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6 Scientific Reasons Why You Should Consider Not Smoking Weed On A Regular Basis

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It’s about being balanced and informed — exploring the upsides and downsides to cannabis.

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We’ve reported many times on the benefits of cannabis and its potential in medical applications, but what about the other side of the coin? Is it fair to say that cannabis carries no risks? When treating people and seeing positive benefits, what negative effects might also arise?

Marijuana has been the subject of intense debate over these last few years. Now legal in multiple states in the U.S. and authorized for medicinal purposes in even more, an increasing number of people are recognizing how arbitrary the line is between legal substances, like alcohol and cigarettes, and illegal ones, like pot — particularly since both alcohol and cigarettes have been shown to be extremely detrimental to the human body, while marijuana has not.

But what’s the reason for that? Obtaining marijuana to study has proven to be incredibly difficult for many researchers and therefore getting study results has been tough. 

So given that this natural substance is being used incredibly regularly, and yet doesn’t have a full scope of research behind it, we wanted to balance the scales and bring awareness to the fact that we should really think twice before using this substance daily for long periods of time, particularly under the misconception that it’s completely safe. The exception here would be if you have been prescribed it for a medical issue, but it’s still good to be aware of the risks.

This article came in response to a recent article that pointed out the many benefits of smoking cannabis without mentioning the risks involved. We feel any research that takes this approach is simply not beneficial to public awareness.

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To be clear, we’re not saying this plant should be illegal. It’s a plant, but we don’t know as much about its safety in daily and long term use as we’d like.

We Can’t Blindly View It as Safe

The massive support for the legalization of marijuana does have its drawbacks, as it leads people to believe that smoking marijuana is completely harmless, and even good for you. While it can be quite therapeutic for dulling pain or alleviating anxiety, so can alcohol and even prescription drugs. It seems like people are willing to see the downsides of those substances, but not do the same for cannabis.

In reality, research shows that smoking cannabis on a regular basis may still be hazardous. Many forget that the plant’s medicinal benefits are most readily accessed when it is ingested, not smoked.

It is admittedly encouraging to see all of the support for the legalization of marijuana, and all of the evidence emerging that smoking it is not as harmful as it was originally said to be. Marijuana legalization threatens many industries, so perhaps this is why it has taken so long to get the ball rolling.

That being said, more of a balance is needed: With so much support out there for marijuana, many people, especially young people, think there are no health consequences involved with smoking it. This is why we’ve decided to put together a list of seven reasons why you should really consider not smoking marijuana on a regular basis.

We are very well aware of the other side of cannabis (articles). For example, we recently published an article showing how cannabis helped cure a girl from cancer. She is one of many examples of people who have benefited immensely from cannabis, and it’s important to raise awareness about how paediatric cannabis is saving lives. You can read that article here.

We have also published a number of articles on why marijuana should be legal, as well as reported on the dozens of health benefits it boasts, from helping people with pain and epilepsy to replacing prescription drugs and more.

A number of studies have been published that show cannabis completely annihilates cancer. We are talking about decades of research (a simple Google search for scholarly articles on cannabis and cancer will show you this). Despite this fact, no human clinical trials have been conducted. Here is an article of a molecular biologist explaining how THC kills cancer.

It’s also important to mention that we are not against smoking weed, but based on the science, wish to convey that smoking weed regularly on a daily basis for a period that lasts more than a year could be harmful to your health.

“Really, the way to do these things, is to do them rarely so that your whole system can reassert itself and come to equilibrium. . . I think the real way to do cannabis is like, once a week. . . . “

— Terrence McKenna (source)

1. Most of the Medicinal Benefits of Marijuana Come From Different Methods of Ingestion, Not Smoking

Cannabinoids are any group of related compounds that include cannabinol and the active constituents of cannabis. They activate cannabinoid receptors that already exist in our body, and our bodies themselves actually produce compounds called endocannabinoids. These play a vital role in the human body, helping to create a healthy environment. Cannabinoids themselves also play an important role in immune system regeneration. Studies have shown that multiple constituents of cannabis can kill cancer cells, repair damaged brain cells, and more. The medical potential of this plant is truly miraculous, and it’s a shame that, despite decades of research showing undeniable results, like its ability to completely annihilate cancer, human clinical trials are only n0w commencing, and only in conjunction with chemotherapy drugs.

