Connect with us

Awareness

Tylenol Damages The Brains of Children, Research Reveals

Published

on

In Brief

  • The Facts:

    Tylenol has a wide range of toxic side effects you should be aware of, especially if you are pregnant or use it with your children. Article written by William Parker, Ph.D for Greenmedinfo.com, published here with permission.

  • Reflect On:

    Why do we keep taking Tylenol and other over-the-counter drugs when it's unquestionable that they do more harm than good? Why don't we ever look into healthy ways to alleviate our symptoms?

Original Article Link

advertisement - learn more

A number of non-peer-reviewed articles have been written and published on the web claiming that there is literally nothing to fear from acetaminophen during pregnancy. There are two types of articles that fall into this category. First, reputable watchdog organizations have weighed in on the issue, declaring acetaminophen use during pregnancy and during childhood to be proven safe. In particular, the National Health Service of the UK and the Center for Accountability in Science have both strongly criticized the Spanish study from 2016 showing a link between acetaminophen use during pregnancy and ADHD/autism.

The second type of article is generally written by a science writer working for an organization that runs a website. Often quoting one to three experts who claim that is perfectly safe and that pregnant women and families should not be concerned, many of these articles are published by reputable sources that are generally trustworthy. Typically, an expert is being asked to comment on one particular publication showing a link between acetaminophen use (usually during pregnancy) and some sort of neuropsychiatric problem (autism, lowered IQ, hyperactivity, and/or social/behavioral problems, depending on the study). There are several important things to consider when evaluating these articles:

1.  There are a number of University Professors who have studied the use of acetaminophen on the developing brain and who are keenly aware of the potential dangers. A partial list of these individuals is provided below.

2.  Being an expert in acetaminophen neurotoxicity during development means that considerable time has been invested in studying the issue. Any true expert in this issue will be aware of basic facts regarding acetaminophen neurotoxicity. These facts include the following:

(a) Studies in animal models (both in mice and in rats) demonstrate that acetaminophen use during a sensitive period of brain development causes long-term alterations in the brain and is manifested as problems with social function.

advertisement - learn more

(b)  Margaret McCarthy, Chair of Pharmacology at the University of Maryland, has worked out the probable mechanism by which acetaminophen-induced brain damage occurs. Her research team has found that the male brain is considerably more sensitive to acetaminophen than the female brain, possibly accounting for the gender bias in autism.

(c) There are (as of January 2017) a total of 8 published studies evaluating the long terms effects on children of acetaminophen use during pregnancy or during childhood. Two of these (one in 2014, one in 2016) were published in JAMA Pediatrics, one of the most highly respected pediatric journals. All studies point toward acetaminophen use being associated with long-term problems with neurological function. Each study design has included some attempt to control for indication. In all studies, acetaminophen use rather than indication has been identified as the key factor associated with cognitive problems. A formal meta-analysis is not currently possible because of the varied outcome measures and study designs, but all 8 studies point in the same direction: Acetaminophen is neurotoxic to the developing brain. The studies are not “cherry picked”, selecting only those which find an effect. All studies point toward a neurotoxic effect of acetaminophen in the developing brain.

(d)   Acetaminophen substantially alters brain chemistry and temporarily impairs awareness of social issues in adult humans.

(e)  Testing of acetaminophen safety in children did not include any evaluation of brain function, and no long-term studies were ever conducted. The primary manufacturer of acetaminophen in the US acknowledges that the drug has never been shown to be safe for brain development when used during pregnancy or in childhood. All safety tests were performed with the assumption that any side effects would be acute in nature (e.g., bleeding or acute organ damage). This assumption was based on observations made with acetaminophen in adults and with aspirin in children. It was not based on any experience with acetaminophen use in children.

3.     Having prescribed tens of thousands of doses of acetaminophen does not make anyone an expert on the neurotoxicity of acetaminophen, any more than eating thousands of pounds of chips makes somebody an expert in the effects of an inflammatory diet. Credentials and certifications that allow physicians to prescribe acetaminophen do not make them experts, and elevated positions in the medical community do not qualify anybody as an expert on the effects of acetaminophen. If somebody does not know those basic facts listed above, then they are not an expert on the neurotoxicity of acetaminophen. Usually, the experts will have published one or more peer-reviewed manuscripts on the topic. Those are the people to ask when an expert is needed.

