children and adults. However, U.S. officialdom persists in making hollow claims that water fluoridation is safe and beneficial, choosing to ignore even its own research! A multimillion-dollar longitudinal study published in Environmental Health Perspectives in September, 2017, for example, was largely funded by the National Institutes of Health and National Institute of Environmental Health Sciences—and the seminal study revealed a strong relationship between fluoride exposure in pregnant women and lowered cognitive function in offspring. Considered in the context of other research, the study’s implications are, according to the nonprofit Fluoride Action Network, “enormous”—“a cannon shot across the bow of the 80 year old practice of artificial fluoridation.”

According to declassified government documents summarized by Project Censored, Manhattan Project scientists discovered early on that fluoride was a leading health hazard to bomb program workers and surrounding communities.

A little history

During World War II, fluoride (a compound formed from the chemical element fluorine) came into large-scale production and use as part of the Manhattan Project. According to declassified government documents summarized by Project Censored, Manhattan Project scientists discovered early on that fluoride was a “leading health hazard to bomb program workers and surrounding communities.” In order to stave off lawsuits, government scientists “embarked on a campaign to calm the social panic about fluoride…by promoting its usefulness in preventing tooth decay.”

To prop up its “exaggerated claims of reduction in tooth decay,” government researchers began carrying out a series of poorly designed and fatally flawed community trials of water fluoridation in a handful of U.S. cities in the mid-1940s. In a critique decades later, a University of California-Davis statistician characterized these early agenda-driven fluoridation trials as “especially rich in fallacies, improper design, invalid use of statistical methods, omissions of contrary data, and just plain muddleheadedness and hebetude.” As one example, a 15-year trial launched in Grand Rapids, Michigan in 1945 used a nearby city as a non-fluoridated control, but after the control city began fluoridating its own water supply five years into the study, the design switched from a comparison with the non-fluoridated community to a before-and-after assessment of Grand Rapids. Fluoridation’s proponents admitted that this change substantially “compromised” the quality of the study.

In 1950, well before any of the community trials could reach any conclusions about the systemic health effects of long-term fluoride ingestion, the U.S. Public Health Service (USPHS) endorsed water fluoridation as official public health policy, strongly encouraging communities across the country to adopt the unproven measure for dental caries prevention. Describing this astonishingly non-evidence-based step as “the Great Fluoridation Gamble,” the authors of the 2010 book, The Case Against Fluorideargue that:

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“Not only was safety not demonstrated in anything approaching a comprehensive and scientific study, but also a large number of studies implicating fluoride’s impact on both the bones and the thyroid gland were ignored or downplayed” (p. 86).

In 2015, Newsweek magazine not only agreed that the scientific rationale for putting fluoride in drinking water was not as “clear-cut” as once thought but also shared the “shocking” finding of a more recent Cochrane Collaboration review, namely, that there is no evidence to support the use of fluoride in drinking water.

Bad science and powerful politics

The authors of The Case Against Fluoride persuasively argue that “bad science” and “powerful politics” are primary factors explaining why government agencies continue to defend the indefensible practice of water fluoridation, despite abundant evidence that it is unsafe both developmentally and after “a lifetime of exposure to uncontrolled doses.” Comparable to Robert F. Kennedy, Jr.’s bookThimerosal: Let the Science Speak, which summarizes studies that the Centers for Disease Control and Prevention (CDC) and “credulous journalists swear don’t exist,” The Case Against Fluoride is an extensively referenced tour de force, pulling together hundreds of studies showing evidence of fluoride-related harm.

… death rates in the ten most fluoridated U.S. states are 5% to 26% higher than in the ten least fluoridated states, with triple the rate of Alzheimer’s disease.

The research assembled by the book’s authors includes studies on fluoride biochemistry; cancer; fluoride’s effects on the brain, endocrine system and bones; and dental fluorosis. With regard to the latter, public health agencies like to define dental fluorosis as a purely cosmetic issue involving “changes in the appearance of tooth enamel,” but the International Academy of Oral Medicine & Toxicology (IAOMT)—a global network of dentists, health professionals and scientists dedicated to science-based biological dentistry—describes the damaged enamel and mottled and brittle teeth that characterize dental fluorosis as “the first visible sign of fluoride toxicity.”

