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Magnesium: The Safe First Line of Defense for Clinical Depression

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This article was written by Ali Le Vere for Greenmedinfo.com. It’s republished here with their permission. For more information from Greenmedinfo, you can sign up for the newsletter here.

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The science supporting the efficacy of magnesium for major depression and other psychiatric disorders, testing for magnesium deficiency, and which forms and dosages are most effective.

Depression, a life-threatening psychiatric disorder, lies at the confluence of biochemical, hormonal, immunological, and neurodegenerative variables, which intersect to generate the pro-inflammatory state with which depression is associated. A major public health issue, depression is estimated to become one of the top three contributors to the global burden of diseases within a few years. Not only does depression consume a sizable portion of health care expenditures, but it is considered to be an independent risk factor for metabolic, cardiovascular, and neuropsychiatric disorders (1).

Current treatments are predicated upon a misguided serotonin theory of depression, and are accompanied by a laundry list of deleterious side effects ranging from sexual dysfunction to homicidality (2, 3, 4). Antidepressant medications likewise significantly increase the risk of all-cause mortality, or death from any cause, as well as heart disease, leading researchers to deem this class of pharmaceuticals as harmful to the general population (5). This, in combination with data indicating that antidepressants are clinically equivalent to placebo, render them an unfavorable option (6), especially considering that they offer little in the way of resolving the root cause.

Magnesium: The Miracle Mineral

Rather than resorting to psychotropic drugs, it would be prudent to explore whether magnesium (Mg) supplementation improves depression, since this essential mineral is implicated in the pathophysiology of this disorder. Magnesium may be indeed branded as miraculous given its essentiality as a cofactor to over three hundred enzymatic reactions (7). It is second only to potassium in terms of the predominant intracellular cations, or ions residing in cells that harbor a positive charge (7).

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Magnesium is fundamentally involved in protein production, synthesis of nucleic acids, cell growth and division, and maintenance of the delicate electrolyte composition of our cells (7). It also imparts stability to the membranes of the energy factories of our cells called mitochondria (7). As articulated by researchers, “The physiological consequences of these biochemical activities include Mg’s central roles in the control of neuronal activity, cardiac excitability, neuromuscular transmission, muscular contraction, vasomotor tone, and blood pressure” (7).

The biological effects of magnesium are widespread. When deficient, magnesium is correlated with systemic inflammation. Not only does magnesium sufficiency promote cardiovascular health, relaxing the smooth muscles that comprise blood vessels and preventing high levels of vascular resistance that cause hypertension, but it also plays a role in musculoskeletal health and prevents sarcopenia, osteoporosis, and fractures (8). Magnesium is essential to regulation of sleep (9) and vitamin D metabolism (10) as well as neural plasticity and cognitive function.

However, food processing and industrial agriculture, including monoculture crop practices and the use of magnesium-devoid fertilizers, have led to soil erosion and depletion of magnesium content in our food (7). Magnesium is likewise removed from most drinking water supplies, rendering magnesium deficiency an inevitability (11). As such, our daily intake of magnesium has steadily declined from 500 milligrams (mg) per day to 175 mg per day (7). The nutrient-poor, energy-dense dietary patterns which have come to dominate the industrialized landscape are also insufficient in the fiber-rich fruits and vegetables which contain magnesium.

Animal Studies Propose a Role for Magnesium in Depression

Preliminary animal studies pointed to a role of magnesium in depression, as depletion of magnesium in the diet of mice lead to enhanced depression- and anxiety-related behavior such as increased immobility time in the forced swim test (12). In the forced swim test, a common assay for examining depression-like behavior in rodents, the animal is confined to a container filled with water and observed as it attempts to escape. The time in which the animal exhibits immobility is used as a barometer of despair, indicating that the animal has succumbed to a fate of drowning (1).

This model is confirmed by studies showing that administering substances with antidepressant properties such as Hypericum perforatum, also known as St. John’s Wort, can significantly decrease the time the animal spends without locomotor activity (12). In addition, the time the animal spends immobilized is influenced by many of the factors that are changed as a consequence of depression in humans, such as drug-withdrawal-induced anhedonia, impaired sleep, and altered food consumption (1).

