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Groundbreaking: China Study Links Immune Activation By Vaccination & Autism

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*This article is a summary of a larger article put together by J.B. Handley at Healthcare in America. It is a conglomeration of a wide body of recent research pieced together by a growing group of concerned scientists. For more information, please visit the website, vaccinepapers.org.

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A study out of China is the first to test the effects of immune activation by vaccination (hep B/BCG) on brain development in rats. Results indicate vaccines containing an aluminum adjuvant (i.e., hep B) spike cytokine levels in the hippocampus region of the brain, in particular the cytokine interleukin-6 (IL-6), the key cytokine known for its dysregulating effect on neuronal circuitry and the key cytokine implicated in autism.

History of research into immune activation and autism

Before we get into the China study, it’s important to understand all of the previous research leading up to it.

In 2006, late Caltech scientist Dr. Paul Patterson and his colleagues were among the first to discover the implications of maternal immune activation and brain development in offspring.

In an article published in the Engineering & Science journal, titled “Pregnancy, Immunity, Schizophrenia, and Autism,” Patterson wrote that “brain-immune conversation actually starts during the development of the embryo, where the state of the mother’s immune system can alter the growth of cells in the fetal brain.”

Patterson and his team built on the work led by Carlos Pardo at Johns Hopkins, which discovered “neural inflammation” in postmortem examination of brains of patients with autism. Strangely, these autistic patients did not die due to any infections that would have caused the inflammation.

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This research was the first to suggest “an ongoing, permanent immune-system activation in the brains of autistic people.” 

In 2007 Patterson took this research further, publishing a study that found the culprit of this chronic brain inflammation — cytokine interleukin-6 (IL-6).

Cytokines are cell signalling molecules that aid cell to cell communication, stimulating the movement of cells toward sites of inflammation, infection, and trauma.

Cytokine interleukin-6

Patterson found that IL-6 was critical for mediating the behavioural and transcriptional changes in the neurology of the rat offspring.

This study was replicated by Patterson in 2012, which was more autism-specific, and reached the same conclusion“These results indicate that [maternal immune activation] MIA yields male offspring with deficient social and communicative behaviour, as well as high levels of repetitive behaviours, all of which are hallmarks of autism.”

In 2014, the M.I.N.D. Institute at UC-Davis replicated Dr. Patterson’s work in rhesus monkeys and found the same results.

Another 2012 study from Neuroscience agreed with Patterson — Brain IL-6 elevation causes neuronal circuitry imbalances and mediates autism-like behaviours .

The next question, then, was what causes immune activation that would lead to increased levels of IL-6 in the brain?

Aluminum bioaccumulates in the brain

Aluminum compounds (Al hydroxide and Al phospate) are currently used in the hepatitis A, hepatitis B, diphtheria-tetanus-pertussis (DTaP, Tdap), Haemophilus influenzae type b (Hib), human papillomavirus (HPV), and pneumococcus (PCV) vaccines.

Aluminum adjuvant “activates” the immune system, which induces long term immunity to antigens in the vaccine.

Dr. Chris Shaw at the University of British Columbia did extensive research on injected aluminum in 2007 and 2009, and found “the results reported mirror previous work that has clearly demonstrated that aluminum, in both oral and injected forms, can be neurotoxic. Potential toxic mechanisms of action for aluminum may include enhancement of inflammation.”

Concerns about the limited understanding of aluminum toxicity were further questioned by Dr. Lucija Tomljenovic in this 2012 paper.

It is somewhat surprising to find that in spite of over 80 years of use, the safety of Al adjuvants continues to rest on assumptions rather than scientific evidence. For example, nothing is known about the toxicology and pharmacokinetics of Al adjuvants in infants and children.9 On the other hand, in adult humans long-term persistence of Al vaccine adjuvants can lead to cognitive dysfunction and autoimmunity.6,10 Yet, in spite of these observations children continue regularly to be exposed to much higher levels of Al adjuvants than adults, via routine childhood vaccination programmes.

In 2013, French scientists demonstrated that aluminum adjuvant, when injected into the body of a mouse, ended up in the brain one year later.

In 2015, another study from Université Paris Est Créteil (UPEC) in France further supported this new view of aluminum adjuvant, showing that Al makes its way to the brain slowly, where it stays there, possibly forever.

