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Brain Regeneration: Can Infrared Light Reverse Parkinson’s & Alzheimer’s?

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This article was written by Ali Le Vere at Greenmedinfo.com. It’s republished here with their permission. For more information from Greenmedinfo, you can sign up for the newsletter here.

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Contrary to conventional wisdom, brain regeneration is possible. One promising therapy that promotes neurogenesis and is effective in pre-clinical studies of Alzheimer’s and Parkinson’s is near infrared light therapy, and it may improve other mental illnesses and neurodegenerative disorders including dementia, stroke, ALS, and traumatic brain injury as well.

Alzheimer’s disease and Parkinson’s disease are the most common neurodegenerative disorders. The former is a type of dementia that occurs secondary to the accumulation of abnormal protein deposits in the brain, including β-amyloid plaques and intraneuronal neurofibrillary tangles made of tau protein (1). Upon neuroimaging studies, gross cerebral cortical atrophy is found, meaning that the part of the brain responsible for executive functions such as learning, memory, language, decision-making, and problem-solving progressively degenerates (1). In addition, gliosis, or brain inflammation, is a hallmark characteristic of Alzheimer’s (1).

One hypothesis that is championed proposes that Alzheimer’s occurs due to self-propagating, prion-like protein assemblies, which interfere with the function of nerve cells (2). An alternate theory is that these so-called proteinopathies occur secondary to a microvascular hemorrhage or brain bleed (3). The brain bleed is believed to be the result of age-induced degradation of cerebral capillaries, which creates neuron-killing protein plaques and tangles (3).

Dysfunction of mitochondria, the energy-generating powerhouses of the cell, is also implicated in Alzheimer’s, as reduced efficacy of these organelles creates oxidative stress-inducing reactive oxygen species, or free radicals, which lead to neuronal cell death (4). Whatever the cause, extensive death of brain cells occurs, which explains the cognitive deficits that occur with Alzheimer’s disease, in addition to symptoms such as impaired judgment, confusion, agitation, linguistic abnormalities, social withdrawal, and even hallucinations (1).

Parkinson’s disease, on the other hand, is characterized by progressive death of dopamine-producing neurons in a region of the brainstem called the substantial nigra, but it can extend to other brain areas such as the locus coeruleus, olfactory bulb, dorsal motor nucleus of the vagal nerve, and even the cortex in late stages (5). As a result, the primary manifestation is that dopamine deficiency appears in the basal ganglia, a set of nuclei embedded deep in the brain hemispheres that is responsible for motor control (6). This leads to the cardinal manifestation of Parkinson’s, namely, a movement disorder that includes bradykinesia or slow movement, loss of voluntary movement, muscular rigidity, and resting tremor (7).

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Not unlike what happens in Alzheimer’s, accumulation of abnormal intracellular protein aggregates known as Lewy bodies, composed of a protein called α-synuclein, is thought to be central to the pathogenesis of Parkinson’s disease (8). Like Alzheimer’s, mitochondrial dysfunction induced by genetic mutations, toxic agents, or damage to blood vessels is also considered to contribute to neuron cell death in Parkinson’s (9). Toxin exposure is especially implicated, as animal studies hint that development of Parkinson’s disease may occur as a byproduct of exposure to neurotoxins such as rotenone or paraquat (10). Impaired blood brain barrier function and damage to the endothelial cells of the vascular system, which line the interior surface of blood vessels, are also thought to play a role in Parkinson’s (10).

Overturning Old Notions of Neuroscience

The central dogma of neuroscience conceived of the central nervous system tissue as “perennial” after the doctrines of Giulio Bizzozero, the most prominent Italian histologist, who decreed that the lifelong cells of the nervous system were devoid of replicative potential (11). In other words, the perennial nature ascribed to the nerve cells of the brain and spinal cord meant that nerve cells were believed to be incapable of undergoing proliferation, or cell division, in the postnatal brain (11). While the early stage of in utero prenatal development known as embryogenesis permits massive neurogenesis, or the ability to create new nerve cells, the scientific consensus up until the end of the twentieth century held that neurogenesis was arrested after birth in mammals.

Santiago Ramon y Cajal, who led the charge in the neuroscience discipline in the later half of the nineteenth century onward and won a Nobel Prize for Medicine and Physiology, in fact stated that: “Once development was ended, the fonts of growth and regeneration of the axons and dendrites dried up irrevocably. In adult centers, the nerve paths are something fixed and immutable: everything may die, nothing may be regenerated” (11). Acknowledgment of the mere possibility of adult neurogenesis was hampered by the fact that scientists lacked the visualization techniques to detect neural stem cells, the precursors to new neurons and means by which neurogenesis occurs, and also did not have access to the molecular markers and microscopy required to observe cells in different cycle phases.

