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How Pharmaceuticals Came To Be The 4th Leading Cause Of Death In America

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lisaPrescription drugs are the 4th leading cause of death in America. (1) People know this to be true, they know it to be appalling, but it’s still seen as incomprehensible and absurd. How could medicine hurt so many people? We all know that side-effects happen, but they are thought to be rare. They must be rare, right? We all know some good, kind, generous, thoughtful doctors who want nothing more for their patients than health and happiness, so they certainly aren’t giving their patients drugs that hurt them, are they? We know that the FDA is a federal bureaucracy, so it must be too restrictive of the pharmaceutical industry, right? And the FDA is supposed to protect consumers, so we’re as safe as we can be, right? And people can sue, so the legal system must be keeping the bad aspects of the medical system in check, right? All of these questions, and many more, bring up some cognitive dissonance for people when they’re faced with the fact that prescription drugs, used as prescribed, kill an inordinate a number of people. It brings up the questions –

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How do prescription drugs get to be the 4th leading cause of death in America? How does that happen?

Here is a tale of how prescription drugs, used as prescribed, kill people.

Kerstin (age 30) comes down with a urinary tract infection. It’s a Saturday so her regular doctor’s office is closed. Urinary tract infections are painful so she knows that she can’t wait ‘til Monday to get treatment. She goes to an Emergency Services Clinic close to her house. She tells them that she has a urinary tract infection and they write her a prescription for Cipro (Ciprofloxacin – a fluoroquinolone antibiotic). They do not culture her urine because they don’t have the time or capacity to do so. It doesn’t matter what kind of bacteria is in her urine though, they know that Cipro will kill it because Cipro is a broad-spectrum antibiotic and it will kill all the offending bacteria in her urinary tract, plus some.

Kerstin is relieved. Her painful urinary tract and bladder are about to be healed.

Kerstin takes two 500 mg. pills of Cipro two times a day for a week. On the 5th day of taking Cipro, Kerstin starts to feel a bit off. Her bladder feels full even when it isn’t, she has dark “floaters” interfering with her vision and she feels anxious. She doesn’t think anything of these things. They’re strange, but not too worrisome. She doesn’t think for a second that they could be due to the antibiotic that she is taking. Kerstin finishes the seven day course of Cipro. Her urinary tract infection is gone and she is pleased about that. Her bladder fullness, floaters and anxiety come and go and she doesn’t think much of them.

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Ten days pass in which Kerstin feels fine. On the eleventh day after she has finished taking Cipro, she starts taking ibuprofen to treat menstrual cramps. On the thirteenth day after she has finished taking the Cipro, it feels as if a bomb goes off in her body. Her hands and feet swell to twice their normal size. It becomes painful for her to walk or to do anything with her hands. Her knees are burning as if every tendon in them is inflamed. She is weak. She develops hives all over her body. Her anxiety levels are sky-high.

Kerstin goes to the doctor. The doctor says that the hives are a result of an allergic reaction and tells her to take Benadryl. Kerstin asks the doctor why she can barely walk when she was going to the gym daily just a few days earlier. The doctor says that she doesn’t know, but that she will run tests.

Kerstin takes Benadryl but it doesn’t seem to help. She goes back to the doctor for something stronger. She is put on prednisone.

The swelling in her hands and feet goes down, but her other symptoms worsen. She develops insomnia. She sprains her wrist while opening a jar. Intermittent pain throughout her body, but especially in her legs, begins. She loses her memory and has trouble concentrating.

Her test results come back. They are all “normal.”

Her pain worsens. She is diagnosed with Fibromyalgia. She asks the doctor who diagnoses her with Fibromyalgia how she could have gone from being healthy and active to being disabled and in pain, now with a diagnosis of Fibromyalgia. The doctor mutters something about mysterious diseases and unexplained symptoms. Kerstin asks if her symptoms could be related to any of the medications that she took – Cipro, ibuprofen and prednisone. The doctor says no. More tests are run to see if there are other causes of Kerstin’s symptoms.

Kerstin is put on Lyrica to help her with her Fibromyalgia pain.

The Lyrica seems to help some of her pain but her mental symptoms get worse while she is on it. In addition to her already existing memory and concentration problems, Kerstin develops brain-fog. She feels slow, stupid and like she is living in a dream. She gains 15 pounds in two months. Her hair starts to fall out. She feels suicidal. She is taken off of Lyrica by her doctor.

Kerstin continues to have problems in her joints, especially her wrists, knees and ankles, so she is not surprised when she is diagnosed with Rheumatoid Arthritis. She starts seeing a Rheumatologist who puts her on Humira. Humira decreases some of her inflammation symptoms but many of her other symptoms remain. She receives Humira treatments for 2 years.

After two years of Humira treatments, Kerstin is diagnosed with hepatosplenic T-cell lymphoma – cancer. She dies on the operating table when her surgeon attempts to remove the lymph nodes on her neck that had been affected by the cancer. She is 34 years old when she passes away.

Kerstin’s story is fictionalized, but it is far from fantasy. Stories like hers happen every day. A large portion of her story is my own and it was both true and horrifying to experience. Stories that are significantly worse, where a doctor’s injection site turn into a staph or MRSA infection to start the whole process, or where anti-psychotic medications that the patient are put on drive her to homicide or suicide. And I didn’t delve into the PAIN that comes with Fluoroquinolone Toxicity (Cipro is a fluoroquinolone and the others are just as bad, if not worse), so it’s a light fictionalized version – with the hope that you’ll find the horror to be believable, because it is very, very real for too many people.

