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The Tail Wagging the Dog: Death Categorization Drives Healthcare Decisions

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By World Mercury Project Guest Contributor Joy M. Fritz, posted here with permission.

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I work with doctors, coroners and the local county registrars every day to create death records. It’s what I do for a living and I wanted to share my thoughts on the mortality rates being thrown around on mainstream and social media regarding the influenza epidemic. Please note: This information I am sharing is not limited to influenza reporting, but rather, serves as a case study of how the mortality rate recording system (mal)functions at large.

I am sorry to say that death rates are NOT as simple or as valid as every news broadcaster with perfectly-trained vocal delivery makes them sound, and they are absolutely not the infallible pillar of medical history that the CDC purports.

This failed mechanism in the mortality rate ‘generator’, if you will, is the same for the hotly debated adverse vaccine reactions. This is the reason you see horrible adverse vaccine reactions and deaths being claimed by parents on social media, but no line item for them in national statistics.

An Imperfect System

Our current system for capturing mortality rates can and does provide a mostly uninvestigated and inaccurate picture of what causes a death. The process for creating and registering causes of death for public records is a complicated, convoluted, and politicized one. It is completely open to both ignorance and the manipulations of personal, professional, and governmental interests.

I have come to realize how greatly this reality becomes a public health issue during this current flu season when every major media outlet is providing us with live updates on the accruing death toll. Seeing these reports caused me concern for my family. My husband and I discussed what preventive treatment we might consider. I started reading the FDA package inserts for different flu immunization options to get informed on which might be safest for our infant and six-year-old. What I ultimately wanted to investigate was the risk of death. My kids getting sick is just part of life; other people getting sick is just part of life; lowering the risk of death to my family and the people around me is what I cared about when it came specifically to the seasonal flu.

The process for creating and registering causes of death . . . is completely open to both ignorance and the manipulations of personal, professional, and governmental interests.

I started researching mortality rates to find the line item in the CDC reports for “deaths due to influenza” vs. “adverse reaction to influenza medications and immunizations”. I found influenza rates, no problem. Flu medications and shots? No deaths reported. Awesome. What a simple decision to make! But, being in the mortuary industry and curious about how they get these reports, I looked at the last full report for 2014, dug deeper, and eventually found that they simply code and reorganize the data that they receive from death records—the very death records that I am typing up and registering every day.

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So my head started to explode. And I felt, and still feel, sick. I realized that without being aware of it, I knew exactly how influenza deaths are recorded, and why there was no line item in the CDC’s mortality rates for adverse reactions to common medical treatments.

Before I continue, please know that I will not be explaining all the ins and outs of my job, nor the incredibly rare reality that medication complications and adverse reactions do get captured (usually in box 112 of the death record, not as the primary underlying cause). Those exceptions are made possible by exceptional, and likely, very principled people, choosing individually to go above and beyond the call of protocol, whether that be the family that is aware of the impact of the legal documentation that occurs after death and stays level-headed and involved mere hours after the death of their loved one, or an insanely humble and honest doctor, in conjunction with the coroner medical-legal officer who trusts and cooperates with the honest doctor and vigilant family to think outside the box of their standard procedures. Almost five years and nearly 5,000 death certificates later, I can say with confidence that that kind of post-death communication concoction is at a statistical percentage point that even the CDC would consider insignificant.

What most people don’t know is that doctors are not allowed to attest to anything that is not a strictly NATURAL cause of death.

Core Considerations

So, in the spirit of very uncomfortable truthfulness, I will share a snapshot of the core issues embedded in the daily procedures of creating the death statistics that we so desperately need to make prudent health decisions for ourselves and our families. I will also include some examples of how these core issues would manifest into faulty statistical analysis at the level of our public health and lead to the miscalculation of the benefits and risks surrounding our individual medical choices.

Core Issue A: Doctors who provide causes have not all been trained the same way, and therefore do not provide standardized responses. This may at first glance seem minor, as it always has to me, but this directly affects the cause that the doctor lists on the death certificate. Some doctors prefer listing the underlying cause of death as the recent complications that occurred in the last days or weeks before death, such as pneumonia or influenza, while leaving out the more chronic illnesses that had led to the decline in health. Other doctors decide they will provide the more long-standing health conditions as the cause of death (for instance, diabetes, asthma or congenital abnormality) while leaving out the more immediate illnesses. Some doctors include both the short-term and long-term diagnoses.

Many factors play a role in which approach doctors choose. These include in what capacity the doctor saw the patient (hospital vs. hospice care for example) or the immediate availability of the complete medical record within the time frame being impressed by the mortuary due to upcoming funeral or cremation services, or simply the way the doctor personally prioritizes information. Furthermore, doctors feel limited as to what they can provide for a cause by the professional context in which they saw the patient, as determined by their specialty. For example, a primary care physician might provide a cause of death as “coronary artery disease” since that was what he/she was prescribing medication to the patient for, whereas the patient could simultaneously be being treated for stage four chronic kidney disease and be on dialysis. In this case, rather than the objectively more serious health condition being listed on the death certificate, the health condition that the doctor is most comfortable attesting to is listed. Again, way too many factors to go into in this piece, but the basic issue of the lack of standardization in cause of death diagnosis and reporting remains.

In the case of a patient who dies after contracting influenza, this patient could have all of the above-mentioned conditions on his/her medical record simultaneously, from influenza to asthma, pneumonia, congenital abnormality, coronary artery disease and chronic kidney disease. Any ONE of those conditions listed is correct and valid, and could be entered as a stand-alone cause which would then be registered by me and the local and state registrar’s offices without a query. It’s the doctor’s preference and his medical opinion—yet the national attention given, medical research dollars, and yearly health choices we all make are swayed by whichever cause this particular doctor, with his/her own particular training and personality, decides to jot down on the worksheet and send back to me to enter into the official record.

CORE ISSUE B) What most people don’t know is that doctors are not allowed to attest to anything that is not a strictly NATURAL cause of death. Falls, medication complications or overdoses, causes with the word “injury” in it, anything that is considered an unnatural or external cause is outside the realm of their jurisdiction as far as the death record is concerned. The coroner would need to be contacted and agree to certify or co-certify a death record that has an unnatural or external cause listed. This is a whole other, very complicated reporting issue that I will not get into in this post. I will say, however, from the perspective of a mortuary representative, that everyone involved (doctor, coroner, registrar and myself) understands that the delay caused by any coroner involvement is highly dreaded and avoided if at all possible due to the amplified grief it can cause the family if they do not want an autopsy or investigation done or have to suffer a delay in services and/or an upset in their own personal closure process.