Human Studies Confirm the Role of Magnesium in Depression

There is a paucity of research on the influence of specific micronutrients in depression and results are inconsistent, but several studies have revealed low serum magnesium in this mood disorder. It is well-documented, for example, that dietary magnesium deficiency in conjunction with stress can lead to neuropathologies and symptoms of psychiatric disorders. Researchers echo this sentiment, stating that, “Dietary deficiencies of magnesium, coupled with excess calcium and stress may cause many cases of other related symptoms including agitation, anxiety, irritability, confusion, asthenia, sleeplessness, headache, delirium, hallucinations and hyperexcitability” (11, p. 362).

The Hordaland Health study in Western Norway illustrated an inverse association between standardized energy-adjusted magnesium intake and depression scores, meaning that people who consumed less magnesium had higher rates of depression (13). When the serum and cerebrospinal fluid of acutely depressed patients diagnosed with major depressive disorder or bipolar patients in a depressive episode were compared to healthy controls, the calcium to magnesium ratio was found to be elevated in the former (14). Calcium and magnesium are minerals which antagonize one another and compete for absorption, since each of these minerals is a divalent cation (a positive ion with a valence of two). Suicidality, one of the primary manifestations of severe depression, is accompanied by low cerebrospinal fluid levels of magnesium despite normal calcium levels, lending credence to the role of magnesium in positive emotionality (15).

Magnesium Effective in Bipolar Disorder, Fibromyalgia, PMS, and Chronic Fatigue Syndrome

A formulation of magnesium aspartate hydrochloride known as Magnesiocard has been shown to invoke mood-stabilizing effects in patients with severe rapid cycling bipolar disorder in one open study label (16). In half of the patients treated, this magnesium preparation had results equivalent to lithium, the standard of care for this patient population, such that the researchers suggested: “The possibility that Magnesiocard could replace or improve the efficacy of lithium as a preventive treatment of manic-depressive illness merits further clinical investigation” (16, p. 171). When used as an adjunctive therapy in severe, therapy-resistant mania, magnesium sulphate infusions significantly reduced the use of lithium, benzodiazepines and neuroleptics, so much so that the researchers concluded that it “may be a useful supplementary therapy for the clinical management of severe manic agitation” (17, p. 239).

In another randomized trial of elderly patients with type 2 diabetes and magnesium deficiency, elemental magnesium administered at 450 mg per day was found to have equivalent efficacy to 50 mg of the antidepressant drug Imipramine in treating depressive symptoms (18). Magnesium citrate taken at 300 mg per day has likewise been shown to decrease depression and other symptoms in patients with fibromyalgia as indicated by significant decreases in the fibromyalgia impact questionnaire (FIQ) and Beck depression scores (19).

Data also indicate that supplementation with 360 mg of magnesium administered to women with premenstrual syndrome (PMS) three times a day in the second half of the cycle is effective for so-called negative affect and other premenstrual-related mood symptoms (20). Lastly, intramuscular magnesium sulphate administered every week for six weeks has been proven to be effective in improving emotional state and other parameters in chronic fatigue syndrome (CFS) (21).

Mechanism of Action for Antidepressant Effects of Magnesium

According to researchers, “Biological systems discussed to be involved in the pathophysiology of affective disorders and the action of mood stabilizing drugs are affected by Mg, such as the activity of the hypothalamus–pituitary–adrenocortical (HPA) system, corticotropin releasing factor (CRF)-, GABA- and glutamatergic (via NMDA receptors) neurotransmission and several transduction pathways including protein kinase C” (12). Not only that, but magnesium elicits similar effects on nocturnal hormonal secretion and sleep brain waves to lithium salts, which are used as a treatment modality for bipolar disorder, supporting the role of magnesium as a mood stabilizer (22).

Magnesium operates as an agonist, or a stimulatory molecule, for γ-aminobutyric acid (GABA) receptors (22). GABA is the main inhibitory neurotransmitter in the central nervous system. By binding to the GABA receptor and replicating the effects of GABA, magnesium may alleviate anxiety. Magnesium may also elicit its antidepressant effects by acting as an inorganic antagonist of N-methyl-d-aspartic acid (NMDA) receptor function (Poleszak et al., 2007). Receptor antagonists are ligands, or substances, which bind to a receptor but inhibit its activity rather than activating it. NMDA receptors, which occur on the surface of nerve cells, are activated in part by glutamate, one of the excitatory amino acids in the brain.