The Explanations, Journal Articles and Facts behind Kerstin’s Story

I don’t expect you to accept the story above as fact without some thorough explanation. Here is the information behind my assertions:

The antibiotic that Kerstin took is Cipro (Ciprofloxacin). Cipro is a fluoroquinolone antibiotic, along with Levaquin, Avelox, Floxin and a few other less commonly used drugs in the fluoroquinolone class. Fluoroquinolone antibiotics are the “big guns” of antibiotics. They are broad spectrum antibiotics that will kill all bacteria in their path. (2) They are frequently prescribed to treat urinary tract infections (3) because they penetrate kidney tissue well (4).

Cipro, and all the other fluroquinolones, have terrible side-effects. Many of the awful side-effects that can be experienced, often all at once, are listed on the Cipro Warning Label. However, many things are left off of the warning label, they are listed on http://www.ciproispoison.com/.

Additionally, here are articles backing up Kerstin’s symptoms:

  • Vision Floaters – The JAMA article entitled “Oral Fluoroquinolones and the Risk of Retinal Detachment” notes that fluoroquinolones increase the incidence of Retinal Detachment (5). If the connective tissue in your eye is damaged, visual disturbances, including floaters, can result.
  • Anxiety – The Journal of Neurosciences in Rural Practices’ article entitled “Levofloacin-induced Acute Anxiety and Insomnia” notes that Levofloxacin (another fluoroquinolone – Levaquin) can induce anxiety and insomnia (6) Cipro/Ciprofloxacin can do the same.
  • Bladder fullness – This is a symptom that I experience, along with many other people suffering from Fluoroquinolone Toxicity. I’m not completely sure what it stems from, but here are a couple of possibilities. This article in the Journal of Urology entitled “Role of Mitochondria in Ciprofloxacin Induced Apoptosis in Bladder Cancer Cells” notes that Cipro disturbs the mitochondria in bladder cells and causes apoptosis (cell death) (7). It is also possible that the feeling of bladder fullness is a result of dysglycemia as it is noted in an article in Medscape Medical News that fluoroquinolones increase the risk of severe dysglycemia in diabetics. Additionally, “one fluoroquinolone antibiotic, gatifloxacin (Tequin, Bristol-Myers Squibb), was already withdrawn from the US market in 2006 due to the risk for severe dysglycemia” (8)
  • Pain and swelling in hands and feet – This symptom can be more succinctly described as peripheral neuropathy. The FDA issued an update to the labels for fluoroquinolones noting that PERMANENT Peripheral Neuropathy is a possibility in August, 2013 (9). This neuropathy may stem from destruction of the Myelin caused by the fluoroquinolone.

There are likely other causes and reasons for Peripheral Neuropathy being a result of Fluoroquinolone Toxicity, including the production of neurotoxins caused by the drugs (10) and the fracturing of DNA (11).

  • Skin problems like hives/uticaria/rashes are listed on the warning label
  • Tendon pain/tear/strain/rupture – This adverse effect is so well documented that fluoroquinolones carry a black box warning about the danger of rupturing a tendon on the top of the warning label. An article in Musculoskeletal Medicine entitled “Musculoskeltal Complications of Fluoroquinolones: Guidelines and Precautions for Usage in the Athletic Population” notes that young, healthy, athletic people’s muscles and tendons are adversely effected by fluoroquinolones (12)
  • Fibromyalgia – Mysterious, sometimes intermittent, sometimes constant, pain is common among those with fluoroquinolone toxicity. The information above about peripheral neuropathy should ring a lot of bells for those diagnosed with Fibromyalgia.  Additionally, Carboxylic Acid is attached to the quinolone molecule (13). It is a known neurotoxin. (14 and 15) Also, a quinolone studied in the article “Cytotoxcicity of Quinolones toward Eukaryotic Cells” notes that quinolones “kills cells by converting the (topoisomerase) type II ezyme into a cellular poison.” (16) Cellular poisons can lead to pain.
  • A diagnosis of Rheumatoid Arthritis – Per Toxicologist, Professor Joe King, “when a cell is malfunctioning (due to a mutation caused by a toxin or radiation) the body deems it alien and begins and autoimmune response as a defense mechanism. Thus producing positive autoimmune antibodies in lab tests, but in actuality you don’t really have the disease, it is bad cells. For example I test positive for rheumatoid arthritis (RA), but I don’t have RA, I have Fibrillan Connective Tissue destruction upon biopsy. But the doctor reads the lab report for RA and recommends anti-inflammatory steroids. Bad diagnosis, because the problem is not RA but Fibrillan and steroids will dissolve the Fibrillan faster.” Also per Professor King, “the cells in your tissue, organs, etc. are not functioning correctly, there is a mutation in there somewhere and the body is reacting to this weird cells as alien, thus producing an inflammatory process (which is painful).” Additionally, it should be noted that Cipro was found to cause chromosomal abnormalities in immune system cells. (17)

I mentioned NSAIDs and steroids. Both NSAIDs and steroids are contraindicated with fluoroquinolones (18 and 12). Please note that Kerstin didn’t take NSAIDs or steroids at the same time as the Cipro. Both NSAIDs and steroids are contraindicated for any person who has ever experienced an adverse reaction to a fluoroquinolone, likely because of the production of acyl glucuronides, “which are chemically reactive electrophiles formed by carboxylic acid-containing drugs” (15) and/or because of the depletion of the CPY450 enzymes by quinolones/fluoroquinolones that leave the body unable to metabolize other drugs (19 and 20).

