Scientists have been aware of aluminum’s neurotoxicity for decades. Although aluminum’s apologists have tried to shroud the metal’s risks in manufactured controversy, a growing number of reports by researchers in the United Kingdom, France, Canada, Israel, the U.S. and elsewhere has furnished substantive evidence linking aluminum to neuropathology, including the epidemics of Alzheimer’s disease (AD) and autism spectrum disorder (ASD).
Dr. Christopher Exley—one of the world’s leading experts on aluminum toxicity—has shown that chronic intoxication with myriad forms of this “ubiquitous and omnipresent metal” is exacting a high price on human health. Dr. Exley and other aluminum experts such as molecular biologist Dr. Lucija Tomljenovic have confirmed that aluminum readily and actively traverses the blood-brain barrier to selectively accumulate in brain tissues, where it induces unwelcome changes in brain biochemistry. As Dr. Exley has noted, “There are no ‘normal’ levels of brain aluminum,” meaning that “its presence in brain tissue, at any level, could be construed as abnormal” [emphasis added].
Documenting Aluminum in the ASD Brain
In light of the fact that even minute amounts of aluminum can have adverse neurological consequences, Dr. Exley’s newest paper—which reports on the first-ever study of aluminum in ASD brain tissue—is groundbreaking. Published in the Journal of Trace Elements in Medicine and Biology, the paper documents some of the highest values for aluminum in human brain tissue ever recorded. Using a two-pronged study design (see box), the researchers measured and characterized aluminum deposits in brain tissues from five to ten ASD donors, most of whom died in their teens or twenties.
|Study DesignQuantitative component: First, the investigators used graphite furnace atomic absorption spectrometry (GRAAS) to measure aluminum content in frozen brain tissue samples. Frozen tissue was available from one female donor (age 44) and four male donors (ages 15, 22, 33 and 50) who, when alive, had a confirmed ASD diagnosis. The researchers quantified aluminum levels in 59 tissue samples representing five different areas of the brain (frontal, parietal, occipital, temporal and hippocampal).
Qualitative component: Using a technique called fluorescence microscopy, the researchers visualized aluminum deposits according to their presence (a) insideversus outside the brain cells and (b) in the two types of brain tissue (grey matterversus white matter). For this component, fixed tissue samples were available for the same five donors plus an additional five donors diagnosed with ASD, including two females (ages 13 and 29) and three males (ages 14, 22 and 29).
What the research team found was startling. The study’s quantitative arm documented “consistently high” aluminum levels representing “some of the highest values for brain aluminum content ever measured in healthy or diseased tissues.” Specifically:
- All five individuals had at least one brain tissue with a “pathologically significant” level of aluminum, defined as greater than or equal to 3.00 micrograms per gram of dry brain weight (μg/g dry wt). (Dr. Exley and colleagues developed categories to classify aluminum-related pathology after conducting other brain studies, wherein older adults who died healthy had less than 1 μg/g dry wt of brain aluminum.)
- Roughly two-thirds (67%) of all the tissue samples displayed a pathologically significant aluminum content.
- Aluminum levels were particularly high in the male brains, including in a 15-year-old boy with ASD who had the study’s single highest brain aluminum measurement (22.11 μg/g dry wt)—many times higher than the pathologically significant threshold and far greater than levels that might be considered as acceptable even for an aged adult.
- Some of the elevated aluminum levels rivaled the very high levels historically reported in victims of dialysis encephalopathy syndrome (a serious iatrogenic disorder resulting from aluminum-containing dialysis solutions).
The study’s qualitative findings were equally concerning:
- Across the 10 donors, the investigators identified 150 aluminum deposits. All 10 donors had aluminum deposits in at least one tissue.
- Aluminum deposits were markedly more prevalent in males than females (129 deposits in seven males, averaging over 18 deposits each, versus 21 deposits in three females, for an average of 7).
- In males, most aluminum deposits were inside cells (80/129), whereas aluminum deposits in females were primarily extracellular (15/21). The majority of intracellular aluminum was inside non-neuronal cells (microglia and astrocytes).