However, the majority of doctors are aware of their own limitation to certify only natural causes of death. And usually in the interest of serving the grieving family, they will provide the simplest natural cause that they know will quickly pass the approval of the local registrar’s office, fulfill their duty as a signing physician, and enable the grieving family to move forward with their scheduled burial or cremation services. It should be noted here that doctors are under an additional pressure since they have a limited time set out by their State Health and Safety Codes to provide causes of death to a funeral home. In California, it is within 15 hours of death, although that is rarely achieved. Delays of more than a few days after death would risk them getting their license reported to the state medical board for lack of compliance.

What Works About This System?

The system is created in such a way that naturally occurring infectious disease (such as influenza) can be and is being reported and recorded in national mortality rates. However, the lack of standardization in the way doctors report it creates an unreliable number to set as the threshold for what constitutes an epidemic.

What Does NOT Work About This system?

It does not report on the true consequential timeline of the patient’s medical treatment, including unnatural and external complications and errors in their medical care and is therefore woefully inadequate as the basis for ANY medical claims or recommendations.

The first example to illustrate the impact of this issue is as follows:

I read a post from a nurse the other day that shared her story of being hospitalized due to complications of the flu. Even though she had gotten the flu shot every year, she had only gotten influenza this year. Five days after experiencing flu symptoms, she went to her medical provider and was prescribed Tamiflu. She went through her course of medication. Her flu symptoms eased but she started getting a tightness in the chest, which further worsened until she needed to be hospitalized for pneumonia and a close call with sepsis. The conclusion of her post—and her medical opinion as a nurse—was that this year’s flu was very dangerous and anyone less healthy than she could have easily died with her symptoms, so she urged everyone to please get the flu shot to prevent the flu from spreading.

The saddest part about reading her story was discovering that she must not have read the Tamiflu manufacturer’s insert, which states that “No influenza vaccine interaction study has been conducted” and “Efficacy of TAMIFLU in patients who begin treatment after 40 hours of symptoms has not been established” and furthermore, “Events reported more frequently in subjects receiving TAMIFLU compared to subjects receiving placebo in prophylaxis studies, and more commonly than in treatment studies, were aches and pains, rhinorrhea, dyspepsia and upper respiratory tract infections.” (emphasis added)

This would lead to an alternate, very feasible medical conclusion that her hospitalization and pneumonia was the result of using a medication that has not been tested on a population of her vaccination status and symptoms duration, which also has the adverse reaction of a URTI.

…the likelihood of influenza causing the death is greater than the medication causing the death because of mortality rates—but they are the ones creating the mortality rates…

But what if someone less healthy than herself with her exact symptoms and medication course HAD died? Her medical opinion, and many other medical care providers’ opinions would have been that it was influenza that had caused the death, instead of the complications of the medication. In the medical provider’s mind, the likelihood of influenza causing the death is greater than the medication causing the death because of mortality rates—but they are the ones creating the mortality rates—so what is considered reasonable likelihood is being created in a closed loop, a regurgitating cycle.

So, whether the attending physician at the hospital was aware of this medical misstep by the other medical provider or not, in this case the hospital physician could simply put “Influenza” on the causes of death worksheet and send it back to me. Influenza would be entered in the death record and be reported in the state and then national database as such with no question from me or the government registrars.

A Public Health Reporting Conundrum

What this has created, then, is a serious public health reporting conundrum. Death due to complications of improperly prescribed medications are NOT being calculated into the national reporting agencies in a real-time setting. Neither would they be communicated in real-time to the public. Instead, people would simply hear of the rising influenza death toll and run for more medication (and likely not be reading the manufacturer’s insert either to verify if they truly are good candidates for that medication).

I have many friends and family in the medical industry and it is easily admitted that legal and personal liability is a factor in the considerations of proper reporting.

In this medication example, as you can imagine, even IF the recorders realize that the medication was prescribed erroneously, it would not be in the professional best interest of the medical provider or medical facility to report this prescription error and its possibly fatal complications to the family or public health officials. I have many friends and family in the medical industry and it is easily admitted that legal and personal liability is a factor in the considerations of proper reporting. However, if and when this possibly fatal prescription misstep was ever reported, it would be in some very passive EMR analysis many months or years later, with no urgency or real-time public health warning. The ability for government to cross-check and minutely examine nearly three million decedent medical records of varying electronic availability—annually—is just not there.

This failed mechanism in the mortality rate “generator”, if you will, is the same for the hotly debated adverse vaccine reactions. This is the reason you see horrible adverse vaccine reactions and deaths being claimed by parents on social media, but no line item for them in national statistics. It is not because they don’t exist or don’t happen. The real-time data reporting system of death recording is not set up to calculate these deaths. The families that become aware of the adverse reactions in time to request investigation (<24 hours after death), and are able to request any relevant pathological specimens to be procured before the burial or cremation of their loved one, would then need to have the time and resources to go through the lengthy reporting and court procedures through the Vaccine Adverse Event Reporting System (VAERS) and the National Vacine Injury Compensation Program (NVICP). A very few families do, and if they can establish enough scientific evidence (like pathology reports) and find and produce enough experts and professional support, they MIGHT eventually get the causes of death amended and compensation for their loss paid out by the allotted government fund. And after five, 10, 15 or 20 years, this passive data capture system might accrue enough statistical information to be reported back to the medical community so that they adjust their recommendations. However, with the HHS claim that only around 1% of vaccine injuries are reported to VAERS, even this may not be realistic.

So, just like in the medication example, any death due to an adverse reaction to the flu shot or for ANY regularly scheduled wellness immunization, would similarly not be captured in the standard process of death recording. As before, the doctor can still provide either influenza or any other natural-occurring immune response as the only cause of death. He would send it to me and I would enter it in, get the state to approve it, and “Voila!”—a thoroughly inaccurate mortality rate reporting. 

Impacting Informed Consent

One of the most difficult realities for me to recognize in examining the mortality rate reporting system that I am a part of, is that the medical community itself is suffering from the ignorance that this kind of circular mortality rate generating system creates. Doctors and coroners are limited by the already existing mortality rates to gauge the likelihood of what caused death. That kind of system can only regurgitate the same causes of death over and over again by forcing its reporters to use the same types of “acceptable” death diagnoses that already exist.