4.     It is tempting to point accusing fingers at physicians who say that acetaminophen is safe when they literally have no grasp whatsoever of the relevant scientific literature. However, this would be a mistake. I have tracked down a few of these individuals who were quoted in a very public format, and one individual, in particular, didn’t even remember having made a comment on the topic. The most likely explanation is that a reporter asked them if acetaminophen was safe, and their response based on their training (not on the knowledge of the literature) was that it is safe. After all, if they didn’t think it was safe, they would not be administering it dozens of times per day. So, if a reporter asks a physician if something is safe, and they provide their knowledge based on what they have been taught and how they practice, then it is hard to blame them. The reporter didn’t ask them to spend days or even weeks reviewing the literature in detail, but rather assumed that any physician administering something dozens of times per day would know the literature. (This is a false assumption. No physician has the time to study all current literature on every drug they administer.) So, in a nutshell, a tragic propagation of incorrect information is occurring despite the best of intentions of all parties involved.

5.     Unless an organization such as the National Health Service has the time to review a topic thoroughly, they should remain silent on an issue. It took a team of us two years to put together our summary of the evidence, both direct and circumstantial, regarding the potential neurotoxicity of acetaminophen during development. It took the NHS only days to publish their recent criticism of the 2016 Spanish study. Offering questionable criticisms of a single paper without reviewing the literature to see how that publication fits into the big picture is a disservice to the public being served.

6. Reading the published quotes from many “experts” who exonerate acetaminophen, it is apparent that the logic falls into one of two categories.

(a) Everybody is doing it, so it must be OK.

(b) This single study is not perfect, so no change in practice should be made.

Neither of these criticisms is logically sound, of course. These two criticisms are often combined and were, in fact, part of the critical comments directed toward the first paper showing that acetaminophen probably has substantial neurotoxicity during development (published in 2008 by Steve Shultz). Further, the evaluation of study weaknesses is usually skewed and not entirely valid. Since the idea that acetaminophen is safe is being embraced, then any merit in the paper is often undermined to make the case. This is certainly true of the published (peer reviewed) criticisms of the 2008 Shultz paper.

7.     Many on-line sources support the view that acetaminophen can be very dangerous to the developing brain. Probably the most reliable source, the FDA, is remaining silent on the topic until something more definitive is done. The FDA knows that this is extremely urgent, but unfortunately, our FDA is not linked well (in a practical manner) with our NIH, and thus they can’t dictate research priorities.

8.     Here is a list (not comprehensive) of experts regarding the neurotoxicity of acetaminophen during brain development.

a) First, I’ll thank the wonderful team of individuals who helped put together our comprehensive review on this topic. Shu Lin, a professor with me in Duke’s Surgery Department, is a very dear and long-time friend of mine who has supported me through countless projects over the past 22 years. Staci Bilbo, director for research on Autism at Harvard, is a friend and collaborator who has helped me understand what causes inflammation and the role of inflammation in brain dysfunction. Chi Dang Hornik, a pediatric pharmacist at Duke, contributed greatly to our understanding of the frequency of acetaminophen administration and the available formulations of the drug. Many thanks to Martha Herbert. As a Harvard professor and clinician, she has a great appreciation for the clinical data obtained from patients with autism. Cindy Nevison, a professor at the University of Colorado at Boulder, rounds out our team, providing critical information about the epidemiology of autism. (Thanks also to our interns (Rasika Rao and Lauren Gentry) and research analyst (Zoie Holzknecht) who were a tremendous help in compiling information and preparing that information for publication.)

b) Margaret McCarthy, chair of Pharmacology at the University of Maryland, it the most knowledgeable person I know regarding the biochemistry of the human brain and how that is affected by acetaminophen and other drugs in that class.

c) Chittaranjan Andrade, Chair of Psychopharmacology at the National Institute of Mental Health and Neurosciences, Bangalore, India, has written a peer reviewed paper on the topic of acetaminophen induced brain damage. He nicely summarized a number of studies looking at the connection between acetaminophen and neurological damage. His final conclusion is that the drug is probably more associated with ADHD than autism, but the conclusion was limited to exposure during pregnancy and his work was conducted before some critical studies were published in 2016.