Last fall, results published in the journal Toxicology sealed the deal on Al adjuvant, revealing that low, consistent doses of Al were most dangerous of all for neurotoxic effects. Larger doses produced granulomas at injection sites, which prevented the Al from spreading. Smaller doses did not produce this effect, causing changes in the brain and behaviour.

The study authors stated that “the present study may suggest that aluminium adjuvant toxicokinetics and safety require reevaluation.”

And just last year, a study out of the Middle East looking at Alzheimer’s in rats found that aluminum produced a four-fold increase in IL-6 in the brain.

So we know that Al adjuvant causes on-going, increased levels of IL-6 in the brain. So what argument do the CDC and FDA use to justify aluminum being safe?

The difference between ingested Al and injected Al adjuvant

Currently, the FDA and CDC state that aluminum in vaccines is safe, based on this 2011 study.

This study erroneously concluded that aluminum from vaccines likely ends up in the body’s skeletal system. However, as the plethora of research previously mentioned shows, Al nanoparticles are not safely excreted or stored, they accumulate in the brain.

Another point to make here is that there is a difference between the aluminum discussed in the 2011 study (linked above) and the aluminum injected in vaccines. The CDC base their conclusions about Al safety on ingested, water-soluble aluminum salts, not the nanoparticle aluminum-hydroxide.

As Vaccine Papers explains, the two couldn’t be any further from the same.

Most vaccines contain aluminum, and aluminum is a proven neurotoxin, in amounts received from vaccines. Vaccines in combination can result in toxic aluminum overload. Even the aluminum in a single vaccine can be harmful because the aluminum is in a form that is more dangerous than ingested aluminum. Specifically, vaccine aluminum is in nanoparticulate form, which is harder for the body to eliminate, and because it is transported around the body differently than ingested aluminum.

It is natural and normal to ingest small doses of aluminum from food and water. Its not good for you, but the body has adequate defenses. Absorption of ingested Al is low, about 0.3%, so about 99.7% is eliminated in feces. Ingested aluminum is in ionic form (individual charged atoms), which is readily removed by the kidneys. Also, ionic aluminum is blocked from entering the brain by the blood brain barrier. The low absorption, rapid elimination by the kidneys and barrier to brain entry adequately protects the brain from aluminum.

However, nanoparticulate aluminum from vaccines cannot be removed by the kidneys. The particles are far too large to be filtered out by the kidneys. The Al nanoparticles do dissolve slowly (converting to ionic aluminum). But long before they can dissolve completely, the Al nanoparticles are “eaten” by immune system cells called macrophages. In other words, the particles wind up inside the macrophages. Once loaded with the Al nanoparticles, the macrophages spread aluminum as they travel through the body. This is dangerous, because the Al-loaded macrophages carry Al nanoparticles to tissues (e.g. the brain) that are damaged by very small amounts of aluminum.

China study links aluminum, IL-6, and autism

In 2015, Li et al. out of Sun Yat-Sen University published a groundbreaking study that tied all of the latter research together.

Li et al. were the first to test the effects of immune activation by vaccination on brain development. All other studies of immune activation before this had used pathological conditions to mimic infection and induce fever, and therefore concerns about the transferability of the data had been in question until this study came out.

The study looked at the effects of bacillus calmette-guerin (BCG) vaccine (for tuberculosis) and hepatitis B vaccine on brain development in infant rats.

J.B. Handley sums up the results:

“There were three different groups of rats:

  1. Rats receiving the BCG vaccine (not given in the U.S.)
  2. Rats receiving the Hepatitis B vaccine (given on day 1 of life in the U.S.)
  3. A control group with no vaccine.

The BCG vaccine does NOT contain aluminum adjuvant and the impact on the rat’s brains from BCG was actually positive!

The Hep B vaccine rats, however, showed the kind of immune activation event we are seeing in autism (high IL-6). This is biological proof of the link between a vaccine — given to a post-natal animal — inducing an immune activation event, including the cytokine marker for autism, IL-6. A scientific first.”

Vaccine Papers further detailed the implications of this study:

An important finding in the Li et al study is that some of the effects of hep B vaccine did not appear until age 8 weeks. This finding undermines claims of vaccine safety, which are almost always based on short-term outcomes of a few days or weeks. 8 weeks is a long time in rat development. 8 week old rats are almost fully mature adults. This suggests that adverse effects of vaccines may take years or decades to appear in humans, or can be life-long. This is consistent with what is known about immune activation and schizophrenia. Immune activation in the fetus can cause schizophrenia 20-30 years later.