And these are the statistics the medical community uses to educate themselves and provide informed consent to the patient on what the most prudent option is for medical care to safeguard health and prevent death.

And, yes, I will take the opportunity here to say that we can logically apply this critical analysis of the lack of proper data capture to those reluctant to vaccinate or use medications. There is no current national data capture system that records the morbidity or mortality rates of those who choose less medical intervention or choose to not vaccinate themselves or their kids. We don’t know what their life expectancy, quality of life or mortality rate is in our modern day, with the advancements in hygiene, technology and post-disease-diagnosis medical care availability being considered. It could absolutely be worse, statistically, but we wouldn’t know.

For nationally reported statistics, we are left then with bad data on one side, and no control group data on the other. Hardly the recipe for safe or settled scientifically guided medical care.

Now where does that leave you and me? Our highly subjective—yet somehow infallible—weaponry of mortality rates, whether from national statistics or the social media horror stories, has us and all our friends and family swinging the manic flag of “People are dying!”

This flu season, for example, some of our friends are saying, “People are dying from flu! Get vaccinated! Take medication!” while other friends are saying, “People are dying from adverse reactions to medications and shots! Don’t get vaccinated! Drink elderberry!” And we are all running for the nearest remedies that we are sure will help us because of statistics—OR because we don’t see statistics reflecting our lived reality, so we do the best we can to discern our health without statistics.

But I’m the one creating these statistics and I offer you this: If you take one thing away from this, take away a healthier skepticism about even the most accepted mainstream, nationally reported, CDC or other “scientific” statistics. Humans who had no concept of their widespread impact made them. The numbers are not hard—they are very, very fluid. And conversely, have a healthier skepticism about all the alternative remedies we welcome as hopeful scientific-ish options. There is no unbiased, century-long, data capture system set up for these choices either.

As a parent, the most painful part of taking a step back and looking at all this, is having to humbly admit—I don’t know what the right thing to do is.

I don’t have the unbiased data I need to make the safest decision for my children.

I don’t know what the right thing to do is for myself, or for my husband.

I don’t know what side of the fence to stand on in the vaccination and mainstream medicine battlefield, and I don’t want to stand on a side: I just want the unbiased, uncorrupted and standardized data needed to accurately assess the benefits vs. the ultimate risks for my family’s health.

For nationally reported statistics, we are left then with bad data on one side, and no control group data on the other. Hardly the recipe for safe or settled scientifically guided medical care.

A Self-Reporting System

In the face of this fallible data capture system, my own resolution that I am willing to publicly recommend—no matter what medical choices you decide are best—would be:

  • Become self-reporters. Keep a health journal for each family member complete with dates and times and severity of symptoms of illness, and track dates and dosages of any medical treatment administered. Track degree of fevers, severity of migraines, frequency of ear infections, changes of behavior, hospitalizations, medication dosages and immunization combinations, etc.
  • Think critically and ask questions when you see inconsistencies in any health recommendations offered to you or your family. Request and encourage a satisfactory discussion of benefits and risks with your medical provider.
  • Download and thoroughly read the manufacturer’s insert provided on the FDA’s website for any medication or immunization you are considering, and verify that you are a good candidate for that medication. If you decide to use that medical treatment, record any minor reactions in the health journal, immediately report any somewhat severe reactions to your medical provider, and ask for that information to be added to your electronic medical record so that it might inform any future medical provider on your individual contraindications you may have in other medication courses. Remember that each of us is liable for our own health choices; you cannot expect a medical provider to be a perfect assessor of what’s best for you.
  • Follow up and make sure proper reporting was done on the medical provider’s part to the appropriate national databases, or report it yourself:  MedWatch reports for medications and VAERS reports for vaccines. This recommendation is less for you and more for others and for the sake of having the appropriate authorities informed so they can eventually take medical treatments off the market and create the demand for safer ones. Those kind of databases can only function well for the populations they serve if they are being used by everyone.

Yes, people are dying. Each and every day. I do their death records every flu season or surfing season. And try as hard as we do—and no matter how absolutely shredded inside I am, especially when I do an infant or child’s death certificate—we will never eradicate death. We CAN work to slowly eradicate and reform bad systems and misinformation. And even though there is no immediate gratification in it, we will probably save more lives when we work intelligently, truthfully and ethically towards a better future. That usually starts with a lot of humility and admitting that change is needed.

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Awareness

Epigenetic Memories Are Passed Down 14 Successive Generations, Game-Changing Research Reveals

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In Brief

  • The Facts:

    It's amazing how much information can be passed on to our offspring. Scientist have discovered that our DNA has memories, and these can also be passed down. We are talking about thoughts, feelings emotions and perceptions.

  • Reflect On:

    Biological changes are shaped by our environment, as well as our thoughts, feelings, emotions and reaction to that environment. Our DNA can be changed with belief, the placebo is a great example. Thoughts feelings and emotions are huge in biology.

This article was written by the Greenmedinfo research group, from Greenmedinfo.com. Posted here with permission.

Until recently, it was believed that our genes dictate our destiny. That we are slated for the diseases that will ultimately beset us based upon the pre-wired indecipherable code written in stone in our genetic material. The burgeoning field of epigenetics, however, is overturning these tenets, and ushering in a school of thought where nurture, not nature, is seen to be the predominant influence when it comes to genetic expression and our freedom from or affliction by chronic disease.

Epigenetics: The Demise of Biological Determinism

Epigenetics, or the study of the physiological mechanisms that silence or activate genes, encompasses processes which alter gene function without changing the sequence of nucleotide base pairs in our DNA. Translated literally to mean “in addition to changes in genetic sequence,” epigenetics includes processes such as methylation, acetylation, phosphorylation, sumolyation, and ubiquitylation which can be transmitted to daughter cells upon cell division (1). Methylation, for example, is the attachment of simple methyl group tags to DNA molecules, which can repress transcription of a gene when it occurs in the region of a gene promoter. This simple methyl group, or a carbon bound to three hydrogen molecules, effectively turns the gene off.