Researchers state that, “Dysfunction of NMDA receptors seems to play a crucial role in the neurobiology of disorders such as Parkinson’s diseaseAlzheimer’s diseaseepilepsy, ischemic stroke, anxiety and depression,” such that, “ligands interacting with different sites of NMDA receptor complex are widely investigated as potential agents for the treatment of a variety of neuropsychiatric disorders” (22). In fact, drug inhibitors at the NMDA receptor complex, such as ketamine, demonstrate antidepressant effects (23, 24), but also induce such severe side effects that their clinical utility is limited (31). Magnesium, on the other hand, may have a similar mechanism of action by interfering with NMDA receptor activation without the adverse consequences of drug-induced NMDA receptor blockade (25).

Recent Study Proves Efficacy of Oral Magnesium for Depression

A recent open-label, randomized, cross-over trial was conducted in outpatient primary care clinics on 126 adults diagnosed with depression (26). During the intervention, 248 mg of elemental magnesium chloride per day, obtained from four 500 mg tablets, was administered for six weeks and compared to six weeks of no treatment, and subjects were evaluated for changes in depressive symptoms (26).

Magnesium administration results in clinically significant improvements in scores on both the Patient Health Questionnaire-9 (PHQ-9), a validated measure of the severity of depression and response to treatment, as well as the Generalized Anxiety Disorders-7 (GAD-7), a sensitive self-reported screening tool for severity of anxiety disorders (26). Impressively, results appeared in as little as two weeks, representing the dramatic improvement that nutrient restoration can facilitate (26). Impressively, however, magnesium exerted anti-depressant effects regardless of baseline magnesium level. It also exhibited efficacy independent of the gender, age, or baseline severity of depression of subjects, as well as their use of antidepressant medications (26). The authors of the study conclude, “Magnesium is effective for mild-to-moderate depression in adults. It works quickly and is well tolerated without the need for close monitoring for toxicity” (26).

Populations At Risk for Magnesium Deficiency

Half of the population of the United States was found to consume less than the recommended amount of magnesium when estimated a decade ago (27). Not only is magnesium lost with certain medical conditions, but this mineral is excreted as a consequence of biological activities such as sweating, urinating, and defecating as well as excess production of stress hormones (7, 11). In addition, because low magnesium has been correlated with various disease states, increasing magnesium status may mitigate risk of these diseases.

For instance, researchers note that, “Low magnesium intakes and blood levels have been associated with type 2 diabetes, metabolic syndrome, elevated C-reactive protein, hypertension, atherosclerotic vascular disease, sudden cardiac death, osteoporosis, migraine headache, asthma, and colon cancer” (27, p. 153). In addition, magnesium deficiency at a cellular level “elicits calcium-activated inflammatory cascades independent of injury or pathogens” (27, p. 153). Low magnesium is associated with systemic inflammation, and inflammation is at the root of most chronic and degenerative diseases.

Testing for Magnesium and Food Sources of Magnesium

While the first inclination of some physicians may be to test magnesium levels for an objective parameter of deficiency, the widely used serum or plasma magnesium does not accurately reflect magnesium levels stored in other tissues (28, 29). In addition, both this hematological index of magnesium status, referred to as total magnesium, and the erythrocyte magnesium level, indicative of the levels of magnesium inside red blood cells, are not negatively affected until severe magnesium deprivation has occurred (7). Therefore, these testing methodologies are not accurate enough to catch preliminary or subclinical magnesium deficiency.

Good food sources of magnesium include pumpkin and squash seed kernels, Brazil nuts, almonds, cashews, peanuts, pine nuts, quinoa, spinach, Swiss chard, beet greens, potatoes, artichoke hearts, dates, bananas, coconut milk, prickly pear, black beans, lima beans, soybeans, and seafood sources including halibut, abalone, anchovy, caviar, conch, crab, oyster, scallop, snail, and pollock. However, it is important to note that magnesium can be leeched from vegetables when food is boiled, and that fiber in excess can decrease magnesium absorption by increasing gastrointestinal motility (7).

Most Bioavailable Forms of Magnesium

As elucidated by the researchers, “Over-the-counter magnesium can be offered as an alternative therapy to those patients hesitant to begin antidepressant treatment and is easily accessible without a prescription” (26). Because the soil is no longer enriched in magnesium, supplementation may be warranted. Organic salts of magnesium, including the acetate, ascorbate, aspartate, bicitrate, gluconate, and lactate forms are more soluble and biologically active over the magnesium mineral salts such as magnesium oxide, magnesium carbonate, magnesium chloride, and magnesium sulfate (7).