It’s important to know that, contrary to popular belief, smoking cannabis does not assist a great deal in treating disease within the body, as therapeutic levels cannot be reached through smoking. Creating oil from the plant or eating the plant is the best way to absorb cannabinoids. Smoking also alters the plant molecules; when cannabis is heated and burned it changes the chemical structure and acidity of the THC, which in turn negates its therapeutic value. The smoke from marijuana is toxic to the body, just as the smoke from any other substance would be. (I will discuss this further on in the article.) Furthermore, anytime you burn something and inhale it, you create oxidation within the body, which is unhealthy and can lead to many issues.

None of the health benefits of marijuana come from smoking it. When one says “cannabis cures cancer,” that doesn’t mean smoking it.

2. Heavy Marijuana Use Is Linked to Lower Dopamine Levels in the Brain

Researchers in the Department of Psychiatry at Columbia University have found that heavy smokers of marijuana could have a compromised dopamine system. When studying heavy smokers, they discovered lower dopamine release in one region of the brain, the striatum, which is the part of the brain that’s involved in working memory, impulsive behaviour, and attention. Several other studies have shown that addiction to other drugs can have similar effects on dopamine release, but this is the first evidence of its kind linking it to smoking cannabis.

The study examined 11 adults between the ages of 21 and 40 who were heavily dependent on cannabis against 12 healthy control subjects. 16 was the average age these individuals started smoking, and they had not stopped since.

Their press release outlines how the study was conducted, and the methods used:

Using positron emission tomography (PET) to track a radiolabelled molecule that binds to dopamine receptors in the brain, the scientists measured dopamine release in the striatum and its subregions, as well as in several brain regions outside the striatum, including the thalamus, midbrain, and globus pallidus. The cannabis users in this study stayed in the hospital for a week of abstinence to ensure that the PET scans were not measuring the acute effects of the drug. Participants were scanned before and after being given oral amphetamine to elicit dopamine release. The percent change in the binding of the radiotracer was taken as an indicator of capacity for dopamine release.

Compared with the controls, the cannabis users had significantly lower dopamine release in the striatum, including subregions involved in associative and sensorimotor learning, and in the globus pallidus. (source)(source)

Anissa Abi-Dargham, MD, a professor of psychiatry (in radiology) at Columbia University Medical Center (CUMC) and a lead author of the paper, said that “the bottom line is that long-term, heavy cannabis use may impair the dopaminergic system, which could have a variety of negative effects on learning and behaviour.” She also went on to emphasize that “these findings add to the growing body of research demonstrating the potentially adverse effects of cannabis, particularly in youth, at the same time that government policies and laws are increasing access and use.” 

Here is another study that was done two years ago that examines the same thing.

While correlation doesn’t always mean causation, it doesn’t mean we should disclude a possible connection. You have to make your own judgements, use the Bradford Hill Criteria, and look at a number of different studies from both sides.

Based on everything I’ve looked at, in my opinion, marijuana smoking does have some sort of effect on the brain in multiple ways, differing from person to person. Whether it’s ‘good’ or ‘bad,’ I’m not sure.

3. Smoking Marijuana Linked to Schizophrenia, But It’s Complicated

A number of studies have linked smoking cannabis to schizophrenia and psychosis. For example, a fairly recent study found that schizophrenia plays a role in a person’s likelihood of smoking weed. The study showed that genetic variants predicting schizophrenia  can also be used to predict a person’s tendency to smoke pot. The study showed that the same genes that predispose people to enjoying smoking cannabis might also predispose some to develop schizophrenia.

Lead author of the study, Robert Power, a genetic psychiatrist at King’s College London, stated that “there is a well-established link between people who use cannabis and schizophrenia.” 

Based on the scientific literature, it’s quite clear that there is a link, and at the same time, it seems to be clear that there isn’t. Mathew Hill, a cell biologist at the University of Calgary, tells us “the relationship is an ongoing debate in the scientific world — at least what the nature of the association is.” He also told us that “there is little evidence that, at a population level, cannabis use during adolescence is a primary contributing factor in the development of psychiatric illness.”

Just because some studies show an associative link does not mean there is one. At the same time, it doesn’t mean that there’s not one. This is the key! Some studies have shown that people who are in the early stages of schizophrenia that also smoke weed experience much larger brain modifications, like changes in white matter, compared to those who are not susceptible to schizophrenia.

