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Fluorine-Based Toxins Accumulate In The Body & Cause Multiple Health Problems

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When you bake muffins in a non-stick tin, do you think about how the non-stick coating is going to affect your health? There has been enough publicity around damaged Teflon that some reading this will likely think about the harm that can come from chipped non-stick coating. After all, who wants to eat that stuff? But I still doubt that most people think about the health consequences of the vapors that are being emitted from the heated non-stick pan or the chemicals that are being leached into their muffins while they bake them.

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Do you think about the vapors coming up from carpet-covered in-floor heating? Do you think about the health consequences of the particularly slippery dental floss that has become so popular? Do you think about how non-clump kitty litter may affect the health of your children? Do you think about how water-repellant packaging chemicals seep into the food they cover?

Most people don’t think about those things. Consumer products with non-stick coatings are such a pervasive part of life that they’re rarely thought about at all. Most people assume that those products are safe and that the Environmental Protection Agency (E.P.A.) and other regulatory agencies wouldn’t let people use them if they were dangerous.

Unfortunately, there is increasing evidence that the chemicals used to make non-stick consumer products, chemicals such as perfluorooctanoic acid (PFOA, a.k.a. C8), are toxic and that they’re related (causally, but that’s difficult to prove) to many diseases. Even more worrisome is the fact that organofluorine compounds like PFOA bioaccumulate — meaning that they are not excreted from the body. Rather, they accumulate in the body with each exposure, and the risk of suffering from ill-health because of them increases as the toxic load on the body increases. Additionally, man-made organofluorine compounds accumulate in the environment and the food-chain. Creatures who are high on the food chain, like humans, get exposed to these chemical compounds that have concentrated our food. We’re also affected by them when they’re in the water that we drink and the air that we breathe.

Whether you think about it or not, you have PFOA and other organofluorine compounds in your blood. Since they started being produced in the 1950s, PFOAs and other organofluorine compounds, have become so pervasive that every person, and every living creature tested, has organofluorines in his or her body (1).

PFOAs, and other organoflurine compounds, are toxic to living creatures. In animal studies, PFOAs have “been found to cause liver toxicity, disruption of lipid metabolism and the immune and endocrine systems, adverse neurobehavioral effects, neonatal toxicity and death, and tumors in multiple organ systems” (2), as well as “cancerous testicular, pancreatic and liver tumors” (3). In humans, elevated levels of PFOAs in the blood have been linked to “testicular cancer, liver cancer, thyroid disease, ulcerative colitis, high cholesterol and pregnancy-induced hypertension” (3), as well as seizures, kidney failure, miscarriage and birth defects (4). DNA damage has been linked to organofluorine compounds (3). The toxicity of organofluorine compounds is widely spread across many organ systems, and the diseases that are related to them are multi-symptom, chronic diseases.

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In addition to being found in thousands of household products like carpeting, non-stick pans, waterproof clothes, dental floss, kitty litter and cosmetics (4), organofluorine compounds are also found in “aerosol propellants, surfactants, refrigerants, plastics, anesthetics, pesticides, plant growth regulators, medicines, adhesives, fire retardants, and even blood substitutes” (5). These products are used daily by millions of people, most of whom aren’t aware that they’re exposing themselves to toxins.

Organofluorine compounds are particularly dangerous because they essentially never biodegrade, and accumulate in the bodies of living beings while moving through the food-chain (6). Organofluorines have been “detected everywhere—produce and beef in American grocery stores, polar bears in the Arctic, children in the remote Faeroe Islands. One analysis of blood banks from around the world showed that nearly all of the blood contained C8. The lone exception was a set of archived samples that had been collected from Korean War veterans before 1952.” (4).

Organofluorine accumulation is getting worse every day, and with each generation. Organofluorine compounds have been found in umbilical cord blood (3) and breast milk, and children throughout the world are exposed to these toxic chemical compounds every day of their lives. As more and more organofluorines accumulate in each person, the toxic burden will get worse, and health consequences will become increasingly dire.