However, case studies have shown remarkably rapid recovery from major depression, in less than seven days, when magnesium glycinate and magnesium taurinate are administered at dosages of 125 to 300 mg with each meal and at bedtime (11). Magnesium threonate may also be explored as a therapeutic option, as it may have better penetrance of the blood brain barrier and restore neurological levels of magnesium. This form, which is delivered directly to the brain, may improve cerebral signaling pathways and synaptic connections between nerve cells as well as support learning and memory, although the studies have been conducted in animal models (30).

Researchers report that magnesium is usually effective for treating depression in general use, and that comorbid conditions occurring in these case studies, including “traumatic brain injury, headache, suicidal ideation, anxiety, irritability, insomnia, postpartum depression, cocaine, alcohol and tobacco abuse, hypersensitivity to calcium, short-term memory loss and IQ loss were also benefited” by magnesium supplementation (11, p. 362). Barring abnormal kidney function, the Institute of Medicine sets the upper tolerable limit for intake at 350 mg of elemental magnesium per day, but there are few adverse side effects documented unless consumed in inordinate doses (26).

Before changing your medication or nutraceutical regimen, always consult a functional or integrative medical doctor for contraindications. However, given the benign nature of magnesium supplementation and the ubiquity of magnesium insufficiency, depressedpatients should be offered this as a first line strategy alongside a holistic root-cause resolution approach to treating depression (26).

For additional research on magnesium, visit our database on the subject. 

References

1. Yankelevitch-Yahav, R. et al. (2015). The Forced Swim Test as a Model of Depressive-like Behavior. Journal of Visualized Experiments,  97, 52587.

2. Srilakshmi, P., & Versi, L. (2012). Review of sexual dysfunction due to selective serotonin repute inhibitors. AP Journal of Psychological Medicine, 13(1), 28-31.

3. Dording, C.M. et al. (2002). The pharmacologic management of SSRI-induced side effects: a survey of psychiatrists. Annals of Clinical Psychiatry, 14(3), 143-147.

4. Moore, T.J., Glenmullen, J., & Furberg, C.D. (2010). Prescription drugs associated with reports of violence towards others. PLoS One, 5, e15337.

5. Maslej, M.M. et al. (2017). The Mortality and Myocardial Effects of Antidepressants Are Moderated by Preexisting Cardiovascular Disease: A Meta-Analysis. Psychotherapy and Psychosomatics, 86, 268-282.

6. Antonuccio, D.O., Burns, D.D., & Danton, W.G. (2002). Antidepressants: A Triumph of Marketing Over Science? Prevention & Treatment, Volume 5(25).

7. Newhouse, I., & Finstad, E.W. (2000). The Effects of Magnesium Supplementation on Exercise Performance. Journal of Sports Medicine, 10(3), 195-200.

8. Welch, A.A., Skinner, J., & Hickson, M. (2017). Dietary Magnesium May Be Protective for Aging of Bone and Skeletal Muscle in Middle and Younger Older Age Men and Women: Cross-Sectional Findings from the UK Biobank Cohort. Nutrients, 9(11), E1189. doi: 10.3390/nu9111189.

9. Abbasi, B. et al. (2012). The effect of magnesium supplementation on primary insomnia in elderly: A double-blind placebo-controlled clinical trial. Journal of Research in Medical Science, 17(12), 1161-1169.

10. Mursu, J. et al. (2015). The association between serum 25-hydroxyvitamin D3 concentration and risk of disease death in men: modification by magnesium intake. European Journal of Epidemiology, 30(4), 343-347.  doi: 10.1007/s10654-015-0006-9.

11. Eby, G.A., & Eby, K.L. (2006). Rapid recovery from major depression using magnesium treatment. Medical Hypotheses, 67(2), 362-370.

12. Singewald, N. et al. (2004). Magnesium-deficient diet alters depression- and anxiety-related behavior in mice–influence of desipramine and Hypericum perforatum extract. Neuropharmacology, 47(8), 1189-1197.

13. Jacka, F.N. et al. (2009). Association between magnesium intake and depression and anxiety in community-dwelling adults: the Hordaland Health Study. Australian and New Zealand Journal of Psychiatry, 43(1), 45-52. doi: 10.1080/00048670802534408.

14. Levine, J. et al. (1999). High serum and cerebrospinal fluid Ca/Mg ratio in recently hospitalized acutely depressed patients. Neuropsychobiology, 39(2), 63-70.