Post-translational modifications of histone proteins is another epigenetic process. Histones help to package and condense the DNA double helix into the cell nucleus in a complex called chromatin, which can be modified by enzymes, acetyl groups, and forms of RNA called small interfering RNAs and microRNAs (1). These chemical modifications of chromatin influence its three-dimensional structure, which in turn governs its accessibility for DNA transcription and dictates whether genes are expressed or not.

We inherit one allele, or variant, of each gene from our mother and the other from our father. If the result of epigenetic processes is imprinting, a phenomenon where one of the two alleles of a gene pair is turned off, this can generate a deleterious health outcome if the expressed allele is defective or increases our susceptibility to infections or toxicants (1). Studies link cancers of nearly all types, neurobehavioral and cognitive dysfunction, respiratory illnessesautoimmune disorders, reproductive anomalies, and cardiovascular disease to epigenetic mechanisms (1). For example, the cardiac antiarrhythmic drug procainamide and the antihypertensive agent hydralazine can cause lupus in some people by causing aberrant patterns of DNA methylation and disrupting signalling pathways (1).

Genes Load the Gun, Environment Pulls the Trigger

Pharmaceuticals, however, are not the only agents that can induce epigenetic disturbances. Whether you were born via vaginal birth or Cesarean section, breastfed or bottle-fed, raised with a pet in the house, or infected with certain childhood illnesses all influence your epigenetic expression. Whether you are sedentary, pray, smoke, mediate, do yoga, have an extensive network of social support or are alienated from your community—all of your lifestyle choices play into your risk for disease operating through mechanisms of epigenetics.

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In fact, the Centers for Disease Control (CDC) states that genetics account for only 10% of disease, with the remaining 90% owing to environmental variables (2). An article published in the Public Library of Science One (PLoS One) entitled “Genetic factors are not the major causes of chronic diseases” echoes these claims, citing that chronic disease is only 16.4% genetic, and 84.6% environmental (3). These concepts make sense in light of research on the exposome, the cumulative measure of all the environmental insults an individual incurs during their life course that determines susceptibility to disease (4)

In delineating the totality of exposures to which an individual is subjected over their lifetime, the exposome can be subdivided into three overlapping and intertwined domains. One segment of the exposome called the internal environment is comprised of processes innate to the body which impinge on the cellular milieu. This encompasses hormones and other cellular messengers, oxidative stress, inflammation, lipid peroxidation, bodily morphology, the gut microbiotaaging and biochemical stress (5).

Another portion of the exposome, the specific external environment, consists of exposures including pathogens, radiation, chemical contaminants and pollutants, and medical interventions, as well as dietary, lifestyle, and occupational elements (5). At an even broader sociocultural and ecological level is the segment of the exposome called the general external environment, which may circumscribe factors such as psychological stress, socioeconomic status, geopolitical variables, educational attainment, urban or rural residence, and climate (5).

Transgenerational Inheritance of Epigenetic Change: Endocrine Disruptors Trigger Infertility in Future Generations

Scientists formerly speculated that epigenetic changes disappear with each new generation during gametogenesis, the formation of sperm and ovum, and after fertilization. However, this theory was first challenged by research published in the journal Science which demonstrated that transient exposure of pregnant rats to the insecticide methoxychlor, an estrogenic compound, or the fungicide vinclozolin, an antiandrogenic compound, resulted in increased incidence of male infertility and decreased sperm production and viability in 90% of the males of four subsequent generations that were tracked (1).

Most notably, these reproductive effects were associated with derangements in DNA methylation patterns in the germ line, suggesting that epigenetic changes are passed on to future generations. The authors concluded, “The ability of an environmental factor (for example, endocrine disruptor) to reprogram the germ line and to promote a transgenerational disease state has significant implications for evolutionary biology and disease etiology” (6, p. 1466). This may suggest that the endocrine-disrupting, fragrance-laden personal care products and commercial cleaning supplies to which we are all exposed may trigger fertility problems in multiple future generations.

Transgenerational Inheritance of Traumatic Episodes: Parental Experience Shapes Traits of Offspring

In addition, traumatic experiences may be transmitted to future generations via epigenetics as a way to inform progeny about salient information needed for their survival (7). In one study, researchers wafted the cherry-like chemical acetophenone into the chambers of mice while administering electric shocks, conditioning the mice to fear the scent (7). This reaction was passed onto two successive generations, which shuddered significantly more in the presence of acetophenone despite never having encountered it compared to descendants of mice that had not received this conditioning (7).

The study suggests that certain characteristics of the parental sensory environment experienced before conception can remodel the sensory nervous system and neuroanatomy in subsequently conceived generations (7). Alterations in brain structures that process olfactory stimuli were observed, as well as enhanced representation of the receptor that perceives the odor compared to control mice and their progeny (7). These changes were conveyed by epigenetic mechanisms, as illustrated by evidence that the acetophenone-sensing genes in fearful mice were hypomethylated, which may have enhanced expression of odorant-receptor genes during development leading to acetophenone sensitivity (7).

The Human Experience of Famine and Tragedy Spans Generations

The mouse study, which illustrates how germ cells (egg and sperm) exhibit dynamic plasticity and adaptability in response to environmental signals, is mirrored by human studies. For instance, exposures to certain stressors such as starvation during the gestational period are associated with poor health outcomes for offspring. Women who undergo famine before conception of her offspring have been demonstrated to give birth to children with lower self-reported mental health and quality of life, for example (8).

Studies similarly highlight that, “Maternal famine exposure around the time of conception has been related to prevalence of major affective disorders, antisocial personality disorders, schizophrenia, decreased intracranial volume, and congenital abnormalities of the central nervous system” (8). Gestational exposure to the Dutch Famine of the mid-twentieth century is also associated with lower perceived health (9), as well as enhanced incidence of cardiovascular disease, hypertension, and obesity in offspring (8). Maternal undernourishment during pregnancy leads to neonatal adiposity, which is a predictor of future obesity (10), in the grandchildren (11).

The impact of epigenetics is also exemplified by research on the intergenerational effects of trauma, which illuminates that descendants of people who survived the Holocaust exhibit abnormal stresshormone profiles, and low cortisol production in particular (12). Because of their impaired cortisol response and altered stress reactivity, children of Holocaust survivors are often at enhanced risk for post-traumatic stress disorder (PTSD), anxiety, and depression (13).