This view of nervous tissue as perennial was also reinforced by clinical observations that patients with chronic neurodegeneration, traumatic brain lesions, and cerebrovascular diseases do not experience functional recovery (11). Prevailing theories posited that adult neurogenesis was an evolutionary unlikelihood, since it would interfere with pre-existing neuronal connections and the fine-tuned electrochemical communication in the nervous system, as well as disrupt memory recall, which was believed to occur via stable neuronal circuits created and encoded during learning (11).

That brain cells are finite, and incapable of regeneration, painted a portrait of doom and gloom and inexorable debilitation for patients suffering from devastating neurodegenerative conditions. However, relatively recent discoveries have overturned these antiquated conceptions by revealing that the brain is plastic, or pliable, and that even neurons in adult higher vertebrates are capable of neurogenesis.

Scientists Discover Neural Regeneration is Possible

In the 1960s, these postulates of the old neurobiology were disproven when Joseph Altman and colleagues performed an experiment where radioactively labelled thymidine, one of the nucleotide base pairs that makes up DNA, was incorporated into a brain area called the dentate gyrus of the hippocampus and integrated into the genetic material of what was later confirmed via electron microscopy to be dividing neurons (12, 13). In essence, this illustrated that neurons were undergoing mitosis, a process of cell division where genetically identical daughter cells are created, and showed that adult neurogenesis is possible.

Another nail in the coffin of this antiquated perception of the nervous system was that neural stem cells, the multipotent, self-renewing progenitors from which new neurons arise, were found in the brains of adult mammals, and discovered to undergo expansion in their populations when prompted by signaling molecules called growth factors and morphogens (11). The multiplication and differentiation of neural stem cells, which are residents of the central nervous system, is essential for neurogenesis (14). Neural stem cells are capable of generating all of the cell types of the nervous system, including astrocytes, glial cells, and what are called oligodendrocytes in the central nervous system and Schwann cells in the peripheral nervous system (11). Researchers Colucci-D’Amato and Bonita in fact state that, “To date neural stem cells have been isolated from nearly all areas of the embryonic brain and in a growing list of adult mammalian brain areas, including cerebellum and cortex” (11, p. 268).

Other advances, such as confocal microscopy and the identification of cellular markers which allowed the phenotype of cells to be characterized all culminated in the realization that neurogenesis occurs continuously in some brain area, such as the hippocampus and subventricolar zone (SVZ), the former of which is responsible for the formation and consolidation of memories (11). To date, neurogenesis has been shown to be influenced by various chemical, pharmacological, and environmental stimuli. For instance, work by researcher Fernando Nottebohm demonstrated the spontaneous replacement of neurons in the adult avian brain (15). In song birds such as canaries, which experience seasonal modification in their songs, new neurons are recruited into their neuronal circuitry in a way that may be dependent upon social and reproductive interactions, territorial defense, migratory patterns and food caching (15).

This all should serve as a beacon of hope for patients experiencing the ravages of neurodegenerative disease, as it may mean that epigenetics, or the way gene expression changes based on lifestyle factors, may lend itself to neurogenesis and the reversal of these scourges of mankind. For example, researchers state that an enriched environment, learning, exercise, exposure to different odorant molecules, and drugs such as antidepressants, steroids, and alcohol can all favorably or unfavorably impact neurogenesis  (11). These newfound revelations are being used in fact as an impetus to find cures for a laundry list of neurodegenerative diseases (11).

Novel Therapy Shown to Grow New Nerve Cells

Despite this research, the prevailing view of neurodegenerative diseases such as Alzheimer’s and Parkinson’s is that their underlying pathophysiology, a relentless progression of neuronal death, remains irreversible (10). Thus far, then, approaches have aimed to slow or stop neuronal cell death or to develop disease-modifying treatments that could stabilize the rate of neurodegeneration (10). One non-pharmacological therapy that may be able to actually regenerate brain cells, however, is light in the near infrared range, also known as low-level laser or light emitting diode (LED) therapy that utilizes wavelengths in the red to infrared spectrum.

Near infrared light therapy has the potential to “mitigate ubiquitous processes relating to cell damage and death,” and may have applications in conditions that “converge on common pathways of inflammation and oxidative stress” (10). This is demonstrated by the widespread efficacy of near infrared light therapy in improving conditions including traumatic brain injury, ischemic stroke, major depression, and age-related macular degeneration (10). In traumatic brain injury, for example, treatment with near infrared light improves social, interpersonal, and occupational functions, reduces symptoms of post-traumatic stress disorder (PTSD), and is helpful for sleep (16).

Because near infrared light treatment improves cognitive and emotional dimensions (17) and enhances short-term memory and measures of sustained attention (18), researchers have long suspected its potential for neuropsychological disorders. In a revolutionary publication, scientists propose that infrared light is superior to pharmacological standard of care for these debilitating conditions given its neuron-saving abilities (10).