The accumulating scientific evidence and the Li et al study in particular strongly suggest that early-life vaccination may cause mental illness. The mental illnesses would emerge years or decades after vaccination of an infant. Vaccines are likely contributing the the rise of mental illnesses in the USA over the last 25 years. The rise in mental illnesses in the USA is coincident with the dramatic increase in vaccination that started in the 1980s.

Is this the proof we’ve been waiting for?

As you’ve just read, there is a growing body of research that paints an undeniable link between immune activation and autism.

Source: HealthcareinAmerica.us

Aluminum adjuvants, given early and continually, stimulate immune activation event after immune activation event, raising levels of IL-6 in pre- and post-natal brains, leading to chronic inflammation and dysregulation of neuronal circuitry and the symptoms associated with autism.

Source: HealthcareinAmerica.us

Chronic brain inflammation would also explain why many autistic children develop enlarged foreheads. It would perhaps explain why these children feel the need to bang their heads against walls, or why they become frustrated easily.

What about the gastrointestinal disorders autistic children frequently experience? If you guessed aluminum was the culprit, you are correct.

Here is a study from Nature that explains how aluminum causes inflammation in the gut and impairs gut function.

Auto-immune disorders? Here is a groundbreaking 2013 study that explains how aluminum adjuvant causes a wide-spectrum of immune disorders.

What about the MMR vaccine?

Since the MMR vaccine does not contain aluminum, why then do parents talk about the MMR vaccine being a trigger for their child’s autism?

J.B. Handley puts it simply:

The MMR vaccine is the first live virus vaccine children receive (it’s typically given between age 12–18 months, most children have received 15–20 vaccines by then), and it’s a triple (measles, mumps, rubella) live virus.

For an immune system bathed in aluminum adjuvant and possibly already simmering with activation events, this triple dose might push a child right over the edge. This might explain the seizures (an extreme immune activation event) that sometimes follow the MMR appointment.

Only one ingredient (thimerosal) in one vaccine (MMR) has been studied in relation to autism in humans.

This picture sums the point up perfectly:

Source: HealthcareinAmerica.us

So where does all of this data leave us? Groundbreaking as all of this is, it is undeniable that there are many more questions waiting to be answered and more research needed.

Certainly we are in for a wild ride these next few years as the body of research for vaccine safety grows and as more people wake up to the fact that they’ve been lied to (get your popcorn ready).

For now, though, our only ally is to find our public voice, to spread the information to our circles, to involve ourselves in the discussions taking place online and in public, to let go of the emotional attacks and let the science boldly speak for itself. That is our moral responsibility.

The rest, I say, we leave to the adage about truth. It may have taken centuries and millions of lives to get here, and somewhere along the way we probably lost hope that it would ever arrive, but in the end the adage held true, that no matter how deep the lie or how ruthless the coverup, the truth always prevails.

*For more information concerning the science around vaccine safety, please visit vaccinepapers.org

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Awareness

Cancer is Now the Leading Cause of Death

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In Brief

  • The Facts:

    Cancer has surpassed heart disease as the No. 1 cause of death in high-income countries, highlighting the urgent need to change the way this disease is prevented and treated.

  • Reflect On:

    Rather than being a random result of DNA mutations, it's possible that cancer could have much deeper roots that would be better targeted with natural therapies than toxicity.

This article was written by the Greenmedinfo Research Group, originally published by Greenmedinfo.com. Published here with permission. 

Cancer has dethroned heart disease to earn the nefarious title of leading cause of death in high-income and certain middle-income countries.[i] While heart disease remains the No. 1 cause of death globally among adults aged 35 to 70, in high-income countries, which included Saudi Arabia, United Arab Emirates, Canada and Sweden, cancer caused twice as many deaths as heart disease.[ii]

Some middle-income countries, which included the Philippines, Iran, South Africa, Colombia, China, Brazil, Malaysia, Turkey, Poland, Argentina and Chile, also saw cancer become the leading cause of death.

While the U.S. was not included in the new analysis, research published in 2018 suggested, “the United States is in the midst of an epidemiologic transition in the leading cause of death,” moving from heart disease to cancer.[iii]

That study, too, found that cancer was quickly outpacing heart disease as the top killer, with high-income counties transitioning first. In fact, while only 21% of U.S. counties had cancer as the leading cause of death in 2003, this rose to 41% in 2015.