15. Banki, C.M. et al. (1995). Cerebrospinal fluid magnesium and calcium related to amine metabolites, diagnosis, and suicide attempts. Biological Psychiatry, 20, 163-171.

16. Chouinard, D. et al. (1990). A pilot study of magnesium aspartate hydrochloride (Magnesiocard) as a mood stabilizer for rapid cycling bipolar affective disorder patients. Progress in Neuro-Psychopharmacology, Biology, and Psychiatry, 14, 171-180.

17. Heiden, A. et al. (1999). Treatment of severe mania with intravenous magnesium sulphate as a supplementary therapy. Psychiatry Research, 3, 239-246.

18. Barragán-Rodríguez, L., Rodríguez-Morán, M., & Guerrero-Romero, F. (2008). Efficacy and safety of oral magnesium supplementation in the treatment of depression in the elderly with type 2 diabetes: a randomized, equivalent trial. Magnesium Research, 21(4), 218-223.

19. Bagis, S. et al. (2013). Is magnesium citrate treatment effective on pain, clinical parameters and functional status in patients with fibromyalgia? Rheumatology International, 33(1), 167-172. doi: 10.1007/s00296-011-2334-8.

20. Facchinetti, F. et al. (1991). Oral magnesium successfully relieves premenstrual mood changes. Obstetrics and Gynecology, 78(2), 177-181.

21. Cox, I.M. et al. (1991). Red blood cell magnesium and chronic fatigue syndrome. The Lancet, 337(8744), 757-760.

22. Held, K. et al. (2002). Oral Mg(2+) supplementation reverses age-related neuroendocrine and sleep EEG changes in humans. Pharmacopsychiatry, 35(4), 135-143.

23. Zarate, C.A. Jr. et al. (2006). A randomized trial of an N-methyl-D-aspartate antagonist in treatment-resistant major depression. Archives of General Psychiatry, 63, 856-864.

24. Berman, R.M. et al. (2000). Antidepressant effect of ketamine in depressed patients. Biological Psychiatry, 47, 351-354.

25. Poleszak, E. et al. (2007). NMDA/glutamate mechanism of antidepressant-like action of magnesium in forced swim test in mice. Elsevier Pharmacology Biochemistry and Behavior, 88(2).

26. Tarleton, E.K. et al. (2017). Role of magnesium supplementation in the treatment of depression: A randomized clinical trial. PLoS One, 12(6), e0180067. doi: 10.1371/journal.pone.0180067.

27. Rosanoff, A., Weaver, C.M., & Rude, R.K. (2012). Suboptimal magnesium status in the United States: are the health consequences underestimated? Nutrition Reviews, 70(3), 153-164. doi: 10.1111/j.1753-4887.2011.00465.x.

28. Altura, B.T. et al. (1994). Characterization of a new ion selective electrode for ionized magnesium in whole blood, plasma, serum, and aqueous samples. Scandinavian Journal of Clinical Lab Investigations, 54(Suppl. 217), 21–36.

29. Weller, E. et al. (1998). Lack of effect of oral Mg-supplementation on Mg in serum, blood cells and calf muscle. Medical Science Sports Exercise, 30, 1584–1591.

30. Slutsky, I. et al. (2010). Enhancement of learning and memory by elevating brain magnesium. Neuron, 65(2), 165-177. doi: 10.1016/j.neuron.2009.12.026.

31. Willetts, J., Balster, R.L., & Leander, J.D. (1990). The behavioral pharmacology of NMDA receptor antagonists. Trends in Pharmacological Science, 11, 423-428.

Ali Le Vere holds dual Bachelor of Science degrees in Human Biology and Psychology, minors in Health Promotion and in Bioethics, Humanities, and Society, and is a Master of Science in Human Nutrition and Functional Medicine candidate. Having contended with chronic illness, her mission is to educate the public about the transformative potential of therapeutic nutrition and to disseminate information on evidence-based, empirically rooted holistic healing modalities. Read more at @empoweredautoimmune on Instagram and at www.EmpoweredAutoimmune.com: Science-based natural remedies for autoimmune disease, dysautonomia, Lyme disease, and other chronic, inflammatory illnesses.

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U. S. Congressman-Elect Tells Constituents Vaccines May Cause Autism

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In Brief

  • The Facts:

    Congressman-Elect Mark Green told his constituents not only that he believes there may be a causal relationship between vaccines and autism, but that the Centers for Disease Control has 'fraudulently managed' their research data on vaccine safety.