- Aluminum was present in both grey matter (88 deposits) and white matter (62 deposits). (The brain’s grey matter serves to process information, while the white matter provides connectivity.)
- The researchers also identified aluminum-loaded lymphocytes in the meninges (the layers of protective tissue that surround the brain and spinal cord) and in similar inflammatory cells in the vasculature, furnishing evidence of aluminum’s entry into the brain “via immune cells circulating in the blood and lymph” and perhaps explaining how youth with ASD came to acquire such shockingly high levels of brain aluminum.
The Importance of Glial Cells
There are three broad categories of non-neuronal (glial) cells, including astrocytes (which support neuronal signaling), oligodendrocytes (which create myelin) and microglia (responsible for repairing damage). In discussing their results, Dr. Exley’s team comments that the intracellular location of most of the aluminum in these non-neuronal cells was the “standout observation” for ASD.
Unlike other brain cells, the microglia (which represent about 10% of brain cells) are dedicated immune cells. Microglia also play a key role in the process known as synaptic pruning that takes place during vital phases of cognitive development in early childhood as well as adolescence, continuing into the late 20s. This process, which some observers have likened to “neural spring cleaning,” allows the maturing brain to shed “weak or redundant [neuronal] connections.” Given this and other important microglial functions, the microglia have attracted considerable research attention as key players in brain disease, including autism. (Astrocytes also have implications for autism, given the role of astrocyte dysfunction in seizures—a condition that is frequently comorbid with ASD.) A pivotal review article published in 2017 observes that “microglia are now known to be active participants in brain function and dysfunction” and notes that “aberrant [synaptic] pruning during critical developmental periods could contribute to neurodevelopmental disorders.” Evidence suggesting that the microglia are dysfunctional in ASD includes findings from postmortem ASD brain studies showing “altered microglial counts, morphology, and neuronal interaction” as well as altered expression of microglia-specific genes.
It is clear to many researchers that environmental factors can alter microglia function, negatively affecting brain development and synaptic connectivity; when this occurs during important developmental periods, there may be “consequences throughout life.” Aluminum exposure undoubtedly constitutes a dangerous environmental exposure, and Dr. Exley observes that “microglia heavily loaded with aluminum…will inevitably be compromised.”
The Most Pervasive Exposure to Aluminum
The study’s results strongly suggest that aluminum is entering the brain in ASD via cells that have become loaded up with aluminum in the periphery. Where is the aluminum coming from? One of the most pervasive routes of modern-day exposure to neurotoxic aluminum is via aluminum adjuvants in vaccines. (Vaccine manufacturers use aluminum adjuvants to intensify the vaccine recipient’s immune response.) Elsewhere, Dr. Exley has described the “migratory capabilities” of aluminum-based adjuvants “at sites distant to the injection site,” including the brain.
In the ASD brain paper, Dr. Exley and coauthors point out that the “burgeoning” use of aluminum-adjuvant-containing childhood vaccines “has been directly correlated with increasing prevalence of ASD.” A 2011 study by Lucija Tomljenovic and Christopher Shaw confirms that aluminum-containing vaccines are having crippling neurological consequences. Their analysis shows that children from countries where ASD prevalence is highest have the highest exposure to aluminum from vaccines; moreover, children’s increased exposure to aluminum adjuvants over the two decades starting in the 1990s significantly correlates with the increase in ASD prevalence in the U.S. Counting the shots now pushed during pregnancy, highly vaccinated American children may receive up to 73 total vaccine doses by age 18, including multiple rounds of injected aluminum.