“The shift to cancer as the leading cause of death was greatest in the highest-income counties,” the researchers explained,[iv] echoing the current study, which also cited “a transition in the predominant causes of deaths in middle-age” in high-income countries.[v]

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“The world is witnessing a new epidemiologic transition among the different categories of noncommunicable diseases, with CVD [cardiovascular disease] no longer the leading cause of death in HIC [high-income countries],” lead author Dr. Gilles Dagenais, professor emeritus, Laval University, Quebec, Canada, said in a statement.[vi]

Why is Cancer a Top Killer?

The study suggested cancer is rising to the top because heart disease is better treated in high-income countries, saving more lives from heart disease and paving the way for cancer deaths to flourish. But perhaps a better question is why cancer continues to kill so many.

Even globally, cancer still comes in as the second leading cause of death behind heart disease, responsible for 26% of deaths worldwide.[vii] In the U.S., Americans have a 1 in 3 risk of developing cancer at some point in their lifetimes, along with a 1 in 5 risk of dying from the disease.[viii]

In early 2019, it was announced that cancer death rates in the U.S. declined 27% since 1991,[ix] a statistic that makes it seem as though we’re winning the “war on cancer.” But most of these declines can be attributed to reductions in smoking — and perhaps a limited measure of increased early detection and treatment — and are not a sign that conventional medicine’s model of surgerychemotherapy and/or radiation to treat cancer is, on the whole, working.

While death rates from certain cancer have declined, others have increased. Overall, cancer deaths in the U.S. in 2016 were similar to those in 1930[x] — despite all the “advances” in detection and treatment.

Changing the Way We Think About Cancer

It’s becoming increasingly clear that in order to conquer cancer, it’s necessary to change the way we think about it. Cancer is found in virtually all animals, suggesting it has evolutionary significance.[xi] It’s possible that cancer is an ancient survival program unmasked — even a process the body undergoes in order to survive nutrient deprivation and exposure to toxins.

Rather than being the result of an accumulation of DNA mutations that create rogue cells that multiply out of control, cancer could be cells that have flipped an epigenetic switch into survival mode in the form of a tumor. In the journal Physical Biology, researchers theorized:[xii]

“[C]ancer is an atavistic [primitive] condition that occurs when genetic or epigenetic malfunction unlocks an ancient ‘toolkit’ of pre-existing adaptations, re-establishing the dominance of an earlier layer of genes that controlled loose-knit colonies of only partially differentiated cells, similar to tumors.”

If this is true, it makes sense that conventional cancer treatments aimed to poison or “kill” the cancerous cells may only make the problem worse by creating an even more toxic environment, which could trigger the cancer to reach back into its “ancient toolkit” to find additional means of survival.

This explanation may be overly simplistic, as there are many factors that contribute to cancer, but there is evidence to suggest that natural substances and therapies that support the body’s overall health can be useful in the fight against cancer.

Nearly 1,000 Natural Substances Have Anti-Cancer Potential

GreenMedInfo has a database of 986 substances that have been researched as potential cancer prevention and treatment strategies. There are undoubtedly many more out there that have yet to be discovered. At the top of the list is curcumin, the active ingredient in the curry spice turmeric, which targets cancer stem cells while leaving normal stem cells unharmed.[xiii]

Another top contender is vitamin D, which you can get for free from adequate sun exposure. Higher vitamin D levels are not only known to lower your cancer risk but also to improve outcomes if you’ve already been diagnosed.[xiv] Fiberresveratrolsulforaphane and vitamin E — all substances you can get from your diet — also show anti-cancer promise, as does coffee, perhaps because it improves the body’s ability to efficiently repair DNA damage.[xv]

So if there was one silver lining to the news that cancer is now the leading cause of death in some countries, it would be that it’s a condition that has many promising natural avenues for prevention and treatment. Current conventional cancer treatments are failing, but that doesn’t mean cancer is unstoppable — it means it’s time to broaden our research into and usage of traditional therapies.

Many natural substances, like noni leaf,[xvi] have even been shown to work better than chemotherapy, highlighting why, if we’re going to win the war against cancer, we’re going to need to do it with nature on our side.

For more on how to naturally fight Cancer, visit the GreenMedInfo database on the subject.