What is clear is that people who already show signs of psychotic illness do experience adverse effects from smoking marijuana.

“There is definitely some kind of genetic basis to increased vulnerability to these adverse effects [in people with schizophrenia] that go beyond the correlational association discussed in this paper”

— Mathew Hill (source)

Again, it’s well-known that marijuana smoking by people with schizophrenia only worsens the disease, and a number of studies have shown that smoking marijuana actually increases the development of schizophrenia in those who might be genetically predisposed to it. So, if you have a family history of psychotic illness, smoking weed is something you might not want to partake in — or if you suffer from any other ailment that’s classified as a mental illness, for that matter.

One thing seems to be certain: Cannabis smoking does affect the brain in various ways, especially at crucial stages of brain development in adolescents.

Below is a great publication and a good summary to find out more information about this topic and why it’s so confusing. Again, this connection is still up for debate in the scientific world, and conflicting studies emerge every single year. This suggests that we simply don’t know enough and therefore should be careful with our habits.

So, the next time someone tells you that there is a link between schizophrenia and marijuana smoking, they’re wrong, and the next time someone tells you there is no link, they are also wrong! The best way to avoid any risk is to just avoid smoking marijuana altogether.

Clearing the smoke: What do we know about adolescent cannabis use and schizophrenia?

4. Smoking Marijuana Changes Your Brain

One recent study found that using marijuana daily for at least four years or longer can create certain anatomical changes in the brain. In this particular study, researchers used magnetic resonance imaging (MRI) to examine the brains of approximately 50 adults who were chronic marijuana users, compared to more than 60 people who didn’t use marijuana at all.

Researchers found that the people who had been smoking daily for at least four years had a smaller volume of gray matter in their orbitofrontal cortex, a phenomenon usually associated with addiction.

Lead author of the study Francesca Filbey, an associate professor in the School of Behavioural Brain Sciences at the University of Texas at Dallas, said that “not only is a change in structure, but there also tends to be a change reflected in the connectivity.” 

This is concerning, especially given the fact that grey matter is a major component of the central nervous system, and is associated with muscle control, sensory perception, memory, emotions, speech, decision making, and self-control. Loss of grey matter has also been implicated in a number of psychiatric disorders, including depression. There is a widespread reduction of grey matter in people who suffer depression, and while smoking marijuana may give temporary relief from depression, if someone doesn’t have it, they might feel depressed.  Marijuana could therefore contribute to depression, and the fact that people feel better when they smoke it might make them think that it helps with their depression — a dangerous pattern that could lead to continual grey matter depletion.

Again, there are a number of studies that show smoking marijuana can drastically change the brain, and also disrupt brain development.

Another recent study also found that marijuana smokers showed signs of damage in the corpus callous, which is a major white matter tract that connects the left side of the brain to the right side. However, the study did mention that the people examined could have had deviant brain structures prior to their use.

A study published a few years ago showed that people who constantly smoke marijuana have abnormal brain structures, but multiple studies have also shown that marijuana smokers show no difference in brain structure.

The list goes on and on, but one thing is for certain: The effects on the brain are unclear. Further research is needed to identify what smoking marijuana does to the brain, because the research is inconclusive and contradictory right now.

5. Smoking Harms the Lungs

Regardless of what you are smoking, smoke is harmful to lung health; this is a no-brainer. Whether it’s burning wood, tobacco, or marijuana, toxins and carcinogens are released from the combustion of materials. According to the American Lung Association:

Smoking marijuana clearly damages the human lung. Research shows that smoking marijuana causes chronic bronchitis and marijuana smoke has been shown to injure the cell linings of the large airways, which could explain why smoking marijuana leads to symptoms such as chronic cough, phlegm production, wheeze and acute bronchitis.

Again, marijuana smoke contains a similar range of harmful chemicals to that of tobacco smoke, and there is no shortage of research suggesting that long-term marijuana smoking leads to an increased risk of some respiratory problems. Despite this proven correlation, there is no association between smoking marijuana and lung cancer.

There are other alternatives to smoking marijuana, however, such as vaporizing or eating it.