Intrauterine exposure to maternal stress in the form of intimate partner violence during pregnancy can also lead to changes in the methylation status of the glucocorticoid receptor (GR) of their adolescent offspring (14). These studies suggest that an individual’s experience of trauma can predispose their descendants to mental illness, behavioral problems, and psychological abnormalities due to “transgenerational epigenetic programming of genes operating in the hypothalamic-pituitary-adrenal axis,” a complex set of interactions among endocrine glands which determine stress response and resilience (14).

Body Cells Pass Genetic Information Directly Into Sperm Cells

Not only that, but studies are illuminating that genetic information can be transferred through the germ line cells of a species in real time. These paradigm-shifting findings overturn conventional logic which postulates that genetic change occurs over the protracted time scale of hundreds of thousands or even millions of years. In a relatively recent study, exosomes were found to be the medium through which information was transferred from somatic cells to gametes.

This experiment entailed xenotransplantation, a process where living cells from one species are grafted into a recipient of another species. Specifically, human melanoma tumor cells genetically engineered to express genes for a fluorescent tracer enzyme called EGFP-encoding plasmid were transplanted into mice. The experimenters found that information-containing molecules containing the EGFP tracer were released into the animals’ blood (15). Exosomes, or “specialized membranous nano-sized vesicles derived from endocytic compartments that are released by many cell types” were found among the EGFP trackable molecules (16, p. 447).

Exosomes, which are synthesized by all plant and animal cells, contain distinct protein repertoires and are created when inward budding occurs from the membrane of multivesicular bodies (MVBs), a type of organelle that serves as a membrane-bound sorting compartment within eukaryotic cells (16). Exosomes contain microRNA (miRNA) and small RNA, types of non-coding RNA involved in regulating gene expression (16). In this study, exosomes delivered RNAs to mature sperm cells (spermatozoa) and remained stored there (15).

The researchers highlight that this kind of RNA can behave as a “transgenerational determinant of inheritable epigenetic variations and that spermatozoal RNA can carry and deliver information that cause phenotypic variations in the progeny” (15). In other words, the RNA carried to sperm cells by exosomes can preside over gene expression in a way that changes the observable traits and disease risk of the offspring as well as its morphology, development, and physiology.

This study was the first to elucidate RNA-mediated transfer of information from somatic to germ cells, which fundamentally overturns what is known as the Weisman barrier, a principle which states that the movement of hereditary information from genes to body cells is unidirectional, and that the information transmitted by egg and sperm to future generations remains independent of somatic cells and parental experience (15).

Further, this may bear implications for cancer risk, as exosomes contain vast amounts of genetic information which can be source of lateral gene transfer (17) and are abundantly liberated from tumor cells (18). This can be reconciled with the fact that exosome-resembling vesicles have been observed in various mammals (15), including humans, in close proximity to sperm in anatomical structures such as the epididymis as well as in seminal fluid (19). These exosomes may thereafter be propagated to future generations with fertilization and augment cancer risk in the offspring (20).

The researchers concluded that sperm cells can act as the final repositories of somatic cell-derived information, which suggests that epigenetic insults to our body cells can be relayed to future generations. This notion is confirmatory of the evolutionary theory of “soft inheritance” proposed by French naturalist Jean-Baptiste Lamarck, whereby characteristics acquired over the life of an organism are transmitted to offspring, a concept which modern genetics previously rejected before the epigenetics arrived on the scene. In this way, the sperm are able to spontaneously assimilate exogenous DNA and RNA molecules, behaving both as vector of their native genome and of extrachromosomal foreign genetic material which is “then delivered to oocytes at fertilization with the ensuing generation of phenotypically modified animals” (15).

Epigenetic Changes Endure Longer Than Ever Predicted

In a recent study, nematode worms were manipulated to harbor a transgene for a fluorescent protein, which made the worms glow under ultraviolet light when the gene was activated (21). When the worms were incubated under the ambient temperature of 20° Celsius (68° Fahrenheit), negligible glowing was observed, indicating low activity of the transgene (21). However, transferring the worms to a warmer climate of 25°C (77° F) stimulated expression of the gene, as the worms glowed brightly (21).

In addition, this temperature-induced alteration in gene expression was found to persist for at least 14 generations, representing the preservation of epigenetic memories of environmental change across an unprecedented number of generations (21). In other words, the worms transmitted memories of past environmental conditions to their descendants, through the vehicle of epigenetic change, as a way to prepare their offspring for prevailing environmental conditions and ensure their survivability.

Future Directions: Where Do We Go From Here?

Taken cumulatively, the aforementioned research challenges traditional Mendelian laws of genetics, which postulate that genetic inheritance occurs exclusively through sexual reproduction and that traits are passed to offspring through the chromosomes contained in germ line cells, and never through somatic (bodily) cells. Effectively, this proves the existence of non-Mendelian transgenerational inheritance, where traits separate from chromosomal genes are transmitted to progeny, resulting in persistent phenotypes that endure across generations (22).

This research imparts new meaning to the principle of seven generation stewardship taught by Native Americans, which mandates that we consider the welfare of seven generations to come in each of our decisions. Not only should we embody this approach in practices of environmental sustainability, but we would be wise to consider how the conditions to which we subject our bodies—the pollution and toxicants which permeate the landscape and pervade our bodies, the nutrient-devoid soil that engenders micronutrient-poor food, the disruptions to our circadian rhythm due to the ubiquity of electronic devices, our divorce from nature and the demise of our tribal affiliations—may translate into ill health effects and diminished quality of life for a previously unfathomed number of subsequent generations.

Hazards of modern agriculture, the industrial revolution, and contemporary living are the “known or suspected drivers behind epigenetic processes…including heavy metals, pesticides, diesel exhaust, tobacco smoke, polycyclic aromatic hydrocarbons, hormones, radioactivity, viruses, bacteria, and basic nutrients” (1, p. A160). Serendipitously, however, many inputs such as exercise, mindfulness, and bioactive components in fruits and vegetables such as sulforaphane in cruciferous vegetables, resveratrol from red grapes, genistein from soy, diallyl sulphide from garlic, curcumin from turmeric, betaine from beets, and green tea catechin can favorably modify epigenetic phenomena “either by directly inhibiting enzymes that catalyze DNA methylation or histone modifications, or by altering the availability of substrates necessary for those enzymatic reactions” (23, p. 8).