How do fluoroquinolone ANTIBIOTICS cause all that harm? The harm that they cause is in the essence of the way they work. They are the “first antibacterial agents that efficiently inhibited DNA replication.” (21) Antibiotics in the penicillin and cephalosporin classes, by comparison, work by disrupting bacterial cell walls, not by doing anything to bacterial, or human, DNA. Fluoroquinolones also form “a poisonous adduct on DNA” (21). Fluoroquinolones cause chromosomal abnormalities in human cells (17) and also have tumor killing qualities (22). While that might sound great on the surface, if you read between the lines you’ll note that if these drugs kill tumor cells, they kill human cells. Fluoroquinolones cause apoptosis, programmed cell death, at a massive rate (23). Patient studies have shown, through a DNA Adduct Mass Spectrogram Analysis, that quinolone molecules have adducted to their DNA. Adducting to and breaking human DNA can cause every single one of the problems that Kerstin experienced, all of the problems listed on the FDA warning label for these drugs, and more. It’s a bad idea to mess with human DNA and chromosomes – a look back at the history of Agent Orange will tell you why this is true.

The consequences of the DNA destruction done by fluoroquinolones is yet to be established. An article was published in Nature in September, 2013 connecting topoisomerase inhibiting drugs (fluoroquinolones inhibit topoisomerases II and IV (24)) with triggering the expression of autism related genes. I wrote about this on CE – http://www.collective-evolution.com/2013/09/18/a-horrifying-cause-of-autism-dna-damage-from-synthetic-antibiotics/ Of course, more studies need to be done to determine the implications of this study.

Studies of the DNA make-up of Gulf War Veterans and their children may also be revealing as all 1991 Gulf War Veterans were given Cipro prophylactically because of fear of anthrax (25). Likewise, in 2001 United States Postal Workers who took Cipro prophylactically, also to prevent anthrax, and any ensuing health issues that they have (57% reported side-effects –26) may be related to their exposure to fluoroquinolones.

Fluoroquinolone antibiotics are dangerous drugs that have been used recklessly since their introduction to the market as a first-choice broad-spectrum antibiotic. They are likely responsible for many of the “mysterious” illnesses that have been on the rise since the early 1980s when Cipro was patented by Bayer and Levaquin was patented by Johnson & Johnson. Everyone who has Fibromyalgia, Chronic Fatigue Syndrome, Thyroid Dysfunction, any Autoimmune Disease, Gulf War Syndrome, Leaky Gut Syndrome, Dysautonomia, etc. should look at their medical records to see if they have ever taken a fluoroquinolone. If a fluoroquinolone is in your past, fractured genes may have resulted, and thus your pain and suffering. Please note that adverse reactions to fluoroquinolones are often delayed for weeks or sometimes months or years after administration of the drugs has stopped and there is a tolerance threshold for metabolism of these drugs (20) so most people do not react to their first dose.

Lyrica and Humira

Here is the warning label for Lyrica – (link – Source 27) Please note that suicidal ideation is one of the acknowledged adverse effects caused by this drug. Weight gain, difficulty concentrating, etc. are also listed on the warning label. Patient reports (these people aren’t lying) can be found on askapatient.com – Lyrica.

Humira, Enbrel and other TNF inhibiting drugs CAUSE CANCER. This is well documented and known. The warning labels for both Humira and Enbrel state in a big black box warning that various cancers are associated with use of those drugs. In case it needs to be spelled out, cancer can be deadly.

Here is an excellent blog post about how Humira can kill, and how it is marketed – http://davidhealy.org/welcome-to-the-humiraverse/

Conclusions

It is often noted as people are bemoaning the unwillingness of the pharmaceutical industry to create more antibiotics, that there isn’t enough money to be made from antibiotics to encourage their production. (28) While there may not be much money to be made in selling antibiotics directly, there is a whole lot of money to be made in treating autoimmune diseases. Humira reached $7.9 Billion in sales (29) in 2011 despite the undisputed fact that it causes cancer. If a class of antibiotics can cause the body to react as it would if it had an autoimmune disease for an extended period of time (the ill effects of fluorouquinolones can be permanent but they typically last from 6 to 36 months), and therefore a person gets diagnosed and treated for an autoimmune disease, though they don’t actually have the autoimmune disease, they actually have an autoimmune reaction to a poisonous drug, the pharmaceutical industry has effectively taken an acute problem, an infection, and converted it into a chronic problem, an autoimmune disease. Chronic conditions mean repeat customers and the pharmaceutical industry makes billions. (I doubt that this process is a conspiracy or even planned on the parts of the people in the pharmaceutical industry. Rather, I think that it is caused by willful ignorance among those in the medical professions, encouraged by greed and a complete lack of checks and balances on the pharmaceutical companies, those that have the most to gain in creating repeat customers.)

People are being hurt by their medicine and it is unacceptable. If harm is impossible to avoid completely, it should be minimized. There is zero effort on the part of Doctors, Pharmacists, the FDA or anyone else to minimize adverse effects of drugs. If an effort were being made, we would not be in the tragic situation that we’re in, with the pharmaceutical industry being the 4th leading cause of death of Americans.