6. Smoking Marijuana Can Increase Your Risk of Serious Cardiovascular Disorders

Multiple studies have connected smoking marijuana with potential cardiovascular disorders. One recent study published in the Journal of the American Heart Association noted:

Several striking cardiovascular complications following cannabis use raised the issue of the possible implication of cannabis in cardiovascular outcomes and the necessary national review. Despite the known underreporting, the rate of cannabis-related cardiovascular complications reported steadily rose during the past 5 years. Cardiovascular disorders represented 2% of the reports related to cannabis, classified into cardiac, cerebral, and peripheral complications. The majority consisted of acute coronary syndromes and peripheral arteriopathies. This result is consistent with previous findings and strengthens the idea that cannabis may be responsible for serious complications, in particular on the cardiovascular system.

Another recent study, published last year, concluded, “The evidence reported in this article point toward an undisputed linkage between cannabis consumption and potentially lethal cardiovascular complications.” (source)

Something to Consider:

Trouble Living Life Without Smoking

As with so many of our other favourite habits, smoking is a vice, and many people who smoke weed have difficulty stopping. Life often feels boring without it; smoking becomes a need and almost a mental addiction (we know there is no physical addiction involved.) And this is obviously problematic. It is rare to find someone who will be able to smoke a joint a couple of times a month, which is the amount many experts in the field of “mind-altering” drugs propose. It’s a substance that is abused and not given the respect it deserves, often being used as an escape — helping the person avoid asking themselves why they feel the need to alter their state so often. If you are a regular smoker and notice that without smoking for a night or two you become bored, anxious, or depressed, or have certain feelings come up because you don’t have a joint in your hand, it may be time to consider reducing your usage, and perhaps facing those negative feelings head on.

If you have a hard time going without smoking a joint, that in itself is a problem. Alternatively, if you are a regular marijuana smoker but can easily stop for weeks at a time, without any desire to smoke, perhaps you don’t have a problem. The main point here is that smoking shouldn’t be used to escape one’s problems because it just prolongs the process of facing them.

How Is it Grown?

These days, it’s hard to find ‘pure’ weed. Most people are unaware of the original source, and marijuana can be grown with harmful pesticides. In fact, it wasn’t after the legalization of marijuana in Colorado that authorities found dangerous pesticides in most of the marijuana being sold. You can read more about that here.

Where do the seeds come from? When Big Pharma takes over, what type of seeds will they be, and how will it be grown? Are they genetically modified? There are still many questions to be asked.

Concluding Comments

So what can you take from all of this? We simply don’t know enough about this plant to smoke it every day and expect no repercussions. A misleading culture surrounding marijuana’s safety has been established and it’s something we need to start questioning.

Many natural substances are harmful to you if you have too much of them or use them too regularly. Even natural medicinal botanicals are not meant to be used every day and come with risks.

Again, we hope this helps to clear up the negative stigma around marijuana, but also the blindly positive stigma around it, too. Balance is important in this case, and what we strive to convey in this report.

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Awareness

Cancer is Now the Leading Cause of Death

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In Brief

  • The Facts:

    Cancer has surpassed heart disease as the No. 1 cause of death in high-income countries, highlighting the urgent need to change the way this disease is prevented and treated.

  • Reflect On:

    Rather than being a random result of DNA mutations, it's possible that cancer could have much deeper roots that would be better targeted with natural therapies than toxicity.

This article was written by the Greenmedinfo Research Group, originally published by Greenmedinfo.com. Published here with permission. 

Cancer has dethroned heart disease to earn the nefarious title of leading cause of death in high-income and certain middle-income countries.[i] While heart disease remains the No. 1 cause of death globally among adults aged 35 to 70, in high-income countries, which included Saudi Arabia, United Arab Emirates, Canada and Sweden, cancer caused twice as many deaths as heart disease.[ii]

Some middle-income countries, which included the Philippines, Iran, South Africa, Colombia, China, Brazil, Malaysia, Turkey, Poland, Argentina and Chile, also saw cancer become the leading cause of death.

While the U.S. was not included in the new analysis, research published in 2018 suggested, “the United States is in the midst of an epidemiologic transition in the leading cause of death,” moving from heart disease to cancer.[iii]

That study, too, found that cancer was quickly outpacing heart disease as the top killer, with high-income counties transitioning first. In fact, while only 21% of U.S. counties had cancer as the leading cause of death in 2003, this rose to 41% in 2015.