This quintessentially underscores that the air we breathe, the food we eat, the thoughts we allow, the toxins to which we are exposed, and the experiences we undergo may persevere in our descendants and remain in our progeny long after we are gone. We must be cognizant of the effects of our actions, as they elicit a ripple effect through the proverbial sands of time.

You can join the Greenmedinfo newsletter here for updates and more information about the world of health

References

1. Weinhold, B. (2006). Epigenetics: The Science of Change. Environmental Health Perspectives, 114(3), A160-A167.

2. Centers for Disease Control and Prevention. (2014). Exposome and Exposomics. Retrieved from https://www.cdc.gov/niosh/topics/exposome/

3. Rappaport, S.M. (2016). Genetic factors are not the major causes of chronic diseases. PLoS One, 11(4), e0154387.

4. Vrijheid, M. (2014). The exposome: a new paradigm to study the impact of environment on health. Thorax, 69(9), 876-878. doi: 10.1136/thoraxjnl-2013-204949.

5. Wild, C.P. (2012). The exposome: from concept to utility. International Journal of Epidemiology, 41, 24–32. doi:10.1093/ije/dyr236

6. Anway, M.D. et al. (2005). Epigenetic transgenerational actions of endocrine disruptors and male fertility. Science, 308(5727), 1466-1469.

7. Dias, B.G., & Ressler, K.J. (2014). Parental olfactory experience influences behavior and neural structure in subsequent generations. Nature Neuroscience, 17(1), 89-98.

8. Stein, A.D. et al. (2009). Maternal exposure to the Dutch Famine before conception and during pregnancy: quality of life and depressive symptoms in adult offspring. Epidemiology, 20(6), doi:  10.1097/EDE.0b013e3181b5f227.

9. Roseboom, T.J. et al. (2003). Perceived health of adults after prenatal exposure to the Dutch famine. Paediatrics Perinatal Epidemiology, 17, 391–397.

10. Badon, S.E. et al. (2014). Gestational Weight Gain and Neonatal Adiposity in the Hyperglycemia and Adverse Pregnancy Outcome Study-North American Region. Obesity (Silver Spring), 22(7), 1731–1738.

11. Veenendaal, M.V. et al. (2013). Transgenerational effects of prenatal exposure to the 1944-45 Dutch famine. BJOG, 120(5), 548-53. doi: 10.1111/1471-0528.

12. Yehuda, R., & Bierer, L.M. (2008). Transgenerational transmission of cortisol and PTSD risk. Progress in Brain Research, 167, 121-135.

13. Aviad-Wilcheck, Y. et al. (2013). The effects of the survival characteristics of parent Holocaust survivors on offsprings’ anxiety and depression symptoms. The Israel Journal of Psychiatry and Related Sciences, 50(3), 210-216.

14. Radke, K.M. et al. (2011). Transgenerational impact of intimate partner violence on methylation in the promoter of the glucocorticoid receptor. Translational Psychiatry, 1, e21. doi: 10.1038/tp.2011.21.

15. Cossetti, C. et al. (2014). Soma-to-Germline Transmission of RNA in Mice Xenografted with Human Tumour Cells: Possible Transport by Exosomes. PLoS One, https://doi.org/10.1371/journal.pone.0101629.

16. Zomer, A. et al. (2010). Exosomes: Fit to deliver small RNA. Communicative and Integrative Biology, 3(5), 447–450.

17. Balaj, L. et al. (2011) Tumour microvesicles contain retrotransposon elements and amplified oncogene sequences. Natural Communications, 2, 180.

18. Azmi, A.S., Bao, B., & Sarkar, F.H. (2013). Exosomes in cancer development, metastasis, and drug resistance: a comprehensive review. Cancer Metastasis Review, 32, 623-643

19. Poliakov, A. et al. (2009). Structural heterogeneity and protein composition of exosomes-like vesicles (prostasomes) in human semen. Prostate, 69, 159-167.

20. Cheng, R.Y. et al. (2004) Epigenetic and gene expression changes related to transgenerational carcinogenesis. Molecular Carcinogenesis, 40, 1–11.

21. Klosin, A. et al. (2017). Transgenerational transmission of environmental information in C. elegans. Science, 356(6335).

22. Lim, J.P., & Brunet, A. (2013). Bridging the transgenerational gap with epigenetic memory. Trends in Genetics, 29(3), 176-186. doi: 10.1016/j.tig.2012.12.008

23. Choi, S.-W., & Friso, S. (2010). Epigenetics: A New Bridge between Nutrition and Health Advances in Nutrition: An International Review Journal, 1(1), 8-16. doi:10.3945/an.110.1004.

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Brain Imaging Shows Autistic Brains Contain HIGH Amounts of Aluminum

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In Brief

  • The Facts:

    A study published early in 2018 identified very high amounts of aluminum lodged in the brains of multiple autistic people.

  • Reflect On:

    We know little about where the heavy metals used as adjuvants in vaccines and where they end up in the body. We now know that injected aluminum doesn't exit the body like aluminum intake from other sources. When injected, it ends up in the brain

A study published earlier in 2018 should have made headlines everywhere, as it discovered historically high amounts of aluminum in autistic brains. The study was conducted by some of the worlds leading scientists in the field.

Five people were used in the study, four males and one female, all between the ages of 14-50. Each of their brains contained unsafe and high amounts of aluminum compared to patients with other diseases where high brain aluminum content is common, like Alzheimer’s disease, for example.

Of course, this caused people to downplay the study, citing a low sample group, but that’s not entirely a valid argument given the reason why this study was conducted. As cited in the study above, recent studies on animals, published within the past few years, have supported a strong connection between aluminum, and aluminum adjuvants used in human vaccinations, and Autism Spectrum Disorder (ASD.)

Studies have also shown that injected aluminum does not exit the body, and can be detected inside the brain even a year after injection. That being said, when we take aluminum in from sources such as food, the body does a great job of getting it out, but there is a threshold. It’s important to acknowledge that the aluminum found in the brain, could be due to the presence of aluminum adjuvants in vaccines. This latest study also identified the location of aluminum in these tissues, and where they end up. This particular study was done on humans, which builds upon, and still supports, the findings of the animal studies.

This is also important because the majority of studies that previously examined human exposure to aluminum have only used hair, blood and urine samples. The study also makes a clear statement regarding vaccines, stating that “Paediatric vaccines that include an aluminum adjuvant are an indirect measure of infant exposure to aluminum and their burgeoning use has been directly correlated with increasing prevalence of ASD.”