The mantra of “all drugs have side-effects” has been so ingrained into the collective consciousness that we have come to think of it as acceptable that drugs have side-effects, and for drug side-effects to be devastating. In accepting this “better someone else than me” / “it can’t happen to me” attitude, we have given permission to the FDA to be inept, incompetent and lazy. In their ineptitude, they have ignored 15 years of research noting that commonly prescribed ANTIBIOTICS are damaging our DNA. We can only hope that this oversight caused by laziness and incompetence is not consequential to us all. Because I can accept the possibility that it may be worth it for society for me to be sacrificed so that we can have powerful antibiotics, but no drug of any sort, no matter what good it does, is worth sacrificing our collective DNA.

Post Script:  The author’s web site, with more information about fluoroquinolones, is www.floxiehope.com

Sources:

1.Donald W. Light, “Risky Drugs: Why the FDA Cannot be Trusted,” Harvard University, The Lab @ Edmond J. Safra Center for Ethics. http://www.ethics.harvard.edu/lab/blog/312-risky-drugs?layout=default#stay-informed

  1. Jane E. Brody, “Popular Antibiotics May Carry Serious Side Effects,” New York Times, September 10, 2012. http://well.blogs.nytimes.com/2012/09/10/popular-antibiotics-may-carry-serious-side-effects/?_r=1
  2. Web MD, Antibiotics for Urinary Tract Infections (UTIs) http://www.webmd.com/a-to-z-guides/antibiotics-for-urinary-tract-infections-utis
  3. DANA E. KING, M.D., ROBB MALONE, PHARM.D., and SANDRA H. LILLEY, PHARM.D., East Carolina University School of Medicine, Greenville, North Carolina, “New Classification and Update on the Quinolone Antibiotics” American Family Physician http://www.aafp.org/afp/2000/0501/p2741.html
  4. Mahyar Etminan, et. al., “Oral Fluoroquinolones and the Risk of Retinal Detachment” JAMA, April 4, 2012—Vol 307, No. 13 http://211.144.68.84:9998/91keshi/Public/File/40/307-13/pdf/joc25028_1414_1419.pdf
  5. Arun Kandasamy and D Srinath, “Levofloxacin-induced acute anxiety and insomnia” J Neurosci Rural Pract. 2012 May-Aug; 3(2): 212–214. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3410005/
  6. OLIVIA ARANHA, et. al., “ROLE OF MITOCHONDRIA IN CIPROFLOXACIN INDUCED APOPTOSIS IN BLADDER CANCER CELLS” The Journal of Urology
Volume 167, Issue 3, Pages 1288-1294, March 2002 http://www.jurology.com/article/S0022-5347(05)65283-4/abstract
  7. Lisa Nainggolan, “Fluoroquinolones Up Risk for Severe Dysglycemia in Diabetes” Medscape Medical News, http://www.medscape.com/viewarticle/809442 Based on this article http://cid.oxfordjournals.org/content/early/2013/07/23/cid.cit439.abstract
  8. 08/15/2013 FDA Drug Safety Communication: FDA requires label changes to warn of risk for possibly permanent nerve damage from antibacterial fluoroquinolone drugs taken by mouth or by injection http://www.fda.gov/Drugs/DrugSafety/ucm365050.htm
  9. David A. Jernigan, “Lyme Toxins The Primary Cause of Your Symptoms” Townsend Letter. April, 2007. http://www.benbrew.com/lb/lyme5.pdf
  10. G. Palu, et. al., “Quinolone binding to DNA is mediated by magnesium ions” Proc. Natl. Acad. Sci. USA Vol. 89, pp. 9671-9675, October 1992 Biochemistry http://www.pnas.org/content/89/20/9671.full.pdf
  11. Mederic M. Hall, MD, Jonathan T. Finnoff, DO, Jay Smith, MD, “Musculoskeletal Complications of Fluoroquinolones: Guidelines and Precautions for Usage in the Athletic Population” 2011 by the American Academy of Physical Medicine and Rehabilitation, Vol. 3, 132-142, February 2011 http://www.levaquinadversesideeffect.com/wp-content/uploads/Documents/Hall-2011.pdf
  12. NAI-XUN CHIN AND HAROLD C. NEU, “Ciprofloxacin, a Quinolone Carboxylic Acid Compound Active Against Aerobic and Anaerobic Bacteria” ANTIMICROBIAL AGENTS AND CHEMOTHERAPY, Mar. 1984, p. 319-326, 1984, American Society for Microbiology http://www.ncbi.nlm.nih.gov/pmc/articles/PMC185508/pdf/aac00192-0027.pdf
  13. José Antonio Vázquez, et. al. “Evaluation of toxic effects of several carboxylic acids on bacterial growth by toxicodynamic modelling” Microbial Cell Factories2011,10:100 http://www.microbialcellfactories.com/content/10/1/100
  14. Urs A. Boelsterli, “Acyl Glucuronides: Mechanistic Role in Drug Toxicity?” Current Drug Metabolism (v.12, #3) p. 213-214 http://www.chemweb.com/journals/journals?type=issue&jid=13892002&iid=12003
  15. Sarah H. Elsea, et. Al, “Cytotoxicity of Quinolones toward Eukaryotic Cells: IDENTIFICATION OF TOPOISOMERASE I1 AS THE PRIMARY CELLULAR TARGET FOR THE QUINOLONE CP-115,953 IN YEAST,” The Journal of Biological Chemistry, Vol. 267, No. 19, Issue of July 5, pp. 13150-13153 http://www.jbc.org/content/267/19/13150.full.pdf+html
  16. PS Ambulkar, et. Al, “Genotoxic and cytotoxic effects of antibacterial drug, ciprofloxacin, on human lymphocytes in vitro” Nepal Med Coll J 2009; 11(3): 147-151 http://www.nmcth.edu/images/gallery/Editorial/xRZVmps_ambulkar.pdf
  17. S. Mannino, et. al., “NSAIDs, Quinolones and Convulsions: An Epidemiologic Approach” Post Marketing Survellance 1992 p. 119-128 http://213.4.18.135:48080/10.pdf
  18. HJ Xie, et. al., “Alteration of pharmacokinetics of cyclophosphamide and suppression of the cytochrome P450 genes by ciprofloxacin” Bone Marrow Transplantation (2003) 31,197–203. http://www.nature.com/bmt/journal/v31/n3/abs/1703815a.html
  19. Dean P. Jones, et. al., “Mechanisms of Pathogenesis in Drug Hepatotoxicity Putting the Stress on Mitochondria” Mol Interv. 2010 April;10(2): 98–111. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2895369/
  20. Arkady B. Khodurskyand Nicholas R. Cozzarelli, “The Mechanism of Inhibition of Topoisomerase IV by Quinolone Antibacterials” October 16, 1998 The Journal of Biological Chemistry, 273, p. 27668-27677.http://www.jbc.org/content/273/42/27668.full
  21. Yi Xia, et. al., “Recent Advances in the Discovery and Development of Quinolones and Analogs As Antitumor Agents” Current Medicinal Chemistry, 1999, p. 179-194 http://books.google.com/books?hl=en&lr=&id=SJoxUN91vK4C&oi=fnd&pg=PA179#v=onepage&q&f=true
  22. V Talla and PR Veerareddy, “Oxidative Stress Induced by Fluoroquinolones on Treatment for Complicated Urinary Tract Infections in Indian Patients” J Young Pharm. 2011 Oct-Dec; 3(4): 304–309. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3249743/?report=printable
  23. 2013 FDA Warning Label for Ciprofloxacin (Cipro) – http://www.accessdata.fda.gov/drugsatfda_docs/label/2013/019537s082,020780s040lbl.pdf
  24. Patricia Kime, “New FDA warnings on Cipro may tie into Gulf War illness” Air Force Times, November 1, 2013. http://www.airforcetimes.com/article/20131101/NEWS/311010018/New-FDA-warnings-Cipro-may-tie-into-Gulf-War-illness
  25. “Postal Workers Sue Bayer Over Cipro” http://www.yourlawyer.com/articles/title/postal-workers-sue-bayer-over-cipro
  26. Lyrica Label http://www.accessdata.fda.gov/drugsatfda_docs/label/2009/021446s013s014lbl.pdf
  27. Garcia Rey-C, “The role of the pharmaceutical industry. Why aren’t new antibiotics being marketed?”  2010 Nov;28 Suppl 4:45-9. http://www.ncbi.nlm.nih.gov/pubmed/21458701
  28. Ben Comer, “Brand of the Year: Humira” PharmExec.com February 1, 2012 http://www.pharmexec.com/pharmexec/article/articleDetail.jsp?id=757392