d) Henrik Viberg is a professor in the Department of Organismal Biology at Uppsala University in Sweden. He has studied how exposure of mice to acetaminophen during development can cause long term brain damage.

e) In 2015, a group of scientists working with Laurence de Fays at the Federal Agency for Medicines and Health Products in Brussels acknowledged the clinical studies and the studies in animal models which indicated that acetaminophen could be dangerous to the developing fetus, but concluded that paracetamol is “still to be considered safe in pregnancy”. At the same time, they state that “additional carefully designed studies are necessary to confirm or disprove the association (between acetaminophen and brain damage to children)”, and that “care should be taken to avoid raising poorly founded concerns among pregnant females”. We very strongly agree with the conclusion that more studies are needed, but very strongly disagree with the conclusion that women should be kept in the dark about the matter. It is important to point out that several more studies have come out since Laurence de Fays’ report. One of those is a 2016 manuscript in JAMA Pediatrics(see the next expert), a highly reputable peer reviewed journal, which addresses the concerns raised by de Fays, so it is possible that de Fays’ group may now have a different opinion.

f) A team of scientists and doctors working with Evie Stergiakouli at the University of Bristol analyzed data from a prospective birth cohort, and concluded that “children exposed to acetaminophen prenatally are at increased risk of multiple behavioral difficulties”. They found considerable evidence indicating that the association was not due to the confounding factors that concerned de Fays’ group (previous expert).

g) Jordi Julvez at the Centre for Research in Environmental Epidemiology in Barcelona, Spain worked with a team of a dozen clinicians and scientists to publish their 2016 study linking acetaminophen with autism and ADHD.

h) Amany A. Abdin, a professor in the Department of Pharmacology, Tanta University, Egypt, wrote a review of the acetaminophen/autism connection and published it in the journal Biochemistry and Pharmacology: Open Access. Her conclusion in 2013 was that the drug is not safe and that the acetaminophen/autism connection should receive attention.

i) The original paper that identified a connection between neuropsychiatric disorders and acetaminophen was published by Steve Shultz while at the University of California at San Diego. Coauthors on the paper included Hillary Klonoff-Cohen, currently an Endowed Professor and Director of the MPH program at the University of Illinois.

j) Four scientists, including research scientist Ragnhild Eek Brandlistuen and professors Hedvig Nordeng and Eivind Ystrom in the Department of Pharmacy at the University of Oslo, coauthored a study showing a connection between adverse neurodevelopment and acetaminophen use during pregnancy.

k) Jorn Olsen, Professor and Chair of the Department of Epidemiology at UCLA, published one of the more recent papers (2016) showing a connection between autism and acetaminophen use during pregnancy.

l) Five professors (John M. D. Thompson, Karen E. Waldie, Clare R. Wall, Rinky Murphy, and Edwin A. Mitchell) from four different departments at The University of Auckland published their findings in PLOSone in 2014 which “strengthen the contention that acetaminophen exposure in pregnancy increases the risk of ADHD-like behaviours. Our study also supports earlier claims that findings are specific to acetaminophen.”

For evidence-based research on the dangers of acetaminophen, visit the GreenMedInfo.com Research Dashboard.\

Read their related article on Tylenol: 

Tylenol Kills Emotions As Well As Pain, Study Reveals

Sign Up For The Greenmedinfo Newsletter HERE.


William Parker is an Associate Professor at Duke University, where he has worked in the Department of Surgery since 1993.  William is currently investigating a number of issues associated with inflammation and Western society, including vitamin D deficiency, heart disease and alteration of the symbionts of the human body (“biota alteration”). He has been interested in “natural” immune function for some time, which has led him down a path that includes the first studies of immune function in wild rats and the discovery of the function of the human appendix.

Start Your Free 7 Day Trial To CETV!

Due to the pressure of mass censorship, we now have our own censorship-free, and ad-free on demand streaming network!

It is the world's first and only conscious media network streaming mind-expanding interviews, news broadcasts, and conscious shows.