Originally published: 2019-09-14

Article Updated: 2019-11-05

References

[i] The Lancet September 3, 2019

[ii] CNN September 3, 2019

[iii] Annals of Internal Medicine December 18, 2018

[iv] Annals of Internal Medicine December 18, 2018

[v] The Lancet September 3, 2019

[vi] Medscape September 3, 2019

[vii] Medscape September 3, 2019

[viii] American Cancer Society, Lifetime Risk of Developing or Dying From Cancer

[ix] CA: A Cancer Journal for Clinicians January 8, 2019

[x] CA: A Cancer Journal for Clinicians January 8, 2019

[xi] Front. Oncol., 10 January 2019

[xii] Physical Biology February 7, 2011

[xiii] Anticancer Res. 2015 Feb ;35(2):599-614.

[xiv] Br J Cancer. 2017 Mar 16. Epub 2017 Mar 16.

[xv] J Nutrigenet Nutrigenomics. 2015 ;8(4-6):174-84.

[xvi] Mol Cell Biochem. 2016 Apr 22. Epub 2016 Apr 22.


For more info from Greenmedinfo, you can join their newsletter by clicking here.

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Man Fasts For 382 Days Straight & Loses 276 Pounds

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In Brief

  • The Facts:

    Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days. He remained completely healthy for the duration of the fast.

  • Reflect On:

    Today, it's firmly established in scientific literature that fasting can have tremendous benefits, if done correctly. It can also be used to treat a variety of diseases. Perhaps it's not emphasized because you can't make money off of not eating?

A study published in the Post Graduate Medical Journal in 1972 brought more attention to a gentleman by the name of Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days and, at the time the study was published, had since maintained his ordinary weight. In his case, “prolonged fasting had no ill effects.” Barbieri’s weight decreased from 456 to 180 pounds during the fast.

This isn’t the only example that’s available in the literature, it’s similar to an earlier patient prior to Barbieri who reduced his weight from 432 to 235 pounds during 350 days of intermittent fasting (Stewart, Fleming & Robertson, 1966). Researchers have also fasted patients for 256 days (Collison, 1967, 1971), 249 and 236 days (Thomson et al., 1966) as well as  210 days (Garnett et al., 1969; Runcie & Thomson, 1970), all of which are cited in the 1972 study.

Since the publication of this time, there are many documented examples of prolonged fasting done by highly obese people. Here’s one recent example of a man who fasted for 50 straight days, while being medically supervised and tested the whole time.

When you fast, your body switches from burning glucose, to burning fat. Fasting lowers insulin levels which allows the body to access its fat stores for energy. When you eat, food is converted into glucose and that’s what we usually burn. This is why fasting has become a therapeutic intervention for many people with type two diabetes, and more doctors, like Dr. Jason Fung, a Toronto Based nephrologist, are having great success with utilizing fasting as an appropriate and necessary health intervention. Fung has many great articles regarding the science of fasting, you can access them here if you’re interested in learning more. This article references some of the leading scientists in the field so you can learn more by looking them up as well.

The graph below depicts what happens to your protein while fasting. Interesting isn’t it? People often believe that if you fast, you will experience a tremendous amount of muscle loss during fasting, but that’s simply not true. This graph is from Kevin Hall, from the NIH in the book “Comparative Physiology of Fasting, Starvation, and Food Limitation.”

“It seems that there are always concerns about loss of muscle mass during fasting. I never get away from this question. No matter how many times I answer it, somebody always asks, “Doesn’t fasting burn your muscle?” Let me say straight up, NO.”  – source Dr. Jason Fung

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But what about Angus Barbieri? Obviously we’re not saying long term fasts for this long are healthy, obviously for many people they will probably be unhealthy and unsafe unless medically supervised. In  the 1972 study doctors measured a number of concentrations within the body. For example, plasma potassium concentrations over the first four months decreased systematically. As a result, they provided a very small daily dose that increased his potassium level. After another 10 weeks, no potassium was given, and from there on in until the end of the fast, plasma potassium levels remained normal. Cholesterol concentrations also remained around 230 mg/ 100 ml until 300 days of fasting, but increased to 370 mg/100 ml during refeeding.

Plasma magnesium levels decreased over the first few weeks of the fast but then went up and stabilized. This is interesting to note as there is nothing going into the body, yet levels still stabilized after the initial decrease.

Normal plasma magnesium concentrations, despite magnesium ‘depletion’ in muscle tissue, have been described (Drenick et al., 1969) during short-term fasting (1-3 months). The only other relevant report is a remark (Runcie & Thomson, 1970) that one patient who fasted 71 days had a normal plasma magnesium level of 2-2 mEq/l at the time when she developed latent tetany. The decrease in the plasma magnesium concentration of our patient was systematic and persistent.