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Awareness

Cancer is Now the Leading Cause of Death

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In Brief

  • The Facts:

    Cancer has surpassed heart disease as the No. 1 cause of death in high-income countries, highlighting the urgent need to change the way this disease is prevented and treated.

  • Reflect On:

    Rather than being a random result of DNA mutations, it's possible that cancer could have much deeper roots that would be better targeted with natural therapies than toxicity.

This article was written by the Greenmedinfo Research Group, originally published by Greenmedinfo.com. Published here with permission. 

Cancer has dethroned heart disease to earn the nefarious title of leading cause of death in high-income and certain middle-income countries.[i] While heart disease remains the No. 1 cause of death globally among adults aged 35 to 70, in high-income countries, which included Saudi Arabia, United Arab Emirates, Canada and Sweden, cancer caused twice as many deaths as heart disease.[ii]

Some middle-income countries, which included the Philippines, Iran, South Africa, Colombia, China, Brazil, Malaysia, Turkey, Poland, Argentina and Chile, also saw cancer become the leading cause of death.

While the U.S. was not included in the new analysis, research published in 2018 suggested, “the United States is in the midst of an epidemiologic transition in the leading cause of death,” moving from heart disease to cancer.[iii]

That study, too, found that cancer was quickly outpacing heart disease as the top killer, with high-income counties transitioning first. In fact, while only 21% of U.S. counties had cancer as the leading cause of death in 2003, this rose to 41% in 2015.

“The shift to cancer as the leading cause of death was greatest in the highest-income counties,” the researchers explained,[iv] echoing the current study, which also cited “a transition in the predominant causes of deaths in middle-age” in high-income countries.[v]

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“The world is witnessing a new epidemiologic transition among the different categories of noncommunicable diseases, with CVD [cardiovascular disease] no longer the leading cause of death in HIC [high-income countries],” lead author Dr. Gilles Dagenais, professor emeritus, Laval University, Quebec, Canada, said in a statement.[vi]

Why is Cancer a Top Killer?

The study suggested cancer is rising to the top because heart disease is better treated in high-income countries, saving more lives from heart disease and paving the way for cancer deaths to flourish. But perhaps a better question is why cancer continues to kill so many.