Click here to start a FREE 7-Day Trial and watch 100's of hours of conscious media videos, that you won't see anywhere else.

Advertisement
advertisement - learn more

Awareness

Cancer is Now the Leading Cause of Death

Published

on

In Brief

  • The Facts:

    Cancer has surpassed heart disease as the No. 1 cause of death in high-income countries, highlighting the urgent need to change the way this disease is prevented and treated.

  • Reflect On:

    Rather than being a random result of DNA mutations, it's possible that cancer could have much deeper roots that would be better targeted with natural therapies than toxicity.

This article was written by the Greenmedinfo Research Group, originally published by Greenmedinfo.com. Published here with permission. 

Cancer has dethroned heart disease to earn the nefarious title of leading cause of death in high-income and certain middle-income countries.[i] While heart disease remains the No. 1 cause of death globally among adults aged 35 to 70, in high-income countries, which included Saudi Arabia, United Arab Emirates, Canada and Sweden, cancer caused twice as many deaths as heart disease.[ii]

Some middle-income countries, which included the Philippines, Iran, South Africa, Colombia, China, Brazil, Malaysia, Turkey, Poland, Argentina and Chile, also saw cancer become the leading cause of death.

While the U.S. was not included in the new analysis, research published in 2018 suggested, “the United States is in the midst of an epidemiologic transition in the leading cause of death,” moving from heart disease to cancer.[iii]

That study, too, found that cancer was quickly outpacing heart disease as the top killer, with high-income counties transitioning first. In fact, while only 21% of U.S. counties had cancer as the leading cause of death in 2003, this rose to 41% in 2015.

“The shift to cancer as the leading cause of death was greatest in the highest-income counties,” the researchers explained,[iv] echoing the current study, which also cited “a transition in the predominant causes of deaths in middle-age” in high-income countries.[v]

advertisement - learn more

“The world is witnessing a new epidemiologic transition among the different categories of noncommunicable diseases, with CVD [cardiovascular disease] no longer the leading cause of death in HIC [high-income countries],” lead author Dr. Gilles Dagenais, professor emeritus, Laval University, Quebec, Canada, said in a statement.[vi]

Why is Cancer a Top Killer?

The study suggested cancer is rising to the top because heart disease is better treated in high-income countries, saving more lives from heart disease and paving the way for cancer deaths to flourish. But perhaps a better question is why cancer continues to kill so many.

Even globally, cancer still comes in as the second leading cause of death behind heart disease, responsible for 26% of deaths worldwide.[vii] In the U.S., Americans have a 1 in 3 risk of developing cancer at some point in their lifetimes, along with a 1 in 5 risk of dying from the disease.[viii]

In early 2019, it was announced that cancer death rates in the U.S. declined 27% since 1991,[ix] a statistic that makes it seem as though we’re winning the “war on cancer.” But most of these declines can be attributed to reductions in smoking — and perhaps a limited measure of increased early detection and treatment — and are not a sign that conventional medicine’s model of surgerychemotherapy and/or radiation to treat cancer is, on the whole, working.

While death rates from certain cancer have declined, others have increased. Overall, cancer deaths in the U.S. in 2016 were similar to those in 1930[x] — despite all the “advances” in detection and treatment.

Changing the Way We Think About Cancer

It’s becoming increasingly clear that in order to conquer cancer, it’s necessary to change the way we think about it. Cancer is found in virtually all animals, suggesting it has evolutionary significance.[xi] It’s possible that cancer is an ancient survival program unmasked — even a process the body undergoes in order to survive nutrient deprivation and exposure to toxins.

Rather than being the result of an accumulation of DNA mutations that create rogue cells that multiply out of control, cancer could be cells that have flipped an epigenetic switch into survival mode in the form of a tumor. In the journal Physical Biology, researchers theorized:[xii]

“[C]ancer is an atavistic [primitive] condition that occurs when genetic or epigenetic malfunction unlocks an ancient ‘toolkit’ of pre-existing adaptations, re-establishing the dominance of an earlier layer of genes that controlled loose-knit colonies of only partially differentiated cells, similar to tumors.”