Furthermore:

The excretion of sodium, potassium, calcium and inorganic phosphate decreased to low levels throughout the first 100 days, but thereafter the excretion of all four urinary constituents, as well as of magnesium, began to increase. During the subsequent 200 days sodium excretion, previously between 2 and 20 mEq daily, reached over 80 mEq/24 hr, potassium excretion increased to 30-40 mEq daily and calcium excretion increased from 10-30 mg/24 hr to 250- 280 mg/24 hr. Magnesium excretion (which was not measured during the first 100 days) reached 10 mEq/ 24 hr between Days 200-300. Phosphate excretion, which had decreased to under 200 mg/24 hr, also increased to around 800 mg/24 hr, even exceeding 1000 mg/24 hr on occasion. Peak excretions of all these constituents were seen around Day 300, after which there was a marginal decrease, but excretion remained high.

Obviously, this is an extreme fast and such fasts have only been tested on people of tremendous obesity, and it shows that people with a high body fat percentage have the ability to fast longer simply because their body has more stores to pull from.

The study concluded in 1972 that:

We have found, like Munro and colleagues (1970), that prolonged supervised therapeutic starvation of the obese patient can be a safe therapy, which is also effective if the ideal weight is reached. There is, however, likely to be occasionally a risk in some individuals, attributable to failures in different aspects of the adaptative response to fasting. Until the characteristics of these variations in response are identified, and shown to be capable of detection in their prodromal stages, extended starvation therapy must be used cautiously. In our view, unless unusual hypokalaemia is seen, potassium supplements are not mandatory. Xanthine oxidase inhibitors (or uricosuric agents) are not always necessary and could even be potentially harmful (British Medical Journal, 1971) perhaps particularly in the long-term fasting situation.

It’s almost 2020, and the literature, studies and research that’s been published since 1972 is vast. We’ve learned a lot more about it and if done correctly it can be extremely beneficial. Shot term fasting  presents minimal to no health risks, and so does long term fasting that lasts more than 24 hours, that is unless a person already has an underlying condition. That being said, it’s not easy to start. Most people are used to eating three meals plus snacks every single day, therefore they are never adapted to burning their fat stores, something that appears the human body was meant to do.

“Why is it that the normal diet is three meals a day plus snacks? It isn’t that it’s the healthiest eating pattern, now that’s my opinion but I think there is a lot of evidence to support that. There are a lot of pressures to have that eating pattern, there’s a lot of money involved. The food industry — are they going to make money from skipping breakfast like I did today? No, they’re going to lose money. If people fast, the food industry loses money. What about the pharmaceutical industries? What if people do some intermittent fasting, exercise periodically and are very healthy, is the pharmaceutical industry going to make any money on healthy people?” – Mark Mattson (source)

Fasting has also been shown to be effective as a therapeutic intervention for cancer. Fasting protects healthy cells while ‘starving’ cancer cells, it’s now being used as an intervention that’s being combined with chemotherapy. Fasting has also been shown to greatly reduce the risk of age related diseases like Parkinson’s Disease, and Alzheimer’s disease. Mark Mattson, one of the foremost researchers of the cellular and molecular mechanisms underlying multiple neurodegenerative disorders has shown through his work that fasting can have a tremendous effect on the brain, and can even reverse the symptoms of multiple neurodegenerative disorders. You can watch his interesting TED talk here.  Scientists have also discovered strong evidence that fasting is a natural intervention for triggering stem cell-based regeneration of an entire organ or system.

Fasting has actually long been known to have an effect on the brain. Children who suffer from epileptic seizures have fewer of them when placed on caloric restriction or fasts. It is believed that fasting helps kick-start protective measures that help counteract the overexcited signals that epileptic brains often exhibit.  (source)

The list goes on and is quite long. At the end of the day if you do your research, fasting, under proper medical supervision, can have tremendous health benefits that go far beyond what’s mentioned in the paragraph above. Every single study that has looked at fasting as a therapeutic intervention for several diseases has shown nothing but positive benefits. Even studies conducted regarding caloric restriction, something completely different than fasting, have shown promising results in all animal models.