Even globally, cancer still comes in as the second leading cause of death behind heart disease, responsible for 26% of deaths worldwide.[vii] In the U.S., Americans have a 1 in 3 risk of developing cancer at some point in their lifetimes, along with a 1 in 5 risk of dying from the disease.[viii]

In early 2019, it was announced that cancer death rates in the U.S. declined 27% since 1991,[ix] a statistic that makes it seem as though we’re winning the “war on cancer.” But most of these declines can be attributed to reductions in smoking — and perhaps a limited measure of increased early detection and treatment — and are not a sign that conventional medicine’s model of surgerychemotherapy and/or radiation to treat cancer is, on the whole, working.

While death rates from certain cancer have declined, others have increased. Overall, cancer deaths in the U.S. in 2016 were similar to those in 1930[x] — despite all the “advances” in detection and treatment.

Changing the Way We Think About Cancer

It’s becoming increasingly clear that in order to conquer cancer, it’s necessary to change the way we think about it. Cancer is found in virtually all animals, suggesting it has evolutionary significance.[xi] It’s possible that cancer is an ancient survival program unmasked — even a process the body undergoes in order to survive nutrient deprivation and exposure to toxins.

Rather than being the result of an accumulation of DNA mutations that create rogue cells that multiply out of control, cancer could be cells that have flipped an epigenetic switch into survival mode in the form of a tumor. In the journal Physical Biology, researchers theorized:[xii]

“[C]ancer is an atavistic [primitive] condition that occurs when genetic or epigenetic malfunction unlocks an ancient ‘toolkit’ of pre-existing adaptations, re-establishing the dominance of an earlier layer of genes that controlled loose-knit colonies of only partially differentiated cells, similar to tumors.”

If this is true, it makes sense that conventional cancer treatments aimed to poison or “kill” the cancerous cells may only make the problem worse by creating an even more toxic environment, which could trigger the cancer to reach back into its “ancient toolkit” to find additional means of survival.

This explanation may be overly simplistic, as there are many factors that contribute to cancer, but there is evidence to suggest that natural substances and therapies that support the body’s overall health can be useful in the fight against cancer.

Nearly 1,000 Natural Substances Have Anti-Cancer Potential

GreenMedInfo has a database of 986 substances that have been researched as potential cancer prevention and treatment strategies. There are undoubtedly many more out there that have yet to be discovered. At the top of the list is curcumin, the active ingredient in the curry spice turmeric, which targets cancer stem cells while leaving normal stem cells unharmed.[xiii]

Another top contender is vitamin D, which you can get for free from adequate sun exposure. Higher vitamin D levels are not only known to lower your cancer risk but also to improve outcomes if you’ve already been diagnosed.[xiv] Fiberresveratrolsulforaphane and vitamin E — all substances you can get from your diet — also show anti-cancer promise, as does coffee, perhaps because it improves the body’s ability to efficiently repair DNA damage.[xv]

So if there was one silver lining to the news that cancer is now the leading cause of death in some countries, it would be that it’s a condition that has many promising natural avenues for prevention and treatment. Current conventional cancer treatments are failing, but that doesn’t mean cancer is unstoppable — it means it’s time to broaden our research into and usage of traditional therapies.

Many natural substances, like noni leaf,[xvi] have even been shown to work better than chemotherapy, highlighting why, if we’re going to win the war against cancer, we’re going to need to do it with nature on our side.

For more on how to naturally fight Cancer, visit the GreenMedInfo database on the subject.

Originally published: 2019-09-14

Article Updated: 2019-11-05

References

[i] The Lancet September 3, 2019

[ii] CNN September 3, 2019

[iii] Annals of Internal Medicine December 18, 2018

[iv] Annals of Internal Medicine December 18, 2018

[v] The Lancet September 3, 2019

[vi] Medscape September 3, 2019

[vii] Medscape September 3, 2019

[viii] American Cancer Society, Lifetime Risk of Developing or Dying From Cancer

[ix] CA: A Cancer Journal for Clinicians January 8, 2019

[x] CA: A Cancer Journal for Clinicians January 8, 2019

[xi] Front. Oncol., 10 January 2019

[xii] Physical Biology February 7, 2011

[xiii] Anticancer Res. 2015 Feb ;35(2):599-614.

[xiv] Br J Cancer. 2017 Mar 16. Epub 2017 Mar 16.

[xv] J Nutrigenet Nutrigenomics. 2015 ;8(4-6):174-84.

[xvi] Mol Cell Biochem. 2016 Apr 22. Epub 2016 Apr 22.


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Awareness

Man Fasts For 382 Days Straight & Loses 276 Pounds

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In Brief

  • The Facts:

    Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days. He remained completely healthy for the duration of the fast.

  • Reflect On:

    Today, it's firmly established in scientific literature that fasting can have tremendous benefits, if done correctly. It can also be used to treat a variety of diseases. Perhaps it's not emphasized because you can't make money off of not eating?

A study published in the Post Graduate Medical Journal in 1972 brought more attention to a gentleman by the name of Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days and, at the time the study was published, had since maintained his ordinary weight. In his case, “prolonged fasting had no ill effects.” Barbieri’s weight decreased from 456 to 180 pounds during the fast.

This isn’t the only example that’s available in the literature, it’s similar to an earlier patient prior to Barbieri who reduced his weight from 432 to 235 pounds during 350 days of intermittent fasting (Stewart, Fleming & Robertson, 1966). Researchers have also fasted patients for 256 days (Collison, 1967, 1971), 249 and 236 days (Thomson et al., 1966) as well as  210 days (Garnett et al., 1969; Runcie & Thomson, 1970), all of which are cited in the 1972 study.

Since the publication of this time, there are many documented examples of prolonged fasting done by highly obese people. Here’s one recent example of a man who fasted for 50 straight days, while being medically supervised and tested the whole time.