If this is true, it makes sense that conventional cancer treatments aimed to poison or “kill” the cancerous cells may only make the problem worse by creating an even more toxic environment, which could trigger the cancer to reach back into its “ancient toolkit” to find additional means of survival.

This explanation may be overly simplistic, as there are many factors that contribute to cancer, but there is evidence to suggest that natural substances and therapies that support the body’s overall health can be useful in the fight against cancer.

Nearly 1,000 Natural Substances Have Anti-Cancer Potential

GreenMedInfo has a database of 986 substances that have been researched as potential cancer prevention and treatment strategies. There are undoubtedly many more out there that have yet to be discovered. At the top of the list is curcumin, the active ingredient in the curry spice turmeric, which targets cancer stem cells while leaving normal stem cells unharmed.[xiii]

Another top contender is vitamin D, which you can get for free from adequate sun exposure. Higher vitamin D levels are not only known to lower your cancer risk but also to improve outcomes if you’ve already been diagnosed.[xiv] Fiberresveratrolsulforaphane and vitamin E — all substances you can get from your diet — also show anti-cancer promise, as does coffee, perhaps because it improves the body’s ability to efficiently repair DNA damage.[xv]

So if there was one silver lining to the news that cancer is now the leading cause of death in some countries, it would be that it’s a condition that has many promising natural avenues for prevention and treatment. Current conventional cancer treatments are failing, but that doesn’t mean cancer is unstoppable — it means it’s time to broaden our research into and usage of traditional therapies.

Many natural substances, like noni leaf,[xvi] have even been shown to work better than chemotherapy, highlighting why, if we’re going to win the war against cancer, we’re going to need to do it with nature on our side.

For more on how to naturally fight Cancer, visit the GreenMedInfo database on the subject.

Originally published: 2019-09-14

Article Updated: 2019-11-05

References

[i] The Lancet September 3, 2019

[ii] CNN September 3, 2019

[iii] Annals of Internal Medicine December 18, 2018

[iv] Annals of Internal Medicine December 18, 2018

[v] The Lancet September 3, 2019

[vi] Medscape September 3, 2019

[vii] Medscape September 3, 2019

[viii] American Cancer Society, Lifetime Risk of Developing or Dying From Cancer

[ix] CA: A Cancer Journal for Clinicians January 8, 2019

[x] CA: A Cancer Journal for Clinicians January 8, 2019

[xi] Front. Oncol., 10 January 2019

[xii] Physical Biology February 7, 2011

[xiii] Anticancer Res. 2015 Feb ;35(2):599-614.

[xiv] Br J Cancer. 2017 Mar 16. Epub 2017 Mar 16.

[xv] J Nutrigenet Nutrigenomics. 2015 ;8(4-6):174-84.

[xvi] Mol Cell Biochem. 2016 Apr 22. Epub 2016 Apr 22.


For more info from Greenmedinfo, you can join their newsletter by clicking here.

Start Your Free 7 Day Trial To CETV!

Due to the pressure of mass censorship, we now have our own censorship-free, and ad-free on demand streaming network!

It is the world's first and only conscious media network streaming mind-expanding interviews, news broadcasts, and conscious shows.

Click here to start a FREE 7-Day Trial and watch 100's of hours of conscious media videos, that you won't see anywhere else.

Continue Reading

Awareness

Man Fasts For 382 Days Straight & Loses 276 Pounds

Published

on

In Brief

  • The Facts:

    Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days. He remained completely healthy for the duration of the fast.

  • Reflect On:

    Today, it's firmly established in scientific literature that fasting can have tremendous benefits, if done correctly. It can also be used to treat a variety of diseases. Perhaps it's not emphasized because you can't make money off of not eating?

A study published in the Post Graduate Medical Journal in 1972 brought more attention to a gentleman by the name of Angus Barbieri, a man who, in June of 1965, began a fast under medical supervision for exactly 382 days and, at the time the study was published, had since maintained his ordinary weight. In his case, “prolonged fasting had no ill effects.” Barbieri’s weight decreased from 456 to 180 pounds during the fast.