 Aluminum, in this case, was found in all four lobes of the brain.

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The aluminum content of brain tissues from donors with a diagnosis of ASD was extremely high (Table 1). While there was significant inter-tissue, inter-lobe and inter-subject variability the mean aluminium content for each lobe across all 5 individuals was towards the higher end of all previous (historical) measurements of brain aluminium content, including iatrogenic disorders such as dialysisencephalopathy[13][15][16][17][18][19]. All 4 male donors had significantly higher concentrations of brain aluminum than the single female donor. We recorded some of the highest values for brain aluminum content ever measured in healthy or diseased tissues in these male ASD donors

We Know, And Have Known, Aluminum Is Not Safe, Yet We Ignore It

When we talk about the ‘safe’ amount of aluminum here, there is no such thing. Aluminum is extremely toxic to any biological process, it’s not meant for us which is why it stayed deep within the Earth until we took it out. It has no place within us, and that’s simply due to the fact that it causes nothing but havoc. This makes it odd that we would put them in vaccinations despite the fact that for 100 years there has been no appropriate safety testing.

Aluminum is an experimentally demonstrated neurotoxin and the most commonly used vaccine adjuvant. Despite almost 90 years of widespread use of aluminum adjuvants, medical science’s understanding about their mechanisms of action is still remarkably poor. There is also a concerning scarcity of data on toxicology and pharmacokinetics of these compounds. In spite of this, the notion that aluminum in vaccines is safe appears to be widely accepted. Experimental research, however, clearly shows that aluminum adjuvants have a potential to induce serious immunological disorders in humans.

The quote above comes from a study published in 2011, it’s 2018 now and we’ve come along way in our understanding. We are starting to see even more research confirming the statement above.

Almost every study you read regarding previous studies on aluminum adjuvants within vaccines emphasized how the nature of its bioaccumulation is unknown, and a serious matter. We now know that it goes throughout the body, into distant organs eventually ends up in the brain.

Another fairly recent study from 2015 points out:

Evidence that aluminum-coated particles phagocytozed in the injected muscle and its draining lymph notes can disseminate within phagocytes throughout the body and slowly accumulate in the brain further suggests that alum safety should be evaluated in the long term.(source)

The pictures below come from the recent 2018 study and show ‘bright spots’ that indicate heavy metals in the brain.

 

The more recent study discussed in this article is adding to that evidence. Below you can watch one of the most recent interviews with Dr. Eric Exly, one of the world’s foremost leading authors on the subject, and one of the authors of this most recent study. He is a Biologist (University of Stirling) with a Ph.D. in the ecotoxicology of aluminum. You can read more about his background here.

Take Away

People need to understand that despite media bullying, it’s ok to question vaccine safety, and there is plenty of reason to. There are many concerns, and heavy metals are one of them. In fact, the persistence and abundant presence of heavy metals in our environment, foods and medications is a concern, one that has been the clear cause for a variety of health ailments, yet it’s one that’s hardly addressed by the medical industry.

You can detox from this with items such as Spirulina, and waters that contain a high Silica content. There are studies that show various methods of detoxing can be used to get this lodged aluminum, or some of it, out of your body, organs and brain. This is where educating yourself regarding the medicinal value of food and nutrition is a key Perhaps this can be a motivation to better your diet, especially if you have, are someone, or know someone with an ASD diagnosis.

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The CDC’s Influenza Math Doesn’t Add Up: Exaggerating the Death Toll to Sell Flu Shots

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In Brief

  • The Facts:

    The flu shot is irresponsibly marketed, unnecessary and in some cases dangerous. This perspective comes from many people and health professionals, yet it's a narrative that's constantly ignored.

  • Reflect On:

    Is a flu shot really necessary? Are our immune systems suffering from a lack of real immunity? Are vaccines doing more harm than good?

Every year at about this time, public health officials and their media megaphones start up the drumbeat to encourage everyone (including half-year-old infants, pregnant women and the invalid elderly) to get a flu shot. Never mind that more often than not the vaccines don’t work, and sometimes even increase the risk of getting sick.

To buttress their alarmist message for 2018-2019, representatives from the Centers for Disease Control and Prevention (CDC) and other health agencies held a press conference and issued a press release on September 27, citing a particularly “record-breaking” (though unsubstantiated) 80,000 flu deaths last year. Having “medical experts and public health authorities publicly…state concern and alarm (and predict dire outcomes)” is part and parcel of the CDC’s documented playbook for “fostering public interest and high…demand” for flu shots. CDC’s media relations experts frankly admit that “framing” the current flu season as “more severe than last or past years” or more “deadly” is a highly effective strategy for garnering strong interest and attention from both the media and the public.

If accurate, 80,000 deaths would represent an enormous (and mystifying) one-year jump—tens of thousands more flu deaths compared to the already inflated numbers presented for 2016 (and every prior year).

Peter Doshi (associate editor at The BMJ and a MIT graduate) has criticized the CDC’s “aggressive” promotion of flu shots, noting that although the annual public health campaigns deliver a “who-in-their-right-mind-could-possibly-disagree message,” the “rhetoric of science” trotted out each year by public health officials has a “shaky scientific basis.” Viewed within the context of Doshi’s remarks, the CDC’s high-flying flu numbers for 2017-2018 raise a number of questions. If accurate, 80,000 deaths would represent an enormous (and mystifying) one-year jump—tens of thousands more flu deaths compared to the already inflated numbers presented for 2016 (and every prior year). Moreover, assuming a roughly six-month season for peak flu activity, the 80,000 figure would translate to an average of over 13,300 deaths per month—something that no newspaper last year came close to reporting.

The CDC’s statistics are impervious to independent verification because they remain, thus far, unpublished—despite the agency’s pledge on its website to base its public health pronouncements on high-quality data derived openly and objectively. Could the CDC’s disappointment with influenza vaccination coverage—which lags far behind the agency’s target of 80%—have anything to do with the opacity of the flu data being used to peddle the unpopular and ineffective vaccines?

Fudging facts

There are a variety of reasons to question the precision with which the CDC likes to imbue its flu statistics. First, although the CDC states that it conducts influenza mortality surveillance with its partner agencies, there is no actual requirement for U.S. states to report adult flu deaths to the CDC. (In public health parlance, adult influenza deaths are not “reportable” or “nationally notifiable.”) In fact, the only “flu-associated deaths” that the CDC requires states and other jurisdictions to report are deaths in children—180 last year.