“The shift to cancer as the leading cause of death was greatest in the highest-income counties,” the researchers explained,[iv] echoing the current study, which also cited “a transition in the predominant causes of deaths in middle-age” in high-income countries.[v]

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“The world is witnessing a new epidemiologic transition among the different categories of noncommunicable diseases, with CVD [cardiovascular disease] no longer the leading cause of death in HIC [high-income countries],” lead author Dr. Gilles Dagenais, professor emeritus, Laval University, Quebec, Canada, said in a statement.[vi]

Why is Cancer a Top Killer?

The study suggested cancer is rising to the top because heart disease is better treated in high-income countries, saving more lives from heart disease and paving the way for cancer deaths to flourish. But perhaps a better question is why cancer continues to kill so many.

Even globally, cancer still comes in as the second leading cause of death behind heart disease, responsible for 26% of deaths worldwide.[vii] In the U.S., Americans have a 1 in 3 risk of developing cancer at some point in their lifetimes, along with a 1 in 5 risk of dying from the disease.[viii]

In early 2019, it was announced that cancer death rates in the U.S. declined 27% since 1991,[ix] a statistic that makes it seem as though we’re winning the “war on cancer.” But most of these declines can be attributed to reductions in smoking — and perhaps a limited measure of increased early detection and treatment — and are not a sign that conventional medicine’s model of surgerychemotherapy and/or radiation to treat cancer is, on the whole, working.

While death rates from certain cancer have declined, others have increased. Overall, cancer deaths in the U.S. in 2016 were similar to those in 1930[x] — despite all the “advances” in detection and treatment.

Changing the Way We Think About Cancer

It’s becoming increasingly clear that in order to conquer cancer, it’s necessary to change the way we think about it. Cancer is found in virtually all animals, suggesting it has evolutionary significance.[xi] It’s possible that cancer is an ancient survival program unmasked — even a process the body undergoes in order to survive nutrient deprivation and exposure to toxins.

Rather than being the result of an accumulation of DNA mutations that create rogue cells that multiply out of control, cancer could be cells that have flipped an epigenetic switch into survival mode in the form of a tumor. In the journal Physical Biology, researchers theorized:[xii]

“[C]ancer is an atavistic [primitive] condition that occurs when genetic or epigenetic malfunction unlocks an ancient ‘toolkit’ of pre-existing adaptations, re-establishing the dominance of an earlier layer of genes that controlled loose-knit colonies of only partially differentiated cells, similar to tumors.”

If this is true, it makes sense that conventional cancer treatments aimed to poison or “kill” the cancerous cells may only make the problem worse by creating an even more toxic environment, which could trigger the cancer to reach back into its “ancient toolkit” to find additional means of survival.

This explanation may be overly simplistic, as there are many factors that contribute to cancer, but there is evidence to suggest that natural substances and therapies that support the body’s overall health can be useful in the fight against cancer.

Nearly 1,000 Natural Substances Have Anti-Cancer Potential

GreenMedInfo has a database of 986 substances that have been researched as potential cancer prevention and treatment strategies. There are undoubtedly many more out there that have yet to be discovered. At the top of the list is curcumin, the active ingredient in the curry spice turmeric, which targets cancer stem cells while leaving normal stem cells unharmed.[xiii]

Another top contender is vitamin D, which you can get for free from adequate sun exposure. Higher vitamin D levels are not only known to lower your cancer risk but also to improve outcomes if you’ve already been diagnosed.[xiv] Fiberresveratrolsulforaphane and vitamin E — all substances you can get from your diet — also show anti-cancer promise, as does coffee, perhaps because it improves the body’s ability to efficiently repair DNA damage.[xv]

So if there was one silver lining to the news that cancer is now the leading cause of death in some countries, it would be that it’s a condition that has many promising natural avenues for prevention and treatment. Current conventional cancer treatments are failing, but that doesn’t mean cancer is unstoppable — it means it’s time to broaden our research into and usage of traditional therapies.

Many natural substances, like noni leaf,[xvi] have even been shown to work better than chemotherapy, highlighting why, if we’re going to win the war against cancer, we’re going to need to do it with nature on our side.

For more on how to naturally fight Cancer, visit the GreenMedInfo database on the subject.