This isn’t the only example that’s available in the literature, it’s similar to an earlier patient prior to Barbieri who reduced his weight from 432 to 235 pounds during 350 days of intermittent fasting (Stewart, Fleming & Robertson, 1966). Researchers have also fasted patients for 256 days (Collison, 1967, 1971), 249 and 236 days (Thomson et al., 1966) as well as  210 days (Garnett et al., 1969; Runcie & Thomson, 1970), all of which are cited in the 1972 study.

Since the publication of this time, there are many documented examples of prolonged fasting done by highly obese people. Here’s one recent example of a man who fasted for 50 straight days, while being medically supervised and tested the whole time.

When you fast, your body switches from burning glucose, to burning fat. Fasting lowers insulin levels which allows the body to access its fat stores for energy. When you eat, food is converted into glucose and that’s what we usually burn. This is why fasting has become a therapeutic intervention for many people with type two diabetes, and more doctors, like Dr. Jason Fung, a Toronto Based nephrologist, are having great success with utilizing fasting as an appropriate and necessary health intervention. Fung has many great articles regarding the science of fasting, you can access them here if you’re interested in learning more. This article references some of the leading scientists in the field so you can learn more by looking them up as well.

The graph below depicts what happens to your protein while fasting. Interesting isn’t it? People often believe that if you fast, you will experience a tremendous amount of muscle loss during fasting, but that’s simply not true. This graph is from Kevin Hall, from the NIH in the book “Comparative Physiology of Fasting, Starvation, and Food Limitation.”

“It seems that there are always concerns about loss of muscle mass during fasting. I never get away from this question. No matter how many times I answer it, somebody always asks, “Doesn’t fasting burn your muscle?” Let me say straight up, NO.”  – source Dr. Jason Fung

advertisement - learn more

But what about Angus Barbieri? Obviously we’re not saying long term fasts for this long are healthy, obviously for many people they will probably be unhealthy and unsafe unless medically supervised. In  the 1972 study doctors measured a number of concentrations within the body. For example, plasma potassium concentrations over the first four months decreased systematically. As a result, they provided a very small daily dose that increased his potassium level. After another 10 weeks, no potassium was given, and from there on in until the end of the fast, plasma potassium levels remained normal. Cholesterol concentrations also remained around 230 mg/ 100 ml until 300 days of fasting, but increased to 370 mg/100 ml during refeeding.

Plasma magnesium levels decreased over the first few weeks of the fast but then went up and stabilized. This is interesting to note as there is nothing going into the body, yet levels still stabilized after the initial decrease.

Normal plasma magnesium concentrations, despite magnesium ‘depletion’ in muscle tissue, have been described (Drenick et al., 1969) during short-term fasting (1-3 months). The only other relevant report is a remark (Runcie & Thomson, 1970) that one patient who fasted 71 days had a normal plasma magnesium level of 2-2 mEq/l at the time when she developed latent tetany. The decrease in the plasma magnesium concentration of our patient was systematic and persistent.

Furthermore:

The excretion of sodium, potassium, calcium and inorganic phosphate decreased to low levels throughout the first 100 days, but thereafter the excretion of all four urinary constituents, as well as of magnesium, began to increase. During the subsequent 200 days sodium excretion, previously between 2 and 20 mEq daily, reached over 80 mEq/24 hr, potassium excretion increased to 30-40 mEq daily and calcium excretion increased from 10-30 mg/24 hr to 250- 280 mg/24 hr. Magnesium excretion (which was not measured during the first 100 days) reached 10 mEq/ 24 hr between Days 200-300. Phosphate excretion, which had decreased to under 200 mg/24 hr, also increased to around 800 mg/24 hr, even exceeding 1000 mg/24 hr on occasion. Peak excretions of all these constituents were seen around Day 300, after which there was a marginal decrease, but excretion remained high.

Obviously, this is an extreme fast and such fasts have only been tested on people of tremendous obesity, and it shows that people with a high body fat percentage have the ability to fast longer simply because their body has more stores to pull from.