When you fast, your body switches from burning glucose, to burning fat. Fasting lowers insulin levels which allows the body to access its fat stores for energy. When you eat, food is converted into glucose and that’s what we usually burn. This is why fasting has become a therapeutic intervention for many people with type two diabetes, and more doctors, like Dr. Jason Fung, a Toronto Based nephrologist, are having great success with utilizing fasting as an appropriate and necessary health intervention. Fung has many great articles regarding the science of fasting, you can access them here if you’re interested in learning more. This article references some of the leading scientists in the field so you can learn more by looking them up as well.

The graph below depicts what happens to your protein while fasting. Interesting isn’t it? People often believe that if you fast, you will experience a tremendous amount of muscle loss during fasting, but that’s simply not true. This graph is from Kevin Hall, from the NIH in the book “Comparative Physiology of Fasting, Starvation, and Food Limitation.”

“It seems that there are always concerns about loss of muscle mass during fasting. I never get away from this question. No matter how many times I answer it, somebody always asks, “Doesn’t fasting burn your muscle?” Let me say straight up, NO.”  – source Dr. Jason Fung

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But what about Angus Barbieri? Obviously we’re not saying long term fasts for this long are healthy, obviously for many people they will probably be unhealthy and unsafe unless medically supervised. In  the 1972 study doctors measured a number of concentrations within the body. For example, plasma potassium concentrations over the first four months decreased systematically. As a result, they provided a very small daily dose that increased his potassium level. After another 10 weeks, no potassium was given, and from there on in until the end of the fast, plasma potassium levels remained normal. Cholesterol concentrations also remained around 230 mg/ 100 ml until 300 days of fasting, but increased to 370 mg/100 ml during refeeding.

Plasma magnesium levels decreased over the first few weeks of the fast but then went up and stabilized. This is interesting to note as there is nothing going into the body, yet levels still stabilized after the initial decrease.

Normal plasma magnesium concentrations, despite magnesium ‘depletion’ in muscle tissue, have been described (Drenick et al., 1969) during short-term fasting (1-3 months). The only other relevant report is a remark (Runcie & Thomson, 1970) that one patient who fasted 71 days had a normal plasma magnesium level of 2-2 mEq/l at the time when she developed latent tetany. The decrease in the plasma magnesium concentration of our patient was systematic and persistent.

Furthermore:

The excretion of sodium, potassium, calcium and inorganic phosphate decreased to low levels throughout the first 100 days, but thereafter the excretion of all four urinary constituents, as well as of magnesium, began to increase. During the subsequent 200 days sodium excretion, previously between 2 and 20 mEq daily, reached over 80 mEq/24 hr, potassium excretion increased to 30-40 mEq daily and calcium excretion increased from 10-30 mg/24 hr to 250- 280 mg/24 hr. Magnesium excretion (which was not measured during the first 100 days) reached 10 mEq/ 24 hr between Days 200-300. Phosphate excretion, which had decreased to under 200 mg/24 hr, also increased to around 800 mg/24 hr, even exceeding 1000 mg/24 hr on occasion. Peak excretions of all these constituents were seen around Day 300, after which there was a marginal decrease, but excretion remained high.

Obviously, this is an extreme fast and such fasts have only been tested on people of tremendous obesity, and it shows that people with a high body fat percentage have the ability to fast longer simply because their body has more stores to pull from.

The study concluded in 1972 that:

We have found, like Munro and colleagues (1970), that prolonged supervised therapeutic starvation of the obese patient can be a safe therapy, which is also effective if the ideal weight is reached. There is, however, likely to be occasionally a risk in some individuals, attributable to failures in different aspects of the adaptative response to fasting. Until the characteristics of these variations in response are identified, and shown to be capable of detection in their prodromal stages, extended starvation therapy must be used cautiously. In our view, unless unusual hypokalaemia is seen, potassium supplements are not mandatory. Xanthine oxidase inhibitors (or uricosuric agents) are not always necessary and could even be potentially harmful (British Medical Journal, 1971) perhaps particularly in the long-term fasting situation.

It’s almost 2020, and the literature, studies and research that’s been published since 1972 is vast. We’ve learned a lot more about it and if done correctly it can be extremely beneficial. Shot term fasting  presents minimal to no health risks, and so does long term fasting that lasts more than 24 hours, that is unless a person already has an underlying condition. That being said, it’s not easy to start. Most people are used to eating three meals plus snacks every single day, therefore they are never adapted to burning their fat stores, something that appears the human body was meant to do.

“Why is it that the normal diet is three meals a day plus snacks? It isn’t that it’s the healthiest eating pattern, now that’s my opinion but I think there is a lot of evidence to support that. There are a lot of pressures to have that eating pattern, there’s a lot of money involved. The food industry — are they going to make money from skipping breakfast like I did today? No, they’re going to lose money. If people fast, the food industry loses money. What about the pharmaceutical industries? What if people do some intermittent fasting, exercise periodically and are very healthy, is the pharmaceutical industry going to make any money on healthy people?” – Mark Mattson (source)

Fasting has also been shown to be effective as a therapeutic intervention for cancer. Fasting protects healthy cells while ‘starving’ cancer cells, it’s now being used as an intervention that’s being combined with chemotherapy. Fasting has also been shown to greatly reduce the risk of age related diseases like Parkinson’s Disease, and Alzheimer’s disease. Mark Mattson, one of the foremost researchers of the cellular and molecular mechanisms underlying multiple neurodegenerative disorders has shown through his work that fasting can have a tremendous effect on the brain, and can even reverse the symptoms of multiple neurodegenerative disorders. You can watch his interesting TED talk here.  Scientists have also discovered strong evidence that fasting is a natural intervention for triggering stem cell-based regeneration of an entire organ or system.