Originally published: 2019-09-14

Article Updated: 2019-11-05

References

[i] The Lancet September 3, 2019

[ii] CNN September 3, 2019

[iii] Annals of Internal Medicine December 18, 2018

[iv] Annals of Internal Medicine December 18, 2018

[v] The Lancet September 3, 2019

[vi] Medscape September 3, 2019

[vii] Medscape September 3, 2019

[viii] American Cancer Society, Lifetime Risk of Developing or Dying From Cancer

[ix] CA: A Cancer Journal for Clinicians January 8, 2019

[x] CA: A Cancer Journal for Clinicians January 8, 2019

[xi] Front. Oncol., 10 January 2019

[xii] Physical Biology February 7, 2011

[xiii] Anticancer Res. 2015 Feb ;35(2):599-614.

[xiv] Br J Cancer. 2017 Mar 16. Epub 2017 Mar 16.

[xv] J Nutrigenet Nutrigenomics. 2015 ;8(4-6):174-84.

[xvi] Mol Cell Biochem. 2016 Apr 22. Epub 2016 Apr 22.


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Awareness

Man Fasts For 382 Days Straight & Loses 276 Pounds

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In Brief

  • The Facts:

    Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days. He remained completely healthy for the duration of the fast.

  • Reflect On:

    Today, it's firmly established in scientific literature that fasting can have tremendous benefits, if done correctly. It can also be used to treat a variety of diseases. Perhaps it's not emphasized because you can't make money off of not eating?

A study published in the Post Graduate Medical Journal in 1972 brought more attention to a gentleman by the name of Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days and, at the time the study was published, had since maintained his ordinary weight. In his case, “prolonged fasting had no ill effects.” Barbieri’s weight decreased from 456 to 180 pounds during the fast.

This isn’t the only example that’s available in the literature, it’s similar to an earlier patient prior to Barbieri who reduced his weight from 432 to 235 pounds during 350 days of intermittent fasting (Stewart, Fleming & Robertson, 1966). Researchers have also fasted patients for 256 days (Collison, 1967, 1971), 249 and 236 days (Thomson et al., 1966) as well as  210 days (Garnett et al., 1969; Runcie & Thomson, 1970), all of which are cited in the 1972 study.

Since the publication of this time, there are many documented examples of prolonged fasting done by highly obese people. Here’s one recent example of a man who fasted for 50 straight days, while being medically supervised and tested the whole time.

When you fast, your body switches from burning glucose, to burning fat. Fasting lowers insulin levels which allows the body to access its fat stores for energy. When you eat, food is converted into glucose and that’s what we usually burn. This is why fasting has become a therapeutic intervention for many people with type two diabetes, and more doctors, like Dr. Jason Fung, a Toronto Based nephrologist, are having great success with utilizing fasting as an appropriate and necessary health intervention. Fung has many great articles regarding the science of fasting, you can access them here if you’re interested in learning more. This article references some of the leading scientists in the field so you can learn more by looking them up as well.

The graph below depicts what happens to your protein while fasting. Interesting isn’t it? People often believe that if you fast, you will experience a tremendous amount of muscle loss during fasting, but that’s simply not true. This graph is from Kevin Hall, from the NIH in the book “Comparative Physiology of Fasting, Starvation, and Food Limitation.”

“It seems that there are always concerns about loss of muscle mass during fasting. I never get away from this question. No matter how many times I answer it, somebody always asks, “Doesn’t fasting burn your muscle?” Let me say straight up, NO.”  – source Dr. Jason Fung

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But what about Angus Barbieri? Obviously we’re not saying long term fasts for this long are healthy, obviously for many people they will probably be unhealthy and unsafe unless medically supervised. In  the 1972 study doctors measured a number of concentrations within the body. For example, plasma potassium concentrations over the first four months decreased systematically. As a result, they provided a very small daily dose that increased his potassium level. After another 10 weeks, no potassium was given, and from there on in until the end of the fast, plasma potassium levels remained normal. Cholesterol concentrations also remained around 230 mg/ 100 ml until 300 days of fasting, but increased to 370 mg/100 ml during refeeding.

Plasma magnesium levels decreased over the first few weeks of the fast but then went up and stabilized. This is interesting to note as there is nothing going into the body, yet levels still stabilized after the initial decrease.

Normal plasma magnesium concentrations, despite magnesium ‘depletion’ in muscle tissue, have been described (Drenick et al., 1969) during short-term fasting (1-3 months). The only other relevant report is a remark (Runcie & Thomson, 1970) that one patient who fasted 71 days had a normal plasma magnesium level of 2-2 mEq/l at the time when she developed latent tetany. The decrease in the plasma magnesium concentration of our patient was systematic and persistent.