The study concluded in 1972 that:

We have found, like Munro and colleagues (1970), that prolonged supervised therapeutic starvation of the obese patient can be a safe therapy, which is also effective if the ideal weight is reached. There is, however, likely to be occasionally a risk in some individuals, attributable to failures in different aspects of the adaptative response to fasting. Until the characteristics of these variations in response are identified, and shown to be capable of detection in their prodromal stages, extended starvation therapy must be used cautiously. In our view, unless unusual hypokalaemia is seen, potassium supplements are not mandatory. Xanthine oxidase inhibitors (or uricosuric agents) are not always necessary and could even be potentially harmful (British Medical Journal, 1971) perhaps particularly in the long-term fasting situation.

It’s almost 2020, and the literature, studies and research that’s been published since 1972 is vast. We’ve learned a lot more about it and if done correctly it can be extremely beneficial. Shot term fasting  presents minimal to no health risks, and so does long term fasting that lasts more than 24 hours, that is unless a person already has an underlying condition. That being said, it’s not easy to start. Most people are used to eating three meals plus snacks every single day, therefore they are never adapted to burning their fat stores, something that appears the human body was meant to do.

“Why is it that the normal diet is three meals a day plus snacks? It isn’t that it’s the healthiest eating pattern, now that’s my opinion but I think there is a lot of evidence to support that. There are a lot of pressures to have that eating pattern, there’s a lot of money involved. The food industry — are they going to make money from skipping breakfast like I did today? No, they’re going to lose money. If people fast, the food industry loses money. What about the pharmaceutical industries? What if people do some intermittent fasting, exercise periodically and are very healthy, is the pharmaceutical industry going to make any money on healthy people?” – Mark Mattson (source)

Fasting has also been shown to be effective as a therapeutic intervention for cancer. Fasting protects healthy cells while ‘starving’ cancer cells, it’s now being used as an intervention that’s being combined with chemotherapy. Fasting has also been shown to greatly reduce the risk of age related diseases like Parkinson’s Disease, and Alzheimer’s disease. Mark Mattson, one of the foremost researchers of the cellular and molecular mechanisms underlying multiple neurodegenerative disorders has shown through his work that fasting can have a tremendous effect on the brain, and can even reverse the symptoms of multiple neurodegenerative disorders. You can watch his interesting TED talk here.  Scientists have also discovered strong evidence that fasting is a natural intervention for triggering stem cell-based regeneration of an entire organ or system.

Fasting has actually long been known to have an effect on the brain. Children who suffer from epileptic seizures have fewer of them when placed on caloric restriction or fasts. It is believed that fasting helps kick-start protective measures that help counteract the overexcited signals that epileptic brains often exhibit.  (source)

The list goes on and is quite long. At the end of the day if you do your research, fasting, under proper medical supervision, can have tremendous health benefits that go far beyond what’s mentioned in the paragraph above. Every single study that has looked at fasting as a therapeutic intervention for several diseases has shown nothing but positive benefits. Even studies conducted regarding caloric restriction, something completely different than fasting, have shown promising results in all animal models.

According to a review of fasting literature conducted in 2003, “Calorie restriction (CR) extends life span and retards age-related chronic diseases in a variety of species, including rats, mice, fish, flies, worms, and yeast. The mechanism or mechanisms through which this occurs are unclear.” Since this study was published, a great amount of research has been conducted from many researchers, and the mechanisms are being discovered and have become more clear. If you want to further your research, apart from the names listed above, Dr. Valter Longo and his research is another great place to start.

The body has a tremendous amount of storage, and it hangs on to what it needs during a fast, and uses up ‘bad’ things, repairs damaged cells, and more. When you fast and deplete all your glycogen, your body is going to start using fat for energy, it’s going to use damaged cells for energy, it’s basically going to use all of the bad things first, before it gets to the good thing…Your body will not burn protein, as protein is not a fuel source while fasting.

I bring this up because it’s interesting to see what the body loses and hangs on to during a fast.

The Takeaway

The truth about fasting is that it’s not dangerous at all. Intermittent fasting and short term fasting can be done by just about anybody. From what we’ve seen with regards to prolonged fasting, it’s also not very dangerous when it comes to obese people doing it under medically supervised conditions. Theoretically, based on the science alone, any relatively healthy human being should be able to do a prolonged fast without any harmful consequences.