U.S. vaccines containing one or more aluminum adjuvants*
|Infection or Illness||Vaccine||Manufacturer or Brand Name|
|Diphtheria-tetanus or tetanus-diphtheria||DT
Tenivac; Mass Biologics
|Diphtheria-tetanus-pertussis or tetanus-diphtheria-pertussis||DTaP
|Haemophilus Influenzaetype B||Hib||PedvaxHib|
|Hepatitis A||Hep A||Havrix; Vaqta|
|Hepatitis B||Hep B||Engerix-B; Recombivax|
|Human papillomavirus||HPV||Gardasil; Gardasil 9|
|Streptococcus pneumoniae||Pneumococcal||PCV 13/Prevnar 13|
Hep A/Hep B
* Aluminum hydroxide, aluminum phosphate, aluminum salts, amorphous aluminum hydroxyphosphate sulfate (AAHS), potassium aluminum sulfate
Crucially, Dr. Exley and coauthors note that what “discriminates [their] data from other analyses of brain aluminum in other diseases is the age of the ASD donors” [emphasis added]. The extreme levels of aluminum found in the brains of the study’s teenage donors have alarming implications for the entire generation of highly aluminum-vaccinated children. Moreover, Dr. Exley’s other research has consistently shown that aluminum is the most significant contributing factor to Alzheimer’s disease. Given that it is no longer unheard of to see Alzheimer’s being diagnosed in people who are in their 20s, 30s, or 40s, it is not unreasonable to worry that a catastrophic new wave of AD may be about to compound children’s already heavy burden of ASD and other neurological disorders. Recognizing the risks, numerous researchers have called for a halt to the use of aluminum salts in vaccines. The powerful results of this study underscore the urgency of heeding this plea as well as eliminating exposure to other sources of neurotoxic aluminum.
How X-Ray Mammography Is Accelerating The Epidemic of Cancer
Article written by Sayer Ji, Founder of Greenmedinfo LLC, posted here with permission.
While a growing body of research now suggests that x-ray mammography is causing more harm than good in the millions of women who subject themselves to breast screenings, annually, without knowledge of their true health risks, the primary focus has been on the harms associated with over-diagnosis and over-treatment, and not the radiobiological dangers of the procedure itself.
In 2006, a paper published in the British Journal of Radiobiology, titled “Enhanced biological effectiveness of low energy X-rays and implications for the UK breast screening programme,” revealed the type of radiation used in x-ray-based breast screenings is much more carcinogenic than previously believed:
Recent radiobiological studies have provided compelling evidence that the low energy X-rays as used in mammography are approximately four times – butpossibly as much as six times – more effective in causing mutational damage than higher energy X-rays. Since current radiation risk estimates are based on the effects of high energy gamma radiation, this implies that the risks of radiation-induced breast cancers for mammography X-rays are underestimated by the same factor.
In other words, the radiation risk model used to determine whether the benefit of breast screenings in asymptomatic women outweighs their harm, underestimates the risk of mammography-induced breast and related cancers by between 4-600%.
The authors continued
Risk estimates for radiation-induced cancer – principally derived from the atomic bomb survivor study (ABSS) – are based on the effects of high energy gamma-rays and thus the implication is that the risks of radiation-induced breast cancer arising from mammography may be higher than that assumed based on standard risks estimates.
This is not the only study to demonstrate mammography X-rays are more carcinogenic than atomic bomb spectrum radiation. There is also an extensive amount of data on the downside of x-ray mammography.
Sadly, even if one uses the outdated radiation risk model (which underestimates the harm done),* the weight of the scientific evidence (as determined by the work of The Cochrane Collaboration) actually shows that breast screenings are in all likelihood not doing any net good in those who undergo them.
In a 2009 Cochrane Database Systematic Review,** also known as the Gøtzsche and Nielsen’s Cochrane Review, titled “Screening for breast cancer with mammography,” the authors revealed the tenuous statistical justifications for mass breast screenings:
Screening led to 30% overdiagnosis and overtreatment, or an absolute risk increase of 0.5%. This means that for every 2000 women invited for screening throughout 10 years, one will have her life prolonged and 10 healthy women, who would not have been diagnosed if there had not been screening, will be treated unnecessarily. Furthermore, more than 200 women will experience important psychological distress for many months because of false positive findings. It is thus not clear whether screening does more good than harm.