Unfortunately, “there are no reported studies of successful interventions to remove PFCs from the human body. Exposure to these commonly used non-stick and stain-resistant compounds is widespread, but excretion from the body is impaired as a result of the chemical nature of some of these agents and their propensity to be re-absorbed in the kidney and the enterohepatic [liver] circulation” (6).

With the progressive accumulation of organofluorine compounds, and as human health outcomes worsen as a result, there will undoubtedly be angry people who want to hold those responsible accountable for the creation of toxic organofluorines. As is often the case, the responsible parties are greedy corporations and failing regulatory agencies.

Corporate and Regulatory Culprits

Chemical giants DuPont and 3M are the primary producers of organofluorines that are used in household products. In their production of these goods, they have made billions in profit, while making people (and animals and plants) sick. For years DuPont covered up evidence of the toxic nature of PFOAs/C8, and rather than ceasing to use toxic organofluorine compounds when the toxicity of PFOA/C8 became clear, they switched from C8 to other organofluorine compounds which are no better for human or environmental health, but are not as well studied as C8 (4).

The regulatory agencies that are supposed to monitor the safety of chemicals in our environment, including the E.P.A., have failed to regulate organofluorine compounds in any meaningful way. “Under the 1976 Toxic Sub­stances Control Act, the E.P.A. can test chemicals only when it has been provided evidence of harm. This arrangement, which largely allows chemical companies to regulate themselves, is the reason that the E.P.A. has restricted only five chemicals, out of tens of thousands on the market, in the last 40 years” (3). Organofluorine compounds are not restricted by the E.P.A., and industrial giants/polluters like DuPont and 3M produce thousands of tons of them each year.

Details about the malfeasance of DuPont, the role of 3M, and the unwillingness and inability of the E.P.A. to protect people from the toxic, harmful effects of PFOA/C8 can be found in the brilliant exposés, “The Lawyer Who Became DuPont’s Worst Nightmare” written by Nathaniel Rich and published in The New York Times Magazine on January 6, 2016, and “Welcome to Beautiful Parkersburg, West Virginia: Home to one of the most brazen, deadly corporate gambits in U.S. history” written by Mariah Blaze and published in The Huffington Post.

Also exemplifying the problem of industry-controlled regulation of organofluorine compounds is the following from the article “Human detoxification of perfluorinated compounds”:

While there is increasing evidence suggesting health sequelae associated with PFC bioaccumulation, there are some authors and publications that have minimized concern about the accrual of these chemical agents. It is imperative to recognize that much of the research related to potentially toxic compounds, including PFCs, is undertaken or funded by the specific companies, industry-supported organizations and affiliated scientists involved with the production of the chemicals being studied. Furthermore, some toxicology journals routinely use reviewers from chemical companies to scrutinize and review manuscript submissions about the compounds their company manufactures. (6)

As long as science and journalism are corrupted by money, and regulatory agencies and politicians are controlled by corporations, citizens of the world have little protection against the toxic chemicals that can accumulate in our bodies and poison us.

Pharmaceuticals

Another source of toxic organofluorine compounds is fluorinated pharmaceuticals. Approximately 20% of the prescription drugs on the market are fluorinated (7), including fluoroquinolone antibiotics (Cipro/ciprofloxacin, Levaquin/levofloxacin, Avelox/moxifloxacin, Floxin/ofloxacin), statins (Lipitor, Crestor and others), antimalarial drugs (Lariam/mefloquine), antidepressants (SSRIs including Prozac/fluoxetine), anesthesias, and chemotherapy drugs. Depending on how the drugs are formulated, and what position fluorine is attached to carbon, they can form poisonous metabolites (8, 9)  that may be responsible for the severe side-effects associated with many of the fluorinated drugs, and they may be contributing to the multi-symptom diseases of modernity. (Many of the drugs listed have been shown to damage mitochondria (10, 11), and damaged mitochondria lead to a cycle of oxidative stress and further damage to mitochondria (12). Fluorine metabolites, such as fluoroacetate, have also been shown to damage mitochondria and increase oxidative stress (13). Many multi-symptom, chronic diseases are linked to both mitochondrial damage and oxidative stress.)