  • Reflect On:

    Does the unusually bold statements of a public official against the CDC's vaccine safety narrative represent the next step in our getting to the truth about vaccines?

If you are someone who has been following the vaccine/autism debate for a while, you are no doubt aware that public figures rarely take on the Big Pharma-CDC Axis, as they are prone to face serious and debilitating consequences at their own peril. Doctors like Andrew Wakefield, who famously published a study in 1998 in The Lancet that linked the MMR vaccine to autism, experienced the full weight of the medical establishment muscle when sales of the MMR vaccine were threatened.

As far as Western Medicine is concerned, Dr. Wakefield’s study has been ‘debunked’ as a result of their concerted campaign to say and do whatever they could to invalidate his main point by attacking ancillary facts that really had nothing to do with the evidence. You can read our recent article ‘A Statistically Strong Relationship Has Been Found Between The MMR Vaccine & Autism‘ to see how Dr. Brian Hooker has resurrected the study to make an argument in favor of the strong correlation found in the study between the MMR vaccine and autism, and judge for yourself.

While the majority of people probably believe that the safety of vaccines has been proven, the veils of mainstream deception are starting to get threadbare. And with this, whistleblowers, researchers and other challengers to the mainstream notion are starting to get bolder and more forthright. Researcher Judy Mikovits and others like her paved the way by standing firmly in the truth of her research and refusing to buckle under the pressure and coercion of the Western Medical Establishment to recant studies that are threatening to the pharmaceutical industry, as detailed in the article ‘Researcher Jailed After Uncovering Deadly Virus Delivered Through Human Vaccines.’

Congressman-Elect Makes Bold Claim

Still, researchers are one thing. Politicians are a whole different kettle of fish. It is still a relatively new occurrence that a politician could speak out against the vaccine industry and not be committing political suicide and open him or herself up to massive attacks from the Western Medical Establishment. It was helpful, perhaps even groundbreaking, that Donald Trump staked his claim on this matter while campaigning for the presidency:

“When I was growing up, autism wasn’t really a factor, and now all of a sudden, it’s an epidemic. Everybody has their theory. My theory, and I study it because I have young children, my theory is the shots. We’ve giving these massive injections at one time, and I really think it does something to the children.” (source)

Now, according to this article in the Tennessean, Congressman-Elect Mark Green told his constituents not only that he believes there may be a causal relationship between vaccines and autism, he suggests the Centers for Disease Control has ‘fraudulently managed’ the research data that the CDC uses to say there is no link between vaccines and autism. As the video below confirms, Green, who by the way is also a licensed doctor, is truly throwing down the gauntlet against the CDC.

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“Let me say this about autism. I have committed to people in my community, up in Montgomery County, to stand on the CDC’s desk and get the real data on vaccines. Because there is some concern that the rise in autism is the result of the preservatives that are in our vaccines.

As a physician, I can make that argument and I can look at it academically and make the argument against the CDC, if they really want to engage me on it. But it appears some of that data has been, honestly, maybe fraudulently managed. So we’ve got to go up there and stand against that and make sure we get that fixed, that issue addressed.”

The Takeaway

The challenges to the official narrative that vaccines are proven to be safe and do not correlate with the incidence of autism is like chipping away at an old brick wall. With each brick that is removed, more and more people see the holes in the mainstream narrative, and opponents to vaccine safety are becoming bolder and more direct with their challenges. I believe that in the not-so-distant future we will look back to this time and history and be amazed that it took us so long to see through the industry-sponsored fraud of vaccine safety.

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In order to stay truly independent, we need your help. We are not going to put up paywalls on this website, as we want to get our info out far and wide. For as little as $3 a month, you can help keep CE alive!

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“5G: The Most Censored Story Of 2018” – Journalist Masterfully Educates Houston City Council

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In Brief

  • The Facts:

    Derrick Broze, a journalist and activist recently presented to Houston about the new proposed 5G network. He points to multiple studies and scientists outlining serious health concerns for all.

  • Reflect On:

    What can you do to mitigate this? There are solutions presented in the article but are you taking any? How can our regulatory agencies approve a technology that's so harmful to human biology? What is going on here?

The 5G network is new, and it’s being accepted, approved and implemented already without any appropriate safety testing nor discussion with the public.