In this review, the basis for estimating unnecessary treatment was the 35% increased risk of surgery among women who underwent screenings. Many of the surgeries, in fact, were the result of women being diagnosed with ductal carcinoma in situ (DCIS), a “cancer” that would not exists as a clinically relevant entity were it not for the fact that it is detectable through x-ray mammography. DCIS, in the vast majority of cases, has no palpable lesion or symptoms, and some experts believe it should be completely reclassified as a non-cancerous condition.
A more recent study published in the British Medical Journal in 2011 titled, “Possible net harms of breast cancer screening: updated modeling of Forrest report,” not only confirmed the Gøtzsche and Nielsen’s Cochrane Review findings, but found the situation likely worse:
This analysis supports the claim that the introduction of breast cancer screening might have caused net harm for up to 10 years after the start of screening.
So, let’s assume that these reviews are correct, and at the very least, the screenings are not doing any good, and at worst, causing more harm than good. The salient question, however, is how much more harm than good? If we consider that, according to data from Journal of the National Cancer Institute (2011), a mammogram uses 4 mSv of radiation vs. the .02 mSv of your average chest x-ray (which is 200 times more radiation), and then, we factor in the 4-600% higher genotoxicity/carcinogenicity associated with the specific “low-energy” wavelengths used in mammography, it is highly possible that beyond the epidemic of over-diagnosis and over-treatment, mammograms are planting seeds of radiation-induced cancer within the breasts of millions of women.***
With the advent of non-ionizing radiation based diagnostic technologies, such as thermography, it has become vitally important that patients educate themselves about the alternatives to x-ray mammography that already exist. Until then, we must use our good sense – and research like this – to inform our decisions, and as far as the unintended adverse effects of radiation go, erring on the side of caution whenever possible.
*This discrepancy in radiation risk models/estimates follows from two fundamental problems: 1) the older risk model was based on higher-energy radiation emissions, such as are given off from atomic bomb blasts 2) it was a crude model, developed before the discovery of DNA and a full understanding of radiotoxicity/genotoxicity.
** Keep in mind that the Cochrane Database Review is at the top of the “food chain” of truth, in the highly touted “evidence-based model” of conventional medicine. Cochrane Database Reviews are produced by The Cochrane Collaboration, which is internationally recognized as the benchmark for high quality, evidence-based information concerning the effectiveness (or lack thereof) of common health care interventions. The organization, comprised of over 28,000 dedicated people from over 100 countries, prides itself on being an “independent” source of information, and historically has not been afraid to point out the corrupting influence of industry, which increasingly co-opts the biomedical research and publishing fields.
***The low-energy wavelengths cause double strand breaks within the DNA of susceptible cells, which the cell can not repair. Through time these mutations result in “neoplastic transformation”; radiation has the ability to induce a cancerous phenotype within formerly healthy cells that has cancer stem cell-like (CSC) properties.
Sayer Ji is founder of Greenmedinfo.com, a reviewer at the International Journal of Human Nutrition and Functional Medicine, Co-founder and CEO of Systome Biomed, Vice Chairman of the Board of the National Health Federation, Steering Committee Member of the Global Non-GMO Foundation.
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Why Water Fluoridation Is A Forced Experiment That Needs To Stop
The United States stands almost entirely alone among developed nations in adding industrial silicofluorides to its drinking water—imposing the community-wide measure without informed consent. Globally, roughly 5% of the population consumes chemically fluoridated water, but more people in the U.S. drink fluoride-adulterated water than in all other countries combined. Within the U.S., just under a third (30%) of local water supplies are not fluoridated; these municipalities have either held the practice at bay since fluoridation’s inception or have won hard-fought battles to halt water fluoridation.
The fluoride chemicals added to drinking water are unprocessed toxic wasteproducts—captured pollutants from Florida’s phosphate fertilizer industry or unregulated chemical imports from China. The chemicals undergo no purification before being dumped into drinking water and often harbor significant levels of arsenic and other heavy metal contamination; one researcher describes this unavoidable contamination as a “regulatory blind spotthat jeopardizes any safe use of fluoride additives.”