Pesticides

In an article published in 1997 (the use of fluorine-containing pesticides has escalated since), it is noted that, “Over the past 15 years, the number of fluorine-containing agricultural chemicals has grown from 4% to approximately 9% of all agrochemicals and has increased in number faster than non-fluorinated agrochemicals. These compounds are primarily used as herbicides (48%), insecticides (23%), and fungicides (18%)” (5). Exposure to these fluorinated pesticides is hazardous to the health of both agricultural workers and consumers (14).

What Can be Done?

In May, 2015, more than 200 scientists signed The Madrid Statement, “which expresses concern about the production of all fluorochemicals, or PFASs, including those that have replaced PFOA” (3). In the Statement, they suggested that the following action take place:

Scientists:

  • Assemble, in collaboration with industry and governments, a global inventory of all PFASs in use or in the environment, including precursors and degradation products, and their functionality, properties, and toxicology.
  • Develop analytical methods for the identification and quantification of additional families of PFASs, including fluorinated alternatives.
  • Continue monitoring for legacy PFASs in different matrices and for environmental reservoirs of PFASs.
  • Continue investigating the mechanisms of toxicity and exposure (e.g., sources, fate, transport, and bioaccumulation of PFASs), and improve methods for testing the safety of alternatives.
  • Bring research results to the attention of policymakers, industry, the media, and the public.

Governments:

  • Enact legislation to require only essential uses of PFASs, and enforce labeling to indicate uses.
  • Require manufacturers of PFASs to conduct more extensive toxicological testing, make chemical structures public, provide validated analytical methods for detection of PFASs, and assume extended producer responsibility and implement safe disposal of products and stockpiles containing PFASs.
  • Work with industry to develop public registries of products containing PFASs.
  • Make public annual statistical data on production, imports, and exports of PFASs.
  • Whenever possible, avoid products containing, or manufactured using, PFASs in government procurement.
  • In collaboration with industry, ensure that an infrastructure is in place to safely transport, dispose of, and destroy PFASs and PFAS-containing products, and enforce these measures.

Chemical manufacturers:

  • Make data on PFASs publicly available, including chemical structures, properties, and toxicology.
  • Provide scientists with standard samples of PFASs, including precursors and degradation products, to enable environmental monitoring of PFASs.
  • Work with scientists and governments to develop safe disposal methods for PFASs.
  • Provide the supply chain with documentation on PFAS content and safe disposal guidelines.
  • Develop nonfluorinated alternatives that are neither persistent nor toxic.

Product manufacturers:

  • Stop using PFASs where they are not essential or when safer alternatives exist.
  • Develop inexpensive and sensitive PFAS quantification methods for compliance testing.
  • Label products containing PFASs, including chemical identity and safe disposal guidelines.
  • Invest in the development and use of nonfluorinated alternatives.

Purchasing organizations, retailers, and individual consumers:

  • Whenever possible, avoid products containing, or manufactured using, PFASs. These include many products that are stain-resistant, waterproof, or nonstick.
  • Question the use of such fluorinated “performance” chemicals added to consumer products.

Indeed, all of those recommendations need to be put into action. Given the accumulation of organofluorine compounds in our bodies and environment, the time for putting these recommendations into action is now–before it’s too late.

Sources:

  1. Environmental Science & Technology, “Production of Perfluorinated Carboxylic Acids (PFCAs) from the Biotransformation of Polyfluoroalkyl Phosphate Surfactants (PAPS):  Exploring Routes of Human Contamination
  2. Environmental Health Perspectives, “The Madrid Statement on Poly- and Perfluoroalkyl Substances (PFASs)
  3. The New York Times Magazine, “The Lawyer Who Became DuPont’s Worst Nightmare
  4. The Huffington Post, “Welcome to Beautiful Parkersburg, West Virginia: Home to one of the most brazen, deadly corporate gambits in U.S. history
  5. Environmental Science & Technology, “Fluorinated Organics in the Biosphere
  6. Public Health, “Human detoxification of perfluorinated compounds
  7. Biomolecular Research & Therapeutics, “Fluorine is Flourishing in Pharmaceuticals
  8. Verdel, B.M., “Mechanism-based drug exposure classification in pharmacoepidemiological studies” Thesis Utrecht University – with ref. – with summary in Dutch, ISBN 978-90-393-5377-6 © 2010 Bertha Maria Verde
  9. British Journal of Cancer, “The anti-cancer drug 5-fluorouracil is metabolized by the isolated perfused rat liver and in rats into highly toxic fluoroacetate.
  10. Molecular Nutrition and Food Research, “Medication-induced mitochondrial damage and disease
  11. Science Translational Medicine, “Bactericidal Antibiotics Induce Mitochondrial Dysfunction and Oxidative Damage in Mammalian Cells
  12. Rejuvination Research, “Reactive oxygen species production in the mitochondrial matrix: implications for the mechanism of mitochondrial mutation accumulation.
  13. Biochemical Pharmacology, “Differential toxicity of fluoroacetate to heart, kidney and brain mitochondria of the living rat
  14. http://fluoridealert.org/researchers/pesticide/

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Awareness

Cancer is Now the Leading Cause of Death

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In Brief

  • The Facts:

    Cancer has surpassed heart disease as the No. 1 cause of death in high-income countries, highlighting the urgent need to change the way this disease is prevented and treated.

  • Reflect On:

    Rather than being a random result of DNA mutations, it's possible that cancer could have much deeper roots that would be better targeted with natural therapies than toxicity.

This article was written by the Greenmedinfo Research Group, originally published by Greenmedinfo.com. Published here with permission. 

Cancer has dethroned heart disease to earn the nefarious title of leading cause of death in high-income and certain middle-income countries.[i] While heart disease remains the No. 1 cause of death globally among adults aged 35 to 70, in high-income countries, which included Saudi Arabia, United Arab Emirates, Canada and Sweden, cancer caused twice as many deaths as heart disease.[ii]

Some middle-income countries, which included the Philippines, Iran, South Africa, Colombia, China, Brazil, Malaysia, Turkey, Poland, Argentina and Chile, also saw cancer become the leading cause of death.

While the U.S. was not included in the new analysis, research published in 2018 suggested, “the United States is in the midst of an epidemiologic transition in the leading cause of death,” moving from heart disease to cancer.[iii]

That study, too, found that cancer was quickly outpacing heart disease as the top killer, with high-income counties transitioning first. In fact, while only 21% of U.S. counties had cancer as the leading cause of death in 2003, this rose to 41% in 2015.

“The shift to cancer as the leading cause of death was greatest in the highest-income counties,” the researchers explained,[iv] echoing the current study, which also cited “a transition in the predominant causes of deaths in middle-age” in high-income countries.[v]

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“The world is witnessing a new epidemiologic transition among the different categories of noncommunicable diseases, with CVD [cardiovascular disease] no longer the leading cause of death in HIC [high-income countries],” lead author Dr. Gilles Dagenais, professor emeritus, Laval University, Quebec, Canada, said in a statement.[vi]

Why is Cancer a Top Killer?

The study suggested cancer is rising to the top because heart disease is better treated in high-income countries, saving more lives from heart disease and paving the way for cancer deaths to flourish. But perhaps a better question is why cancer continues to kill so many.

Even globally, cancer still comes in as the second leading cause of death behind heart disease, responsible for 26% of deaths worldwide.[vii] In the U.S., Americans have a 1 in 3 risk of developing cancer at some point in their lifetimes, along with a 1 in 5 risk of dying from the disease.[viii]

In early 2019, it was announced that cancer death rates in the U.S. declined 27% since 1991,[ix] a statistic that makes it seem as though we’re winning the “war on cancer.” But most of these declines can be attributed to reductions in smoking — and perhaps a limited measure of increased early detection and treatment — and are not a sign that conventional medicine’s model of surgerychemotherapy and/or radiation to treat cancer is, on the whole, working.