For instance, in mouse models of traumatic brain injury, near infrared light increases levels of brain-derived neurotrophic factor (BDNF), a protein which helps dying nerve cells survive (19). In addition, infrared light both improves neurological performance and increases the numbers of neuroprogenitor cells, the precursors to new neurons, in areas of the brain such as the dentate gyrus of the hippocampus and the sub ventricular zone (20).

Near Infrared Light Therapy in Alzheimer’s and Parkinson’s

Although human trials have not been yet conducted in Alzheimer’s disease, mouse studies show that near infrared treatment reduces its characteristic proteinopathies, decreasing brain levels of β-amyloid plaques and neurofibrillary tangles of tau proteins, while also ameliorating cognitive deficits (10). Cellular energy production, as indicated by levels of ATP, were increased in these studies alongside bolstered mitochondrial function and (10). In transgenic mouse models of Alzheimer’s, application of non-thermal near infrared light reversed significant deficits in working memory and significantly improved cognitive performance (21).

In animal models of Parkinson’s, near infrared treatment has been shown to rescue dopaminergic neurons, the subset that degenerate in this condition, from death (10). In addition, near infrared light treatment corrects the abnormal firing activity of neurons in deep subthalamic brain regions that occurs in parkinsonian conditions (22). Various animal models of Parkinson’s disease shown improved motor control and locomotor activity, as measured by both mobility and velocity, after near infrared is applied (10).

In a macaque monkey model of Parkinson’s, an optical fiber device that administered near infrared to the midbrain largely prevented the development of clinical signs of Parkinson’s when the animals were injected with a chemical known to induce this disorder (23). It also preserved a greater number of dopaminergic nigral cells compared to the monkeys that had not received infrared treatment (23). Limited case reports in humans have shown that near infrared administered through an intranasal apparatus improves symptoms in the majority of Parkinson’s patients, and that its application to the back of the head and upper neck reduced signs of Parkinson’s in one patient (10). Other reports indicate that gait, speech, cognitive function, and freezing episodes were improved in late-stage Parkinson’s patients who undertook this therapy (24), but the study was low-quality (10).

Mechanism of Action: How Near Infrared Promotes Neurogenesis

The ways in which near infrared promotes neurogenesis are multi-fold. There is evidence that near infrared light exerts a hormetic effect, acting as an adaptive or positive stressor. Another example of a hormetic effect is that exhibited by phytonutrients in fruits and vegetables, which act as antioxidants by paradoxically stimulating oxidative damage via a pro-oxidant mechanism. This in turn up-regulates our endogenous antioxidant defense system. Similarly, near infrared light activates cellular stress response systems by targeting a key enzyme in the electron transport chain which is responsible for mitochondrial-based energy production called cytochrome c oxidase, an enzyme that is fundamental to the cellular bioenergetics of nerve cells (25).

By accepting light in the near infrared range of the electromagnetic spectrum, this enzyme induces a change in the electrochemical potential of the mitochondrial membrane, jump-starting production of the cellular energy currency called adenosine triphosphate (ATP) and causing a mild burst in the synthesis of reactive oxygen species (ROS) (10). As a result, downstream signaling pathways are triggered which induce reparative and neuroprotective mechanisms, including neurogenesis, the creation of new synapses, and brain-based antioxidant and metabolic effects (25).

Restoration of mitochondrial function in the endothelial cells lining cerebral blood vessels may also help neurons survive by repairing the blood-brain barrier and vascular network which is compromised in neurogenerative conditions (10). Impressively, “This modulation of multiple molecular systems appears capable of both conditioning neurons to resist future damage and accelerating repair of neurons damaged by a previous or continuing insult” (10).

On the other hand, the application of near infrared light has been shown to elicit systemic effects, possibly via circulating molecular factors (10). In other words, light in the near infrared spectrum applied to a local area elicits benefits in distal tissues remote from the initial site, perhaps by stimulating immune cells that have a neuroprotective role (10). Another way in which near infrared light activates global effects in the body is by up-regulating the production of signaling molecules known as anti-inflammatory cytokines, while down-regulating pro-inflammatory cytokines (26).

Near infrared also mobilizes tissue repair processes by improving the migration of white blood cells to wounds, increasing neovascularization, or the formation of new blood vessels, and facilitating formation of collagen (27). There is also evidence that near-infrared light exposure causes stem cells from the bone marrow to navigate to the site of damage and to release so-called trophic factors such as BDNF, which enhances nerve cell function and survival (28). Lastly, a system of communication between the mitochondria in the brain and the mitochondria in the tissues may be at play, so that application of near infrared light at a point in the body far from the brain can lead to neural regeneration (10).