Dozens of studies and reviews—including in top-tier journals such as The Lancet—have shown that fluoride is neurotoxic and lowers children’s IQ. Fluoride is also associated with a variety of other health risks in both children and adults. However, U.S. officialdom persists in making hollow claims that water fluoridation is safe and beneficial, choosing to ignore even its own research! A multimillion-dollar longitudinal study published in Environmental Health Perspectives in September, 2017, for example, was largely funded by the National Institutes of Health and National Institute of Environmental Health Sciences—and the seminal study revealed a strong relationship between fluoride exposure in pregnant women and lowered cognitive function in offspring. Considered in the context of other research, the study’s implications are, according to the nonprofit Fluoride Action Network, “enormous”—“a cannon shot across the bow of the 80 year old practice of artificial fluoridation.”
A little history
During World War II, fluoride (a compound formed from the chemical element fluorine) came into large-scale production and use as part of the Manhattan Project. According to declassified government documents summarized by Project Censored, Manhattan Project scientists discovered early on that fluoride was a “leading health hazard to bomb program workers and surrounding communities.” In order to stave off lawsuits, government scientists “embarked on a campaign to calm the social panic about fluoride…by promoting its usefulness in preventing tooth decay.”
To prop up its “exaggerated claims of reduction in tooth decay,” government researchers began carrying out a series of poorly designed and fatally flawed community trials of water fluoridation in a handful of U.S. cities in the mid-1940s. In a critique decades later, a University of California-Davis statistician characterized these early agenda-driven fluoridation trials as “especially rich in fallacies, improper design, invalid use of statistical methods, omissions of contrary data, and just plain muddleheadedness and hebetude.” As one example, a 15-year trial launched in Grand Rapids, Michigan in 1945 used a nearby city as a non-fluoridated control, but after the control city began fluoridating its own water supply five years into the study, the design switched from a comparison with the non-fluoridated community to a before-and-after assessment of Grand Rapids. Fluoridation’s proponents admitted that this change substantially “compromised” the quality of the study.
In 1950, well before any of the community trials could reach any conclusions about the systemic health effects of long-term fluoride ingestion, the U.S. Public Health Service (USPHS) endorsed water fluoridation as official public health policy, strongly encouraging communities across the country to adopt the unproven measure for dental caries prevention. Describing this astonishingly non-evidence-based step as “the Great Fluoridation Gamble,” the authors of the 2010 book, The Case Against Fluoride, argue that:
“Not only was safety not demonstrated in anything approaching a comprehensive and scientific study, but also a large number of studies implicating fluoride’s impact on both the bones and the thyroid gland were ignored or downplayed” (p. 86).
In 2015, Newsweek magazine not only agreed that the scientific rationale for putting fluoride in drinking water was not as “clear-cut” as once thought but also shared the “shocking” finding of a more recent Cochrane Collaboration review, namely, that there is no evidence to support the use of fluoride in drinking water.
Bad science and powerful politics
The authors of The Case Against Fluoride persuasively argue that “bad science” and “powerful politics” are primary factors explaining why government agencies continue to defend the indefensible practice of water fluoridation, despite abundant evidence that it is unsafe both developmentally and after “a lifetime of exposure to uncontrolled doses.” Comparable to Robert F. Kennedy, Jr.’s book, Thimerosal: Let the Science Speak, which summarizes studies that the Centers for Disease Control and Prevention (CDC) and “credulous journalists swear don’t exist,” The Case Against Fluoride is an extensively referenced tour de force, pulling together hundreds of studies showing evidence of fluoride-related harm.
The research assembled by the book’s authors includes studies on fluoride biochemistry; cancer; fluoride’s effects on the brain, endocrine system and bones; and dental fluorosis. With regard to the latter, public health agencies like to define dental fluorosis as a purely cosmetic issue involving “changes in the appearance of tooth enamel,” but the International Academy of Oral Medicine & Toxicology (IAOMT)—a global network of dentists, health professionals and scientists dedicated to science-based biological dentistry—describes the damaged enamel and mottled and brittle teeth that characterize dental fluorosis as “the first visible sign of fluoride toxicity.”