The important 2017 study that showed decrements in IQ following fluoride exposure during pregnancy is far from the only research sounding the alarm about fluoride’s adverse developmental effects. In his 2017 volumePregnancy and Fluoride Do Not Mix, John D. MacArthur pulls together hundreds of studies linking fluoride to premature birth and impaired neurological development (93 studies), preelampsia (77 studies) and autism (110 studies). The book points out that rates of premature birth are “unusually high” in the United States. At the other end of the lifespan, MacArthur observes that death rates in the ten most fluoridated U.S. states are 5% to 26% higher than in the ten least fluoridated states, with triple the rate of Alzheimer’s disease. A 2006 report by the National Research Council warned that exposure to fluoride might increase the risk of developing Alzheimer’s.

The word is out

Pregnancy and Fluoride Do Not Mix shows that the Institute of Medicine, National Research Council, Harvard’s National Scientific Council on the Developing Child, Environmental Protection Agency (EPA) and National Toxicology Program all are well aware of the substantial evidence of fluoride’s developmental neurotoxicity, yet no action has been taken to warn pregnant women. Instead, scientists with integrity, legal professionals and the public increasingly are taking matters into their own hands. A Citizens Petitionsubmitted in 2016 to the EPA under the Toxic Substances Control Act requested that the EPA “exercise its authority to prohibit the purposeful addition of fluoridation chemicals to U.S. water supplies.” This request—the focus of a lawsuit to be argued in court later in 2019—poses a landmark challenge to the dangerous practice of water fluoridation and has the potential to end one of the most significant chemical assaults on our children’s developing bodies and brains.

Sign up for free news and updates from Robert F. Kennedy, Jr. and the Children’s Health Defense. CHD is planning many strategies, including legal, in an effort to defend the health of our children and obtain justice for those already injured. Your support is essential to CHD’s successful mission.

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Doctor Explains Why She Never Recommends The ‘Ketogenic Diet’

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In Brief

  • The Facts:

    Michelle McMacken, an internal medicine physician, shares why she does not recommend the ketogenic diet at all for her patients.

  • Reflect On:

    Why are we so quick to jump on bandwagons, especially when it comes to health topics, without ever really looking into it deeper? The ketogenic diet has many health benefits, but it may not be as healthy as many think.

The ketogenic diet has gained a tremendous amount of popularity over the past few years, and it’s become a trend that many people are adopting without doing their own research first. We’ve written multiple articles on the ketogenic diet, a diet that promotes a high fat/low carb intake in order to prolong the production of ketone bodies in one’s blood. The release of these ketone bodies happens when we fast and deplete our glucose reserves, which develop from eating carbohydrates that turn into sugar. One can prolong the production of these ketones by sticking to a low carbohydrate/high fat diet, and essentially run off of fat instead of their glucose reserves.

The ketogenic diet is being used as an intervention for cancer, and there are multiple studies showing how ketones can actually kill cancer. It’s becoming well known that cancer cells cannot efficiently process ketone bodies for energy. Essentially, the cell starves itself, and ketones help slow the proliferation of tumor cells. Dietary ketones have been shown to completely halt metastasis. For example, a study titled “The Ketogenic Diet & Hyperbaric Oxygen Therapy Prolong Survival in Mice with Systemic Metastatic Cancer” explains how it’s already known that the ketogenic diet elevates blood ketones and has been shown to slow cancer progression in both animals and humans. The study also revealed that the ketogenic diet “significantly decreased blood glucose, slowed tumor growth, and increased mean survival time by 56.8 percent in mice with systemic metastatic cancer.”

Fasting (when you fast you produce ketones) is also being used for cancer intervention, seizure prevention (epilepsy), and as a potential therapy for alzheimer’s disease, parkinson’s disease, and other neurodegenerative disorders.

A TEDx talk given by Mark Mattson, the current Chief of the Laboratory of Neuroscience, at the National Institute on Aging goes into detail about fasting, ketones, and how beneficial it is for the brain. He is also a professor of Neuroscience at Johns Hopkins University and one of the foremost researchers of the cellular and molecular mechanisms underlying multiple neurodegenerative disorders.