Recent research has revealed that the frequencies utilized in crowd control weapons are the same as the frequencies used in the 5G network, and there is absolutely no question about the fact that these electromagnetic frequencies impact our biology in multiple harmful ways. With more than 2000 peer-reviewed studies on the subject, thousands of scientists raising multiple causes for concern, hundreds of scientists petitioning the United Nations, and absolutely no oversight, regulation or safety testing, how is it that this type of thing is legal and allowed to be approved?

Well, the 5G, and the entire global network of wireless technology is controlled by a few people and corporations. This highlights the relationship that western corporations have with government regulatory agencies. These corporations sit above the government, and through lobbying, corporations provide instructions to government regulatory agencies. Our regulatory health agencies are a cesspool of corruption as well, so much to the point where those who work within these agencies are actually starting to have a shift in consciousness and are speaking out. The problem has become so big and widespread that they cannot remain silent. The SPIDER papers from multiple CDC scientists was an excellent example, outlining the grave concern about the CDC’s relationship with corporations and the stranglehold these corporations have over them.

Multiple countries around the world have banned WiFi and the building of cell phone towers near primary schools and nurseries, among many other places due to the evidence that shows they are not safe and can implicate the health of young children and adults.

Dr. Devra Lee Davis,  founding director of the board on Environmental Studies and Toxicology of the U.S. National Research Council, National Academy of Sciences, founding director of the Center for Environmental Oncology, University of Pittsburgh Cancer Institute, and President of the Environmental Health Trust stated:

“If you are one of the millions who seek faster downloads of movies, games and virtual pornography, a solution is at hand, that is, if you do not mind volunteering your living body in a giant uncontrolled experiment on the human population. At this moment, residents of the Washington, DC region – like those of 100 Chinese cities – are about to be living within a vast experimental Millimeter wave network to which they have not consented – all courtesy of American taxpayers,”

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Journalist Presents to Houston City Council

That’s why the video below is so important, it’s a video of Derrick Broze, founder of the Conscious Resistance network.  Not only are scientists speaking out about this issue, and continuing to publish eye-opening research, much of which can be found at The Environmental Health Trust, but citizens and activists are getting involved too.

The speech below takes place at a hearing in Houston. This, like so many other health issues we are facing, is important to raise awareness about together. The more people speaking up and creating awareness, the more chances we have of that this awareness leads to action or at the very least, a deep realization by council members that they are being bullied by corporations, much like we’re seeing in France.

A Little More On 5G

Dr. Sharon Goldberg, an internal medicine physician and professor also recently gave her testimony regarding the dangers of electromagnetic radiation. She says:

Wireless radiation has biological effects. Period. This is no longer a subject for debate when you look at PubMed and the peer-review literature. These effects are seen in all life forms; plants, animals, insects, microbes. In humans, we have clear evidence of cancer now: there is no question We have evidence of DNA damage, cardiomyopathy, which is the precursor of congestive heart failure, neuropsychiatric effects…5G is an untested application of a technology that we know is harmful; we know it from the science. In academics, this is called human subjects research.” – Goldberg

You can watch her testimony and read more about it here.

Again, if you want to look at the science/research, a good place to start is with the Environmental Health Trust.

The Takeaway

There are multiple solutions for reducing your exposure to EMF radiation. You could have a wired internet connection at home, which is actually faster. You could unplug your devices before bed, you can purchase electromagnetic radiation shielded clothing from multiple providers. You can also purchase small devices that go right on your phone that help protect against this radiation. Do your research on ‘EMF protection devices’ to find what works for you.

You could also mitigate some effects by living a more healthy lifestyle. This includes diet, nature exposure, limiting screen and phone time and other wellness practices.

The key thing here is to recognize that, in a world where our voice is constantly being silenced and information is swept under the rug for the sake of profit and control (among other reasons), we do still have a voice, and we have to use it.

We are so caught up in our own lives, doing our own thing that we’ve neglected the planet and fail to even look into what’s going on. We’ve given our consciousness away to others who are manipulating it. It’s time to take it back, to wake up, and to start thinking for ourselves instead of relying on a group of powerful people to disseminate information.

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A Statistically Strong Relationship Has Been Found Between The MMR Vaccine & Autism

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In Brief

  • The Facts:

    Dr. Brian Hooker, one of multiple people who received committed data from a CDC senior scientists regarding a 2005 MMR autism vaccine study has done a reanalysis that clearly shows a statistically significant relationship.