While death rates from certain cancer have declined, others have increased. Overall, cancer deaths in the U.S. in 2016 were similar to those in 1930[x] — despite all the “advances” in detection and treatment.

Changing the Way We Think About Cancer

It’s becoming increasingly clear that in order to conquer cancer, it’s necessary to change the way we think about it. Cancer is found in virtually all animals, suggesting it has evolutionary significance.[xi] It’s possible that cancer is an ancient survival program unmasked — even a process the body undergoes in order to survive nutrient deprivation and exposure to toxins.

Rather than being the result of an accumulation of DNA mutations that create rogue cells that multiply out of control, cancer could be cells that have flipped an epigenetic switch into survival mode in the form of a tumor. In the journal Physical Biology, researchers theorized:[xii]

“[C]ancer is an atavistic [primitive] condition that occurs when genetic or epigenetic malfunction unlocks an ancient ‘toolkit’ of pre-existing adaptations, re-establishing the dominance of an earlier layer of genes that controlled loose-knit colonies of only partially differentiated cells, similar to tumors.”

If this is true, it makes sense that conventional cancer treatments aimed to poison or “kill” the cancerous cells may only make the problem worse by creating an even more toxic environment, which could trigger the cancer to reach back into its “ancient toolkit” to find additional means of survival.

This explanation may be overly simplistic, as there are many factors that contribute to cancer, but there is evidence to suggest that natural substances and therapies that support the body’s overall health can be useful in the fight against cancer.

Nearly 1,000 Natural Substances Have Anti-Cancer Potential

GreenMedInfo has a database of 986 substances that have been researched as potential cancer prevention and treatment strategies. There are undoubtedly many more out there that have yet to be discovered. At the top of the list is curcumin, the active ingredient in the curry spice turmeric, which targets cancer stem cells while leaving normal stem cells unharmed.[xiii]

Another top contender is vitamin D, which you can get for free from adequate sun exposure. Higher vitamin D levels are not only known to lower your cancer risk but also to improve outcomes if you’ve already been diagnosed.[xiv] Fiberresveratrolsulforaphane and vitamin E — all substances you can get from your diet — also show anti-cancer promise, as does coffee, perhaps because it improves the body’s ability to efficiently repair DNA damage.[xv]

So if there was one silver lining to the news that cancer is now the leading cause of death in some countries, it would be that it’s a condition that has many promising natural avenues for prevention and treatment. Current conventional cancer treatments are failing, but that doesn’t mean cancer is unstoppable — it means it’s time to broaden our research into and usage of traditional therapies.

Many natural substances, like noni leaf,[xvi] have even been shown to work better than chemotherapy, highlighting why, if we’re going to win the war against cancer, we’re going to need to do it with nature on our side.

For more on how to naturally fight Cancer, visit the GreenMedInfo database on the subject.

Originally published: 2019-09-14

Article Updated: 2019-11-05

References

[i] The Lancet September 3, 2019

[ii] CNN September 3, 2019

[iii] Annals of Internal Medicine December 18, 2018

[iv] Annals of Internal Medicine December 18, 2018

[v] The Lancet September 3, 2019

[vi] Medscape September 3, 2019

[vii] Medscape September 3, 2019

[viii] American Cancer Society, Lifetime Risk of Developing or Dying From Cancer

[ix] CA: A Cancer Journal for Clinicians January 8, 2019

[x] CA: A Cancer Journal for Clinicians January 8, 2019

[xi] Front. Oncol., 10 January 2019

[xii] Physical Biology February 7, 2011

[xiii] Anticancer Res. 2015 Feb ;35(2):599-614.

[xiv] Br J Cancer. 2017 Mar 16. Epub 2017 Mar 16.

[xv] J Nutrigenet Nutrigenomics. 2015 ;8(4-6):174-84.

[xvi] Mol Cell Biochem. 2016 Apr 22. Epub 2016 Apr 22.


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Awareness

Man Fasts For 382 Days Straight & Loses 276 Pounds

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In Brief

  • The Facts:

    Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days. He remained completely healthy for the duration of the fast.