Obviously, prolonged fasts that are not medically supervised can be very detrimental. We are obviously not recommending this and you must do a lot of research and talk to your doctor if you’re interested in fasting, before trying it. For starters, a little bit of intermittent fasting here and there is a no brainer, and not dangerous at all if you have no underlying health conditions, but everybody’s body is different.

Fasting is making a lot of noise, and has been making a lot of noise within the health community, but it’s still not appropriately taught and used by the mainstream medical industry. Why is this so? The answer is simple, you can’t make money off of fasting.

Start Your Free 7 Day Trial To CETV!

Due to the pressure of mass censorship, we now have our own censorship-free, and ad-free on demand streaming network!

It is the world's first and only conscious media network streaming mind-expanding interviews, news broadcasts, and conscious shows.

Click here to start a FREE 7-Day Trial and watch 100's of hours of conscious media videos, that you won't see anywhere else.

Continue Reading

Alternative News

Thousands Gather To Mark The 33rd Anniversary of the National Childhood Vaccine Injury Act

Published

on

Government’s gift to Pharma of liability-free vaccines puts children’s health at risk states Children’s Health Defense (CHD) Chairman, Robert F. Kennedy, Jr.

Washington, DC – Thousands of advocates for children’s health will gather Thursday at the Vaccine Injury Epidemic (VIE) Event on the National Mall to mark the 33rd anniversary of National Childhood Vaccine Injury Act (NCVIA). The rally on Nov. 14th will spotlight the devastating impact NCVIA has had upon the state of children’s health. While children continue to be injured by vaccines daily, vaccine makers cannot be held accountable, thereby eliminating incentive for vaccine safety.

In his remarks, RFK, Jr. will address the ramifications of NCVIA and honor those whose lives have been impacted by vaccine injury and death. “It’s time to call out Congress, the CDC, and drug companies for allowing industry profits to trump children’s health,” said Kennedy. “There is no crisis more urgent than the epidemics of chronic health conditions among our nation’s children.”

Following NCVIA’s passage creating the National Vaccine Injury Compensation Program (NVICP), the childhood vaccine market sparked a gold rush for Pharma as more vaccines for routine childhood illnesses were developed. Coterminous with the burgeoning vaccine schedule, chronic health conditions in children rose from 12% to 54%. As vaccine industry profits grew to $50 billion annually, so did diagnoses of asthmaautismADHDallergiesanxietydepressiondiabetesobsessive-compulsive disorder and auto-immune diseases.  Here are the facts:

  • An HHS-funded study found only 1% of vaccine injuries are reported.
  • Despite NVICP’s high burden of proof and two out of three claims dismissed, over $4.2 billion has been paid for claims of vaccine injury or death.
  • The vaccine-injured find NVICP to be a years-long, litigious program with no jury, discovery and precedent. While medical bills mount, the injured are up against DOJ lawyers and HHS “Special Masters” that act as judges.
  • The Department of Justice and the NVICP are accused of fraud and obstruction of justice in the Autism Omnibus Proceeding.
  • The Institute of Medicine reports that the vaccine schedule as recommended has never been studied for long-term health effects despite independent research suggesting that unvaccinated children are healthier.
  • Modern medicine acknowledges that not everyone responds the same to vaccination and the “one size fits all” vaccine policy is not science based.

Children’s Health Defense’s created these six steps to vaccine safety. RFK, Jr. interviews are available upon request.

Sign up for free news and updates from Robert F. Kennedy, Jr. and the Children’s Health Defense. CHD is planning many strategies, including legal, in an effort to defend the health of our children and obtain justice for those already injured. Your support is essential to CHD’s successful mission.

advertisement - learn more

Start Your Free 7 Day Trial To CETV!

Due to the pressure of mass censorship, we now have our own censorship-free, and ad-free on demand streaming network!

It is the world's first and only conscious media network streaming mind-expanding interviews, news broadcasts, and conscious shows.

Click here to start a FREE 7-Day Trial and watch 100's of hours of conscious media videos, that you won't see anywhere else.

Continue Reading
advertisement - learn more
advertisement - learn more

Video

Pod

Censorship is hiding us from you.

Get breaking conscious news articles sent directly to your inbox!

Choose your topics of interest below:

You have Successfully Subscribed!