In 1923, scientist Otto Warburg hypothesized that cancer was caused by a metabolic process whereby cancer cells fuel their growth “by swallowing up enormous amounts of glucose [blood sugar] and breaking it down without oxygen.” Coined the Warburg Effect, the theory was considered controversial at the time, but the past few decades have sparked new interest in it and oncologists now use the dependence on glucose that cancer cells have to locate tumours within a patient’s body.

Warburg made his discovery around the same time the ketogenic diet was found to be beneficial for epilepsy. Studies have shown that when the body produces ketones, they form a protective barrier around the brain, which is why more and more paediatricians are recommending the diet for children with epilepsy. It has a huge success rate, but since fasting is neither marketable nor profitable, it receives little mainstream attention.

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All of these are specific interventions for certain diseases, and they can be healthy. On a personal level, I believe fasting a few times a month is extremely healthy and can be very beneficial for the body. All of the studies in human and animal models show nothing but benefits.  Keep in mind that while you fast, you also get the benefits of ketones.

This is far different from continuing on with a no carb, high fat diet where you are constantly producing ketones and burning fat. It doesn’t seem normal unless you have to do it for a specific intervention, like cancer. Despite the potential health benefits, the ‘ketogenic diet’ has become a fad with potential dangers that people should also be aware of.

The Five Reasons

Below is a list of points regarding the ketogenic diet from Michelle McMacken, an internal medicine physician, Assistant Professor of Medicine at the NYU School of Medicine, and Director of Bellevue Hospital Weight Management Clinic.

I came across these via her Instagram, which make it clear she does not support the diet:

1. That we know of, no population in history has ever thrived on a very-low-carb/high-fat diet. There is exactly zero scientific evidence that a keto diet is conducive to longevity & longstanding vitality – unlike a plant-centric diet, the foundation of the longest-lived people on earth.

2. A keto diet may cause short-term weight loss, but possibly at a serious price. A 2010 review found that low-carb, animal-based diets increased cardiovascular death by 14%, cancer death by 28%, & all-cause mortality by 23%- trends confirmed in other large studies.

3. A keto diet hasn’t been shown to prevent, control, or reverse type 2 diabetes in the long run. Avoiding carbs will temporarily lower your blood sugar if you have diabetes. But this simply masks the underlying problem, which is insulin resistance – ie. glucose in our blood can’t enter our cells & the liver overproduces sugar. This is NOT the fault of carbs from healthy foods – whole grains, legumes, fruit, or even starchy vegetables. In fact, a high-carb, high-fiber, plant-based diet is exceptionally protective against diabetes & can actually REVERSE insulin resistance & lower diabetes complications. In contrast, low-carb diets can promote diabetes over time, as they foster inflammation & fat buildup in our cells, causing insulin resistance.

4. Keto diet research is in its infancy, focusing on short-term blood results & body weight – not actual rates of disease or death. And some findings are concerning. LDL cholesterol levels tend to rise (or at best, stay the same) on keto diets. An overwhelming wealth of research shows that the higher the LDL, the higher the risk of cardiovascular disease.

5. A keto diet is low in refined grains & added sugar, which is great. But it also can be low in phytonutrients, antioxidants, & fiber, all of which have profound benefits, and it forbids some of the most powerfully health-promoting foods on earth – whole grains, legumes, & many fruits. To me, that’s just not good medicine.

Her references:


The Takeaway

It’s great to see the world becoming more health conscious, it’s one of multiple contributing factors in raising our vibrational frequency, and feeling more ‘alive.’ That being said, a lot of ‘fads’ seem to pop up in this field, which are coupled with a great misunderstanding of how these specific diets, like the ketogenic diet, is supposed to be used. At the end of the day, balance is key, and it’s best to incorporate more organic fruits and vegetables in your diet, and completely cut out all processed foods, and substances like high fructose corn syrup etc. Being healthy is not hard, and it’s not complicated. If you’re going to incorporate a specific diet into your lifestyle, just make sure it’s not one that’s specifically designed to combat certain diseases, like the ketogenic diet.

Related CE Articles:

Study Shows What A Ketogenic Diet Did To Mice With Systemic Metastatic Cancer

Doctor Explains What Happens To The Body When It Goes Into Ketosis

The Biggest Misconception About The Ketogenic Diet…You Don’t Actually Have To Follow It

Ending The Debate About The Ketogenic Diet – 9 Studies You Must Be Aware Of 


 

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