According to a review of fasting literature conducted in 2003, “Calorie restriction (CR) extends life span and retards age-related chronic diseases in a variety of species, including rats, mice, fish, flies, worms, and yeast. The mechanism or mechanisms through which this occurs are unclear.” Since this study was published, a great amount of research has been conducted from many researchers, and the mechanisms are being discovered and have become more clear. If you want to further your research, apart from the names listed above, Dr. Valter Longo and his research is another great place to start.

The body has a tremendous amount of storage, and it hangs on to what it needs during a fast, and uses up ‘bad’ things, repairs damaged cells, and more. When you fast and deplete all your glycogen, your body is going to start using fat for energy, it’s going to use damaged cells for energy, it’s basically going to use all of the bad things first, before it gets to the good thing…Your body will not burn protein, as protein is not a fuel source while fasting.

I bring this up because it’s interesting to see what the body loses and hangs on to during a fast.

The Takeaway

The truth about fasting is that it’s not dangerous at all. Intermittent fasting and short term fasting can be done by just about anybody. From what we’ve seen with regards to prolonged fasting, it’s also not very dangerous when it comes to obese people doing it under medically supervised conditions. Theoretically, based on the science alone, any relatively healthy human being should be able to do a prolonged fast without any harmful consequences.

Obviously, prolonged fasts that are not medically supervised can be very detrimental. We are obviously not recommending this and you must do a lot of research and talk to your doctor if you’re interested in fasting, before trying it. For starters, a little bit of intermittent fasting here and there is a no brainer, and not dangerous at all if you have no underlying health conditions, but everybody’s body is different.

Fasting is making a lot of noise, and has been making a lot of noise within the health community, but it’s still not appropriately taught and used by the mainstream medical industry. Why is this so? The answer is simple, you can’t make money off of fasting.

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Thousands Gather To Mark The 33rd Anniversary of the National Childhood Vaccine Injury Act

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Government’s gift to Pharma of liability-free vaccines puts children’s health at risk states Children’s Health Defense (CHD) Chairman, Robert F. Kennedy, Jr.

Washington, DC – Thousands of advocates for children’s health will gather Thursday at the Vaccine Injury Epidemic (VIE) Event on the National Mall to mark the 33rd anniversary of National Childhood Vaccine Injury Act (NCVIA). The rally on Nov. 14th will spotlight the devastating impact NCVIA has had upon the state of children’s health. While children continue to be injured by vaccines daily, vaccine makers cannot be held accountable, thereby eliminating incentive for vaccine safety.

In his remarks, RFK, Jr. will address the ramifications of NCVIA and honor those whose lives have been impacted by vaccine injury and death. “It’s time to call out Congress, the CDC, and drug companies for allowing industry profits to trump children’s health,” said Kennedy. “There is no crisis more urgent than the epidemics of chronic health conditions among our nation’s children.”

Following NCVIA’s passage creating the National Vaccine Injury Compensation Program (NVICP), the childhood vaccine market sparked a gold rush for Pharma as more vaccines for routine childhood illnesses were developed. Coterminous with the burgeoning vaccine schedule, chronic health conditions in children rose from 12% to 54%. As vaccine industry profits grew to $50 billion annually, so did diagnoses of asthmaautismADHDallergiesanxietydepressiondiabetesobsessive-compulsive disorder and auto-immune diseases.  Here are the facts:

  • An HHS-funded study found only 1% of vaccine injuries are reported.
  • Despite NVICP’s high burden of proof and two out of three claims dismissed, over $4.2 billion has been paid for claims of vaccine injury or death.
  • The vaccine-injured find NVICP to be a years-long, litigious program with no jury, discovery and precedent. While medical bills mount, the injured are up against DOJ lawyers and HHS “Special Masters” that act as judges.
  • The Department of Justice and the NVICP are accused of fraud and obstruction of justice in the Autism Omnibus Proceeding.
  • The Institute of Medicine reports that the vaccine schedule as recommended has never been studied for long-term health effects despite independent research suggesting that unvaccinated children are healthier.
  • Modern medicine acknowledges that not everyone responds the same to vaccination and the “one size fits all” vaccine policy is not science based.

Children’s Health Defense’s created these six steps to vaccine safety. RFK, Jr. interviews are available upon request.

Sign up for free news and updates from Robert F. Kennedy, Jr. and the Children’s Health Defense. CHD is planning many strategies, including legal, in an effort to defend the health of our children and obtain justice for those already injured. Your support is essential to CHD’s successful mission.

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