Fasting has actually long been known to have an effect on the brain. Children who suffer from epileptic seizures have fewer of them when placed on caloric restriction or fasts. It is believed that fasting helps kick-start protective measures that help counteract the overexcited signals that epileptic brains often exhibit.  (source)

The list goes on and is quite long. At the end of the day if you do your research, fasting, under proper medical supervision, can have tremendous health benefits that go far beyond what’s mentioned in the paragraph above. Every single study that has looked at fasting as a therapeutic intervention for several diseases has shown nothing but positive benefits. Even studies conducted regarding caloric restriction, something completely different than fasting, have shown promising results in all animal models.

According to a review of fasting literature conducted in 2003, “Calorie restriction (CR) extends life span and retards age-related chronic diseases in a variety of species, including rats, mice, fish, flies, worms, and yeast. The mechanism or mechanisms through which this occurs are unclear.” Since this study was published, a great amount of research has been conducted from many researchers, and the mechanisms are being discovered and have become more clear. If you want to further your research, apart from the names listed above, Dr. Valter Longo and his research is another great place to start.

The body has a tremendous amount of storage, and it hangs on to what it needs during a fast, and uses up ‘bad’ things, repairs damaged cells, and more. When you fast and deplete all your glycogen, your body is going to start using fat for energy, it’s going to use damaged cells for energy, it’s basically going to use all of the bad things first, before it gets to the good thing…Your body will not burn protein, as protein is not a fuel source while fasting.

I bring this up because it’s interesting to see what the body loses and hangs on to during a fast.

The Takeaway

The truth about fasting is that it’s not dangerous at all. Intermittent fasting and short term fasting can be done by just about anybody. From what we’ve seen with regards to prolonged fasting, it’s also not very dangerous when it comes to obese people doing it under medically supervised conditions. Theoretically, based on the science alone, any relatively healthy human being should be able to do a prolonged fast without any harmful consequences.

Obviously, prolonged fasts that are not medically supervised can be very detrimental. We are obviously not recommending this and you must do a lot of research and talk to your doctor if you’re interested in fasting, before trying it. For starters, a little bit of intermittent fasting here and there is a no brainer, and not dangerous at all if you have no underlying health conditions, but everybody’s body is different.

Fasting is making a lot of noise, and has been making a lot of noise within the health community, but it’s still not appropriately taught and used by the mainstream medical industry. Why is this so? The answer is simple, you can’t make money off of fasting.

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Alternative News

Thousands Gather To Mark The 33rd Anniversary of the National Childhood Vaccine Injury Act

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Government’s gift to Pharma of liability-free vaccines puts children’s health at risk states Children’s Health Defense (CHD) Chairman, Robert F. Kennedy, Jr.

Washington, DC – Thousands of advocates for children’s health will gather Thursday at the Vaccine Injury Epidemic (VIE) Event on the National Mall to mark the 33rd anniversary of National Childhood Vaccine Injury Act (NCVIA). The rally on Nov. 14th will spotlight the devastating impact NCVIA has had upon the state of children’s health. While children continue to be injured by vaccines daily, vaccine makers cannot be held accountable, thereby eliminating incentive for vaccine safety.

In his remarks, RFK, Jr. will address the ramifications of NCVIA and honor those whose lives have been impacted by vaccine injury and death. “It’s time to call out Congress, the CDC, and drug companies for allowing industry profits to trump children’s health,” said Kennedy. “There is no crisis more urgent than the epidemics of chronic health conditions among our nation’s children.”

Following NCVIA’s passage creating the National Vaccine Injury Compensation Program (NVICP), the childhood vaccine market sparked a gold rush for Pharma as more vaccines for routine childhood illnesses were developed. Coterminous with the burgeoning vaccine schedule, chronic health conditions in children rose from 12% to 54%. As vaccine industry profits grew to $50 billion annually, so did diagnoses of asthmaautismADHDallergiesanxietydepressiondiabetesobsessive-compulsive disorder and auto-immune diseases.  Here are the facts:

  • An HHS-funded study found only 1% of vaccine injuries are reported.
  • Despite NVICP’s high burden of proof and two out of three claims dismissed, over $4.2 billion has been paid for claims of vaccine injury or death.
  • The vaccine-injured find NVICP to be a years-long, litigious program with no jury, discovery and precedent. While medical bills mount, the injured are up against DOJ lawyers and HHS “Special Masters” that act as judges.
  • The Department of Justice and the NVICP are accused of fraud and obstruction of justice in the Autism Omnibus Proceeding.
  • The Institute of Medicine reports that the vaccine schedule as recommended has never been studied for long-term health effects despite independent research suggesting that unvaccinated children are healthier.
  • Modern medicine acknowledges that not everyone responds the same to vaccination and the “one size fits all” vaccine policy is not science based.

Children’s Health Defense’s created these six steps to vaccine safety. RFK, Jr. interviews are available upon request.

Sign up for free news and updates from Robert F. Kennedy, Jr. and the Children’s Health Defense. CHD is planning many strategies, including legal, in an effort to defend the health of our children and obtain justice for those already injured. Your support is essential to CHD’s successful mission.

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