  • Reflect On:

    Today, it's firmly established in scientific literature that fasting can have tremendous benefits, if done correctly. It can also be used to treat a variety of diseases. Perhaps it's not emphasized because you can't make money off of not eating?

A study published in the Post Graduate Medical Journal in 1972 brought more attention to a gentleman by the name of Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days and, at the time the study was published, had since maintained his ordinary weight. In his case, “prolonged fasting had no ill effects.” Barbieri’s weight decreased from 456 to 180 pounds during the fast.

This isn’t the only example that’s available in the literature, it’s similar to an earlier patient prior to Barbieri who reduced his weight from 432 to 235 pounds during 350 days of intermittent fasting (Stewart, Fleming & Robertson, 1966). Researchers have also fasted patients for 256 days (Collison, 1967, 1971), 249 and 236 days (Thomson et al., 1966) as well as  210 days (Garnett et al., 1969; Runcie & Thomson, 1970), all of which are cited in the 1972 study.

Since the publication of this time, there are many documented examples of prolonged fasting done by highly obese people. Here’s one recent example of a man who fasted for 50 straight days, while being medically supervised and tested the whole time.

When you fast, your body switches from burning glucose, to burning fat. Fasting lowers insulin levels which allows the body to access its fat stores for energy. When you eat, food is converted into glucose and that’s what we usually burn. This is why fasting has become a therapeutic intervention for many people with type two diabetes, and more doctors, like Dr. Jason Fung, a Toronto Based nephrologist, are having great success with utilizing fasting as an appropriate and necessary health intervention. Fung has many great articles regarding the science of fasting, you can access them here if you’re interested in learning more. This article references some of the leading scientists in the field so you can learn more by looking them up as well.

The graph below depicts what happens to your protein while fasting. Interesting isn’t it? People often believe that if you fast, you will experience a tremendous amount of muscle loss during fasting, but that’s simply not true. This graph is from Kevin Hall, from the NIH in the book “Comparative Physiology of Fasting, Starvation, and Food Limitation.”

“It seems that there are always concerns about loss of muscle mass during fasting. I never get away from this question. No matter how many times I answer it, somebody always asks, “Doesn’t fasting burn your muscle?” Let me say straight up, NO.”  – source Dr. Jason Fung

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But what about Angus Barbieri? Obviously we’re not saying long term fasts for this long are healthy, obviously for many people they will probably be unhealthy and unsafe unless medically supervised. In  the 1972 study doctors measured a number of concentrations within the body. For example, plasma potassium concentrations over the first four months decreased systematically. As a result, they provided a very small daily dose that increased his potassium level. After another 10 weeks, no potassium was given, and from there on in until the end of the fast, plasma potassium levels remained normal. Cholesterol concentrations also remained around 230 mg/ 100 ml until 300 days of fasting, but increased to 370 mg/100 ml during refeeding.

Plasma magnesium levels decreased over the first few weeks of the fast but then went up and stabilized. This is interesting to note as there is nothing going into the body, yet levels still stabilized after the initial decrease.

Normal plasma magnesium concentrations, despite magnesium ‘depletion’ in muscle tissue, have been described (Drenick et al., 1969) during short-term fasting (1-3 months). The only other relevant report is a remark (Runcie & Thomson, 1970) that one patient who fasted 71 days had a normal plasma magnesium level of 2-2 mEq/l at the time when she developed latent tetany. The decrease in the plasma magnesium concentration of our patient was systematic and persistent.

Furthermore:

The excretion of sodium, potassium, calcium and inorganic phosphate decreased to low levels throughout the first 100 days, but thereafter the excretion of all four urinary constituents, as well as of magnesium, began to increase. During the subsequent 200 days sodium excretion, previously between 2 and 20 mEq daily, reached over 80 mEq/24 hr, potassium excretion increased to 30-40 mEq daily and calcium excretion increased from 10-30 mg/24 hr to 250- 280 mg/24 hr. Magnesium excretion (which was not measured during the first 100 days) reached 10 mEq/ 24 hr between Days 200-300. Phosphate excretion, which had decreased to under 200 mg/24 hr, also increased to around 800 mg/24 hr, even exceeding 1000 mg/24 hr on occasion. Peak excretions of all these constituents were seen around Day 300, after which there was a marginal decrease, but excretion remained high.