Practical Application of Near Infrared Light Therapy

The key to mitigating the burden of chronic illness lies in physiological regeneration, which is emerging as a physiological inevitability, even in regions of the body where it was previously not thought possible. The ability to regenerate, secondary to normal biological processes of cellular erosion and decay, is programmed into our body in order for us to regain homeostasis.

So-called “photobiomodulation,” which includes near infrared light therapy, has limitless possible applications, and has even been shown to improve animal models of wound healing, heart attack, spinal cord injury, stroke, arthritis, familial amylotropic lateral sclerosis (FALS), diabetic ulcers, carpal tunnel syndrome, major depression, generalized anxiety disorder, frontotemporal dementia (29) and traumatic brain injury (27).

The biggest obstacle with infrared light therapy in neurodegenerative disease is targeting the zone of pathology, “when there are many intervening body tissues, namely skin, thick cranium, and meninges, and brain parenchyma,” since there is considerable dissipation of the signal across each millimeter of brain tissue (10). This is less problematic in Alzheimer’s, where the target regions are more superficial structures, but less easily rectified in the case of Parkinson’s, where there is significant distance from cranium to the brainstem where neurodegeneration takes place (10).

With Alzheimer’s, optimal delivery would be a near infrared light-emitting helmet worn over the entire cranium (10). Parkinson’s patients can achieve symptomatic relief when near infrared is applied in this fashion, as this would influence the abnormal neural circuitry in the cortex. However, to circumvent the problem of the sheer distance to the region of pathology in the brainstem, researchers propose that the minimally invasive surgical implantation of an optical fiber device near the brain parenchyma would be ideal, which would deliver therapeutic levels of near infrared (10). Until these options are commercially available, photobiomodulation devices or near infrared saunas may be a viable option, although human studies have not proved their efficacy.

Given its large margin of safety and lack of adverse effects, near infrared light therapy should be offered as an option for patients suffering from a myriad of chronic conditions, but is especially promising for neurodegenerative diseases including Alzheimer’s and Parkinson’s and may even have future use in multiple sclerosis. Near infrared therapy is superior to the mainstay drug treatments for these diseases since pre-clinical studies have demonstrated proof-of-concept that near infrared either arrests or slows the underlying pathology of these disease processes, and leads to the birth of new neurons, rather than merely mitigating symptoms (10).


References

1. Bird, T.D. (1998). Alzheimer disease overview. GeneReviews® [Internet]. Retrieved from https://www.ncbi.nlm.nih.gov/books/NBK1161/

2. Goedert, M. (2015). Alzheimer’s and Parkinson’s diseases: the prion concept in relation to assembled Aβ, tau, and α-synuclein. Science, 349, 1255555.

3. Stone, J. (2008). What initiates the formation of senile plaques? The origin of Alzheimer-like dementias in capillary haemorrhages. Medical Hypotheses, 71, 347–359.

4. Gonzalez-Lima, F., Barksdale B.R., & Rojas J.C. (2014). Mitochondrial respiration as a target for neuroprotection and cognitive enhancement. Biochemical Pharmacology, 88, 584–593. 10.1016/j.bcp.2013.11.010

5. Bergman, H., & Deuschl, G. (2002). Pathophysiology of Parkinson’s disease: from clinical neurology to basic neuroscience and back. Movement Disorders, 7(Suppl. 3), S28–S40.

6. Lanciego, J.L., Luquin, N., & Obeso, J.A. (2012). Functional Neuroanatomy of the Basal Ganglia. Cold Springs Harbor Perspectives in Medicine, 2(12), a009621.

7. De Virgilio, A. et al. (2016). Parkinson’s disease: Autoimmunity and neuroinflammation. Autoimmunity Reviews, 15(10), 1005-1011. doi: 10.1016/j.autrev.2016.07.022.

8. Gitler A.D. et al. (2009). Alpha-synuclein is part of a diverse and highly conserved interaction network that includes PARK9 and manganese toxicity. Natural Genetics, 41, 308–315.

9. Exner, N. et al. (2012). Mitochondrial dysfunction in Parkinson’s disease: molecular mechanisms and pathophysiological consequences. EMBO Journal, 31, 3038–3062. 10.1038/emboj.2012.170

10. Johnstone, D.M. et al. (2015). Turning On Lights to Stop Neurodegeneration: The Potential of Near Infrared Light Therapy in Alzheimer’s and Parkinson’s Disease. Frontiers in Neuroscience, 9, 500. doi:  10.3389/fnins.2015.00500

11. Colucci-D’Amato, L., & Bonavita, V. (2006). The end of the central dogma of neurobiology: stem cells and neurogenesis in adult CNS. Neurological Science, 27(4), 266-270.