Furthermore:

The excretion of sodium, potassium, calcium and inorganic phosphate decreased to low levels throughout the first 100 days, but thereafter the excretion of all four urinary constituents, as well as of magnesium, began to increase. During the subsequent 200 days sodium excretion, previously between 2 and 20 mEq daily, reached over 80 mEq/24 hr, potassium excretion increased to 30-40 mEq daily and calcium excretion increased from 10-30 mg/24 hr to 250- 280 mg/24 hr. Magnesium excretion (which was not measured during the first 100 days) reached 10 mEq/ 24 hr between Days 200-300. Phosphate excretion, which had decreased to under 200 mg/24 hr, also increased to around 800 mg/24 hr, even exceeding 1000 mg/24 hr on occasion. Peak excretions of all these constituents were seen around Day 300, after which there was a marginal decrease, but excretion remained high.

Obviously, this is an extreme fast and such fasts have only been tested on people of tremendous obesity, and it shows that people with a high body fat percentage have the ability to fast longer simply because their body has more stores to pull from.

The study concluded in 1972 that:

We have found, like Munro and colleagues (1970), that prolonged supervised therapeutic starvation of the obese patient can be a safe therapy, which is also effective if the ideal weight is reached. There is, however, likely to be occasionally a risk in some individuals, attributable to failures in different aspects of the adaptative response to fasting. Until the characteristics of these variations in response are identified, and shown to be capable of detection in their prodromal stages, extended starvation therapy must be used cautiously. In our view, unless unusual hypokalaemia is seen, potassium supplements are not mandatory. Xanthine oxidase inhibitors (or uricosuric agents) are not always necessary and could even be potentially harmful (British Medical Journal, 1971) perhaps particularly in the long-term fasting situation.

It’s almost 2020, and the literature, studies and research that’s been published since 1972 is vast. We’ve learned a lot more about it and if done correctly it can be extremely beneficial. Shot term fasting  presents minimal to no health risks, and so does long term fasting that lasts more than 24 hours, that is unless a person already has an underlying condition. That being said, it’s not easy to start. Most people are used to eating three meals plus snacks every single day, therefore they are never adapted to burning their fat stores, something that appears the human body was meant to do.

“Why is it that the normal diet is three meals a day plus snacks? It isn’t that it’s the healthiest eating pattern, now that’s my opinion but I think there is a lot of evidence to support that. There are a lot of pressures to have that eating pattern, there’s a lot of money involved. The food industry — are they going to make money from skipping breakfast like I did today? No, they’re going to lose money. If people fast, the food industry loses money. What about the pharmaceutical industries? What if people do some intermittent fasting, exercise periodically and are very healthy, is the pharmaceutical industry going to make any money on healthy people?” – Mark Mattson (source)

Fasting has also been shown to be effective as a therapeutic intervention for cancer. Fasting protects healthy cells while ‘starving’ cancer cells, it’s now being used as an intervention that’s being combined with chemotherapy. Fasting has also been shown to greatly reduce the risk of age related diseases like Parkinson’s Disease, and Alzheimer’s disease. Mark Mattson, one of the foremost researchers of the cellular and molecular mechanisms underlying multiple neurodegenerative disorders has shown through his work that fasting can have a tremendous effect on the brain, and can even reverse the symptoms of multiple neurodegenerative disorders. You can watch his interesting TED talk here.  Scientists have also discovered strong evidence that fasting is a natural intervention for triggering stem cell-based regeneration of an entire organ or system.

Fasting has actually long been known to have an effect on the brain. Children who suffer from epileptic seizures have fewer of them when placed on caloric restriction or fasts. It is believed that fasting helps kick-start protective measures that help counteract the overexcited signals that epileptic brains often exhibit.  (source)

The list goes on and is quite long. At the end of the day if you do your research, fasting, under proper medical supervision, can have tremendous health benefits that go far beyond what’s mentioned in the paragraph above. Every single study that has looked at fasting as a therapeutic intervention for several diseases has shown nothing but positive benefits. Even studies conducted regarding caloric restriction, something completely different than fasting, have shown promising results in all animal models.