Obviously, this is an extreme fast and such fasts have only been tested on people of tremendous obesity, and it shows that people with a high body fat percentage have the ability to fast longer simply because their body has more stores to pull from.

The study concluded in 1972 that:

We have found, like Munro and colleagues (1970), that prolonged supervised therapeutic starvation of the obese patient can be a safe therapy, which is also effective if the ideal weight is reached. There is, however, likely to be occasionally a risk in some individuals, attributable to failures in different aspects of the adaptative response to fasting. Until the characteristics of these variations in response are identified, and shown to be capable of detection in their prodromal stages, extended starvation therapy must be used cautiously. In our view, unless unusual hypokalaemia is seen, potassium supplements are not mandatory. Xanthine oxidase inhibitors (or uricosuric agents) are not always necessary and could even be potentially harmful (British Medical Journal, 1971) perhaps particularly in the long-term fasting situation.

It’s almost 2020, and the literature, studies and research that’s been published since 1972 is vast. We’ve learned a lot more about it and if done correctly it can be extremely beneficial. Shot term fasting  presents minimal to no health risks, and so does long term fasting that lasts more than 24 hours, that is unless a person already has an underlying condition. That being said, it’s not easy to start. Most people are used to eating three meals plus snacks every single day, therefore they are never adapted to burning their fat stores, something that appears the human body was meant to do.

“Why is it that the normal diet is three meals a day plus snacks? It isn’t that it’s the healthiest eating pattern, now that’s my opinion but I think there is a lot of evidence to support that. There are a lot of pressures to have that eating pattern, there’s a lot of money involved. The food industry — are they going to make money from skipping breakfast like I did today? No, they’re going to lose money. If people fast, the food industry loses money. What about the pharmaceutical industries? What if people do some intermittent fasting, exercise periodically and are very healthy, is the pharmaceutical industry going to make any money on healthy people?” – Mark Mattson (source)

Fasting has also been shown to be effective as a therapeutic intervention for cancer. Fasting protects healthy cells while ‘starving’ cancer cells, it’s now being used as an intervention that’s being combined with chemotherapy. Fasting has also been shown to greatly reduce the risk of age related diseases like Parkinson’s Disease, and Alzheimer’s disease. Mark Mattson, one of the foremost researchers of the cellular and molecular mechanisms underlying multiple neurodegenerative disorders has shown through his work that fasting can have a tremendous effect on the brain, and can even reverse the symptoms of multiple neurodegenerative disorders. You can watch his interesting TED talk here.  Scientists have also discovered strong evidence that fasting is a natural intervention for triggering stem cell-based regeneration of an entire organ or system.

Fasting has actually long been known to have an effect on the brain. Children who suffer from epileptic seizures have fewer of them when placed on caloric restriction or fasts. It is believed that fasting helps kick-start protective measures that help counteract the overexcited signals that epileptic brains often exhibit.  (source)

The list goes on and is quite long. At the end of the day if you do your research, fasting, under proper medical supervision, can have tremendous health benefits that go far beyond what’s mentioned in the paragraph above. Every single study that has looked at fasting as a therapeutic intervention for several diseases has shown nothing but positive benefits. Even studies conducted regarding caloric restriction, something completely different than fasting, have shown promising results in all animal models.

According to a review of fasting literature conducted in 2003, “Calorie restriction (CR) extends life span and retards age-related chronic diseases in a variety of species, including rats, mice, fish, flies, worms, and yeast. The mechanism or mechanisms through which this occurs are unclear.” Since this study was published, a great amount of research has been conducted from many researchers, and the mechanisms are being discovered and have become more clear. If you want to further your research, apart from the names listed above, Dr. Valter Longo and his research is another great place to start.