The important 2017 study that showed decrements in IQ following fluoride exposure during pregnancy is far from the only research sounding the alarm about fluoride’s adverse developmental effects. In his 2017 volume, Pregnancy and Fluoride Do Not Mix, John D. MacArthur pulls together hundreds of studies linking fluoride to premature birth and impaired neurological development (93 studies), preelampsia (77 studies) and autism (110 studies). The book points out that rates of premature birth are “unusually high” in the United States. At the other end of the lifespan, MacArthur observes that death rates in the ten most fluoridated U.S. states are 5% to 26% higher than in the ten least fluoridated states, with triple the rate of Alzheimer’s disease. A 2006 report by the National Research Council warned that exposure to fluoride might increase the risk of developing Alzheimer’s.
The word is out
Pregnancy and Fluoride Do Not Mix shows that the Institute of Medicine, National Research Council, Harvard’s National Scientific Council on the Developing Child, Environmental Protection Agency (EPA) and National Toxicology Program all are well aware of the substantial evidence of fluoride’s developmental neurotoxicity, yet no action has been taken to warn pregnant women. Instead, scientists with integrity, legal professionals and the public increasingly are taking matters into their own hands. A Citizens Petitionsubmitted in 2016 to the EPA under the Toxic Substances Control Act requested that the EPA “exercise its authority to prohibit the purposeful addition of fluoridation chemicals to U.S. water supplies.” This request—the focus of a lawsuit to be argued in court later in 2019—poses a landmark challenge to the dangerous practice of water fluoridation and has the potential to end one of the most significant chemical assaults on our children’s developing bodies and brains.
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Doctor Explains Why She Never Recommends The ‘Ketogenic Diet’
- The Facts:
Michelle McMacken, an internal medicine physician, shares why she does not recommend the ketogenic diet at all for her patients.
- Reflect On:
Why are we so quick to jump on bandwagons, especially when it comes to health topics, without ever really looking into it deeper? The ketogenic diet has many health benefits, but it may not be as healthy as many think.
The ketogenic diet has gained a tremendous amount of popularity over the past few years, and it’s become a trend that many people are adopting without doing their own research first. We’ve written multiple articles on the ketogenic diet, a diet that promotes a high fat/low carb intake in order to prolong the production of ketone bodies in one’s blood. The release of these ketone bodies happens when we fast and deplete our glucose reserves, which develop from eating carbohydrates that turn into sugar. One can prolong the production of these ketones by sticking to a low carbohydrate/high fat diet, and essentially run off of fat instead of their glucose reserves.
The ketogenic diet is being used as an intervention for cancer, and there are multiple studies showing how ketones can actually kill cancer. It’s becoming well known that cancer cells cannot efficiently process ketone bodies for energy. Essentially, the cell starves itself, and ketones help slow the proliferation of tumor cells. Dietary ketones have been shown to completely halt metastasis. For example, a study titled “The Ketogenic Diet & Hyperbaric Oxygen Therapy Prolong Survival in Mice with Systemic Metastatic Cancer” explains how it’s already known that the ketogenic diet elevates blood ketones and has been shown to slow cancer progression in both animals and humans. The study also revealed that the ketogenic diet “significantly decreased blood glucose, slowed tumor growth, and increased mean survival time by 56.8 percent in mice with systemic metastatic cancer.”
Fasting (when you fast you produce ketones) is also being used for cancer intervention, seizure prevention (epilepsy), and as a potential therapy for alzheimer’s disease, parkinson’s disease, and other neurodegenerative disorders.
A TEDx talk given by Mark Mattson, the current Chief of the Laboratory of Neuroscience, at the National Institute on Aging goes into detail about fasting, ketones, and how beneficial it is for the brain. He is also a professor of Neuroscience at Johns Hopkins University and one of the foremost researchers of the cellular and molecular mechanisms underlying multiple neurodegenerative disorders.