According to a review of fasting literature conducted in 2003, “Calorie restriction (CR) extends life span and retards age-related chronic diseases in a variety of species, including rats, mice, fish, flies, worms, and yeast. The mechanism or mechanisms through which this occurs are unclear.” Since this study was published, a great amount of research has been conducted from many researchers, and the mechanisms are being discovered and have become more clear. If you want to further your research, apart from the names listed above, Dr. Valter Longo and his research is another great place to start.

The body has a tremendous amount of storage, and it hangs on to what it needs during a fast, and uses up ‘bad’ things, repairs damaged cells, and more. When you fast and deplete all your glycogen, your body is going to start using fat for energy, it’s going to use damaged cells for energy, it’s basically going to use all of the bad things first, before it gets to the good thing…Your body will not burn protein, as protein is not a fuel source while fasting.

I bring this up because it’s interesting to see what the body loses and hangs on to during a fast.

The Takeaway

The truth about fasting is that it’s not dangerous at all. Intermittent fasting and short term fasting can be done by just about anybody. From what we’ve seen with regards to prolonged fasting, it’s also not very dangerous when it comes to obese people doing it under medically supervised conditions. Theoretically, based on the science alone, any relatively healthy human being should be able to do a prolonged fast without any harmful consequences.

Obviously, prolonged fasts that are not medically supervised can be very detrimental. We are obviously not recommending this and you must do a lot of research and talk to your doctor if you’re interested in fasting, before trying it. For starters, a little bit of intermittent fasting here and there is a no brainer, and not dangerous at all if you have no underlying health conditions, but everybody’s body is different.

Fasting is making a lot of noise, and has been making a lot of noise within the health community, but it’s still not appropriately taught and used by the mainstream medical industry. Why is this so? The answer is simple, you can’t make money off of fasting.

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Thousands Gather To Mark The 33rd Anniversary of the National Childhood Vaccine Injury Act

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Government’s gift to Pharma of liability-free vaccines puts children’s health at risk states Children’s Health Defense (CHD) Chairman, Robert F. Kennedy, Jr.

Washington, DC – Thousands of advocates for children’s health will gather Thursday at the Vaccine Injury Epidemic (VIE) Event on the National Mall to mark the 33rd anniversary of National Childhood Vaccine Injury Act (NCVIA). The rally on Nov. 14th will spotlight the devastating impact NCVIA has had upon the state of children’s health. While children continue to be injured by vaccines daily, vaccine makers cannot be held accountable, thereby eliminating incentive for vaccine safety.

In his remarks, RFK, Jr. will address the ramifications of NCVIA and honor those whose lives have been impacted by vaccine injury and death. “It’s time to call out Congress, the CDC, and drug companies for allowing industry profits to trump children’s health,” said Kennedy. “There is no crisis more urgent than the epidemics of chronic health conditions among our nation’s children.”

Following NCVIA’s passage creating the National Vaccine Injury Compensation Program (NVICP), the childhood vaccine market sparked a gold rush for Pharma as more vaccines for routine childhood illnesses were developed. Coterminous with the burgeoning vaccine schedule, chronic health conditions in children rose from 12% to 54%. As vaccine industry profits grew to $50 billion annually, so did diagnoses of asthmaautismADHDallergiesanxietydepressiondiabetesobsessive-compulsive disorder and auto-immune diseases.  Here are the facts:

  • An HHS-funded study found only 1% of vaccine injuries are reported.
  • Despite NVICP’s high burden of proof and two out of three claims dismissed, over $4.2 billion has been paid for claims of vaccine injury or death.
  • The vaccine-injured find NVICP to be a years-long, litigious program with no jury, discovery and precedent. While medical bills mount, the injured are up against DOJ lawyers and HHS “Special Masters” that act as judges.
  • The Department of Justice and the NVICP are accused of fraud and obstruction of justice in the Autism Omnibus Proceeding.
  • The Institute of Medicine reports that the vaccine schedule as recommended has never been studied for long-term health effects despite independent research suggesting that unvaccinated children are healthier.
  • Modern medicine acknowledges that not everyone responds the same to vaccination and the “one size fits all” vaccine policy is not science based.

Children’s Health Defense’s created these six steps to vaccine safety. RFK, Jr. interviews are available upon request.

Sign up for free news and updates from Robert F. Kennedy, Jr. and the Children’s Health Defense. CHD is planning many strategies, including legal, in an effort to defend the health of our children and obtain justice for those already injured. Your support is essential to CHD’s successful mission.

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