12. Altman, J. (1962). Are new neurons formed in the brains of adult mammals? Science, 135, 1127-1128.

13. Kaplan, M.S., & Hinds, J.W. (1977). Neurogenesis in the adult rat: electron microscopic analysis of light radioautographs. Science, 197, 1092-1094.

14. Martino, G. et al. (2011). Brain regeneration in physiology and pathology: the immune signature driving therapeutic plasticity of neural stem cells. Physiological Reviews, 91(4), 1281-1304.

15. Nottebohm, F. (2002). Why are some neurons replaced in adult brain? Journal of Neuroscience, 22(3), 624-628.

16. Naeser, M.A. et al. (2014). Significant improvements in cognitive performance post-transcranial, red/near-infrared light-emitting diode treatments in chronic, mild traumatic brain injury: open-protocol study. Journal of Neurotrauma, 31,(11), 1008-1017.  doi: 10.1089/neu.2013.3244.

17. Barrett, D.W., & Gonzalez-Lima, F. (2013). Transcranial infrared laser stimulation produces beneficial cognitive and emotional effects in humans. Neuroscience, 230, 13-23.  doi: 10.1016/j.neuroscience.2012.11.016.

18. Blanco, N.J., Maddox, W.T., & Gonzalez-Lima, F. (2015). Journal of Neuropsychology, 11(1),14-25. doi: 10.1111/jnp.12074.

19. Xuan, W. et al. (2013). Transcranial low-level laser therapy improves neurological performance in traumatic brain injury in mice: effect of treatment repetition regimen. PLoS ONE, 8, e53454.

20. Xuan, W. et al. (2014). Transcranial low-level laser therapy enhances learning, memory, and neuroprogenitor cells after traumatic brain injury in mice. Journal of Biomedical Optics, 191(10), 108003.

21. Michalikova, S. et al. (2008). Emotional responses and memory performance of middle-aged CD1 mice in a 3D maze: effects of low infrared light. Neurobiology of Learning and Memory, 89(4), 480-488.

22. Shaw, V.E. et al. (2012). Patterns of Cell Activity in the Subthalamic Region Associated with the Neuroprotective Action of Near-Infrared Light Treatment in MPTP-Treated Mice. Parkinsonian Disease, 2012, 29875. doi: 10.1155/2012/296875.

23. Darlot, F. et al. (2016). Near-infrared light is neuroprotective in a monkey model of Parkinson disease. Annals of Neurology, 79(1), 59-65. doi: 10.1002/ana.24542.

24. Maloney, R., Shanks, S., & Maloney J. (2010). The application of low-level laser therapy for the symptomatic care of late stage Parkinson’s disease: a non-controlled, non-randomized study. American Society of Laser Medicine and Surgery, 185.

25. Rojas, J.C., & Gonzalez-Lima, F. (2011). Low-level light therapy of the eye and brain. Eye and Brain, 3, 49–67.

26. Muili, K.A. et al. (2012). Amelioration of experimental autoimmune encephalomyelitis in C57BL/6 mice by photobiomodulation induced by 670 nm light. PLoS ONE, 7, e30655.

27. Chung, H. et al. (2012). The Nuts and Bolts of Low-level Laser (Light) Therapy. Annals of Biomedical Engineering, 40(2), 516-533.gma

28. Hou, S.T. et al. (2008). Permissive and Repulsive Cues and Signalling Pathways of Axonal Outgrowth and Regeneration. International Review of Cell and Molecular Biology, 267, 121-181.

29. Purushothuman, S. et al. (2013). The impact of near-infrared light on dopaminergic cell survival in a transgenic mouse model of parkinsonism. Brain Research, 1535, 61–70.

Free David Wilcock Screening: Disclosure & The Fall of the Cabal

We interviewed David about what is happening within the cabal and disclosure. He shared some incredible insight that is insanely relevant to today.

So far, the response to this interview has been off the charts as people are calling it the most concise update of what's happening in our world today.

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Will Cyrptocurrencies Disrupt Central Banking? US Congress Meets To Discuss

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In Brief

  • The Facts:

    Witnesses are testifying at a House Financiancial Services subcommittee on whether the government should consider cryptocurrencies as money and their uses. This could be a monumental moment in central banking disruption.

  • Reflect On:

    Are you aware of how fiat currency currently works? Are you aware of what the central banking system is? Cryptocurrency has the potential to be a bridge that disrupts these enslaving systems and brings power back to people.

The buzz about cryptocurrencies seems to be as volatile as cryptocurrencies themselves, there is always a ‘war for our perception’ when it comes to something so threatening to the current financial system, and the way we’ve been doing things for years with regards to finance. Everybody from billionaires like tech investor Tim Draper, to ex-Facebook executive Chamath Palipitaya, to entertainers like Akon and more, have all urged the masses to look into Bitcoin, the current dominant cryptocurrency.