In 1923, scientist Otto Warburg hypothesized that cancer was caused by a metabolic process whereby cancer cells fuel their growth “by swallowing up enormous amounts of glucose [blood sugar] and breaking it down without oxygen.” Coined the Warburg Effect, the theory was considered controversial at the time, but the past few decades have sparked new interest in it and oncologists now use the dependence on glucose that cancer cells have to locate tumours within a patient’s body.
Warburg made his discovery around the same time the ketogenic diet was found to be beneficial for epilepsy. Studies have shown that when the body produces ketones, they form a protective barrier around the brain, which is why more and more paediatricians are recommending the diet for children with epilepsy. It has a huge success rate, but since fasting is neither marketable nor profitable, it receives little mainstream attention.
All of these are specific interventions for certain diseases, and they can be healthy. On a personal level, I believe fasting a few times a month is extremely healthy and can be very beneficial for the body. All of the studies in human and animal models show nothing but benefits. Keep in mind that while you fast, you also get the benefits of ketones.
This is far different from continuing on with a no carb, high fat diet where you are constantly producing ketones and burning fat. It doesn’t seem normal unless you have to do it for a specific intervention, like cancer. Despite the potential health benefits, the ‘ketogenic diet’ has become a fad with potential dangers that people should also be aware of.
The Five Reasons
Below is a list of points regarding the ketogenic diet from Michelle McMacken, an internal medicine physician, Assistant Professor of Medicine at the NYU School of Medicine, and Director of Bellevue Hospital Weight Management Clinic.
I came across these via her Instagram, which make it clear she does not support the diet:
1. That we know of, no population in history has ever thrived on a very-low-carb/high-fat diet. There is exactly zero scientific evidence that a keto diet is conducive to longevity & longstanding vitality – unlike a plant-centric diet, the foundation of the longest-lived people on earth.
2. A keto diet may cause short-term weight loss, but possibly at a serious price. A 2010 review found that low-carb, animal-based diets increased cardiovascular death by 14%, cancer death by 28%, & all-cause mortality by 23%- trends confirmed in other large studies.
3. A keto diet hasn’t been shown to prevent, control, or reverse type 2 diabetes in the long run. Avoiding carbs will temporarily lower your blood sugar if you have diabetes. But this simply masks the underlying problem, which is insulin resistance – ie. glucose in our blood can’t enter our cells & the liver overproduces sugar. This is NOT the fault of carbs from healthy foods – whole grains, legumes, fruit, or even starchy vegetables. In fact, a high-carb, high-fiber, plant-based diet is exceptionally protective against diabetes & can actually REVERSE insulin resistance & lower diabetes complications. In contrast, low-carb diets can promote diabetes over time, as they foster inflammation & fat buildup in our cells, causing insulin resistance.
4. Keto diet research is in its infancy, focusing on short-term blood results & body weight – not actual rates of disease or death. And some findings are concerning. LDL cholesterol levels tend to rise (or at best, stay the same) on keto diets. An overwhelming wealth of research shows that the higher the LDL, the higher the risk of cardiovascular disease.
5. A keto diet is low in refined grains & added sugar, which is great. But it also can be low in phytonutrients, antioxidants, & fiber, all of which have profound benefits, and it forbids some of the most powerfully health-promoting foods on earth – whole grains, legumes, & many fruits. To me, that’s just not good medicine.
It’s great to see the world becoming more health conscious, it’s one of multiple contributing factors in raising our vibrational frequency, and feeling more ‘alive.’ That being said, a lot of ‘fads’ seem to pop up in this field, which are coupled with a great misunderstanding of how these specific diets, like the ketogenic diet, is supposed to be used. At the end of the day, balance is key, and it’s best to incorporate more organic fruits and vegetables in your diet, and completely cut out all processed foods, and substances like high fructose corn syrup etc. Being healthy is not hard, and it’s not complicated. If you’re going to incorporate a specific diet into your lifestyle, just make sure it’s not one that’s specifically designed to combat certain diseases, like the ketogenic diet.
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