The body has a tremendous amount of storage, and it hangs on to what it needs during a fast, and uses up ‘bad’ things, repairs damaged cells, and more. When you fast and deplete all your glycogen, your body is going to start using fat for energy, it’s going to use damaged cells for energy, it’s basically going to use all of the bad things first, before it gets to the good thing…Your body will not burn protein, as protein is not a fuel source while fasting.

I bring this up because it’s interesting to see what the body loses and hangs on to during a fast.

The Takeaway

The truth about fasting is that it’s not dangerous at all. Intermittent fasting and short term fasting can be done by just about anybody. From what we’ve seen with regards to prolonged fasting, it’s also not very dangerous when it comes to obese people doing it under medically supervised conditions. Theoretically, based on the science alone, any relatively healthy human being should be able to do a prolonged fast without any harmful consequences.

Obviously, prolonged fasts that are not medically supervised can be very detrimental. We are obviously not recommending this and you must do a lot of research and talk to your doctor if you’re interested in fasting, before trying it. For starters, a little bit of intermittent fasting here and there is a no brainer, and not dangerous at all if you have no underlying health conditions, but everybody’s body is different.

Fasting is making a lot of noise, and has been making a lot of noise within the health community, but it’s still not appropriately taught and used by the mainstream medical industry. Why is this so? The answer is simple, you can’t make money off of fasting.

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Thousands Gather To Mark The 33rd Anniversary of the National Childhood Vaccine Injury Act

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Government’s gift to Pharma of liability-free vaccines puts children’s health at risk states Children’s Health Defense (CHD) Chairman, Robert F. Kennedy, Jr.

Washington, DC – Thousands of advocates for children’s health will gather Thursday at the Vaccine Injury Epidemic (VIE) Event on the National Mall to mark the 33rd anniversary of National Childhood Vaccine Injury Act (NCVIA). The rally on Nov. 14th will spotlight the devastating impact NCVIA has had upon the state of children’s health. While children continue to be injured by vaccines daily, vaccine makers cannot be held accountable, thereby eliminating incentive for vaccine safety.

In his remarks, RFK, Jr. will address the ramifications of NCVIA and honor those whose lives have been impacted by vaccine injury and death. “It’s time to call out Congress, the CDC, and drug companies for allowing industry profits to trump children’s health,” said Kennedy. “There is no crisis more urgent than the epidemics of chronic health conditions among our nation’s children.”

Following NCVIA’s passage creating the National Vaccine Injury Compensation Program (NVICP), the childhood vaccine market sparked a gold rush for Pharma as more vaccines for routine childhood illnesses were developed. Coterminous with the burgeoning vaccine schedule, chronic health conditions in children rose from 12% to 54%. As vaccine industry profits grew to $50 billion annually, so did diagnoses of asthmaautismADHDallergiesanxietydepressiondiabetesobsessive-compulsive disorder and auto-immune diseases.  Here are the facts:

  • An HHS-funded study found only 1% of vaccine injuries are reported.
  • Despite NVICP’s high burden of proof and two out of three claims dismissed, over $4.2 billion has been paid for claims of vaccine injury or death.
  • The vaccine-injured find NVICP to be a years-long, litigious program with no jury, discovery and precedent. While medical bills mount, the injured are up against DOJ lawyers and HHS “Special Masters” that act as judges.
  • The Department of Justice and the NVICP are accused of fraud and obstruction of justice in the Autism Omnibus Proceeding.
  • The Institute of Medicine reports that the vaccine schedule as recommended has never been studied for long-term health effects despite independent research suggesting that unvaccinated children are healthier.
  • Modern medicine acknowledges that not everyone responds the same to vaccination and the “one size fits all” vaccine policy is not science based.

Children’s Health Defense’s created these six steps to vaccine safety. RFK, Jr. interviews are available upon request.

Sign up for free news and updates from Robert F. Kennedy, Jr. and the Children’s Health Defense. CHD is planning many strategies, including legal, in an effort to defend the health of our children and obtain justice for those already injured. Your support is essential to CHD’s successful mission.

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