What makes cryptocurrencies so popular is not their potential to make a lot of money, but, from a financial perspective, it’s simply another option from “autocratic regimes and baking infrastructure that we know is corrosive to how the world needs to work properly.” Said Palipitaya about cryptocurrency a couple of years ago. “You cannot have Central Banks infinitely printing currency, you cannot have folks with misguided and misdirected monetary and fiscal policy…It’s a con game.”

Anything that can disrupt modern day banking is huge, given that this is the control hub of the Deep State and the ‘Cabal’ that seems to have its tentacles spread over all aspects of humanity, preventing solutions from being implemented quicker. Christine Lagarde, head of the International Monetary Fund has even cautioned that cryptocurrencies could displace central banks and conventional banking in the long term.

Clearly a good thing for us, but not the financial elite who continue to benefit from a system that only serves a select few.

Cryptocurrencies can change this.

It’s About Disrupting Old Outdated Ways

Whether you know much about cryptocurrencies, and Bitcoin or not, know that it threatens something so central, and something that has been holding humanity back in many ways for a long time. Banking.

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Any disruption to our current banking system is a great leap forward, in any form, but ultimately, any type of currency with any type of value is not really needed here on planet Earth. But that’s a transition to take place over a period of multiple years if we choose to head the right direction as a collective.

This is why it would not be a surprise to see some big-time restrictions and regulation put on cryptocurrencies, like Bitcoin, in the United States and other parts of the world. There already has been, but at this point, Bitcoin doesn’t really seem like it can be stopped and looks like something that has a bright future, according to many. That being said, a lot of mainstream media and some big-time popular financial figures have constantly bashed and criticized it.

And others:

“Bitcoin is money outside of government, controlled by the people who become their own sovereignty. Bitcoin will do the nation-state what the printing press did to the church…The use-case as stateless money drives adoption because it’s universally appealing and fills a void every human being feels in their soul: to be free.”  – Max Kieser (source)

The thing is, can the government really do anything about cryptocurrency? The story goes that it was created without their permission, it went past the gatekeepers, are they really needed anymore? Is cryptocurrency beyond their reach? Do they have no choice but to perhaps join in and participate somehow?

Important questions… While some feel there can be a backdoor into some popular cryptocurrencies, it’s important to note that either way, people are beginning to learn about central banking and how enslaving the current system truly is.

Could It Be Used Worldwide Soon?

Right now, witnesses are testifying at a House Financiancial Services subcommittee on whether the government should consider cryptocurrencies as money and their uses.

Rodney Garratt, a professor of economics at the University of California, Santa Barbara, claimed that the banks have to decide whether they want “to withdraw completely from providing a payment device for the general public,” or whether they prefer to adopt a digital alternative. Which, in turn, could be “some form of crypto.”

This seems to suggest some are pushing for the mainstream world to create a competitor to Bitcoin? If they wanted to make a cryptocurrency popular, the ‘gatekeepers’ and the Deep State no doubt could.

An article written for Cointelegraph explains:

Alex Pollock, senior fellow at the R Street Institute, argued that “to have a central bank digital currency is one of the worst financial ideas of recent times, but still it’s quite conceivable…” Pollock said that central bank digital currencies would only increase the size, role, and power of the bank, adding that the Federal Reserve adopting a CBDC would result in it become the “overwhelming credit allocator of the U.S. economic and financial system.”

The hearing includes a lot of people, not just academics, but engineers, entrepreneurs and more from the industry, one that seems to be growing rapidly with more adoption of cryptocurrencies.

Again, this is great that the financial world is being shaken up, and there is a big discussion to be had, but nothing can justify what we have today remains the same. Things are changing, and as always, heavy criticism and harsh resistance follow.

The Need For A Use Case

You hear a lot about new coins popping up, with ICO’s that reach within the millions. People buy in banking on the rise of the coin based on emotion alone. But what use does the new coin truly have? Or simply, is the coin tied to service, value or commodity of sorts that may suggest a greater value than simply emotion?

For example, Bitcoin is an old outdated version of cryptocurrencies already. What?! Yes. It’s payment processing times are slow, making it very difficult to be used as a means for everyday payment. It’s blockchain simply can’t scale to manage this sort of task. It also does not have any use case or service tied to it that makes it useful beyond being held as an investment. In other words, the only thing holding up Bitcoin’s value is emotion.

Whereas, if you look at a new crypto and new blockchain called TimiCoin (also TimiHealth and TimiDNA), you would find that not only is their blockchain robust enough to service many transactions at a time, but there is a powerful ‘use-case’ tied to the token that creates a ton of value. The use-case is simple: the Timi ecosystem allows people to access their health records instantly – and monetize their own data! This, instead of having corporations keep hold of your records, and having them sold to third parties to be monetized, where you get nothing, with your records, your data, you should be able to monetize it.