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…when actual death certificates are tallied, influenza deaths on average are little more than 1,000 yearly.

How did the CDC reach its as-yet-unpublished conclusion—widely shared with the media—that 79,820 American adults in addition to 180 children died from the flu in 2017-2018? The agency states that it relies on death certificate data. However, members of the Cochrane research community have observed that “when actual death certificates are tallied, influenza deaths on average are little more than 1,000 yearly.”

Other knowledgeable individuals have also noted that the death records system in the U.S. is subjective, incomplete and politicized, and have suggested that citizens should adopt a “healthy skepticism about even the most accepted, mainstream, nationally reported CDC or other ‘scientific’ statistics.” This skepticism may be especially warranted for the influenza stats, which are so inextricably intertwined with the CDC’s vaccination agenda that the statistical techniquesand assumptions that the agency uses focus specifically on “project[ing] the burden of influenza that would have occurred in the absence of vaccination.”

skepticism may be especially warranted for the influenza stats, which are so inetricably intertwined with the CDC’s vaccination agenda.

Notwithstanding its incessant use of influenza statistics to justify its flu vaccine policies, the CDC tries to have it both ways, cautioning that because “influenza activity reporting…is voluntary,” influenza surveillance in the U.S. “cannot be used to ascertain how many people have become ill with influenza during the influenza season.” A larger problem is that the vital statistics that form the basis of the CDC’s surveillance data conflate deaths from pneumonia and influenza (P&I). The CDC concedes that this conflation complicates the challenge of specifically estimating flu deaths:

The system “tracks the proportion of death certificates processed that list pneumonia or influenza as the underlying or contributing cause of death. This system…does not provide an exact number of how many people died from flu” [emphasis added].

Curiously, the CDC presented its cause-of-death data slightly differently prior to 2015. Through 2014, the agency’s annual National Vital Statistics Reports included tables showing influenza deaths and pneumonia deaths as separate line items. Those reports made it abundantly clear that pneumonia deaths (at least as transmitted by death certificates) consistently and dramatically outstripped influenza deaths. The table below illustrates this pattern for 2012-2014.

Starting in 2015, the annual vital statistics reports began displaying P&I together and eliminated the distinct line items. At present, only one tool remains to examine mortality associated with influenza as distinct from pneumonia—the CDC’s interactive FluView dashboard—which provides weekly national breakdowns. The dashboard shows the same general pattern as in the annual reports—that is, lower numbers of influenza deaths and much higher numbers of pneumonia deaths. Bearing in mind all the shortcomings and potential biases of death certificate data, dashboard reports for the first week of March (week 9) for the past three years show 257 influenza deaths versus 4,250 pneumonia deaths in 2016, and 534 and 736 flu deaths (versus over 4,000 annual pneumonia deaths) in 2017 and 2018, respectively.

When clinicians in outpatient settings do order testing, relatively few of the “flu” specimens—sometimes as low as 1%—actually test positive for influenza.

Semantic shenanigans

Semantics also play a key role in the CDC’s slippery communications about “flu.” For example, CDC’s outpatient surveillance focuses on the broad category of “influenza-like illness” (ILI)—an almost meaningless term describing general symptoms (fever, cough and/or sore throat) that any number of non-influenza viruses are equally capable of triggering. Cochrane lists several problems with the reliance on ILI to make inferences about influenza:

  • There is “no reliable system to monitor and quantify the epidemiology and impact of ILI” and no way of knowing what proportion of ILI is caused by influenza.
  • There are almost no reliable data on the number of ILI-related physician contacts or hospitalizations—and no one knows what proportion of ILI doctor visits and hospitalizations are due to influenza.

“Pneumonia,” too, is a catch-all diagnosis covering lung infections caused by a variety of different agents: viruses (non-influenza as well as influenza), bacteriafungiair pollutants and many others. Interestingly, hospitalization is a common route of exposure to pneumonia-causing pathogens, and mortality from hospital-acquired pneumonia exceeds 60%. In a plausible scenario, an adult hospitalized for suspected (but unconfirmed) “flu” could acquire a lethal pneumonia bug in the hospital, and their death might be chalked up to “flu” regardless of the actual facts, particularly because clinicians do not necessarily order influenza testing. When clinicians in outpatient settings do order testing, relatively few of the “flu” specimens—sometimes as low as 1%—actually test positive for influenza. Over the past couple of decades, the proportion of specimens testing positive has averaged around 15%—meaning that about 85% of suspected “flu” specimens are not, in fact, influenza.

Roughly four-fifths of the vaccine injury and death cases settled through the National Vaccine Injury Compensation Program are flu-vaccine-related.

Propaganda with a purpose

It takes little subtlety to recognize that the principal reason for flu hyperbole is to sell more vaccines. However, more and more people—even infectious disease specialists—are realizing that flu shots are fraught with problems. Roughly four-fifths of the vaccine injury and death cases settled through the National Vaccine Injury Compensation Program are flu-vaccine-related. A University of Toronto-based expert recently stated, “We have kind of hyped this vaccine so much for so long we are starting to believe our own hype.”

Pro-flu-vaccination studies—through their skillful placement in prestigious journals—tend to drown out other influenza studies that should be ringing warning bells. Published peer-reviewed studies show that:

  • Previous influenza vaccination, particularly in those who get a flu shot every year, diminishes or “blunts” the already low effectiveness of flu shots.
  • Getting vaccinated against influenza increases susceptibility to other severe respiratory viruses and also to other strains of influenza.
  • Mothers who receive influenza vaccines during pregnancy face an increased risk of miscarriages and their offspring face elevated risks of birth defects and autism.

A systematic review of influenza vaccine trials by Cochrane in 2010 urges the utmost caution. Noting that “studies funded from public sources [have been] significantly less likely [than industry-funded studies] to report conclusions favorable to the vaccines,” and citing evidence of “widespread manipulation of conclusions,” the Cochrane reviewers’ bottom line is that “reliable evidence on influenza vaccines is thin.” We should all keep those words in mind the next time the CDC and the media try to mischaracterize flu facts and science.

CHD is planning many strategies, including legal, in an effort to defend the health of our children and obtain justice for those already injured.  Your support is essential to CHD’s successful mission. Please visit our crowdfunding page.

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