  • Reflect On:

    Why are negative aspects and important research/testimony regarding vaccines completely ignored? Why are people believing that vaccines are safe and effective if all of the evidence points otherwise? Why is the only response ridicule?

After four long years, CHD Board Member, Dr. Brian Hooker‘sreanalysis of the CDC’s MMR-autism data from the original DeStefano et al. 2004 Pediatrics paper has been republished in the Winter 2018 Edition of the Journal of American Physicians and Surgeons. The data, when properly analyzed, using the CDC’s own study protocol, show a strong, statistically significant relationship between the timing of the first MMR vaccine and autism, specifically in African American males. In addition, a relationship also exists in the timing of the MMR vaccine and those individuals who were diagnosed with autism without mental retardation. These relationships call into question the conclusion of the original DeStefano et al. 2004 paper which dismissed a connection between the MMR vaccine and autism.

Main Points from Reanalysis:

  • The rate of autism diagnoses has increased alarmingly in the U.S., and is about 25 percent higher in black children. Boys are far more likely than girls to receive this diagnosis.
  • As early as 2001, the Centers for Disease Control and Prevention (CDC) had data showing an increased rate of autism diagnoses in black male school children in Atlanta who received their first measles-mumps-rubella (MMR) vaccination before 36 months of age.
  • The original publication concerning the data downplayed the association, and no follow-up was conducted.
  • Dr. Hooker noted that the CDC deviated from its original data analysis plan, possibly because of unwanted results.
  • The relationship loses its statistical significance if the analysis is restricted to children with a Georgia birth certificate, which decreases the sample size by about 40 percent.
  • Dr. Hooker reanalyzed the same data set using the same methodology of conditional logistic regression but didn’t exclude children lacking a Georgia birth certificate.
  • By stratifying data for African-American males by birth year, Dr. Hooker also found a statistically significant higher risk of an autism diagnosis in children who had received the first MMR vaccine 1 year earlier, only in children born in 1990 or later. Thimerosal exposure increased in the early 1990s, and it was not removed from most pediatric vaccines until 2001-2004. Dr. Hooker suggests the possibility that there may be some interaction between increased mercury exposure and early MMR vaccination. Further study would be needed to explore this possibility.
  • Dr. Hooker’s interest was sparked, he reports, by communication with a CDC whistleblower, a senior scientist, who had retained some of the original analyses.
  • Dr. Hooker concludes that failure to follow-up on these observations represents a huge lost opportunity to understand possible reasons for the enormous increase in this devastating neurological disability.

Introduction from Dr. Hooker’s article:

“This study is a re-analysis of Centers for Disease Control and Prevention (CDC) data pertaining to the relationship of autism incidence and the age at which children got their first measles-mumps-rubella (MMR) vaccine. Statistically significant relationships were observed when African-American males were considered separately while looking at those individuals who were vaccinated prior to and after a 36-month age cut-off. CDC officials observed very similar relationships as early as November 2001, but failed to report them in their final publication. In addition, a relationship is seen when specifically considering children who received a diagnosis of autism without mental retardation. Although this was reported in the original 2004 paper, it was not discussed, nor was any follow-up study conducted. Preliminary results also suggest the possibility of a synergism between thimerosal exposure and MMR timing leading to a greater risk of autism.”

Conclusion from Dr. Hooker’s article:

“The first data set used by DeStefano et.al represents a huge lost opportunity to understand any role between the timing of the first MMR vaccine and autism. The re-analysis presented here elucidates effects that should at least merit further investigation. Specifically, increased risks of earlier vaccination are observed for African-American males and among cases of autism without MR. Both phenomena deserve additional study that could yield important clues regarding the current enormous increase in autism.”

Dr. Hooker’s Reanalysis of CDC Data on Autism Incidence and Time of First MMR Vaccination was published December 7, 2018 in the Journal of American Physicians and Surgeons.

Important Reminder From Collective Evolution

Dr. William Thompson (senior CDC scientist), who is  mentioned above as co-author of this study, blew the whistle and admitted that he was pressured to omit statistically significant data, and that there is a connection between this vaccine and autism. He released this statement in an official capacity, as explained by the Congressman in the video below. This story was an has been completely ignored by mainstream media.

Dr. Hooker and Thompson were in touch, Hooker was the one who did the reanalysis as you can see above.

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