Timicoin is a cryptocurrency for the people. It allows them to take power back from corporations on something important and gives you the power to monetize.

Cryptocurrency, in general, has the potential to empower the people by keeping control out of the hands of government and corporations.

We recognize there have been doubts, and that crypto is simply part of an ‘elite‘ plan, but ultimately, we need a change in how we deal in currency, and this has the makings of it. There are both positive and negative tones coming out of this hearing, and that’s to be expected.

Free David Wilcock Screening: Disclosure & The Fall of the Cabal

We interviewed David about what is happening within the cabal and disclosure. He shared some incredible insight that is insanely relevant to today.

So far, the response to this interview has been off the charts as people are calling it the most concise update of what's happening in our world today.

Watch the interview here.
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8 Incredible Reasons Witch Hazel Should Be Added To Your Medicine Cabinet

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In Brief

  • The Facts:

    There are many harmful chemicals lurking in most personal care products. There are enough natural alternatives out there, such as witch hazel that could eliminate your need to use these chemical-containing products at all.

  • Reflect On:

    How important is it for you to limit the number of harmful chemicals that you, your family and the environment are exposed to?

Witch Hazel is an ancient Native American remedy for a wide variety of ailments and people are starting to see why. Generally, in stores, we see witch hazel available in a clear liquid form in a bottle, but prior to that, it starts off as a shrub with yellow flowers.

There are two ways of getting the extract from the witch hazel bark, one as a distillation, this is commonly found in stores and generally has alcohol added to act as a preservative, or as a decoction. Many herbalists agree that the decoction method is superior because it makes a more concentrated version which means higher levels of tannins, which is the component that gives witch hazel its superior astringent properties. The distilled version is more shelf-stable, but just be aware of the alcohol that’s added, as some are sensitive to this.

Witch hazel can typically be used in the place of alcohol for treatment of many different skin irritations. It is recognized worldwide as a natural cleanser and toner, it’s clear that this product is absolutely great for the skin, so what benefits does it provide?

1. Clear Up Acne

A study, which was published in the Journal of Aesthetic and Clinical Dermatology showed that Witch Hazel was one of the most effective acne treatments out of 52 tested products.

This liquid can slow bacterial growth, speed up healing of scabs and reduce inflammation, redness, oil production and even the development of blackheads and whiteheads.

2. Protect From Skin Cancer

A study that was published in Chemical Research in Toxicology found that the polyphenols and tannins found naturally in witch hazel reduced the number of free radicals and stopped the growth of melanoma cells.

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3. Cleanse Hair & Hydrate Dry Scalp

Witch Hazel is a common ingredient in many homemade, all-natural shampoos. It can reduce oil build up on the hair, which could leave your hair clean, shiny and potentially add more volume.

According to this study, witch hazel was able to reduce symptoms of a dry, itchy and irritated scalp.

4. Reduce Degenerative Effects Of The Skin

Because of the anti-inflammatory and anti-oxidant properties of witch hazel, it can help to protect collagen and the skin’s elasticity. This provides a super cheap alternative to many of the expensive wrinkle creams, cleansers and toners out there. Try mixing Witch Hazel with vitamin E oil and some herbs or flowers such as rose or lavender for your very own home-made anti-aging serum.

5. A Useful Addition To Homemade Baby Wipes

If you are someone who is crafty, economical and practical then the idea of homemade baby wipes might be exciting for you. Witch Hazel is a perfectly safe ingredient to use in homemade wipes to wipe your babies bum clean. This is a great alternative to whatever chemicals may be lurking in those store-bought wipes. Making your own is also much better for the environment as homemade wipes don’t come packed in plastic.

6. Heal Stretch Marks?

Some claim that a witch hazel spray applied directly onto stretch marks once a day can help to eliminate the appearance of stretch marks. It’s so simple so at least it’s worth a try, considering it assists with inflammation and scarring this only makes sense.

7. Itch Remedy

Witch Hazel can be an excellent remedy for anything from bug bites, to eczema to chickenpox and poison ivy. You can put some in a spray bottle and add a few drops of lavender essential oil and find relief next time you or your children have an itch that scratching won’t help.

8. Heal Mouth Sores & Freshen Breath

Canker sores, bit lip or cheek? Simply swish some witch hazel around your mouth to help reduce irritation. This is also a great natural mouthwash that can help freshen breath. You can add a drop or peppermint essential oil for a more powerful fresh feeling, just be careful not to swallow.

All Natural Products, All The Way

With so many all-natural alternatives to our everyday products, one might begin to wonder why we even use mainstream store-bought products that are laden with chemicals and preservatives in the first place? It seems that the plant kingdom has a remedy for almost anything that ails us; it’s just that a lot of this knowledge has been lost and unfortunately it’s not a focal point for most manufacturers. Aside from countless recipes for natural homemade personal care and cleaning products online, some retailers are starting to understand our desire to eliminate chemicals from our lives and offer many alternatives, which are generally found at health food stores.

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Awareness

Study Reveals Popular Vaccine “May Kill More Children From Other Causes Than It Saves”

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In Brief

  • The Facts:

    Multiple studies have shown and emphasized that the DTP vaccine may actually kill more children than it protects from DTP. This is one of the latest to show it, known as the Mogensen study.

  • Reflect On:

    Reflect on the fact that this information is never really brought up within the mainstream medical community. All it takes is one CDC study to "debunk" several studies that show opposite results. What's really going on here? Is our health a priority?

There are numerous vaccine safety issues. It can boggle your mind how health authorities and pharmaceutical companies can deem them to be completely safe, necessary, and responsible for saving millions of lives. When people hear this, they usually just believe it without ever looking into it and doing their research, and don’t realize they are only presented with one side of the story. If you have 100 studies raising an issue with a vaccine, all it takes is one study from the CDC to say it’s safe, and that’s the research medical associations dish out to doctors as well as medical schools. After all, the pharmaceutical companies are the ones paying for the whole shebang; what they say, goes.

“The case against science is straightforward: much of the scientific literature, perhaps half, may simply be untrue.” – Richard Horton, current Editor-in-chief of The Lancet 

Heavy Metals

One example would be the vaccine ingredients themselves. Heavy metals, like aluminum and mercury, have been added to vaccines for approximately 100 years without any appropriate safety testing. Numerous studies point this out.  You can access some of those studies and see some examples here.

Fast forward to 2017: researchers have now identified, in animal models, that the aluminum from a vaccine does not exit the body like aluminum from, let’s say, our food; it actually stays in the body, travels to distant organs and eventually ends up in the brain. Not only that, researchers also found some of the highest brain aluminum content ever measured in autopsies of the brains of people who were autistic.

You can read more about that and access those studies in the articles linked below:

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Study Shows Where ‘Almost 100 Percent of Aluminum From Vaccines Could Go Inside A Baby’s Body’

‘Some of the Highest Values For Brain Aluminum Content Ever Measured’ Found In People With Autism

It is simply no longer possible to believe…or to rely on the judgement of trusted physicians or authoritative medical guidelines. I take no pleasure in this conclusion, which I reached slowly and reluctantly over my two decades as an editor of The New England Journal of Medicine. –  Dr. Marcia Angell

Questioning Vaccine Safety Is Suppressed

With all of these issues, why is there such a harsh reaction to questioning vaccine safety? Why can’t a doctor or professor keep their job if they question vaccine safety? Isn’t science about openly questioning? The day we stop questioning, when there are clearly multiple concerns and questions to be asked and addressed, is the day we abandon the possibility of doing real science.

“The medical profession is being bought by the pharmaceutical industry, not only in terms of the practice of medicine, but also in terms of teaching and research. The academic institutions of this country are allowing themselves to be the paid agents of the pharmaceutical industry. I think it’s disgraceful.”  – Arnold Seymour Relman (1923-2014), Harvard professor of medicine and former Editor in Chief of The New England Medical Journal (source)

The issue here is, vaccines are marketed as completely safe, and anybody who questions vaccines is made out to look crazy, dumb, or unscientific. This couldn’t’ be further from the truth, and there are a number of valid reasons why parents should not be forced to vaccinate their child. 

The DTP Vaccine

Robert F. Kennedy Jr. has long been a vocal critic of the lack of scientifically-based vaccine testing:

The public in both poor and rich countries has a right to scientifically-based evidence that international vaccine programs are as safe as possible and that they have been thoroughly safety-tested.  The best metrics for measuring safety are studies comparing health outcomes of vaccinated versus unvaccinated cohorts.  Yet, both the CDC and the WHO have aggressively discouraged the pursuit of such studies. – RFK Jr.

He wrote an article that goes into more detail about the DTP vaccine, it’s history, and what the current research suggests. It is becoming difficult to avoid the conclusion that the DTP vaccine is causing more harm than good.

Study Finds Higher Mortality In Infants Who Received The DTP Vaccine Compared To Those Who Didn’t

In the video below, I go into more detail about the DTP vaccine. HERE is the study I reference in the video.

Free David Wilcock Screening: Disclosure & The Fall of the Cabal

We interviewed David about what is happening within the cabal and disclosure. He shared some incredible insight that is insanely relevant to today.

So far, the response to this interview has been off the charts as people are calling it the most concise update of what's happening in our world today.

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